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1 Elevated temperature and CO2 also impaired freezing tolerance.
2 ay present opportunities for improving plant freezing tolerance.
3 s the major process that prepares plants for freezing tolerance.
4 whereas overexpression of PLDdelta increased freezing tolerance.
5 naling has been postulated to play a role in freezing tolerance.
6 that the SFR2 gene product is essential for freezing tolerance.
7 in transgenic tomato plants did not increase freezing tolerance.
8 rgeted genes, the CBF regulon, that increase freezing tolerance.
9 F regulon, resulting in an increase in plant freezing tolerance.
10 significant reduction in plant chilling and freezing tolerance.
11 ecrease in endogenous raffinose and impaired freezing tolerance.
12 genes), which contributes to an increase in freezing tolerance.
13 However, this comes at the cost of impaired freezing tolerance.
14 pression of CBF-targeted genes that increase freezing tolerance.
15 absent under long photoperiod, and increased freezing tolerance.
16 limation, define genes that are required for freezing tolerance.
17 uces COR gene expression and increases plant freezing tolerance.
18 cleus to activate genes needed for increased freezing tolerance.
19 of several cold-regulated genes involved in freezing tolerance.
20 ow that BRs participate in the regulation of freezing tolerance.
21 affected specifically in the development of freezing tolerance.
22 raising the question of SFR2 function beyond freezing tolerance.
23 regulon, which contribute to an increase in freezing tolerance.
24 han 100 genes, the CBF regulon, which impart freezing tolerance.
25 gene, inducing flowering with a reduction of freezing tolerance.
26 which reprograms gene expression to increase freezing tolerance.
27 e proteins had on global gene expression and freezing tolerance.
28 nes that are required for the acquisition of freezing tolerance.
29 r subunits, as does cold acclimation-induced freezing tolerance.
30 tional activators of gene pathways imparting freezing tolerance.
31 es showed improved germination and increased freezing tolerance.
32 outside the CBF pathway, and increase plant freezing tolerance.
33 osome underlying important traits, including freezing tolerance.
34 leading from cold perception to chilling and freezing tolerance.
35 induction of CBF-targeted genes that impart freezing tolerance.
36 of the CBF regulon results in an increase in freezing tolerance.
37 camta1 camta3 mutant plants are impaired in freezing tolerance.
38 ssion of the CBF gene regulon, which imparts freezing tolerance.
39 nce, whereas MPK3/MPK6 activation attenuates freezing tolerance.
40 e been shown to control genetic pathways for freezing tolerance.
41 cold acclimatization and the acquisition of freezing tolerance.
42 ng stress whereas its overexpression reduces freezing tolerance.
43 ation of CBF genes and in the development of freezing tolerance.
44 to chilling stress and defective in acquired freezing tolerance.
45 anscription factor required for chilling and freezing tolerance.
46 gulon) that act in concert to increase plant-freezing tolerance.
47 lon, which act to bring about an increase in freezing tolerance.
50 ions as a negative regulator of constitutive freezing tolerance and cold acclimation in Arabidopsis t
51 ith ET biosynthesis to modulate constitutive freezing tolerance and cold acclimation in Arabidopsis.
52 nd that MYB88 and MYB124 positively regulate freezing tolerance and cold-responsive gene expression i
56 uercus series Virentes) were associated with freezing tolerance and growth rate, and whether species
58 cold-responsive gene transcription, acquired freezing tolerance and plant resistance to chilling unde
60 ity is that Arabidopsis accessions differ in freezing tolerance and that those collected from colder
61 in Suc solution in the dark at 2 degrees C, freezing tolerance and the incidence of freeze-induced l
62 based on root elongation, quantification of freezing tolerance and the use of electrolyte leakage ex
64 ed photosynthetic rates, leaf carbohydrates, freezing tolerance, and proteins involved in photosynthe
67 se in CBF expression is sufficient to confer freezing tolerance at temperatures higher than those req
69 pha-Gal in petunia results in an increase in freezing tolerance at the whole-plant level in nonacclim
70 mote proper cold-induced gene expression and freezing tolerance before and after cold acclimation.
71 so reduces the capacity of plants to develop freezing tolerance but does not impair the vernalization
72 sis at low temperature did not contribute to freezing tolerance, but had a major role in configuring
75 disordered protein that contributes to leaf freezing tolerance by stabilizing cellular membranes.
76 nt growth and development, and for a part of freezing tolerance, by affecting the activity of genes i
77 ortion of the variance for the nonacclimated freezing tolerance can be best explained by an additive-
80 ited stunted growth, poor yield, and greater freezing tolerance compared to non-transformed 'Golden P
81 also have strong constitutive heat shock and freezing tolerance compared with mountain plants, where
82 nts initiate their reproductive development, freezing tolerance decreases, suggesting a connection be
83 ts native to cold climates acquire increased freezing tolerance during exposure to low nonfreezing te
84 ate climate zones are able to increase their freezing tolerance during exposure to low, above-zero te
89 E TO FREEZING 2 (SFR2), a gene essential for freezing tolerance in Arabidopsis, encodes a galactolipi
90 /lhy-21 double mutation resulted in impaired freezing tolerance in both nonacclimated and cold-acclim
94 -regulation of the CBF pathway and increased freezing tolerance in preparation for coming cold temper
96 During cold acclimation plants increase in freezing tolerance in response to low non-freezing tempe
97 e process whereby certain plants increase in freezing tolerance in response to low nonfreezing temper
98 tion, the process whereby plants increase in freezing tolerance in response to low nonfreezing temper
100 y plants, including Arabidopsis, increase in freezing tolerance in response to low, nonfreezing tempe
102 s resulted in a 5.5 degrees C improvement in freezing tolerance in the absence of cold acclimation.
103 mponents of this survival in crop plants are freezing tolerance in the nonacclimated state and cold a
104 xpression of the Nicotiana PK1 gene enhances freezing tolerance in transgenic maize plants that are n
105 pression of CBF-targeted genes and increased freezing tolerance indicating that LeCBF1 encodes a func
106 aused a small, but reproducible, increase in freezing tolerance, indicating a role for the ZAT12 regu
107 da') were compared with a goal to reveal how freezing tolerance is achieved in this distinctive overw
109 other than CBF1, CBF2 and CBF3, and whether freezing tolerance is dependent on a functional CBF-CRT/
113 ecotypes that condition local adaptation and freezing tolerance map to a region that includes the C-r
114 t ADA2b may directly or indirectly repress a freezing tolerance mechanism that does not require the e
115 estion thus raised is whether differences in freezing tolerance might contribute to local adaptation
116 2.6 degreesC by cold acclimation whereas the freezing tolerance of 26 mutant lines ranged from -6.8 d
117 We screened for mutations deleterious to the freezing tolerance of Arabidopsis thaliana (L.) Heynh. e
118 idopsis CBF-targeted genes and increases the freezing tolerance of both nonacclimated and cold-acclim
121 nduced COR gene expression and increased the freezing tolerance of nonacclimated Arabidopsis plants.
123 The minimum source temperature predicted the freezing tolerance of populations under temperate condit
127 T CBF2 protein also contributes to the lower freezing tolerance of the IT plants compared with the SW
129 that of 'Golden Promise' and paralleled the freezing tolerance of the winter hardy barley 'Dicktoo'.
131 suggesting that quantitative trait loci for freezing tolerance previously mapped on this chromosome
133 mated conditions, sfr4 protoplasts possessed freezing tolerance similar to that of wild type, with th
134 lay central roles in low-temperature-induced freezing tolerance, spike architecture and hormone metab
136 ession strongly correlated with increases in freezing tolerance, suggesting its involvement in the de
137 -1 is sufficient to determine differences in freezing tolerance, suggesting that quantitative trait l
138 ezing temperatures results in an increase in freezing tolerance that involves action of the C-repeat
139 hly interconnected; and that the increase in freezing tolerance that occurs with cold acclimation is
140 Mutations in the ESK1 gene provide strong freezing tolerance through genetic regulation that is ap
141 ER OF CBF EXPRESSION1, a master regulator of freezing tolerance, thus implicating a potential link be
146 e process whereby certain plants increase in freezing tolerance upon exposure to low temperature.
150 cts of growth temperature and photoperiod on freezing tolerance were most pronounced in plants grown
151 plants like Arabidopsis thaliana increase in freezing tolerance when exposed to low nonfreezing tempe
152 mutant plants are less capable of developing freezing tolerance when treated with low non-freezing te
153 creased expression of CBF genes and enhanced freezing tolerance, whereas constitutive activation of t
154 d the mpk3 mpk6 double mutants show enhanced freezing tolerance, whereas MPK3/MPK6 activation attenua
155 revealed that the WXP1 plants had increased freezing tolerance while the WXP2 plants were more sensi
156 on of one pathway can result in considerable freezing tolerance without activation of other pathways.
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