ライフサイエンスコーパス: fractionated radiation

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1 s with activated c-Myc after single-dose and fractionated radiation.

2 ed between arm A (the control), single daily fractionated radiation (70 Gy at 2 Gy/d); arm B, identic

3 liferation does not diminish the efficacy of fractionated radiation and suggest that disruption of ke

4 3; and arm C, a split course of single daily fractionated radiation and three cycles of concurrent in

5 FC for 12 days, and tumors were treated with fractionated radiation at a dose of 5 Gy/day to a total

6 cervical carcinomas in patients suggest that fractionated radiation can lower tumor IFP and increase

7 The fractionated radiation combined with ATO showed a simila

8 effects are observed in both single-dose and fractionated radiation experiments.

9 oundation for clinical testing of sequential fractionated radiation followed by sorafenib in gastroin

10 al of breast cancer cells treated with hyper-fractionated radiation (HFR), which is used clinically f

11 on would yield equivalent profiles following fractionated radiation in different tumor cell lines.

12 diosensitive than normal cells, but only for fractionated radiation in plateau phase.

13 rate that TPZ potentiates tumor cell kill by fractionated radiation in three murine tumors (SCCVII, R

14 ase inhibitor, also enhanced the efficacy of fractionated radiation in U87 spheroids.

15 odels of radioresistance through exposure to fractionated radiation is an increasingly used approach

16 Fractionated radiation lowered IFP by 2.5 mm Hg when the

17 mycin significantly enhanced the efficacy of fractionated radiation of established U87 xenografts in

18 ithin 2 years of diagnosis, and conventional fractionated radiation remains the standard treatment.

19 agent cisplatin to conventional single daily fractionated radiation significantly improves survival,

20 the third week postinjury, with protocols of fractionated radiation similar to those for treating hum

21 of monoclonal antibodies following single or fractionated radiation that has been reported in the lit

22 to III NSCLC requiring a definitive dose of fractionated radiation therapy (RT) were eligible.

23 d to decrease acute and late xerostomia with fractionated radiation therapy alone for head and neck c

24 2 years have reported the efficacy of using fractionated radiation therapy alone.

25 Intetumumab can potentiate the efficacy of fractionated radiation therapy in human cancer xenograft

26 r model was used to study tumor responses to fractionated radiation therapy in mice with anemia induc

27 ection of radioresistant cancer cells during fractionated radiation therapy may have implications in

28 The effect of DA on tumor responses to fractionated radiation therapy was observed when DA was

29 We test a hypothesis that fractionated radiation therapy within a therapeutic dose

30 activation and repopulation, as occur during fractionated radiation therapy, can lead to distribution

31 When combined with fractionated radiation therapy, DA increased the tumor g

32 When combined with fractionated radiation therapy, intetumumab significantl

33 or-bearing mice and sensitize tumor cells to fractionated radiation therapy.

34 s an important rationale for the widely used fractionated radiation therapy.

35 tumor response during standard clinical dose-fractionated radiation therapy.

36 We found that fractionated radiation to the thorax significantly reduc

37 c division or a fast cycle of GSCs following fractionated radiation treatment is required to yield re

38 this study was to investigate the effect of fractionated radiation treatment, given in daily doses s

39 prostate cancer cells rendered resistant to fractionated radiation treatment.

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