ライフサイエンスコーパス: frontal lobe

1 (k(f)) of the creatine kinase enzyme in the frontal lobe.

2 gradients, with particular enrichment in the frontal lobe.

3 of behavior mediated in part by the maturing frontal lobe.

4 rmation is conveyed from the parietal to the frontal lobe.

5 plexity specific to the human lineage in the frontal lobe.

6 ow grade gliomas (LGG) localized in the left frontal lobe.

7 rietal and occipital regions compared to the frontal lobe.

8 s that from any of the premotor areas in the frontal lobe.

9 the temporal lobes, right basal ganglia and frontal lobe.

10 nd/or myelin abnormalities, primarily in the frontal lobe.

11 ontal craniotomy with resection of the right frontal lobe.

12 homotopic sites and adjoining cortex in the frontal lobe.

13 are seen in the insular region and inferior frontal lobe.

14 austrum, and the white matter underneath the frontal lobe.

15 l diagnosis to recurrence, and tumor outside frontal lobe.

16 ng from within the parietal lobe or from the frontal lobe.

17 g for additional maps, including some in the frontal lobe.

18 s have implicated abnormal activities in the frontal lobe.

19 sample, where lamin B1 was increased in the frontal lobe.

20 es, temporal lobe, parietal lobe, and medial frontal lobe.

21 umor type arising with high frequency in the frontal lobe.

22 atients showed increased connectivity in the frontal lobes.

23 he subarachnoid space, particularly over the frontal lobes.

24 that it involved relative enlargement of the frontal lobes.

25 regions predominantly influenced activity in frontal lobes.

26 ng action selection signals generated in the frontal lobes.

27 ignificant hypometabolism in the temporal or frontal lobes.

28 d towards subtle abnormalities in the medial frontal lobes.

29 stro-caudal hierarchical organization of the frontal lobes.

30 right parietal cortices and bilateral medial frontal lobes.

31 e first time, substantial involvement of the frontal lobes.

32 ional control centers from regulation by the frontal lobes.

33 associated with abnormal connectivity of the frontal lobes.

34 he corpus callosum connecting left and right frontal lobes.

35 redict the level of functional access to the frontal lobes.

36 e encephalopathy predominantly affecting the frontal lobes, 1 hemorrhagic shock and encephalopathy, 1

37 elirium was associated with smaller superior frontal lobe (-2.11 cm(3) [-3.89, -0.32]; p = .03) and h

38 an GBM with KPS 80 and tumor confined to the frontal lobe (25.7 months; 95% CI, 18.7 to 52.5), and wa

39 er, other atypical regions involved were the frontal lobes (30.4%), temporal lobes (8.69%), basal gan

40 8.89 [1.1] cm3; cortical lesion count in the frontal lobe, 5.0 [5.7]; r = -0.60; P < .01).

41 hite matter water diffusion metrics from the frontal lobe accounted for poor performance on both memo

42 esponse characterized by inferior and middle frontal lobe activation (right>left) which bears some si

43 animal's decision was reliably maintained in frontal lobe activity through a selective state sequence

44 We predicted that frontal lobe activity would decrease, whereas posterior

45 unilateral and unilobar focal lesions in the frontal lobe affecting the cortical grey matter or the c

46 localised disease, frequently located in the frontal lobes, affecting the cortical grey matter and ad

47 Damage to the ventromedial frontal lobe also impaired learning about the relevant d

48 Decision-making ability in the frontal lobe (among other brain structures) relies on th

49 th lesions to dissociated regions within the frontal lobe, amongst other regions.

50 olumetric/area measurements obtained for the frontal lobe, amygdala, and hippocampus and subregions o

51 ociated with structural abnormalities of the frontal lobe, amygdala, and insula.

52 sonance imaging studies have highlighted the frontal lobes, amygdala and cerebellum as pathological i

53 genes differentially expressed within human frontal lobe and a striking increase in transcriptional

54 Compared to listening to words, greater frontal lobe and decreased temporal lobe gamma activity

55 abnormalities extended rostrally towards the frontal lobe and dorsocaudally towards the superior temp

56 sconsin Card Sorting Test, a classic test of frontal lobe and executive function in humans, to assess

57 rformance on neuropsychological tests of the frontal lobe and executive functioning, the Trail Making

58 ludes all of the cortical motor areas in the frontal lobe and portions of somatosensory cortex.

59 ity is elicited by top-down signals from the frontal lobe and recapitulates the bottom-up pattern nor

60 ion for imagined words in the temporal lobe, frontal lobe and sensorimotor cortex, consistent with pr

61 ed gray matter in distributed regions of the frontal lobe and significantly smaller brainstem volume.

62 the patient group, most pronouncedly in the frontal lobe and temporal cortex.

63 injection or needle insertion into the right frontal lobe and were euthanized at different times to e

64 In humans, Broca's area in the frontal lobe and Wernicke's area in the temporal lobe ar

65 th premotor (PM) and motor (M1) areas of the frontal lobe and with other areas of parietal and occipi

66 volume difference of approximately 8% in the frontal lobes and 12% in the occipital lobes-these regio

67 at Aegyptopithecus had relatively unexpanded frontal lobes and a brain-to-body mass ratio lower than

68 erns of coherence are identified between the frontal lobes and between frontal and distant brain regi

69 puncture after enucleation and biopsied the frontal lobes and optic nerves of a macaque experimental

70 t there are no inhibitory modules within the frontal lobes and that behavioural inhibition is an emer

71 rtical structures including bilateral medial frontal lobes and the right inferior frontal and tempora

72 more likely in subnetworks that include the frontal lobe, and can be opposite in nature depending on

73 er WMH load, specifically in the subcortical frontal lobe, and periventricular frontal and parietal c

74 articularly those located in the subcortical frontal lobe, and the periventricular frontal and pariet

75 netic methods reveals that the size of human frontal lobes, and of specific frontal regions, is as ex

76 Since larger ECVs, expanded frontal lobes, and reduced olfactory bulbs are already p

77 We found that stimulation of the frontal lobes [anode electrode at the left dorsolateral

78 reases of grey matter were identified in the frontal lobes, anterior cingulate gyrus, left temporal l

79 and cerebral microdialysis monitoring (right frontal lobe, apparently normal tissue) combined with ce

80 and that event-related potentials above the frontal lobe are affected by both word reversal and the

81 r cingulate cortex in the medial wall of the frontal lobe are the source of this increased theta powe

82 The frontal lobes are necessary for cognitive control at all

83 data have motivated the hypothesis that the frontal lobes are organized hierarchically, such that co

84 butyric acid (GABA), particularly within the frontal lobes, are some of the most reliable alterations

85 al white matter fiber tracts associated with frontal lobe areas critical in cognitive processing has

86 rather significantly in the hippocampus and frontal lobe as a function of learning and memory.

87 as correlated with cortical thickness in the frontal lobe as a whole.

88 one subtype showing grey matter loss in the frontal lobes as well as loss of the temporal lobes (fro

89 cluding the cingulate cortex, operculum, and frontal lobe, as well as in the temporal lobe.

90 It is typically associated with frontal lobe atrophy, although patterns of atrophy are h

91 superior temporal gyrus (BA21), dorsolateral frontal lobe (BA9), and dorsal parietal lobe (BA7) of ea

92 e anatomical and functional abnormalities in frontal lobe, basal ganglia and the limbic system.

93 uced perfusion in the watershed areas of the frontal lobes bilaterally compared with controls.

94 or CTx-), with prominent alterations in the frontal lobes bilaterally.

95 obacterium necrophorum who developed a right frontal lobe brain abscess.

96 ignificant white matter tract changes in the frontal lobe, brainstem and hippocampal regions of the A

97 in four major brain structures (hippocampus, frontal lobe, brainstem, and cerebellum) using a techniq

98 uman primates indicate that the ventromedial frontal lobe carries information about both losses and g

99 orrelated with volumetric differences in the frontal lobes, caudate nucleus and cerebellum, and hyper

100 ical ROI and links were observed in superior frontal lobe, cingulate cortex, fusiform, putamen, and m

101 aboratory, there is also evidence that other frontal lobe circuits have evolved to make other types o

102 hip between striatal dopamine processing and frontal lobe cognitive function.

103 Impairment of frontal lobe cognitive functions has been reported in pa

104 atients with enuresis performed worse on two frontal lobe cognitive tests [Letter Fluency (t = 1.97,

105 and control groups, whilst the structure of frontal lobe connections showed relationships with execu

106 However, uptake in the frontal lobe continued to increase with age but it decre

107 We explored frontal lobe contributions to cue-induced drug craving u

108 These results provide insight into frontal lobe contributions to procedural knowledge.

109 The frontal lobes control wide-ranging cognitive functions;

110 serve receptive and expressive speech, with frontal lobe 'corollary discharges' suppressing low-leve

111 y to studies of HCS and the pH values in the frontal lobe correlated negatively with the YMRS values.

112 ded neurons from different somatosensory and frontal lobe cortices of monkeys performing a vibrotacti

113 y linked by a cascade of transient events in frontal lobe cortices.

114 ling Test clinical measures are sensitive to frontal lobe damage although only the suppression error

115 ds (impulsivity) characterizes subjects with frontal lobe damage and behavioral disorders including s

116 Patients with frontal lobe damage are impaired on response sequencing

117 Participants with left lateral frontal lobe damage attributed rewards to irrelevant dim

118 Patients with focal frontal lobe damage completed a demanding reward learnin

119 est but it has been primarily used to detect frontal lobe damage.

120 we describe the performance of a group of 23 frontal lobe damaged patients in an oculomotor rule swit

121 e diseases such as Parkinson's disease (PD), frontal lobe dementia (FLD) and diffuse Lewy-body dement

122 esearch has demonstrated that performance on frontal lobe-dependent tasks is associated with dopamine

123 ive effects of socioeconomic disadvantage on frontal lobe development (with implications for function

124 ject via the occipitofrontal fascicle to the frontal lobe (dorsal area 6, and areas 8Ad, 8B, and 46);

125 s that migrate and integrate widely into the frontal lobe during infancy.

126 tex during incremental learning and the left frontal lobe during retrieval in the patients.

127 such cognitive deficits are attributable to frontal lobe dysfunction as a direct consequence of impa

128 sistently, anatomical studies have indicated frontal lobe dysfunction in the disease.

129 is slow with frequent protracted symptoms of frontal lobe dysfunction.

130 n implicated in autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) and epilepsy of infancy w

131 Autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) is linked with high penet

132 Frontal lobe epilepsy (FLE) surgery is the second most c

133 novel brain pathology in young patients with frontal lobe epilepsy (FLE) that is distinct from focal

134 reported in patients with schizophrenia and frontal lobe epilepsy and may contribute to their associ

135 Autosomal dominant nocturnal frontal lobe epilepsy can be caused by the alpha4 mutati

136 associated with autosomal dominant nocturnal frontal lobe epilepsy have been found to show spontaneou

137 of seizures in autosomal dominant nocturnal frontal lobe epilepsy mice remain unknown.

138 o show that the autosomal dominant nocturnal frontal lobe epilepsy mutations are associated with larg

139 nrapid eye movement parasomnia and nocturnal frontal lobe epilepsy remain.

140 ions in nAChRs subunits have been related to frontal lobe epilepsy, neurodegenerative diseases, and o

141 e been noted in autosomal dominant nocturnal frontal lobe epilepsy.

142 enrichment of methylation QTLs (P<0.001) and frontal lobe expression quantitative trait loci (eQTLs)

143 mary sensorimotor cortices and with enriched frontal lobe expression.

144 l lobe, left posterior cingulate gyrus, left frontal lobe expressive language regions, and left basal

145 lateral, dorsal, and mesial surfaces of the frontal lobe, extending into primary sensory, superior p

146 omplex mixture extracted from a normal fetal frontal lobe (FL37).

147 lobes for the 100-130 ms timeframe, and the frontal lobe for the 130-160 ms timeframe.

148 ulate cortex and orbitofrontal cortex in the frontal lobe from a total of 90 elderly subjects (age ra

149 pe showing loss relatively restricted to the frontal lobes (frontal-dominant subtype).

150 s in 60 (45%) of 133 patients on the test of frontal lobe function (FAB), and in between 13 (10%) and

151 dence of a positive effect of simvastatin on frontal lobe function and a physical quality-of-life mea

152 FA was not associated with frontal lobe function or visuospatial perception.

153 unger adults who performed worse on tests of frontal lobe function showed greater FMT signal in right

154 that damage to frontostriatal loops impairs frontal lobe function, leading to predominant impairment

155 FMT signal related to a cognitive measure of frontal lobe function.

156 Critically, abnormal frontal-lobe function was correlated with the number of

157 orrelation between left frontal lobe NAA and frontal lobe functional activation was found in the ASD

158 ry modality and information domain influence frontal lobe functional organization, they also demonstr

159 ous research has associated abnormalities in frontal lobe functioning with alcohol relapse.

160 ntial underlying mechanism for impairment of frontal lobe functions in both patients and siblings, an

161 an developmental in vivo evidence confirming frontal lobe GABA maturation, which was linked to impuls

162 ystem, also matures during adolescence, with frontal lobe GABA receptors reaching adult levels late i

163 diffusivity [RD, AxD, MD, respectively]) of frontal lobe, genu, and splenium of the corpus callosum

164 s of 5-HTTLPR genotype on total cortical and frontal lobe gray matter volumes were 10% and 16%, respe

165 gray matter, total neocortical white matter, frontal lobe gray matter, frontal lobe white matter, and

166 rved on within-subject changes in volumes of frontal lobe gray matter, lateral ventricles, and sulcal

167 ical gray matter: F(1,24) = 14.11, P = .001; frontal lobe gray matter: F(1,24) = 13.20, P = .001).

168 rtical gray matter: F(1,38) = 5.73, P = .02; frontal lobe gray matter: F(1,38) = 11.73, P = .002).

169 subregions in the UNC sample of 29 children (frontal lobe gray matter: F(2,23) = 6.36, P = .01).

170 The frontal lobes had high PiB retention with little grey ma

171 rain structures, such as the hippocampus and frontal lobe, has only been observed in humans.

172 how state desirability signals decoded from frontal lobe hemodynamics, as measured with near-infrare

173 Significant frontal lobe hyperactivation to support WM was found in

174 re parieto-temporal lobe, cerebellar cortex, frontal lobe, hypothalamus and striatum.

175 ribed structural abnormalities in the medial frontal lobe in juvenile myoclonic epilepsy.

176 and enhanced connectivity within the dorsal frontal lobe in subjects with psychopathy.

177 egulation that is normally controlled by the frontal lobe in the intact brain.

178 of Interest) was defined in 7 locations: the frontal lobe in the right and left hemispheres, the basa

179 niqueness should therefore focus less on the frontal lobes in isolation and more on distributed neura

180 king circuits working in parallel within the frontal lobes in order to make different types of decisi

181 ed with impaired neuronal functioning in the frontal lobes in the ASD group.

182 ion revealed a pathological mass in the left frontal lobe, in close topographical relationship to the

183 ciated with decreased GM density in the left frontal lobe, in the right frontoparietal cortex and in

184 d increased GM predominantly in temporal and frontal lobes, including cingulate and fusiform gyri and

185 isual and auditory attention networks in the frontal lobe, independent of sensory modality.

186 spontaneous long-term recovery from isolated frontal lobe injury and isolated frontoparietal injury.

187 studies using drug cue paradigms have shown frontal lobe involvement in this contextual influence on

188 Frontal lobe involvement is characteristic of CBD, and i

189 rcuits control executive function within the frontal lobe involving the dorsolateral prefrontal corte

190 The human frontal lobe is important for social behavior and execut

191 A fundamental challenge in studying the frontal lobe is to parcellate this cortex into "natural"

192 Because white matter in the frontal lobes is more prone to the effects of aging than

193 bs of high connectivity (many located in the frontal lobe) is associated with amyloid accumulation.

194 CC), which lies on the medial surface of the frontal lobes, is important in regulating cognitive cont

195 disorder with dysfunction and atrophy of the frontal lobes leading to changes in personality, behavio

196 male Vietnam combat veterans consisting of a frontal lobe lesion group with focal penetrating head in

197 ity of lesion or correlation between lateral frontal lobe lesions and impairment on any task.

198 rocess which is known to be impaired by both frontal lobe lesions and PD while the VMT is a control t

199 ation approaches developed for patients with frontal lobe lesions and pharmacotherapy approaches desi

200 Also, right frontal lobe lesions have been implicated, but typically

201 Larger frontal lobe lesions were associated with greater number

202 ue studies in an experiment with humans with frontal lobe lesions, asking whether behavioral impairme

203 some but not all "executive" deficits after frontal lobe lesions.

204 cits similar to those found in patients with frontal lobe lesions.

205 ter numbers of terminal boutons than smaller frontal lobe lesions.

206 By orthogonal coding, the frontal lobe may control transitions between the discret

207 l spatial codes are used in conjunction with frontal lobe mechanisms to plan routes during navigation

208 includes areas on the medial surface of the frontal lobe, medial orbital areas, and two caudolateral

209 by considering the role of the anterior and frontal lobes, mesolimbic connections and the right hemi

210 t recent evidence suggests that parts of the frontal lobe might also play a key role.

211 sitive semi-partial correlation between left frontal lobe NAA and frontal lobe functional activation

212 rily located in the posterior portion of the frontal lobe, near the controlling site, and non-linear

213 l or executive control processes mediated by frontal lobe networks.

214 otheses that (i) cerebellar efferents target frontal lobe neurons involved in forming action represen

215 g firing rates and correlated variability of frontal lobe neurons while monkeys perform a vibrotactil

216 the temporal lobe with predilection for the frontal lobes.New type III is one of the above dysplasia

217 brain has principal and arcuate sulci of the frontal lobe not seen in the stem catarrhine Aegyptopith

218 is procedure to 17 injections, placed in the frontal lobe of nine marmosets as part of earlier studie

219 t parietal and occipital regions, the mesial frontal lobe of the right hemisphere, and the cuneus and

220 ng direct inoculation of M002 into the right frontal lobes of A. nancymae; (iii) there was no histopa

221 stone acetylation in the white matter of the frontal lobes of aged subjects and in patients with chro

222 rons in brain tissues from basal ganglia and frontal lobes of HIV encephalitic patients.

223 cid (DHA) accumulates in the hippocampus and frontal lobes of the fetal brain during the last trimest

224 lower FA and significantly higher RD in the frontal lobes of the RHU group, relative to the TS group

225 uced perfusion in the watershed areas of the frontal lobes on SPECT scanning analyzed by the SSP tech

226 The ventral frontal lobe, or inferior frontal gyrus, plays an import

227 of the basal ganglia, thalamus, hippocampus, frontal lobe, orbitofrontal cortex, and gyrus rectus.

228 ly in the left internal capsule and the left frontal lobe (P = .02-.05).

229 magnetic resonance imaging with an executive frontal lobe paradigm, we investigated cortical activati

230 ial diffusivity, and radial diffusivity from frontal lobes, parietal lobes, and in the frontostriatal

231 rom 12 patients with temporal, occipital, or frontal lobe partial onset seizures.

232 ing associated cognitive abilities, in other frontal lobe pathologies; however, in clinical trials, M

233 actional anisotropy in regions that included frontal lobe pathways.

234 rnally using brain stimulation, and that the frontal lobes play a causal role in mind-wandering behav

235 The frontal lobes play a key role in sequential organization

236 ng in 19% of the whole cortex and 24% of the frontal lobes, primarily in the right superior, medial o

237 neurons in several areas of the human medial frontal lobe prior to voluntary movement.

238 in spatial working memory while the ventral frontal lobe processes language and communication signal

239 nsisted of significantly increased CT within frontal lobe regions and reduced SA in the orbitofrontal

240 argeted effects of aging on the integrity of frontal lobe regions and selective temporal lobe areas a

241 ormation about the complex interplay between frontal lobe regions in mediating these processes and ac

242 antly smaller gray matter volume was seen in frontal lobe regions in patients homozygous for the A al

243 focus on recent evidence that suggests four frontal lobe regions make distinct contributions to rewa

244 phase coding in four temporal lobe and four frontal lobe regions of the human brain using local fiel

245 receptive aphasia; (iii) widespread temporal/frontal lobe regions of the left hemisphere and expressi

246 We propose that frontal lobe regions that contribute to complex decision

247 These functional changes correspond to frontal lobe regions where we previously reported struct

248 brain regional analysis demonstrated only 3 frontal lobe regions with an SUV estimation bias of 5% o

249 activity was augmented in the left temporal/frontal lobe regions, as well as left inferior-parietal

250 g the middle and anterior temporal lobe with frontal lobe regions, including the pars triangularis.

251 Significance statement: Frontal lobe-related function is vital for a variety of

252 s under dynamic conditions, with the lateral frontal lobe required for selecting the relevant dimensi

253 associate with reward, and the ventromedial frontal lobe required to learn the reward association it

254 ting PLA2/5-LOX/LTB4 cascade using a partial frontal lobe resection SBI rat model.

255 A biopsy specimen from the right frontal lobe revealed demyelination and perivascular lym

256 lated with oral naming speed in both lateral frontal lobes (rho = 0.48 and 0.47 for right and left fr

257 Certain seizure types, such as frontal lobe seizures, may mimic nonepileptic seizure se

258 y- and deoxyhemoglobin concentrations in the frontal lobe show differences in response to both primar

259 s were recorded from temporal, parietal, and frontal lobe sites in 12 pediatric patients undergoing e

260 ce demonstrating that circuits involving the frontal lobe, striatum, temporal lobe, and cerebellum ar

261 processes mediated by anatomically distinct frontal lobe subregions are both critical for adaptive c

262 we tested the critical involvement of human frontal lobe subregions in a probabilistic, multidimensi

263 , we examined the necessary contributions of frontal lobe subregions in attributing feedback to relev

264 Findings were principally in the frontal lobe, superior parietal lobes, and in the parame

265 e, including gray matter of the total brain, frontal lobe, temporal lobe, and hippocampus.

266 Regions in the frontal lobe, temporal lobe, and parietal lobe exhibited

267 and may occur in other locations such as the frontal lobes, thalami, basal ganglia and brainstem.

268 Smaller superior frontal lobes, thalamus, and cerebellar volumes at 3 mon

269 tex or less cortical folding in temporal and frontal lobes than control subjects.

270 theta band, involving mainly regions in the frontal lobe that were sending information to a more dis

271 gions, there is extensive involvement of the frontal lobes that has been missed by past ROI analyses.

272 Several areas in the frontal lobe, the anterior cingulate and a parietal netw

273 e occurrence of 5hmC in DNA from human brain frontal lobe tissue.

274 le contributions of different regions of the frontal lobes to probabilistic learning.

275 tion of the right hemisphere, especially the frontal lobe, to reading comprehension performance on th

276 speech information from the temporal to the frontal lobe utilizes two different streams and conducti

277 riety of executive functions governed by the frontal lobes via actions on D1 and D2 receptors.

278 The ventromedial frontal lobe (VMF) has been implicated in learning in dy

279 ypes were characterized by a large amount of frontal lobe volume loss, with one subtype showing grey

280 ion, and smaller medial, lateral and orbital frontal lobe volumes showed faster rates of decline.

281 of altered variability of caudate nucleus or frontal lobe volumes.

282 The frontal lobe was involved in 14 patients (77.8%) and the

283 feedback attribution, while the ventromedial frontal lobe was necessary for learning the value of fea

284 The left lateral frontal lobe was required for filtering option dimension

285 comprising the precuneus, cingulate & middle frontal lobe was significantly higher in psALS and affec

286 Society of Paris and announced that the left frontal lobe was the seat of speech.

287 thinning in children primarily in the right frontal lobes was associated with exposure to prenatal m

288 ration change and the beta band power in the frontal lobe were found to differ the most between the t

289 Volumetric reductions of frontal lobes were largest in the ADHD+ODD group, possib

290 ng on the orbital and medial surfaces of the frontal lobes were most closely connected with limbic st

291 nificantly fewer correlated areas within the frontal lobes when compared to term controls.

292 ated by anatomically distinct regions of the frontal lobes when reinforcement contingencies are assur

293 Abnormal ERPs were recorded over the frontal lobes, where FXTAS patients showed both P300 amp

294 sions with a hypoechogenic halo in the right frontal lobe, which could correspond to brain abscesses.

295 a predilection for iron accumulation in the frontal lobes, which when combined with the subcortical

296 ical white matter, frontal lobe gray matter, frontal lobe white matter, and the hippocampus in a cros

297 eneration were limited to focal areas in the frontal lobe white matter.

298 fornix, global mean white-matter FA and left frontal lobe white-matter FA.

299 l as in distinct regions of the temporal and frontal lobes with increasing age.

300 , various cortical areas in the parietal and frontal lobes work together effortlessly to analyze obje