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1 udate, left inferior frontal gyrus, and left frontal pole.
2 eft rostral middle frontal regions, and left frontal pole.
3 stinctively human component in ventrolateral frontal pole.
4 unterexamples to conclusions recruited right frontal pole.
5 BOLD activation in left cerebellum and right frontal pole.
6 3) in which SD was repeatedly evoked in the frontal pole.
7 human-specific patterns of expression in the frontal pole.
8 atter between the anterior cingulate and the frontal poles.
9 nd dorsal to the principal sulcus and in the frontal pole; 3) a caudolateral network (CLPFC) in and r
10 e to which the latter two regions influenced frontal pole activity correlated with participant-specif
12 connect with largely separate regions of the frontal pole and more medial premotor and dorsal prefron
13 ilateral reductions of cortical thickness in frontal pole and superior frontal gyrus, and similar eff
14 d decreased with the orbital frontal cortex, frontal pole and the dorsal ACC, suggesting a down-regul
15 ral gyrus, middle and superior frontal gyri, frontal pole, and cingulate gyrus in S-allele carriers c
16 t cortex was reciprocally connected with the frontal pole (area 10), rostral principal sulcus (area 4
17 s consolidate the left medial portion of the frontal pole as particularly altered in major depression
19 ostructurally informed subdivisions of human frontal pole between depressed patients and comparison s
20 rtex (Brodmann's area 8), the right inferior frontal pole (Brodmann's area 10), and the right lateral
21 ization of neurons in four cortical regions (frontal pole [Brodmann's area 10], primary motor [area 4
22 ribe their comparison of transcriptomes from frontal pole, caudate nucleus, and hippocampus of multip
23 ulo-opercular control network, including the frontal pole, cingulate cortex, and anterior insula.
25 l activation than comparison subjects in the frontal pole, dorsolateral and ventrolateral prefrontal
27 nalysis found a significantly smaller medial frontal pole in depressed patients, with a negative corr
30 scribe the intracerebral hemodynamics at the frontal pole in the circumscribed period between wakeful
31 occurred in the medial prefrontal cortex and frontal pole in the patients who responded positively to
33 r with grey matter volume asymmetries in the frontal pole, lateral occipital pole or temporal pole.
34 pars opercularis, rostral middle frontal and frontal pole), left supramarginal gyrus, and right trans
35 their functional coupling patterns with the frontal pole, medial prefrontal, and dorsal prefrontal c
36 mplicating the bilateral anterior cingulate, frontal pole, medial temporal lobe, opercular cortex and
37 ral anterior cingulate cortex (rACC), medial frontal pole (mFP) and periaquiduct grey (PAG) are signi
38 uman region sometimes referred to as lateral frontal pole more closely resembled area 46, rather than
41 Group 2 animals (n = 9), the (non-ischemic) frontal pole of the ipsilateral hemisphere was electrica
43 y in the right superior, medial orbital, and frontal pole regions of the prefrontal cortex (p < .01).
44 cingulate cortex, parahippocampal gyri, and frontal pole, that exhibited activity uniquely associate
45 ntral prefrontal regions including the right frontal pole, the medial and lateral orbitofrontal gyrus
46 as well as increased thickness of the right frontal pole, the right lateral parietal lobules, and th
47 analysis showed that hippocampal volume and frontal pole thickness differed between patients who ach
48 (socio-affective) versus lateral (cognitive) frontal pole to major depression pathogenesis is current
49 ds, subregion specificity in the left medial frontal pole volume in depressed patients was demonstrat
52 -0.40, P=0.002) between serum VEGF and total frontal pole volume was found in patients with schizophr
53 ater activation in the left caudate and left frontal pole was associated with abstinence-induced subj
55 tion of emotion (inferior frontal cortex and frontal pole) was negatively correlated with both the nu
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