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1 epsy in humans, autosomal dominant nocturnal frontal lobe epilepsy.
2 e been noted in autosomal dominant nocturnal frontal lobe epilepsy.
3 a4 receptors in autosomal dominant nocturnal frontal-lobe epilepsy.
4  been linked to autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) and co-expressed them wit
5 n implicated in autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) and epilepsy of infancy w
6                 Autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) is linked with high penet
7 tions linked to autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) occur in the M2 region of
8 lpha4) found in autosomal-dominant nocturnal frontal lobe epilepsy (ADNFLE) were expressed along with
9 ndrome known as autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE).
10 ently linked to autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE).
11  genetically to autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE).
12 cal features of autosomal dominant nocturnal frontal lobe epilepsy and concussive seizures are review
13  reported in patients with schizophrenia and frontal lobe epilepsy and may contribute to their associ
14                 Autosomal dominant nocturnal frontal lobe epilepsy can be caused by the alpha4 mutati
15  map, including autosomal dominant nocturnal frontal lobe epilepsy (ENFL2; also known as ADNFLE), a s
16             Fourteen patients had unilateral frontal lobe epilepsy, five occipital lobe epilepsy (OLE
17                                              Frontal lobe epilepsy (FLE) surgery is the second most c
18 novel brain pathology in young patients with frontal lobe epilepsy (FLE) that is distinct from focal
19 associated with autosomal dominant nocturnal frontal lobe epilepsy have been found to show spontaneou
20  of seizures in autosomal dominant nocturnal frontal lobe epilepsy mice remain unknown.
21 o show that the autosomal dominant nocturnal frontal lobe epilepsy mutations are associated with larg
22 ions in nAChRs subunits have been related to frontal lobe epilepsy, neurodegenerative diseases, and o
23 nrapid eye movement parasomnia and nocturnal frontal lobe epilepsy remain.

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