ライフサイエンスコーパス: free fatty acid

1 -oxidation, resulting in increased levels of free fatty acid.

2 ction profiles but shared elevated levels of free fatty acids.

3 epatic uptake of adipose-derived circulating free fatty acids.

4 tabolites in muscle and elevated circulating free fatty acids.

5 despite normal insulin suppression of plasma free fatty acids.

6 the hydrolysis of TGs to diacylglycerols and free fatty acids.

7 y hydrolyses glycerophospholipids to release free fatty acids.

8 as a control group with elevated circulating free fatty acids.

9 acids, trans-isomers, and specific released free fatty acids.

10 of the nascent acyl chains to the medium as free fatty acids.

11 mones, and circulating levels of glucose and free fatty acids.

12 ial role of RpfB is uptake and activation of free fatty acids.

13 l metabolites and increased levels of plasma free fatty acids.

14 inantly triacylglycerols, sterol esters, and free fatty acids.

15 mpanied by the increase of phospholipids and free fatty acids.

16 uring this period, changes in the content of free fatty acids (1.4-3.8 mg/g oil), TBARS values (8.8-1

17 or of their actions is the G protein-coupled free fatty acid 2 (FFA2) receptor, and this has been sug

18 of function of these on the closely related free fatty acid 2 receptor, and a dearth of FFA3-selecti

19 oles of the short chain fatty acid receptor, free fatty acid 3 receptor (FFA3), has been severely lim

20 hermogenesis and can consume, in addition to free fatty acids, a very high amount of glucose from the

21 In pxa1 mutants, in which free fatty acids accumulated rapidly under extended dark

22 In addition to being nutrients, free fatty acids act as signaling molecules by activatin

23 ssue (VAT), intrahepatic lipid (IHL), plasma free fatty acids, adipose cell diameter, and percentage

24 , but the ability of this solvent to extract free fatty acids also decreases.

25 Culturing renal proximal tubular cells with free fatty acid and FXR agonists showed that FXR activat

26 onary sinus blood sampled to measure cardiac free fatty acid and glucose oxidation and lactate uptake

27 more impaired GT, lower plasma T3 and higher free fatty acid and hepatic triglycerides compared with

28 isted aqueous process had a lower content of free fatty acid and lower color imparting components tha

29 on, and growth hormone levels increased, and free fatty acids and 3-hydroxybutyrate concentrations an

30 nd their deaminated metabolites, and lowered free fatty acids and acylcarnitines.

31 ations in glucose metabolism, divergences in free fatty acids and carnitine conjugated lipid levels,

32 ding the first reaction, oxidative terpenes, free fatty acids and carotenoids degradation products we

33 cium (Ca(2+)) depletion induced by saturated free fatty acids and cytokines causes beta-cell ER stres

34 Levels of secondary bile acids, glucose, free fatty acids and dipeptides decrease, whereas those

35 gether with triacylglycerides, diglycerides, free fatty acids and ergosterol in salmon oil.

36 inflammation markers, as well as individual free fatty acids and free amino acids, were measured in

37 hondrial dysfunction; however, the impact of free fatty acids and glucose on mitochondrial traffickin

38 d in HF because baseline oxidation levels of free fatty acids and glucose were, respectively, >70% lo

39 lipases are added to transform the fat into free fatty acids and glycerol, which elute at the end of

40 that hydrolyze triacylglycerols (TAGs) into free fatty acids and glycerol.

41 s also suggest that an accumulation of liver free fatty acids and hepatic lipotoxicity marked by an e

42 to ambient UFP (diameter < 180 nm) increased free fatty acids and lipid metabolites in the mouse smal

43 ugh generation of small neoantigens, such as free fatty acids and lysophospholipids, from common phos

44 rolyzes mammalian cell membranes, liberating free fatty acids and lysophospholipids.

45 Conversely, free fatty acids and monoacylglycerides significantly in

46 ols in rapeseed oil by the esterification of free fatty acids and monoacylglycerols.

47 easant taste and odor) due to the binding of free fatty acids and peroxide compounds.

48 An adsorption mechanism of free fatty acids and peroxides is proposed.

49 ilic molecules are observed in obesity (e.g. free fatty acids and phosphatidic acid) and plastics exp

50 els of cytotoxic lipid intermediates such as free fatty acids and phosphatidic acid, suggesting a buf

51 However, treatment did reduce total free fatty acids and phospholipids and was dose dependen

52 mediates stemming from exposure to saturated free fatty acids and rescues hepatocytes from death.

53 Activation of endogenous free fatty acids and the subsequent reduction of fatty a

54 s was observed by analysing amount of formed free fatty acids and their antioxidant capacities.

55 ease also elevates the levels of circulating free fatty acids and thus causes IR in insulin-sensitive

56 actions: polar lipids (PL), diacylglycerols, free fatty acids and triacylglycerols (TAG) using thin l

57 Circulating free fatty acids and triglycerides decrease while bile a

58 oups and decreased the circulating levels of free fatty acids and triglycerides in the HFD-FBX4w anim

59 iers of GCK mutations showed lower levels of free fatty acids and triglycerides than healthy control

60 sphingolipids, amino acids and derivatives, free fatty acids, and bile acid.

61 atrix consist of ceramides, cholesterol, and free fatty acids, and can form two coexisting crystallin

62 ters, squalene, ceramides, free cholesterol, free fatty acids, and cholesterol and retinyl esters.

63 , pyrimidines, phospholipids, sphingolipids, free fatty acids, and glycerolipids) which were mapped t

64 ized triglyceride monomers, diglycerides and free fatty acids, and induction period based on Rancimat

65 Elevated levels of glucose, free fatty acids, and inflammatory cytokines due to diab

66 o lipid biosynthesis, including cholesterol, free fatty acids, and neutral and acidic phospholipids.

67 lesterol, cholesterol esters, triglycerides, free fatty acids, and phosphatidylcholine, which is bloc

68 hepatocyte death upon exposure to saturated free fatty acids, and secretion of both IL-8 and TNFalph

69 increase in fasting plasma LDL cholesterol, free fatty acids, and total ketone bodies by 25, 49, and

70 dothelium treated with exogenous unsaturated free fatty acid (arachidonic acid).

71 d acid lipase expression and accumulation of free fatty acids are also present in a Pex19-deficient p

72 Exogenously added free fatty acids are rapidly adsorbed by mLDs and concur

73 the strong UV absorption properties of this free fatty acid as well as of the glycerophospholipids h

74 f4)-induced lipotoxicity and accumulation of free fatty acids as the cause for mitochondrial damage i

75 nalyses indicate that TAG lipolysis releases free fatty acids at a time that correlates well with mei

76 nificantly different between groups included free fatty acids, bile acids, and amino acid metabolites

77 phatidic acid into lysophosphatidic acid and free fatty acid (both implicated in metastasis), was inc

78 -L-methionine (SAM) dependent methylation of free fatty acids, but FAME production by this route has

79 consumption, fasted blood glucose or plasma free fatty acids, but fasted plasma insulin and the home

80 e tissue insulin sensitivity (suppression of free fatty acids by insulin) showed a continuous worseni

81 We also investigated whether circulating free fatty acid (cFFA) levels predicted the magnitude of

82 To evaluate the impact of free fatty acids compared to hyperglycemia on mitochondr

83 ctor, and the ability of the lipase to alter free fatty acid composition and sensory characteristics

84 Maternal EGP per unit insulin and plasma free fatty acid concentration during hyperinsulinemia mo

85 dams also had lower serum glucose and higher free fatty acid concentrations than controls on GD 21.

86 The increases in phospholipid and free fatty acid content were observed with hydrolysis ti

87 Sensitivity analysis showed that free fatty acid content, peroxide value, L( *)Cab( *)hab

88 duce hepatic diacylglycerol, cholesterol, or free fatty acid content; improve histologic measures of

89 The free fatty acids content of sesame oil after processing

90 gnificant increase in the diacylglycerol and free fatty acid contents as well as in the number of deg

91 e, C-peptide, fructosamine, triglyceride and free fatty acid contents decreased by 44.7%, 46.2%, 4.0%

92 Free fatty acid contents of sprouted soybean oil were fo

93 fication indexes, beyond moisture, lipid and free fatty acids contents, and density.

94 es to the refining process, as the amount of free fatty acids could be reduced significantly from 2%

95 oblasts derived from ATGL KO mice, exogenous free fatty acids did not affect insulin sensitivity.

96 The use of molar excesses of the free fatty acids did not improve direct esterification r

97 to their coenzyme A derivatives, accumulates free fatty acids during the stationary phase of growth.

98 Acute free fatty acid elevation therefore induces a redistribu

99 l that the coexistence of hypoxia along with free fatty acids exacerbates macrophage-mediated inflamm

100 h and inflammatory responses after saturated free fatty acid exposure by activating NF-kappaB through

101 esterification of vanillyl alcohol (VA) with free fatty acids (FA) and coconut oil (CO) as acyl donor

102 The obtained data showed esterified and free fatty acids, fatty alcohol, sterols, alkanes and ar

103 (TG) levels and HOMA-IR and positively with free fatty acid (FFA) and HDL after control for age and

104 ffects of high as opposed to low circulating free fatty acid (FFA) and triglyceride levels in patient

105 with excess body weight and elevated plasma free fatty acid (FFA) concentrations.

106 release, triglyceride and glycogen contents, free fatty acid (FFA) content and release, and cholester

107 fatty acid and triacylglycerol composition, free fatty acid (FFA) content, peroxide index, thermal p

108 cycle were analyzed for 3-MCPD esters, GEs, free fatty acid (FFA) contents, specific extinction at 2

109 tal volatile base-nitrogen (TVB-N) value and free fatty acid (FFA) formation.

110 nistration significantly reduced insulin and free fatty acid (FFA) levels (P < 0.001) and ameliorated

111 increased adipose lipolysis, elevated plasma free fatty acid (FFA) levels, and impaired insulin signa

112 of CD36/fatty acid translocase and elevated free fatty acid (FFA) levels.

113 oncomitant increased 'lyso'-lipid as well as free fatty acid (FFA) levels.

114 Insulin resistance and dysregulation of free fatty acid (FFA) metabolism are core defects in typ

115 irectly or indirectly, determine the rate of free fatty acid (FFA) oxidation.

116 cell function and dysfunction in a model of free fatty acid (FFA) palmitate-induced oxidative stress

117 atty acids from chylomicrons into the plasma free fatty acid (FFA) pool is an important source of FFA

118 of biologic responses through members of the free fatty acid (FFA) receptor family, which includes FF

119 , finding several triacylglyceride (TAG) and free fatty acid (FFA) species to be significantly increa

120 nstrated that CD36 facilitates cell membrane free fatty acid (FFA) transport, but its role in human m

121 Alterations in hepatic free fatty acid (FFA) uptake and metabolism contribute t

122 p4 and Fabp5 (Fabp4/Fabp5) impairs exogenous free fatty acid (FFA) uptake by CD8(+) TRM cells and gre

123 When applied to free fatty acid (FFA) uptake in 3T3-L1 adipocytes, this

124 (FATP1) to plasma membrane, and thus greater free fatty acid (FFA) uptake, in adipocyte cell models.

125 ted receptor alpha (PPARalpha) activation by free fatty acid (FFA), and cAMP response element-binding

126 Neprilysin contributes to free fatty acid (FFA)-induced cellular dysfunction in no

127 at this defect would be exacerbated by acute free fatty acid (FFA)-induced insulin resistance.

128 Apoptosis and free fatty acid (FFA)-induced lipotoxicity are important

129 hox activation of calpain-1 degrades Erk5 in free fatty acid (FFA)-stressed cardiomyocytes, whereas t

130 sis of ceramide to produce sphingosine and a free fatty acid (FFA).

131 centration of triacylglycerols, DAG, MAG and free fatty acids (FFA) and the concentration of saturate

132 pression of SRA inhibits ATGL expression and free fatty acids (FFA) beta-oxidation.

133 ucted: (i) fish oil extraction, (ii) Omega-3 free fatty acids (FFA) concentration (low temperature wi

134 Free fatty acids (FFA) content of beer affects the abili

135 pothesis that an inherent increase in plasma free fatty acids (FFA) in the HFD together with an HFD-i

136 We measured the incorporation of systemic free fatty acids (FFA) into circulating very low-density

137 es (TBARS), fluorescence compounds (OFR) and free fatty acids (FFA) were evaluated throughout the sto

138 culating and tissue levels of triglycerides, free fatty acids (FFA), and leptin?

139 on, for total lipid content and composition, free fatty acids (FFA), thiobarbituric acid reactive sub

140 d, largely due to a reduction in circulating free fatty acids (FFA).

141 uated Adipo-IR (fasting and mean OGTT plasma free fatty acid [FFA] x insulin concentrations), periphe

142 crophages results in increased intracellular free fatty acids (FFAs) and elevated expression of uncou

143 with a higher abundance of saturated C16-C20 free fatty acids (FFAs) and long polyunsaturated complex

144 Free fatty acids (FFAs) are known to induce lipoapoptosi

145 In cultured podocytes, the presence of free fatty acids (FFAs) associated with serum albumin st

146 Nutrients such as free fatty acids (FFAs) contribute to precise regulation

147 ch in n-3 were produced by extraction of the free fatty acids (FFAs) from flaxseed oil, concentration

148 Saturated free fatty acids (FFAs) have complex effects on the isle

149 ary damage, as well as increased circulating free fatty acids (FFAs) in NAFLD, we hypothesized the in

150 The importance of free fatty acids (FFAs) in wort has been known for a lon

151 evated concentrations of the polyunsaturated free fatty acids (FFAs) linoleic and alpha-linolenic aci

152 Serum free fatty acids (FFAs) profile is highlighted in its as

153 We investigated the mechanism by which free fatty acids (FFAs) regulate MIR122 expression and t

154 s in response to an elevated plasma ratio of free fatty acids (FFAs) to albumin when proteinuria reac

155 scribe longitudinal relations of serum total free fatty acids (FFAs) to insulin resistance (IR) and c

156 Physiologically relevant free fatty acids (FFAs) were analyzed by UV-laser desorp

157 Circulating free fatty acids (FFAs) were less suppressed in IR than

158 flecting chylomicrons of intestinal origin), free fatty acids (FFAs), insulin, glucose, glucagon, glu

159 s is responsible for the slow suppression of free fatty acids (FFAs), which in turn is responsible fo

160 nied by dyslipidemia with elevated levels of free fatty acids (FFAs).

161 0, were measured in plasma phospholipids and free fatty acids (FFAs).

162 gnize and are activated by nonesterified or "free" fatty acids (FFAs).

163 The chemical components (hydrocarbons - HCs, free fatty acids - FFAs, free fatty alcohols - FALs and

164 beta-carotene) and lipid digestion products (free fatty acids, FFAs, and monoacylglycerides, MAGs) du

165 dance of biomarkers such as HDL cholesterol, free fatty acids, FGF21, bilirubin, and lactate depend o

166 at autophagy is necessary for the release of free fatty acids from intracellular stores within neutro

167 (2)H5]glycerol turnover rate and circulating free fatty acids, glycerol, and triglycerides), lipid ox

168 (TAG) and glucose uptake decreased, and the free fatty acid/glycerol ratio increased during the anta

169 the local regulation of ANGPTL4 via AMPK and free fatty acids have key roles in governing lipid homeo

170 obese, remained insulin sensitive, had lower free fatty acid in plasma, were protected against athero

171 nd triglycerides to generate cholesterol and free fatty acids in cellular lysosomes.

172 compounds particularly medium and long-chain free fatty acids in cooked cured pork ham exudates.

173 nal processes also enabling the reduction of free fatty acids in crude oils.

174 bryos, which resulted in the accumulation of free fatty acids in dry seeds.

175 ell as SMc01003 contribute to the release of free fatty acids in S. meliloti, neither one can use pho

176 The defect in the mobilization of free fatty acids in the elderly is accompanied by increa

177 inor components including diacylglycerol and free fatty acids in the heated samples.

178 ough transport phenomena not yet understood, free fatty acids in the oil binding medium migrate throu

179 Neutralization of free fatty acids in the vegetable oil before the deodori

180 yao, has been shown to bind leukotrienes and free fatty acids in vitro Therefore, here we assessed wh

181 It is established that long-chain free fatty acids includingomega-3 fatty acids mediate an

182 oup, the EPA and DHA contributions to plasma free fatty acids increased (P = 0.0003 and P = 0.003, re

183 ity, plasmatic and hepatic triglycerides and free fatty acids, increased adiponectin-to-leptin ratio,

184 found that stimulation of A2B ARs suppressed free fatty acid-induced deleterious inflammatory and met

185 We also defined the role of FSP27 in free fatty acid-induced insulin resistance in adipocytes

186 Additionally, BBR decreased the free fatty acid-induced lipid accumulation and tunicamyc

187 wed that FXR activation protected cells from free fatty acid-induced oxidative stress and endoplasmic

188 Free fatty acid induction of inflammation and cell death

189 a radical increase in the esterification of free fatty acids into triacylglycerol.

190 thioester bonds by thioesterases to produce free fatty acids is important for dictating the diversit

191 enzyme responsible for de novo synthesis of free fatty acids, is up-regulated in many cancers.

192 encompass increased 1) circulating levels of free fatty acid, ketone bodies, and long-chain acylcarni

193 ent and selective agonist for the long chain free fatty acid (LCFA) receptor 4 (FFA4; previously G pr

194 r the first time that long chain unsaturated free fatty acids (LCUFAs) present in Salmonella growth m

195 se increased responsiveness of host cells to free fatty acid, leading to a radical increase in the es

196 ase activity results in a reduction of serum free fatty acids, leading to improved peripheral insulin

197 3 phosphorylation positively correlated with free fatty acid level and measures of insulin sensitivit

198 ns, metabolic function, and triglyceride and free fatty acid levels and reverses weight gain in T2DM.

199 s, and chronically elevated blood long chain free fatty acid levels are thought to contribute to beta

200 olytic effect of insulin, and reduces plasma free fatty acid levels during OGTT.

201 Consistently, free fatty acid levels were reduced in portal but not in

202 al fat were positively correlated with serum free fatty acid levels, suggesting that collagen alpha1

203 leaner with reduced plasma triglyceride and free fatty acid levels.

204 alter growth hormone, cortisol, glucagon, or free fatty acid levels.

205 mplex lipids with relatively small amount of free fatty acids (<15%).

206 six triacylglycerides (m/z 726-860) and six free fatty acids (m/z 271-345).

207 hanced lipolysis, elevated concentrations of free fatty acids, maximal beta-oxidation, and mitochondr

208 itga1(-/-) mice was associated with altered free fatty acid metabolism.

209 nolenoylethanolamides, but not corresponding free fatty-acid metabolites, contributed to the reversib

210 ssiveness were identified, including various free fatty acids, metabolites, and complex lipids such a

211 to characterize the molecular speciation of free fatty acids, monoacylglycerol species, unmodified a

212 rated a negative association between fasting free fatty acids (NEFAs) and insulin secretion.

213 nsidered that accumulation of nonesterified (free) fatty acids (NEFAs) in the first trimester of preg

214 with the fastest release and lower amount of free fatty acids observed at 100 MPa.

215 eptors are those that respond to short-chain free fatty acids of carbon chain length 2-6.

216 an adipocytes against the adverse effects of free fatty acids on insulin signaling.

217 ma BCAs in both insulin groups but increased free fatty acids only in the high insulin group, however

218 h a 41.3+/-6.7% and 32.5+/-10.9% increase in free fatty acid oxidation and a 31.3+/-9.2% and 41.4+/-8

219 abolism in normal dogs, whereas they enhance free fatty acid oxidation and reduce glucose oxidation i

220 patic de novo lipogenesis, decreased hepatic free fatty acid oxidation, or decreased very-low-density

221 In these subjects fasting levels of the free fatty acid palmitate are raised.

222 We investigated effects of a saturated free fatty acid, palmitic acid (PA), on endothelial cell

223 f 32.6%, 46 ppm total phenolic content, 0.7% free fatty acids, peroxide value of 0.2 and 11.5 h of Ra

224 The following analyses were carried out: free fatty acids, peroxide value, specific extinction at

225 rous cow milk fat was evaluated by measuring Free Fatty Acids, peroxide value, Thiobarbituric Acid va

226 method was developed for elucidation of the free fatty acid profile in mussel samples, avoiding a pr

227 to determine oxidation (headspace analysis, free fatty acids profile, peroxide value and anisidine v

228 eted metabolomic measurement of serum BA and free fatty acid profiles was applied to sera of 381 indi

229 Indeed, whereas the plasma free fatty acid Ra and Rd were twice those of YL in both

230 the absence of biliary CA leads to increased free fatty acids reaching the ileal L cells.

231 colon, the involvement of the SCFA receptor free fatty acid receptor (FFA)3, one of the free fatty a

232 The contribution of an SCFA receptor, free fatty acid receptor (FFA)3, to the enteric nervous

233 Free fatty acid receptor (FFAR) 1 (FFA1), which binds lo

234 The free fatty acid receptor 1 (FFA1 or GPR40) is establishe

235 The free fatty acid receptor 1 (FFA1/GPR40) is a potential t

236 in-coupled receptor 40 (GPR40) also known as free fatty acid receptor 1 (FFAR1) is highly expressed i

237 Moreover, we identify a lipid sensor, free fatty acid receptor 1 (Ffar1), that curbs glucose u

238 lin secretion from isolated human islets via free fatty acid receptor 1 (FFAR1/GPR40).

239 second long-chain free fatty acid receptor, free fatty acid receptor 1, the simple chemical structur

240 ot the other candidate transporters CD36 and free fatty acid receptor 1.

241 Free Fatty Acid Receptor 2 (FFA2) is a beta cell-express

242 Free fatty acid receptor 2 (FFA2) is expressed on entero

243 The free fatty acid receptor 2 (FFA2/GPR43) is considered a

244 idue produced a signalling-biased variant of Free Fatty Acid Receptor 2 in which Gi-mediated signalli

245 Free Fatty Acid Receptor 2 is a GPCR activated by short

246 s to homology models of both human and mouse Free Fatty Acid Receptor 2 suggested that a single lysin

247 d SCFAs, propionate, activates ileal mucosal free fatty acid receptor 2 to trigger a negative feedbac

248 The free fatty acid receptor 4 (FFA4 or GPR120) has appeared

249 there is great interest in the potential of free fatty acid receptor 4 (FFA4) as a novel therapeutic

250 The free fatty acid receptor 4 (FFA4, previously GPR120) is

251 Next, we hypothesize that free fatty acid receptor 4 (Ffar4), a functional recepto

252 free fatty acid receptor (FFA)3, one of the free fatty acid receptor family members, has not been cl

253 By demonstrating the expression of the free fatty acid receptor G-coupled protein receptor 40 (

254 upled receptor 40 (GPR40), a Galphaq-coupled free fatty acid receptor linked to MAPK networks and glu

255 614 did not antagonize the second long-chain free fatty acid receptor, free fatty acid receptor 1, th

256 Free fatty acids receptor 3 (FFA3, GPR41) and 2 (FFA2, G

257 Human GPR40 receptor (hGPR40), also known as free fatty-acid receptor 1 (FFAR1), is a G-protein-coupl

258 Free fatty acid receptors (FFARs) for long-chain fatty a

259 Free fatty acid receptors 1 and 4 (FFAR1 and FFAR4) are

260 -derived short-chain fatty acids (SCFAs) and free fatty acid receptors including GPR43 are thought to

261 receptors 1 and 4 (FFAR1 and FFAR4) are two free fatty acid receptors under increasing investigation

262 ycerol, glycerol, and medium- and long-chain free fatty acids, reflective of lipid mobilization and c

263 Free fatty acids regulate numerous cellular processes, l

264 rom SGA newborns, whereas GPR120 (related to free fatty acid regulation) was hypomethylated in placen

265 The rate of free fatty acid release during lipid digestion decreased

266 ype cytokine signaling in adipocytes induces free fatty acid release from visceral adipocytes, thereb

267 Free fatty acid release profiles suggested that the stab

268 Free fatty acids released from the plastid need to be co

269 d digestion were examined by determining the free fatty acids released from various oil-in-water emul

270 In the wild type, in which free fatty acids remained low and unchanged under dark t

271 The incorporation of excess saturated free fatty acids (SFAs) into membrane phospholipids with

272 e cholesterol, phospholipids, ceramides, and free fatty acids showed distributions in good agreement

273 Furthermore, the peroxide value and free fatty acids showed that the quality of the food pro

274 emulsion (MPE) droplets comprising particle-free fatty acid-stabilized apertures.

275 FLD, causes more oxidative damage than other free fatty acids such as palmitic acid, and mediates sel

276 weight, serum cholesterol, triglycerides and free fatty acids, suggesting altered lipid metabolism in

277 Free fatty acid supplementation produced small but signi

278 lights the critical role of plasma saturated free fatty acids that are abundant in the WD with respec

279 In this study, we report that saturated free fatty acids that are elevated in obesity alter reso

280 ulated metabolic process called lipolysis to free fatty acids that serve as energy substrates for bet

281 se (FAAH) degrades NAE into ethanolamine and free fatty acid to terminate its signaling function.

282 ing a hybrid fatty acid synthase shifted the free fatty acids to a medium chain-length scale.

283 otein-coupled receptor that binds long-chain free fatty acids to enhance glucose-dependent insulin se

284 y by catalysing uptake and activation of the free fatty acids to give acyl-CoAs that can be utilized

285 wn adipose tissue (BAT) utilizes glucose and free fatty acids to produce heat, thereby increasing ene

286 ated process that maintains plasma levels of free fatty acids to supply energy to cells.

287 w that autophagy-mediated lipolysis provides free fatty acids to support a mitochondrial respiration

288 A2 (PLA2) to mobilize lysophospholipids and free fatty acids to sustain fatty acid oxidation and oxi

289 of Sinorhizobium meliloti, unable to convert free fatty acids to their coenzyme A derivatives, accumu

290 layed a decrease in fasting plasma levels of free fatty acid, triglyceride, and insulin as well as im

291 hat cathepsin B regulates VLDL secretion and free fatty acid uptake via cleavage of LFABP, which occu

292 cising muscle is mediated by elevated plasma free fatty acids via peroxisome proliferator-activated r

293 nhanced fat oxidation, circulating levels of free fatty acids were reduced in the HFD-fed AdSod2 KO m

294 ties responsible for the generation of these free fatty acids were unknown in rhizobia.

295 ow volatility, such as medium and long-chain free fatty acids, whereas compounds with high volatility

296 other legumes, LOX from lupin only converted free fatty acids, whereas trilinolein and beta-carotene

297 to endogenous lipase-dependent generation of free fatty acids, which destroy the viral lipid envelope

298 The effect of free fatty acids with different chain lengths or unsatur

299 C2C12 myotubes exposed to excess free fatty acids with or without UT were also evaluated

300 ow hypoxia modulates the response of ATMs to free fatty acids within obese adipose tissue is limited.