ライフサイエンスコーパス: fratricide

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1 ptide-specific lysis by neighboring T cells (fratricide).

2 he pancreas, resulting in Fas/Fas-L-mediated fratricide.

3 e of residual CD7 expression and the ensuing fratricide.

4 l and malignant cells, leading to CAR T-cell fratricide.

5 ility of cytotoxic lymphocytes to suicide or fratricide after degranulation.

6 and neoplastic T cells, undergo only limited fratricide and can be expanded long-term ex vivo.

7 age increase in load that is attributable to fratricide and determine the parameters that should be m

8 ic disruption of the CD7 gene prevented this fratricide and enabled expansion of CD7 CAR T cells with

9 ve cytotoxicity and proliferation because of fratricide and not due to the absence of a 2B4-dependent

10 cell death in Th1/Th2 effectors include both fratricide and suicide.

11 tion-associated genes related to competence, fratricide, and the transparent colony phenotype.

12 Here, we describe fratricide as the governing principle behind gelatinase

13 Fratricide between CD8(+) T lymphocytes is known to occu

14 We suggest that Tm-cell fratricide by Fas-mediated apoptosis results in a densit

15 We show that, surprisingly, fratricide can lead either to an increase or a decrease

16 Importantly, however, the fratricide conferred by SLAMF7-CAR T cells spares the SL

17 2B4-mediated inhibition of NK-cell fratricide explains some of the paradoxes of 2B4 functio

18 it is not known what effect, if any, T-cell fratricide has on the course of infection.

19 ve HTLV-I-positive patients considered here, fratricide has probably caused an increase in equilibriu

20 tion and are susceptible to NK cell-mediated fratricide in a perforin- and NKG2D-dependent manner.

21 We also show that NK-NK fratricide in the absence of 2B4-CD48 interaction is dep

22 Fratricide killing, prevalent in wild-type cells, is cal

23 This process of glial 'fratricide' may provide a basis for the secondary propag

24 multiple turnovers, resulting in "molecular fratricide." N-bromoacetyltryptamine should serve as a u

25 ets (RIP-Fas-L) as a result of Fas-dependent fratricide of beta-cells after transfer of diabetogenic

26 mic disruption of a target antigen overcomes fratricide of CAR T cells and establishes the feasibilit

27 nd malignant T cells, potentially leading to fratricide of CAR T cells or profound immunodeficiency.

28 m APCs in an Ag-specific fashion, leading to fratricide of programmed death 1-expressing, neighboring

29 T cells and show that they induce selective fratricide of SLAMF7(+/high) NK cells, CD4(+) and CD8(+)

30 lls, failed to trigger a self-MHC-restricted fratricide of T cells, and was associated with toxicity

31 t the qualitative and quantitative effect of fratricide on HTLV-I equilibrium proviral load.

32 We also investigate the effect of fratricide on the probability of viral clearance.

33 Finally, we show that fratricide reduces the probability of viral clearance.

34 o T cell-bound HLA-I molecules, allowing for fratricide representation and activation.

35 ver-infiltrating cells, pointing to death by fratricide that causes almost complete disappearance of

36 We derive the conditions necessary for fratricide to cause a decrease in load and deduce that,

37 athematical techniques to investigate T-cell fratricide with particular reference to HTLV-I infection

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