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1 lation between abuse and low birth weight in full term infants.
2 that differentiate them from sham stimuli in full-term infants.
3 value was high despite a low recall rate in full-term infants.
4 (< 2,500 g), and birth weight (grams) among full-term infants.
5 ociation between PFOS and birth weight among full-term infants.
6 tion may adversely affect birth weight among full-term infants.
7 as issues pertaining to feeding preterm and full-term infants.
8 m the umbilical cord veins of their healthy, full-term infants.
9 yse associated factors in former preterm and full-term infants.
10 d cerebral palsy or cPVL in both preterm and full-term infants.
11 n in RA women after disease onset) and small full-term infants.
12 compared with results from our database for full-term infants.
13 t formula affects the iron status of healthy full-term infants.
14 ity of poor cognitive outcomes compared with full-term infants.
17 association between PFOS and birth weight in full-term infants (-29 g per log unit increase; 95% CI:
19 (mean +/- SD, 26.1 +/- 13.8%) compared with full-term infants (7.3 +/- 8.2%; P = 0.020) and prematur
22 ed in a forward-masking paradigm in healthy, full-term infants aged 6 weeks (n = 111) and 9 months (n
25 ity of handheld SD OCT imaging of the ONH in full-term infants and children without anaesthesia or se
26 erate the number of T cells was performed on full-term infants and preterm infants when they reached
27 microbiome colonization patterns relative to full-term infants, and it is speculated that the hospita
28 of birth is recognised in about one in 4000 full-term infants, and may present with neurological and
30 included in the original study (n = 382) as full-term infants born after a low-risk pregnancy were i
33 The results of this study suggest that for full-term infants, breast-feeding is associated with enh
34 nt risk factors for low birth weight for the full term infants but not the preterm infants on a bivar
37 received non-selenium-fortified preterm and full-term infant formulas containing 0.12 and 0.11 mumol
38 2 g) received selenate-fortified preterm and full-term infant formulas containing 0.36 and 0.22 mumol
42 why disease severity differs among healthy, full-term infants; however, virus titers, inflammation,
43 ping of the umbilical cord of normal-weight, full-term infants improved iron and haematological statu
44 lies in Honduras, 3) low-birth-weight (LBW), full-term infants in Honduras, and 4) infants in Ghana.
45 The probability of low birth weight among full-term infants in the population was estimated using
48 e dramatically different from those in which full-term infants mature and thus likely impact the deve
51 ency of miscarriage, premature births, small full-term infants, perinatal deaths, and births of live
53 ent RNFL thickness measurements for healthy, full-term infants that may serve as normative data for f
54 children who had been normal birthweight and full-term infants, the cromolyn without steroids group h
57 wever, adequate to meet the calcium needs of full-term infants when the formula's calcium content is
58 cits of cerebral palsy (CP) in premature and full-term infants who present with neonatal encephalopat
60 nd feasibility phase 1/2 trial, we recruited full-term infants younger than 48 h who had hypoxic isch
61 brile (>/=38 degrees C), previously healthy, full-term infants younger than 60 days for whom blood cu
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