ライフサイエンスコーパス: fume

1 nts present in simulated road paving asphalt fumes.

2 g simulated occupational exposure to asphalt fumes.

3 jor component of cigarette smoke and cooking fumes.

4 ught to be mediated by manganese (Mn) in the fumes.

5 MMA-HS) or gas metal arc-mild steel (GMA-MS) fumes.

6 o green upon grinding and green to blue upon fuming.

7 population, occupational exposure to welding fumes accounted for approximately 4% of lung cancer case

8 ned high porosity and ultrastability even in fuming acids.

9 ulus and show diminished avoidance of acidic fumes, ammonia, and carbon dioxide.

10 nual job/industry-specific estimates of lead fume and lead dust exposure, derived from a statistical

11 379) to estimate cumulative exposure to lead fume and lead dust.

12 al-fold, as do other exposures such as metal-fume and wood-dust exposure.

13 , cells at higher voltages exhibited sparks, fumes and fire.

14 tional agents, such as epoxy resins, welding fumes and hand-arm vibration, have been investigated, bu

15 posure of Homo to these elements, via fires, fumes and their ashes, which could have played certain r

16 time occupational history, exposure to metal fume, and potential confounding factors.

17 coryza, and exposure to cigarettes, cooking fumes, and other children in the home were each signific

18 xposure to diesel exhaust, solvents, welding fumes, and other respiratory irritants.

19 oking of meats at high temperatures produces fumes, and these fumes can be suspended as aerosols via

20 es to biological dusts, mineral dusts, gases/fumes, and vapors, gases, dusts, or fumes (VGDF) (high,

21 es to biological dusts, mineral dusts, gases/fumes, and VGDF were associated with incidence of COPD o

22 odels indicate that condensates from asphalt fumes are genotoxic and can promote skin tumorigenesis.

23 LCINS, including exposure to radon, cooking fumes, asbestos, heavy metals, and environmental tobacco

24 dicate that this technique could treat CH3Br fumes at approximately $5/kg, roughly one-third of the c

25 Repeated instillations of the two fumes at doses that mimic approximately 1 to 5 yr of wor

26 Exposure to the OF or ether fumes both produced increases in plasma corticosterone (

27 de high temperature environments and exhaust fumes, but electromagnetic fields have not been implicat

28 Behavioral avoidance of acidic fumes, but no increased labeling in the trigeminal pain

29 ation of total organic matter of the asphalt fume by electron impact ionization of isotope dilution g

30 clic aromatic hydrocarbons (PAHs) in asphalt fume by selected ion monitoring.

31 In addition, topical application of asphalt fumes by painting the tail skin of mice increased AP-1 a

32 irst report showing that exposure to asphalt fumes can activate AP-1 and intracellular signaling that

33 high temperatures produces fumes, and these fumes can be suspended as aerosols via the vapor-to-part

34 These results demonstrate that asphalt fume composition could be characterized and specific pri

35 Generated at 150 degrees C, the asphalt fume concentration in the animal exposure chamber ranged

36 With the developed method, asphalt fumes could be characterized into three fractions: (1) f

37 sult of their exposures, and therefore these fumes could not be a hazard to the general public's heal

38 profiles, whereas asthma related to welding fumes differed.

39 nfertility and exposure to shift work, metal fumes, electromagnetic fields, solvents, lead, paint, pe

40 Flight crews complain of illness following a fume event in aircraft.

41 ed (p < 0.001) in the lung tissue of asphalt-fume-exposed mice relative to tissue from control animal

42 elevated (p < 0.001) in the urine of asphalt fume-exposed rats relative to controls.

43 In the urine of the asphalt fume-exposed rats, benzo[a]pyrene and its metabolites of

44 hydroxy metabolites in the urine of asphalt fume-exposed rats.

45 is study investigated the effects of welding fume exposure on correlates of oxidative stress in the s

46 applications: a study on the effect of metal fume exposure on immune response and a study of gene exp

47 step to study the health effects of asphalt fume exposure, an analytical method was developed to cha

48 g, the odds ratios for persons with dust and fume exposures for chronic cough, chronic phlegm, persis

49 laces are constructed such that they have no fume extraction system, and so all of the gases from com

50 umented in a variety of exposures, including fumes from flavoring plants, smoke from burn pits, and e

51 Capture of CH3Br fumes from fumigation chambers onto GAC, and electrolyti

52 ing individual particulate PhIP as simulated fumes from meat cooking, were constantly produced via co

53 Behaviorally, naked mole-rats did not avoid fumes from moderately high concentrations of acetic acid

54 nition of Gulf War illness, with exposure to fumes from munitions having the highest odds ratio (odds

55 He stated that the fumes from such establishments were not hazardous.

56 dried for reuse to capture and destroy CH3Br fumes from the next fumigation.

57 by other members of their households and to fumes from the use of gas as a cooking fuel.

58 Occupational exposure to gas, dust, and fumes (GDF) increases the risk of asthma and eczema.

59 nd validated for characterization of asphalt fume generated under simulated road paving conditions.

60 A dynamic asphalt fume generation system was modified to provide consisten

61 In the process of fume generation, asphalt was initially preheated in an o

62 reused for multiple runs, and avoids use of fuming HCl.

63 The procedures are performed in a typical fume hood using ordinary laboratory glassware.

64 nt of half the size of a standard laboratory fume hood.

65 d at room temperature (20-25 degrees C) in a fume hood.

66 rs, crystallizers, and filters in laboratory fume hoods.

67 mice were exposed daily (4h/day) to asphalt fume in a whole-body inhalation chamber for 10 days; 16

68 with reported occupational exposure to metal fume in the previous year (adjusted odds ratio (OR) = 1.

69 with 8 as controls and 16 exposed to asphalt fumes in a whole-body inhalation chamber for 10 days (4

70 Occupational exposure to dust and fumes in men and women is similarly associated with airf

71 ing was greater in those exposed to dust and fumes in men and women.

72 biological dust, mineral dust, and gases and fumes in relation to FEV1 levels.

73 potential involvement of exposure to asphalt fumes in skin carcinogenesis.

74 Although occupational exposure to dust and fumes is considered a risk factor for chronic obstructiv

75 Occupational exposure to asphalt fumes may pose a health risk.

76 t to assess the effect of mild steel welding fumes (MS-WF) on PAFR-dependent pneumococcal adhesion an

77 s also observed in dye 5, where grinding and fuming of a solid sample gave blue- and red-shifted emis

78 nonporous surfaces consists of cyanoacrylate fuming of the fingerprint material, followed by impregna

79 vestigated the effect of exposure to asphalt fumes on AP-1 activation in mouse JB6 P+ epidermal cells

80 risk of chronic cough seen with occupational fumes or smoke exposure disappeared after adjusting for

81 without prior development with cyanoacrylate fuming or Vacuum Metal Deposition, was also examined.

82 bar pneumonia and recent exposure to ferrous fume (OR = 2.3, 95% CI: 1.2, 4.3).

83 bability of exposures to dusts, gas, vapors, fumes, or sensitizers also contributed significantly to

84 Exposure to dusts, gas, vapors, fumes, or sensitizers was associated with a significantl

85 with both previous exposure (PE) to dust and fumes (P = 0.006) and airflow limitation (AFL) (P = 0.03

86 8 (95% CI: 1.01, 1.38) was found for welding fumes (prevalence controls: 22.8%), increasing to 1.38 f

87 Exposure to asphalt fumes promoted basal as well as epidermal growth factor-

88 n task for many workers, exposure to welding fumes represents an important risk factor for lung cance

89 test the hypothesis that inhalation of metal fume reversibly increases susceptibility to pneumonia, t

90 thesis that ferrous and possibly other metal fumes reversibly predispose to infectious pneumonia.

91 Exposure to asphalt fumes significantly increased AP-1 activity in JB6 P+ ce

92 ere less than those measured on a nonporous, fumed silica (Cabosil) and were also found to decrease a

93 Specifically, we find for fumed silica a positive correlation of toxicity with hyd

94 atic interactions of the silanol surfaces of fumed silica aggregates with the extracellular plasma me

95 lial and macrophage cells, we discovered for fumed silica an important toxicity relationship to posts

96 ensive routes to high-purity precipitated or fumed silica and compounds with single Si-C bonds.

97 ica zeolite ITQ-12 has been synthesized with fumed silica as the silica source in the presence of 1,3

98 eated equal and that the unusual toxicity of fumed silica compared to that of colloidal silica derive

99 The stability of the fumed silica derivative, however, is greatly compromised

100 al silica into aggregates mimicking those of fumed silica had no effect on cell viability or hemolysi

101 Easy to prepare solid materials based on fumed silica impregnated with polyethylenimine (PEI) wer

102 to generate hydroxyl radicals for Stober and fumed silica NPs with comparable primary particle sizes

103 ation of various nitric oxide (NO)-releasing fumed silica particles (0.2-0.3 microm) are reported.

104 urther shown that the resulting NO-releasing fumed silica particles can be embedded into polymer film

105 physical dispersion of nonporous, nanoscale, fumed silica particles in glassy amorphous poly(4-methyl

106 bstantially more stable derivative made from fumed silica possess equivalent local framework wall str

107 e sodium - free mesostructure assembled from fumed silica retains an open framework under the same hy

108 We propose fumed silica toxicity stems from its intrinsic populatio

109 silica) or high-temperature pyrolysis (e.g., fumed silica) routes.

110 xtural properties and chemical reactivities (fumed silica, amorphous silica and MCM-41) was evaluated

111 l radicals generated by the strained 3MRs in fumed silica, but largely absent in colloidal silicas, m

112 lystyrene), various silica precursors (TEOS, fumed silica, or zeolite seed), and many oils (decane, p

113 conventional filled polymer systems, reflect fumed silica-induced disruption of polymer chain packing

114 A highly active and durable fumed silica-supported heterogeneous molybdenum(VI) cata

115 n nanotubes, graphene, carbon black, Ag, and fumed SiO2 nanoparticles.

116 estimates of occupational exposure to dust, fumes, smoke, and gas.

117 sitol 3-kinase activation eliminated asphalt fume-stimulated AP-1 activation and formation of anchora

118 Fuming sulfuric acid charges SWNTs and promotes their or

119 arbon nanotube fibers were swollen in oleum (fuming sulfuric acid), and organic spacer groups were co

120 Characterization of the asphalt fume test atmospheres included the following: (1) determ

121 llergens, (ii) isocyanates and (iii) welding fumes the day after relevant exposure.

122 in part mediated by the capacity of welding fumes to increase PAFR-dependent pneumococcal adhesion a

123 Asphalt fumes transiently activated c-Jun NH2-terminal kinases w

124 iously, we described a system in which CH3Br fumes vented from fumigation chambers could be captured

125 s, gases/fumes, and vapors, gases, dusts, or fumes (VGDF) (high, low, or unexposed as reference).

126 rted job exposure to vapors, gases, dust, or fumes (VGDF) (PR 4.3; 95% CI 2.2 to 8.6).

127 tional exposure to vapors, gases, dusts, and fumes (VGDF) and pesticides is associated with a lower l

128 pational exposures to vapors, gas, dust, and fumes (VGDF) are associated with high-attenuation areas

129 The fume was conducted from the generator to an exposure cha

130 The asphalt fume was generated at 180 degrees C and the concentratio

131 Total organic matter of the asphalt fume was quantified over the 5 exposure days.

132 Occupational exposure to both dust and fumes was reported by 47.9% of men and 20.1% of women.

133 reatened the closure of many factories whose fumes were considered hazardous to the public's health.

134 Asphalt fumes were generated from a dynamic generation system th

135 An important health concern of welding fume (WF) exposure is neurological dysfunction akin to P

136 at associated with another irritant, ammonia fumes, which elicited an increase in trigeminal but not

137 responses of this species to airborne acidic fumes, which would be expected to affect the trigeminal