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1  integrin (Mac-1) expression, in response to formylmethionylleucylphenylalanine.
2 partially blocked the exocytosis response to formylmethionylleucylphenylalanine and the platelet-acti
3 -Nle-Leu-Phe-Nle-Tyr-Lys (Nle = Norleucine), formylmethionylleucylphenylalanine, and peptide W reveal
4 body-mediated ligation of SIRPbeta1 enhanced formylmethionylleucylphenylalanine-driven PMN transepith
5 orbol 12-myristate 13-acetate/ionomycin, and formylmethionylleucylphenylalanine each induced the rele
6 ochalasin D, latrunculin B, 8-bromo-cGMP, or formylmethionylleucylphenylalanine, FAK and TrxR reassoc
7 induced by the chemotactic N-formylpeptide N-formylmethionylleucylphenylalanine (fMLF), suggesting a
8 ceptor phosphorylation in FPR processing and formylmethionylleucylphenylalanine (fMLF)-mediated chemo
9 meric G protein-coupled receptors, including formylmethionylleucylphenylalanine (fMLP), interleukin-8
10 hingomyelinases was investigated in resting, formylmethionylleucylphenylalanine (FMLP)-activated, and
11 er, this same concentration slowed human PMN formylmethionylleucylphenylalanine (FMLP)-stimulated che
12 e plasma membrane following stimulation with formylmethionylleucylphenylalanine (fMLP).
13 helial monolayers toward the chemoattractant formylmethionylleucylphenylalanine (fMLP).
14 A); it also blocked cross-phosphorylation by formylmethionylleucylphenylalanine (fMLP).
15 lly) and subsequent perfusion ex vivo with n-formylmethionylleucylphenylalanine (fMLP, 10(-7) M).
16 f complement (C5a) or n-formylated peptides (formylmethionylleucylphenylalanine, fMLP).
17                                    Moreover, formylmethionylleucylphenylalanine induced pertussis tox
18 xA1, Ac2-26, and the classical nFPR agonist, formylmethionylleucylphenylalanine, induced intracellula
19                   In contrast, chemotaxis to formylmethionylleucylphenylalanine, leukotriene B(4), an
20 nction, and these changes can be reversed by formylmethionylleucylphenylalanine or 8-bromo-cyclic GMP
21   Alternatively, incubating neutrophils with formylmethionylleucylphenylalanine or 8-bromo-cyclic GMP
22 n of either exogenous Rac1 or Rac2 increased formylmethionylleucylphenylalanine- or phorbol ester-sti
23  (lactoferrin) of maximal when compared with formylmethionylleucylphenylalanine plus cytochalasin B s
24 e activation with concomitant acquisition of formylmethionylleucylphenylalanine-stimulatable and stre
25 was not down-regulated when co-cultured with formylmethionylleucylphenylalanine-stimulated neutrophil
26 meability model, cell-free supernatants from formylmethionylleucylphenylalanine-stimulated PMNs were
27 d to the cell surface after chemoattractant (formylmethionylleucylphenylalanine) stimulation.
28 e A downstream of lipopolysaccharide but not formylmethionylleucylphenylalanine was shown using the s

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