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1 or patients with decompensated cirrhosis and fulminant liver failure.
2 rosis factor (TNF)-mediated murine models of fulminant liver failure.
3 tation is wide, varying from mild disease to fulminant liver failure.
4  neurological disability, then developed sub-fulminant liver failure.
5 ed in a variety of diseases, including acute fulminant liver failure.
6  history of a febrile illness presented with fulminant liver failure.
7 eutic strategy for hepatic disorders such as fulminant liver failure.
8                      The patient died due to fulminant liver failure 14 months later; the histopathol
9  massive hepatocyte apoptosis and subsequent fulminant liver failure and death in D-GalN-sensitized m
10 ients with Wilson's disease who present with fulminant liver failure and for patients unresponsive to
11 r transplantation (LT) between patients with fulminant liver failure and those with cirrhosis and sev
12 pression can lead to severe acute hepatitis, fulminant liver failure, and death.
13 dinedione sometimes, although rarely, led to fulminant liver failure, and ultimately the drug was rem
14            We previously reported that acute fulminant liver failure associated with B19 virus infect
15  is currently the only effective therapy for fulminant liver failure, but its use is limited by the s
16 nitive treatment for end-stage cirrhosis and fulminant liver failure, but the lack of available donor
17 h is frequently up-regulated in HCCs, delays fulminant liver failure in mice by inhibiting apoptosis,
18 followed by 6 hours of reperfusion induced a fulminant liver failure in WT and Stat6 KO mice, as asse
19 patient who is admitted urgently with acute (fulminant) liver failure in whom consent may not be poss
20 SCs) protect from death in a lethal model of fulminant liver failure induced by intraperitoneal injec
21                      These data suggest that fulminant liver failure may potentially benefit from tre
22 rnative medicine agents are a major cause of fulminant liver failure necessitating liver transplantat
23 ed liver injury has been described involving fulminant liver failure or death.
24  or definite AIH) among the 70 patients with fulminant liver failure was 24% for simplified criteria
25 cluding a subset of patients presenting with fulminant liver failure who required liver transplant.
26  DNA, but not control non-CpG DNA, induces a fulminant liver failure with subsequent shock-mediated d
27 nt treatment also prevents TNFalpha-mediated fulminant liver failure without affecting liver regenera

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