ライフサイエンスコーパス: frontal

1 neocortical VOI, 1.84% for MUBADA, 1.46% for frontal, 1.98% for temporal, 2.28% for parietal, and 3.2

2 The effects on response times and on frontal action selection mechanisms were similar in pati

3 view that striatal neurons integrate medial frontal activity and are consistent with drift-diffusion

4 as correlated with and dependent upon medial frontal activity.

5 B, although the degree of involvement of the frontal and anterior temporal lobes and the presence of

6 tary cognitive processing that requires both frontal and cerebellar networks that are disrupted in pa

7 icient, 119.8-275; P < .001) in the superior frontal and frontal polar cortices.

8 outcome emerged in cortical (right inferior frontal and inferior parietal/precuneus) and cerebellar

9 res in regions both vulnerable in the middle frontal and inferior temporal gyri (MFG and ITG) and res

10 od disadvantage on the development of dorsal frontal and lateral orbitofrontal cortices as well as th

11 etwork, including auditory sensory, but also frontal and parietal brain regions involved in controlli

12 urbations to topographically organized human frontal and parietal cortex during WM maintenance cause

13 nd information about task representations in frontal and parietal cortex, but there was no difference

14 ats, we examined the responses of neurons in frontal and parietal cortices during a pulse-based accum

15 n striatal nodes and specific regions within frontal and parietal cortices.

16 nitive control, characterized by less robust frontal and parietal ERP distributions across the respon

17 found in multimodal associative areas in the frontal and parietal lobe than primary regions of sensor

18 quency, whereas connections originating from frontal and parietal regions peaked at beta frequency.

19 ft-dominant activation in the rostral middle-frontal and pars orbitalis inferior-frontal regions arou

20 ared in OSA throughout areas of the superior frontal and prefrontal, and superior and lateral parieta

21 s following the appearance of the "A" and at frontal and right temporal sites during the appearance o

22 ion in two anterior nodes of the DMN (medial frontal and superior frontal) in the non-rest pleasant s

23 cription of a role in stuttering to inferior frontal and superior temporal gyri, caudate, and other s

24 linkage of left perisylvian language areas (frontal and temporal cortex) by way of the arcuate fasci

25 used electrocorticography data obtained from frontal and temporal cortices, permitting detailed spati

26 Findings of structural abnormalities in frontal and temporal lobes, amygdala, and insula are les

27 nalyses revealed that damage predominated in frontal and temporal lobes.

28 erative disorder predominantly affecting the frontal and temporal lobes.

29 rehension engages a cortical network of left frontal and temporal regions.

30 These results reveal how signals from frontal and visual cortex could interact to facilitate o

31 asymmetric synapses does not differ between frontal and visual cortices in either species, but is si

32 rikingly, activity in limbic, right inferior frontal, and inferior parietal areas during imitation of

33 left pars orbitalis inferior-frontal, middle-frontal, and inferior-parietal regions preceded by high-

34 ons of lateral frontal, insular, dorsomedial frontal, and parietal cortex.

35 ntegrity in the uncinate fasciculus, ventral frontal, and right cerebellum regions; and amygdala func

36 her found differential connectivity from the frontal area to the valuation area depending on whether

37 interneurons, whereas association, including frontal, areas are dominated by input-modulating somatos

38 isms (hypertelorism, depressed nasal bridge, frontal bossing), and postaxial polydactyly.

39 stigate a novel strategy to normalize medial frontal brain activity by stimulating cerebellar project

40 reductions in total gray matter and (mainly) frontal brain areas.

41 and 10/12, respectively) than those without frontal brain lesions (1/11, P's < 0.05).

42 onstrated reduced functional connectivity in frontal brain regions and increased functional connectiv

43 outh showed increased NAcc connectivity with frontal brain regions involved in mentalizing.

44 might be effectively coordinated by distant frontal brain regions through synchronized activity in t

45 temporoparietal as well as the left inferior frontal brain regions.

46 f functional connectivity alterations in the frontal brain supports previous investigations postulati

47 l magnetic stimulation to a subject-specific frontal-cingulate reward pathway, this pattern of result

48 e broader involvement of distributed thalamo-frontal circuits in cognition and point to the MD as a p

49 Alzheimer's disease (AD), via frontal compensatory processes, may affect PNS regulatio

50 ge competition recruited a larger network of frontal control and basal ganglia regions than within-la

51 stimulation experiment, where subjects whose frontal control was temporarily disrupted made biased ch

52 tuttering participants in the right inferior frontal cortex (-7.3%; P = .02), inferior frontal white

53 o support emotion perception is the inferior frontal cortex (IFC).

54 FC structures.SIGNIFICANCE STATEMENT Lateral frontal cortex (LFC) is known to play a number of critic

55 y minutes of inphase stimulation over medial frontal cortex (MFC) and right lateral prefrontal cortex

56 nuated delta activity (1-4 Hz) in the medial frontal cortex (MFC) during interval timing.

57 mechanisms of reward signaling by the medial frontal cortex (MFC) have not been resolved.

58 s among orbital frontal cortex (OFC), medial frontal cortex (MFC), and amygdala are thought to underl

59 Orbitofrontal cortex (OFC), medial frontal cortex (MFC), and amygdala mediate stimulus-rewa

60 ltered functional interactions among orbital frontal cortex (OFC), medial frontal cortex (MFC), and a

61 hypothesize that the close coupling between frontal cortex activity and this natural, active primate

62 that this social context-dependent change in frontal cortex activity is supported by several mechanis

63 nd surrounding astrocytic processes in mouse frontal cortex after 6-8 h of sleep, spontaneous wake, o

64 to play a prominent role in ASD, such as the frontal cortex and cerebellum.

65 rting with neural signaling molecules in the frontal cortex and ending in the modulation of developme

66 of cortical surface area are low toward the frontal cortex and high toward the caudo-medial (occipit

67 h and loss of gray matter, especially in the frontal cortex and hippocampus, some focus in drug devel

68 al in areas of early amyloidosis such as the frontal cortex and hippocampus.

69 ierarchical organization of posterior medial frontal cortex and its interaction with the basal gangli

70 recorded single neurons in the human medial frontal cortex and medial temporal lobe while subjects h

71 -specific long noncoding RNA RP1-269M15.3 in frontal cortex and nucleus accumbens basal ganglia, resp

72 cortical thinning in parietal, occipital and frontal cortex and reduced hippocampal volume.

73 levels of tau and TDP-43 were inverse in the frontal cortex and the cerebellum.

74 ated with increased activation in the medial frontal cortex beneath the anode; showing a positive cor

75 for retrospective sensory information, while frontal cortex codes for prospective action.

76 In addition, we found that increased NAcc-frontal cortex connectivity in typically developing yout

77 sting CBF and regional CBF of right superior frontal cortex correlated positively with creatinine-nor

78 nscriptional co-expression (P<0.0001) in the frontal cortex during fetal development and in the tempo

79 its and provides insight into how the medial frontal cortex exerts top-down control of cognitive proc

80 Functional anatomy in frontal cortex has been elusive and controversial.

81 RNA sequencing (RNAseq) analysis of the frontal cortex identified UPF3B-regulated RNAs, includin

82 logopenic variant, within the left inferior frontal cortex in patients with the non-fluent/agrammati

83 ed across cortex revealed a crucial role for frontal cortex in triggering this cortex-wide phenomenon

84 om thalamic neurons projecting to the medial frontal cortex indicated that this phenomenon originates

85 mping activity are disrupted when the medial frontal cortex is inactivated.

86 how critically reconsidering the role of the frontal cortex may further delineate NCCs, (3) advocate

87 cial signaling, we recorded the responses of frontal cortex neurons as freely moving marmosets engage

88 or a novel pattern of neural activity in the frontal cortex of freely moving, naturally behaving, mar

89 uch flexibility, we recorded from the medial frontal cortex of nonhuman primates trained to produce d

90 the GNGT, enhanced response in the inferior frontal cortex of the poly-drug group was found.

91 rticle reviews the effects of lesions to the frontal cortex on the ability to carry out active though

92 edial prefrontal cortex, and lateral orbital frontal cortex predicted treatment response.

93 We analyzed 128 suitable frontal cortex samples, from prion-affected patients (va

94 together with vibrissal motor cortex, vM1 (a frontal cortex target of vS1), while rats compared the i

95 ral areas of parietal, occipitotemporal, and frontal cortex that exhibit action category codes that a

96 e identify a pull-push inhibitory circuit in frontal cortex that originates in vasoactive intestinal

97 en rs31746 and PCDH-beta8 mRNA expression in frontal cortex tissue (P<1 x 10(-5)).

98 combination of cultured cell lines, primate frontal cortex tissue and two human adenocarcinomas, and

99 kami et al. (2017) relate neural activity in frontal cortex to stochastic and deterministic component

100 ed gene expression in the human dorsolateral frontal cortex using RNA- Seq to populate a whole gene c

101 However, activity in the posterior medial frontal cortex was elevated in AN following punishment.

102 y, and perivascular AQP4 localization in the frontal cortex were evaluated.

103 in the cingulate region of the mouse medial frontal cortex, an associative region that matures durin

104 the chromatin landscape in the hypothalamus, frontal cortex, and amygdala of socially challenged mice

105 m three cerebral regions (angular gyrus, mid-frontal cortex, and anterior cingulate gyrus).

106 across regions of the amygdala, hippocampus, frontal cortex, and hypothalamus.

107 d that a node within MDC, located in midline frontal cortex, becomes active during the early stage of

108 rease in inhibitory neurotransmission in the frontal cortex, but not the somatosensory cortex, sugges

109 as applied to one of two targets in the left frontal cortex, one functionally connected (target 1) an

110 eased binding of (11)C-PIB with aging in the frontal cortex, parietotemporal cortex, hippocampus, and

111 nificantly related to patient's outcome were frontal cortex, posterior cingulate cortex, thalamus, pu

112 MV in the anterior cingulate cortex, orbital frontal cortex, temporal pole, and insula, which were co

113 se genes are expressed in the human superior frontal cortex, that heritable genetic factors influence

114 ons were functionally connected to the right frontal cortex, the region most activated in functional

115 ) in the PVN, but not in the hippocampus and frontal cortex, was significantly higher in SHRs than in

116 activity was strong in temporal and inferior frontal cortex, while during low SNR strong entrainment

117 explains fMRI responses in posterior-medial frontal cortex.

118 rly in parietal/sensory regions and later in frontal cortex.

119 l delta rhythms between 1-4 Hz in the medial frontal cortex.

120 l connectivity between temporal and inferior frontal cortex.

121 s are correlated with activity of the medial frontal cortex.

122 pitotemporal, left premotor, and left middle frontal cortex.

123 and 21 human neuronal subpopulations in the frontal cortex.

124 milar inflammatory profile in CHMP2B patient frontal cortex.

125 o examine allele-specific DNA methylation in frontal cortex.

126 rect current stimulation applied to the left frontal cortex.

127 entrainment emerged in premotor and superior frontal cortex.

128 ion of tonic inhibition and performance in a frontal-cortex-dependent reversal-learning task.

129 ffers a potential novel approach to altering frontal cortical activity and exerting pro-cognitive eff

130 y modulating theta phase coupling of distant frontal cortical areas and can contribute to the develop

131 increase of functional connectivity between frontal cortical areas and the motor region of the stria

132 tional control over behavior, which requires frontal cortical cholinergic activity.

133 h of hypoxia caused an expected increase in frontal cortical grey matter perfusion but unexpected pe

134 related probands were investigated to assess frontal cortical inhibition.

135 ue contributions to the variance in superior frontal cortical thickness among all participants.

136 than encoding in medial, superior and middle frontal cortices for novel faces.

137 neus, temporal parietal junction, and medial frontal cortices, there were large differences in neural

138 nnectivity between limbic structures and pre-frontal cortices.

139 hierarchy, from primary sensory and motor to frontal cortices.

140 more in several neocortical areas, including frontal cortices.

141 most sites in superior temporal and inferior frontal cortices.

142 vioral and electrophysiological evidence for frontal delta-mediated top-down control of posterior alp

143 nce of CSS was associated with attention and frontal dysfunction.

144 Frontal electrocorticography [duration: 54 h (34, 66)] f

145 ttention, language, visuospatial, memory and frontal executive functions while presence of CSS was as

146 The frontal eye field (FEF) in prosimian primates was identi

147 The frontal eye field (FEF) is a key brain region to study v

148 in part via its direct projections from the frontal eye field (FEF), an area involved in selective a

149 refrontal cortex (rVLPFC) and the bi-lateral frontal eye field (FEF).

150 utions at a late stage of visual processing [frontal eye field (FEF)] and as a comparison, an early s

151 We examined these contributions in frontal eye field (FEF; N = 51) and as a comparison, an

152 d electrical microstimulation of the macaque frontal eye fields (FEF) modulates the pupillary light r

153 saccadic thresholds of the directly adjacent Frontal Eye Fields (FEF), saccades were only rarely evok

154 Neural activity within the frontal eye fields (FEFs) and intermediate layers of the

155 E STATEMENT The superior colliculus (SC) and frontal eye fields (FEFs) are two of the best-studied ar

156 ger analysis, we further show that the right frontal-eye field (rFEF) exerted feedback control of the

157 being associated with the degree of reduced frontal fast sleep spindle density.

158 The magnitude of the loss of frontal fast sleep spindles in older adults was predicte

159 ottom-up causality correlate with heightened frontal gamma power, they also correlated with increased

160 -up inhibition deficits (reduced posterior-->frontal Granger causality).

161 ue-alpha) and the left dorsolateral superior frontal gyri (item-gamma) on permutation test, where the

162 nd brain activity in the inferior and middle frontal gyri, precuneus, cingulate cortex, caudate, and

163 The inferior frontal gyrus (IFG) is a key cortical hub in the circuit

164 eurocognitive function in the right inferior frontal gyrus (IFG)-one node in a corticothalamic inhibi

165 connectivity (iFC), highlighting the middle frontal gyrus (MFG) for both competencies.

166 ight dorsal anterior cingulate cortex/medial frontal gyrus (R dACC/MFG).

167 rsFC between the DLPFC and the left superior frontal gyrus (SFG) and anterior cingulate cortex; and 3

168 ingulate cortex (ACC) and the right superior frontal gyrus (SFG).

169 otential losses; and increased left inferior frontal gyrus activation when experiencing an actual los

170 as significantly higher in the left inferior frontal gyrus and insula, while GMV was significantly lo

171 ificant activation over left dorsal inferior frontal gyrus and left premotor cortex, children who stu

172 es in DMN connectivity in the right inferior frontal gyrus and supramarginal gyrus.

173 lementary motor area, and the right inferior frontal gyrus as part of the PFC.

174 xhibited less activation in the right middle frontal gyrus during the inhibition task reported more e

175 gyrus or opercular part of the left inferior frontal gyrus has been reported to transiently impair th

176 the human brain, and that the right inferior frontal gyrus hosts a confidence-based statistical learn

177 and underline a distinctive role of inferior frontal gyrus in natural speech comprehension.

178 The decreased connectivity in left middle frontal gyrus of CEN was associated with clinical severi

179 d connectivity was identified in left middle frontal gyrus of CEN, left precuneus and bilateral super

180 f CEN, left precuneus and bilateral superior frontal gyrus of DMN, and right anterior insula of SN.

181 d towards reduced TSPO binding in the middle frontal gyrus of patients with recent-onset schizophreni

182 mulation group, with no change in the middle frontal gyrus or parietal cortices.

183 l lobe post-cocaine and in the left superior frontal gyrus post-saline.

184 y higher activation within the left inferior frontal gyrus relative to nonanxious controls during the

185 "coarse" speech representations in inferior frontal gyrus typically associated with high-level langu

186 tween lifetime alcohol drinking and superior frontal gyrus volume.

187 Smaller inferior frontal gyrus volumes and poor emotional awareness seque

188 on experiment: subjects whose right inferior frontal gyrus was temporarily disrupted made biased choi

189 in frontal regions (medial OFC and superior frontal gyrus) and primary and higher-order visual, soma

190 (dorsolateral prefrontal cortex and inferior frontal gyrus), the medial prefrontal cortex, and the do

191 n = 8) or not including (n = 9) the inferior frontal gyrus, a core mirror neuron system region, and c

192 insula, caudate, anterior cingulate, medial frontal gyrus, and dorsolateral prefrontal cortex.

193 Within the middle frontal gyrus, decrements in linoleic acid, linolenic ac

194 egion of the prefrontal cortex, the inferior frontal gyrus, in children aged 6 to 12 years; and emoti

195 producing biased choices, the right inferior frontal gyrus, often implicated in inhibiting prepotent

196 seen by either direct damage to the inferior frontal gyrus, or via damage to dorsal lateral prefronta

197 The network measures of the left superior frontal gyrus, pars orbitalis (r = -0.40, p = 0.009), le

198 fluent speech production including inferior frontal gyrus, premotor cortex, and superior temporal gy

199 tissues representing three brain regions-the frontal gyrus, the lateral substantia, and the medial su

200 signals were combined in the right inferior frontal gyrus, where they operated in agreement with the

201 nterior cingulate cortex and in the superior frontal gyrus.

202 l language processing areas such as inferior frontal gyrus.

203 ng the intraparietal sulcus and the inferior frontal gyrus.

204 -B(OH)2 syn-syn conformer through a pair of frontal H-bonds with the relevant AA partner.

205 y who presented with a three-week history of frontal headache, and 'blurriness' in the left side of h

206 REM sleep frontal high delta power was a negative correlate of int

207 s dissociating limbic, associative and motor frontal hyper-direct connectivity with anterior and post

208 ents with MS, N-back accuracy improved while frontal hyperactivation (seen at baseline relative to HC

209 that the compensatory processes accompanying frontal hyperactivation appear to be responsible for the

210 Entorhinal tau was associated with frontal hypometabolism, but this dysfunction was not ass

211 odes of the DMN (medial frontal and superior frontal) in the non-rest pleasant stimuli condition.

212 ts with frontal infarcts only and those with frontal infarcts and/or intracerebral haemorrhage were b

213 Patients with frontal infarcts only and those with frontal infarcts an

214 tion of a widespread brain network (superior frontal, insula, middle and superior temporal, putamen,

215 (MD) network, comprising regions of lateral frontal, insular, dorsomedial frontal, and parietal cort

216 Our results demonstrate a role of auditory-frontal interactions in visual speech representations an

217 saliency and expectations likely encoded in frontal "late" regions on perceptual processes occurring

218 ng to a summary distribution volume ratio of frontal, lateral temporal and parietal, and retrosplenia

219 rformance on neuropsychological tests of the frontal lobe and executive functioning, the Trail Making

220 ive effects of socioeconomic disadvantage on frontal lobe development (with implications for function

221 dence of a positive effect of simvastatin on frontal lobe function and a physical quality-of-life mea

222 ue studies in an experiment with humans with frontal lobe lesions, asking whether behavioral impairme

223 l spatial codes are used in conjunction with frontal lobe mechanisms to plan routes during navigation

224 otheses that (i) cerebellar efferents target frontal lobe neurons involved in forming action represen

225 receptive aphasia; (iii) widespread temporal/frontal lobe regions of the left hemisphere and expressi

226 activity was augmented in the left temporal/frontal lobe regions, as well as left inferior-parietal

227 of altered variability of caudate nucleus or frontal lobe volumes.

228 ration change and the beta band power in the frontal lobe were found to differ the most between the t

229 ociated with structural abnormalities of the frontal lobe, amygdala, and insula.

230 er, other atypical regions involved were the frontal lobes (30.4%), temporal lobes (8.69%), basal gan

231 puncture after enucleation and biopsied the frontal lobes and optic nerves of a macaque experimental

232 riety of executive functions governed by the frontal lobes via actions on D1 and D2 receptors.

233 Volumetric reductions of frontal lobes were largest in the ADHD+ODD group, possib

234 This work demonstrates that higher-level frontal mechanisms for cognitive and behavioural flexibi

235 ssociated with reduced cortical thickness in frontal, medial parietal and occipital regions.

236 entation in the left pars orbitalis inferior-frontal, middle-frontal, and inferior-parietal regions p

237 itive transcranial magnetic stimulation of a frontal midline node of the cingulo-opercular MDC affect

238 tential, followed by a driving effect in the frontal module between 140-180 ms, suggesting that the d

239 than in the Shape condition in occipital and frontal modules during the encoding period of the right

240 d with a decrease in ERP amplitude of a late frontal negativity (LFN) elicited by the isolated word.

241 ht into how the cerebellum influences medial frontal networks and the role of the cerebellum in cogni

242 Frontal neurodegeneration does not prevent the perceptua

243 Past work has implicated medial frontal neurons expressing D1 dopamine receptors (D1DRs)

244 sure, men had significantly higher all-night frontal NREM sleep slow-wave activity (SWA: 2-4 Hz), tha

245 ustained attention performance and increased frontal NREM SWA.

246 ere related to reduced perfusion in the left frontal operculum and insula, whereas fear symptoms were

247 on and that compensatory processes linked to frontal overactivation might be responsible for those al

248 hyperactivity and disinhibition give rise to frontal overload and disrupt executive control, fuelling

249 ated linear increase in the amplitude of the frontal P3 event-related component was also observed in

250 as compared within and across the subcortex, frontal, parietal and occipital lobes.

251 mance and greater task-related activation in frontal, parietal, and hippocampal regions compared with

252 glia, and a stepping stone appearance in the frontal pericallosal region.

253 difference is related to visual capacity and frontal placement of eyes, we injected retrograde tracer

254 jects, we assessed gray matter volume in the frontal polar area, a region that has been linked to vis

255 8-275; P < .001) in the superior frontal and frontal polar cortices.

256 Variability of frontal polar volume correlated with individual differen

257 related with left dorsal anterior insula and frontal pole atrophy.

258 as well as increased thickness of the right frontal pole, the right lateral parietal lobules, and th

259 ocortical regions including insular, lateral frontal, posterior temporal and opercular cortex.

260 ose MPH-mediated FC reductions restricted to frontal-prefrontal sites following the appearance of the

261 ure of recognition recruited first the right-frontal region and subsequently the right-parietal ones.

262 ecially strong with SERPING1 in the superior frontal region, consistent with the pattern of disruptio

263 dynamic response during encoding in the left-frontal region, which was associated with a progressive

264 rtical thickness) and regional reductions in frontal regions (medial OFC and superior frontal gyrus)

265 cture, characterized by loss of dominance of frontal regions and emergence of nonfrontal regions, cor

266 bilateral precentral/left posterior superior-frontal regions and speech arrest.

267 l middle-frontal and pars orbitalis inferior-frontal regions around stimulus offset.

268 ons: the interplay between temporal and left-frontal regions during encoding and between temporo-pari

269 oding and between temporo-parietal and right-frontal regions during recognition of speech sounds.

270 functional connectivity between parietal and frontal regions in the crossed versus uncrossed hand pos

271 n in parietal and visual areas, and later in frontal regions with orbitofrontal cortex emerging last.

272 y testing two competing hypotheses involving frontal regions' activity (neurodegeneration vs. compens

273 ate functional magnetic resonance imaging of frontal regions' spontaneous activation, and an electroc

274 pies ml(-1) ) and the two overlapped only in frontal regions.

275 probands had small reductions, primarily in frontal regions.

276 herence to sign language, but entrainment at frontal sites is reduced relative to fluent signers.

277 sing multiple superior temporal and inferior frontal sites, high-gamma power increased with each succ

278 erges simultaneously in parietal/sensory and frontal sources and later than mean reward encoding.

279 aking within previously identified, specific frontal, striatal and parietal networks we suggest that

280 sensorimotor decision making within specific frontal, striatal and parietal networks; we conclude tha

281 sed left-dominant activation in the anterior frontal structures in the older age group may reflect de

282 nt cognitive impairment (n = 42), those with frontal subcortical (FSC) dysfunction (n = 29), those wi

283 emales) identifies early perceptual and late frontal subsystems that are selective to the categorical

284 recentral sulcus (tgPCS) and caudal inferior frontal sulcus (cIFS).

285 l region, above and anterior to the anterior frontal sulcus, from which saccadic eye movements were e

286 ormance based), and DB was measured with the Frontal Systems Behavior Scale (FrSBe: caregiver-report

287 e speech intelligibility for fixed-location, frontal targets, it is currently not known whether these

288 was more pronounced EEG theta activity over frontal, temporal and parietal regions in response to ne

289 In BD, cortical gray matter was thinner in frontal, temporal and parietal regions of both brain hem

290 d plaques in postmortem tissue sections from frontal, temporal, and occipital neocortices in 40 cases

291 ristics of the healthy neonatal brain in the frontal, temporal, and parietal lobes.

292 expanded to comprise a wide-spread bilateral frontal, temporal, and parietal network.

293 r imaging and increase resting-state midline frontal theta activity.

294 Modulation of the coupling from frontal to early visual areas was common to both percept

295 d resolution of the temporal area toward the frontal visual field may facilitate grazing, while resol

296 2) ) that affords enhanced resolution in the frontal visual field.

297 or frontal cortex (-7.3%; P = .02), inferior frontal white matter (-11.4%; P < .001), and caudate (-1

298 ly in late myelinating brain regions such as frontal white matter and the genu of the corpus callosum

299 more than three concussions had lower FA in frontal white matter compared with those with zero to on

300 scores correlated with diffusion measures of frontal white matter.