ライフサイエンスコーパス: fruit body

1 t spore inside a domed-shaped, multicellular fruiting body.

2 D mounds, and finally sporulation within the fruiting body.

3 gnals progression from the slug stage to the fruiting body.

4 C-signal that directs the morphogenesis of a fruiting body.

5 helps to pattern cell movement and shape the fruiting body.

6 of the outer basal disc and lower cup of the fruiting body.

7 anterior-like cells to the upper cup of the fruiting body.

8 de synthase for antrocamphin biosynthesis in fruiting body.

9 ng early and incorporate bacteria into their fruiting bodies.

10 and production of multicellular biofilms or fruiting bodies.

11 hat results in the formation of spore-filled fruiting bodies.

12 shown to restrict bacterial contamination of fruiting bodies.

13 ir range in the nearly still air surrounding fruiting bodies.

14 generate three-dimensional aggregates called fruiting bodies.

15 sites, where they culminate to form sessile fruiting bodies.

16 re differentiation inside Myxococcus xanthus fruiting bodies.

17 aggregates, ensuring that spores form within fruiting bodies.

18 s of PilA and pili as pilT fibA mutants form fruiting bodies.

19 h culminates in the assembly of spore-filled fruiting bodies.

20 stem mutants affect the shape of these early fruiting bodies.

21 oncentration and form aggregates that become fruiting bodies.

22 regate during the formation of multicellular fruiting bodies.

23 ain as migrating slugs when they should form fruiting bodies.

24 the formation of spore-filled, multicellular fruiting bodies.

25 centration; they form aggregates that become fruiting bodies.

26 sulting in small, numerous, and disorganized fruiting bodies.

27 ams that enlarge tiny random aggregates into fruiting bodies.

28 mentally resistant myxospores encased within fruiting bodies.

29 to form larger aggregates that develop into fruiting bodies.

30 during their starvation-induced formation of fruiting bodies.

31 esulting in a small colony covered by sexual fruiting bodies.

32 progression ensures that spores form inside fruiting bodies.

33 xpression with the cell movements that build fruiting bodies.

34 proceeds through development to form mature fruiting bodies.

35 lti-tipped aggregates that mature into small fruiting bodies.

36 on nitrocellulose filters, forming defective fruiting bodies.

37 n and development ending with small, gnarled fruiting bodies.

38 tative medium, but is no longer able to form fruiting bodies.

39 leaves, germlings form structures resembling fruiting bodies.

40 riminate against nonkin, leading to chimeric fruiting bodies.

41 er in complex ways to form well proportioned fruiting bodies.

42 e C-signaling, which increases as cells form fruiting bodies.

43 t nutrients caused partial disaggregation of fruiting bodies.

44 ortholog, and results in formation of barren fruiting bodies.

45 n low-calorie diets, just like the mushrooms fruiting bodies.

46 entiation, in particular in the formation of fruiting bodies.

47 eriod of vigorous motility leading to raised fruiting bodies (8 to 16 h), and a period of maturation

48 s indicates that they are capable of forming fruiting body aggregates in the presence of prey, demons

49 bacteria are provided as a nutrient source, fruiting body aggregation is more organized, such that f

50 characteristic toxins present in MC) in both fruit bodies and spent.

51 n alert monkeys to achromatic images (faces, fruit, bodies and places) and colored gratings.

52 In total, 212 fruiting bodies and 106 underlying topsoil samples were

53 ed soil), very efficiently accumulated Hg in fruiting bodies and concentration levels were at 3.7+/-1

54 pression of the operon occurs within nascent fruiting bodies and depends in part on C signaling.

55 ong the fungal communities identified in the fruiting bodies and external mycelial cortices of Chines

56 erence in the fungal communities between the fruiting bodies and external mycelial cortices of Chines

57 a and fluorescence was visible in transgenic fruiting bodies and GFP was detectable in planta.

58 naB(A116V) mutant was unable to develop into fruiting bodies and produced fewer myxospores than the w

59 o erect aerial filaments, which develop into fruiting bodies and spore-bearing structures.

60 estored the ability of a bsgA mutant to form fruiting bodies and spores and, with one exception, rest

61 gene during vegetative growth, formation of fruiting bodies and spores on semi-rich nutrient medium

62 erium that feeds on other bacteria and forms fruiting bodies and spores, depends on poly P for motili

63 he temporal and spatial formation of complex fruiting bodies and sporulation of M. xanthus.

64 is a morphogen that controls the assembly of fruiting bodies and the differentiation of myxospores.

65 ractions suffice to explain the formation of fruiting bodies and the differentiation of spores within

66 of a bsgA mutant and was capable of forming fruiting bodies and viable spores in the absence of the

67 the three isolates produced the confirmatory fruiting bodies and was thus classified as N. pseudofisc

68 ifferentially expressed between mycelium and fruiting body and 242 proteins in the mevalonate pathway

69 ly regulates the steps that together build a fruiting body and differentiate spores within it.

70 Cell-bound C-signal guides the building of a fruiting body and triggers the differentiation of myxosp

71 pping of Con A receptors, and development to fruiting bodies) and does not inhibit growth on plates,

72 l and phenolic compounds (also higher in its fruiting body) and stronger antioxidant activity than P.

73 21, respectively) from Grifola frondosa (GF) fruiting bodies, and evaluating their effects on nitric

74 illuminated from the lamella side, in sliced fruiting bodies, and in the stipes.

75 ing body formation, the morphology of mature fruiting bodies, and the efficiency of sporulation.

76 he lower cup and the outer basal disc of the fruiting body, and DimB retains a high nuclear concentra

77 n in both vegetative structure (thallus) and fruiting body (apothecia) of anthraquinones, secondary m

78 nd mulch-associated fungus with a splash cup fruiting body appearing like a miniature bird's nest of

79 Most Agaricomycete fruiting bodies are ephemeral, and their fossil record i

80 Fruiting bodies are extended vertically in a series of t

81 Although fruiting bodies are relatively large structures that con

82 Fruiting bodies are smaller and produce fewer spores, wh

83 liding movements to build mounds that become fruiting bodies as some cells differentiate into spores.

84 iaca cells were incapable of building normal fruiting bodies but formed clumps and fungus-like struct

85 o use peptides to trigger sporulation within fruiting bodies, but their sequences have not been defin

86 telium discoideum, which forms multicellular fruiting bodies by aggregation and utilizes two polymorp

87 Myxobacteria build their species-specific fruiting bodies by cell movement and then differentiate

88 The formation of spore-filled fruiting bodies by myxobacteria is a fascinating case of

89 roduction of asexual spores (conidia) within fruiting bodies called conidiomata.

90 nges in productivity and phenology of fungal fruit bodies can give clues to changes in fungal activit

91 s that results in the formation of tree-like fruiting bodies capable of producing spores.

92 Peripheral cells do less C-signaling than fruiting body cells, because they have a different spati

93 Sexual fruiting bodies (cleistothecia) can be formed in both ho

94 lta gpgA mutant was unable to produce sexual fruiting bodies (cleistothecia) in self-fertilization an

95 A (dimethylarsinic acid) in Xerocomus badius fruiting bodies collected from selected Polish forests f

96 ails of the internal structure of M. xanthus fruiting bodies consisting of interconnected pockets of

97 adients to the surface of the soil to form a fruiting body consisting of a stalk supporting a spore h

98 discoideum during the formation of chimeric fruiting bodies, consisting of dead stalk cells and viab

99 Extract of processed C. cibarius fruiting bodies contained l-tryptophan, 5-methyltryptoph

100 telium cells aggregate to form multicellular fruiting bodies containing spores that germinate when tr

101 um and, upon starvation, aggregation to form fruiting bodies containing spores.

102 r mounds that differentiate and develop into fruiting bodies containing spores.

103 l process that results in the formation of a fruiting body containing environmentally resistant myxos

104 toxicity similar (sometimes superior) to its fruiting bodies, contrarily to S. bellinii.

105 Previous studies have demonstrated that fruiting body-derived Myxococcus xanthus myxospores cont

106 Of particular interest, fruiting body-derived myxospores contain a specific two-

107 ronmental conditions: (i) starvation-induced fruiting body development and (ii) predation of other or

108 ing body stages to identify genes regulating fruiting body development and develop EST-SSR markers as

109 cessed, target genes critical for M. xanthus fruiting body development and EPS production in a RAMP-d

110 FruA is a transcription factor essential for fruiting body development and is thought to play a key r

111 waves, termed ripples, during multicellular fruiting body development and predation on other bacteri

112 of progress has been made in the studies of fruiting body development and social gliding in Myxococo

113 ons, but is also required for Pxr to prevent fruiting body development by a developmentally proficien

114 thus is a bacterium displaying multicellular fruiting body development during which approximately 80%

115 molecules per cell and drives the process of fruiting body development forward.

116 een 384 chemicals for complete inhibition of fruiting body development in M. xanthus.

117 ruA is an essential transcription factor for fruiting body development in M. xanthus.

118 Fruiting body development in Myxococcus xanthus is a mul

119 tify an sRNA, Pxr, that negatively regulates fruiting body development in Myxococcus xanthus.

120 -like activator gene nla18 causes defects in fruiting body development in Myxococcus xanthus.

121 ls in the early stages of starvation-induced fruiting body development migrate in a highly organized

122 endospore formation by Bacillus subtilis and fruiting body development of Myxococcus xanthus have rev

123 Microcinematography was used to examine fruiting body development of Myxococcus xanthus.

124 Given the elaborate fruiting body development of this bacterial species, M.

125 In this report, we analyze how M. xanthus fruiting body development proceeds in a coculture with s

126 us xanthus that is proficient at cooperative fruiting body development to evolve while repeatedly enc

127 er cell rises 100-fold from the beginning of fruiting body development to the end, when spores are fo

128 Then, as myxobacteria begin fruiting body development, a rising level of C-signal in

129 H CsgA is responsible for C signaling during fruiting body development, although the mechanism is unc

130 te to perform group functions highlighted by fruiting body development, an obligate multicellular fun

131 Surface motility, biofilm formation, fruiting body development, and host invasion are some of

132 ional studies indicate that SRY drives early fruiting body development, and hybrid MatA protein carry

133 includes motility, predation, multicellular fruiting body development, and sporulation.

134 ve progress in identifying genes controlling fruiting body development, cell behaviors and cell-cell

135 nscription factor for fruA expression during fruiting body development, was identified using a genomi

136 Later in fruiting body development, waves are replaced by streams

137 ns (EBPs) control the temporal expression of fruiting body development-associated genes in Myxococcus

138 , including group motility and multicellular fruiting body development.

139 s produced minor but reproducible defects in fruiting body development.

140 ting a stringent response and for initiating fruiting body development.

141 events that occur during Myxococcus xanthus fruiting body development.

142 tant cells is altered in the early stages of fruiting body development.

143 ive growth resulting in significantly faster fruiting body development.

144 polysaccharide O-antigen and is required for fruiting body development.

145 they were inactivated to look for effects on fruiting body development.

146 uch as sporulation, heterocyst formation and fruiting body development.

147 ertion mutagenesis as an essential locus for fruiting body development.

148 ies of different NtrC-like activators during fruiting body development.

149 at they are deficient in social motility and fruiting body development.

150 nthus is essential for biofilm formation and fruiting body development.

151 differentiation of early sexual tissues, or fruiting body development.

152 Cells inside fruiting bodies differentiate into round, nonmotile, env

153 Cells inside fruiting bodies differentiate into round, nonmotile, env

154 isproportionate number of spores in chimeric fruiting bodies do not actually gain higher fitness as a

155 g in the formation of many aggregates called fruiting bodies, each of which contains up to 100,000 sp

156 y, resulting in formation of a spore-bearing fruiting body, evolved at least six times independently

157 With the exception of linoleic acid in cut fruiting bodies, fatty acid concentrations remained almo

158 rowth and multicellular development, forming fruiting bodies filled with spores.

159 including 14-alpha-demethylase (CYP51F1) in fruiting body for converting lanostane to ergostane trit

160 ody aggregation is more organized, such that fruiting bodies form specifically after a step-down or l

161 Che-like Frz pathway, which is essential for fruiting body formation and differentiation.

162 nslocate from the cytoplasm to the membrane, fruiting body formation and EPS production were restored

163 The deletion of fdgA resulted in defective fruiting body formation and reduced sporulation efficien

164 t the identification of a novel inhibitor of fruiting body formation and sporulation, beta-d-allose.

165 netic locus, mrp, which is required for both fruiting body formation and sporulation.

166 Fruiting body formation depended on regulatory genes req

167 We conclude that fruiting body formation does not occur exclusively in re

168 SigF is required for fruiting body formation during development as well as so

169 pores, whereas DeltagprADeltagprB eliminated fruiting body formation in homothallic conditions.

170 480, and asgC767 and improved the quality of fruiting body formation in the asgB480 mutant.

171 (sRNA) Pxr negatively controls multicellular fruiting body formation in the bacterium Myxococcus xant

172 Furthermore, inhibition of fruiting body formation occurs only when beta-d-allose i

173 Fruiting body formation of Myxococcus xanthus requires t

174 Fruiting body formation of Myxococcus xanthus, like biof

175 ch media and for cellular aggregation during fruiting body formation on starvation media.

176 t tan cells may not require yellow cells for fruiting body formation or starvation-induced sporulatio

177 defective in PKB activation, chemotaxis, and fruiting body formation upon nutrient deprivation.

178 pB deletion mutant exhibited a 24 h delay in fruiting body formation, accumulated less glycogen in th

179 hus includes co-ordinated group movement and fruiting body formation, and requires directed motility

180 When expressed in M. xanthus, NafA restored fruiting body formation, EPS production, and S-motility

181 n protein on the cell surface, is delayed in fruiting body formation, produces fewer spores, is delay

182 During this process of fruiting body formation, short-range C-signaling between

183 ting events in high CO2 but not later steps (fruiting body formation, sporulation), indicating a majo

184 n in the EBP gene nla4 affects the timing of fruiting body formation, the morphology of mature fruiti

185 etion of the pfk-pkn4 operon did not inhibit fruiting body formation, the spore yield was low.

186 the tan vegetative cells that contributed to fruiting body formation.

187 processes involving vegetative swarming and fruiting body formation.

188 signals to ensure appropriate timing during fruiting body formation.

189 cycle that includes swarming, predation and fruiting body formation.

190 tants on single cell reversals, swarming and fruiting body formation.

191 reversals required for directed motility and fruiting body formation.

192 s requires gliding motility for swarming and fruiting body formation.

193 reas a step-up in prey availability inhibits fruiting body formation.

194 rtant clues about the mechanisms involved in fruiting body formation.

195 red for beta-d-allose-mediated inhibition of fruiting body formation.

196 n timing events during the initial stages of fruiting body formation.

197 al model that reproduces the early stages of fruiting body formation.

198 nd reversal rates during the early stages of fruiting body formation.

199 us is essential for social (S-) motility and fruiting body formation.

200 ing that both SfaD and GpgA are required for fruiting body formation.

201 eas regions were defective in S-motility and fruiting body formation.

202 ed with the initiation of asexual and sexual fruiting body formation.

203 rdinates its motility during aggregation and fruiting body formation.

204 ort directional motility during swarming and fruiting body formation.

205 ding social motility, predatory rippling and fruiting body formation.

206 outside colonized D. discoideum spores after fruiting body formation; this observation, together with

207 one continuous simulation all the stages of fruiting-body formation that have been experimentally ob

208 ination and cheating behavior during asexual fruiting-body formation.

209 cycle that includes vegetative swarming and fruiting-body formation.

210 enced the 18S rRNA genes of the coprophilic, fruiting body-forming amoeba Guttulinopsis vulgaris and

211 and patterns of diversity within and across fruiting body groups were examined.

212 The organization of Myxococcus xanthus into fruiting bodies has long been studied not only as an imp

213 three-dimensional structure of myxobacteria fruiting bodies has previously presented a challenge due

214 te determination during morphogenesis of the fruiting body; however, transcriptomic and proteomic stu

215 ol (vitamin D(2)) formation was immediate in fruiting bodies illuminated from the lamella side, in sl

216 gulates the size of Dictyostelium discoideum fruiting bodies in part by regulating cell-cell adhesion

217 gation in phase 2, and disintegration of the fruiting bodies in the third phase.

218 can aggregate and develop into multicellular fruiting bodies in which many die altruistically as they

219 cells glide to aggregation centers and form fruiting bodies in which rod-shaped cells differentiate

220 This slug forms a fruiting body in which about a fifth of cells die to for

221 cells coordinate their movements to build a fruiting body in which spores form.

222 A in alrA- cells causes cells to form normal fruiting bodies, indicating that AlrA affects group size

223 he world, but those studies focused on their fruiting bodies instead of other presentations, such as

224 king and the self-organization of cells into fruiting bodies is an active area of research.

225 ons, the spacing and location of the nascent fruiting bodies is determined by the wavelength and patt

226 The inability to form fruiting bodies is not due to a loss of S-motility, but

227 We also show that the myxobacterial fruiting body is more resistant to predation by worms th

228 Ten M. xanthus fruiting bodies isolated from soil were surveyed for var

229 As in Cremini was distributed throughout the fruiting body, it was localized to the hymenophore regio

230 anscript are required for differentiation of fruiting bodies, karyogamy, meiosis, and efficient forma

231 mpA and ecmB in the lower cup and the mutant fruiting bodies lack a basal disc.

232 systems to self-organize into multicellular fruiting bodies, large mounds in which cells differentia

233 iding motility and initially is able to form fruiting bodies like the wild type when starved for nutr

234 with complex architectural features, such as fruiting-body-like aerial projections whose tips serve a

235 amoebae aggregate upon starvation to form a fruiting body made of dead stalk cells and reproductive

236 ossibly chimeric (genetically heterogeneous) fruiting body made of dead stalk cells and spores.

237 oncentrations of arsenic in C. pulverulentus fruit-bodies may reach 1300mgkg(-1) dry weight.

238 Mutants exhibiting defects in fruiting body morphogenesis also produce fewer viable sp

239 bA active site residue E342 is important for fruiting body morphogenesis in the absence of PilA.

240 group 2 PdsA gene resulted in disruption of fruiting body morphogenesis, but left aggregation unaffe

241 to respond to PE and the observed defects in fruiting body morphogenesis.

242 mutant, but was more effective at restoring fruiting body morphogenesis.

243 A5 and pktB8 with respect to aggregation and fruiting body morphology, but that pktA5 and pktB8 were

244 indole compounds identified in the processed fruiting bodies of A. mellea.

245 One knockout mutant produced fruiting bodies of abnormal shape that depended on the c

246 the levels of eight metallic elements in the fruiting bodies of Bay Bolete (Boletus badius; current n

247 Along with the abundance of cospin in fruiting bodies of C. cinerea and the lack of trypsin-li

248 tely from the external mycelial cortices and fruiting bodies of Chinese Cordyceps from different samp

249 was to evaluate the chemical composition of fruiting bodies of P. ostreatus grown on blank and print

250 Methanolic extracts of processed fruiting bodies of six edible mushroom species (Basidiom

251 y associated with development of the asexual fruiting bodies of the fungus on certain substrates.

252 The fruiting bodies of the fungus Phycomyces blakesleeanus a

253 Fruiting bodies of the oyster mushroom Pleurotus ostreat

254 g multicellular aggregates that develop into fruiting bodies of viable spores and dead stalk cells.

255 The mature fruiting body of Dictyostelium consists of stalk and spo

256 ing does not spread to neighboring asci in a fruiting body of mixed genetic constitution.

257 nutritional and chemical composition of the fruiting bodies; optimize the preparation of bioactive p

258 ed by starvation inside cell aggregations of fruiting bodies or is induced artificially by glycerol i

259 and secrete cellulose coats but do not form fruiting bodies or significant numbers of viable spores.

260 The fruiting bodies, or mushrooms, of terrestrial fungi have

261 ally to mat A males (conidia) or form mature fruiting bodies (perithecia) or meiotic progeny (ascospo

262 ing sonication-resistant spores, and compact fruiting bodies persisted after nutrient addition.

263 ng, but for two of the strains the resulting fruiting bodies remained flattened mounds of cells.

264 Sporulation within the nascent fruiting body requires signaling between moving cells in

265 Chemical analysis of fruit bodies revealed that they are rich in proteins (27

266 ns (Deltapkn8 and Deltapkn14) developed into fruiting bodies significantly faster than that of the pa

267 complex of polypeptides, inhibits group and fruiting body size.

268 production of nutrient rich and saponin free fruit bodies/spent.

269 e photosensory input for phototropism of the fruiting body sporangiophores, but the madC gene has rem

270 vegetative cells with the proteome of mature fruiting body spores.

271 s during Bailinggu's mycelia, primordia, and fruiting body stages to identify genes regulating fruiti

272 molyte in certain mushrooms to help maintain fruiting body structure.

273 vior is the aggregation of cells into raised fruiting body structures in which cells differentiate in

274 n fruiting bodies was much lower than across fruiting bodies, suggesting that migration across even s

275 e resulting spore population of a M. xanthus fruiting body than the tan vegetative cells that contrib

276 deum when migrating slugs differentiate into fruiting bodies that contain persistent spores on top of

277 larly by reducing chimerism in multicellular fruiting bodies that develop near colony-territory borde

278 length determines the spacing and pattern of fruiting bodies that will rise up presaging sporulation.

279 rate into defined tissues and develop into a fruiting body that consists of a stalk and spores.

280 at eventually produced aerial structures, or fruiting bodies, that served as preferential sites for s

281 oach the top of the stalk in a Dictyostelium fruiting body, they rapidly encapsulate in response to t

282 phatidylethanolamine and to form well-spaced fruiting bodies, though substantial aggregation was obse

283 millimeters of nearly still air surrounding fruiting bodies to reach dispersive air flows.

284 s adenylyl cyclase can develop so as to form fruiting bodies under some conditions if PKA is made con

285 Upon starvation they build multicellular fruiting bodies using a developmental program that progr

286 and arsenic concentrations in the associated fruit-bodies was found.

287 However, genetic variation within fruiting bodies was much lower than across fruiting bodi

288 Within the fruit-bodies, we found the majority of arsenic accumulat

289 Bay Bolete fruiting bodies were collected from the forest area near

290 Eight of the 10 fruiting bodies were found to be internally diverse, wit

291 ucibly induce hundreds of randomly localized fruiting bodies when exposed to low nutrient availabilit

292 ents are plentiful and to form developmental fruiting bodies when nutrients are limiting.

293 lls that grow into complex structures called fruiting bodies, where they later sporulate.

294 y some of the initial aggregates mature into fruiting bodies, whereas others disperse, merge, or spli

295 but it has never been observed to produce a fruiting body, which calls to question its capacity for

296 lation of myxobacteria aggregates to build a fruiting body whose shape is species-specific and within

297 m discoideum, a social slime mold that forms fruiting bodies with spores, depends on inorganic polyph

298 nisms but aggregate upon starvation and form fruiting bodies with viable spores and dead stalk cells.

299 owed reduced aggregation and failure to form fruiting bodies with viable spores.

300 earn how myxococcus builds its multicellular fruiting body within which it differentiates spores.