ライフサイエンスコーパス: fumigatus

1 vailable in 1 patient and showed Aspergillus fumigatus.

2 llowing subchronic inhalation exposure to A. fumigatus.

3 ugmented in animals repeatedly exposed to A. fumigatus.

4 the opportunistic human pathogen Aspergillus fumigatus.

5 le in antifungal defense against Aspergillus fumigatus.

6 d against GBS serotypes for reactivity to A. fumigatus.

7 tan (GAG) contributes to the virulence of A. fumigatus.

8 or Asp f 13 null (13) strains of Aspergillus fumigatus.

9 lergic immunological response to Aspergillus fumigatus.

10 ue transcriptional regulatory pathways in A. fumigatus.

11 ed individuals that is caused by Aspergillus fumigatus.

12 -ficolin modulates the immune response to A. fumigatus.

13 ents with asthma who are IgE sensitized to A fumigatus.

14 , in the human pathogenic fungus Aspergillus fumigatus.

15 active against a third pathogen, Aspergillus fumigatus.

16 the opportunistic human pathogen Aspergillus fumigatus.

17 might harbor triazole-resistant Aspergillus fumigatus.

18 esulting in azole-resistant phenotypes of A. fumigatus.

19 gents Listeria monocytogenes and Aspergillus fumigatus.

20 pounds affected cell wall biosynthesis in A. fumigatus.

21 cial role in propagation and virulence of A. fumigatus.

22 ) pathway in the fungal pathogen Aspergillus fumigatus.

23 es involved in innate responses to viable A. fumigatus.

24 cell transplantation, especially regarding A fumigatus.

25 uding real-time visualization of Aspergillus fumigatus (5 d for culturing, 1-2 d for imaging).

26 responses against fungi, such as Aspergillus fumigatus, a major fungal pathogen in humans.

27 pores of the environmental mould Aspergillus fumigatus, a major opportunistic pathogen.

28 Using Aspergillus fumigatus, a respiratory pathogen, we characterize the i

29 Aspergillus fumigatus, a ubiquitous human fungal pathogen, produces

30 Conditioned media (CM) from A. fumigatus, A. niger, and A. flavus cultures also reduced

31 In contrast to Aspergillus fumigatus, A. terreus infections are associated with hig

32 e when paired with the MAT1-1 isolates of A. fumigatus, A. viridinutans, A. felis, A. pseudoviridinut

33 In Aspergillus fumigatus, AcuM governs gluconeogenesis and iron acquisi

34 mothers, the magnitude of HDM or Aspergillus fumigatus (AF) extract-induced airway hyperresponsivenes

35 e sensitised and challenged with Aspergillus fumigatus (Af).

36 n to ubiquitous fungi, typically Aspergillus fumigatus (Af).

37 with ABPA with the classically described A. fumigatus allergens Aspf1, Aspf2, Aspf3, and Aspf4, as w

38 globulin E and G4 directed to recombinant A. fumigatus allergens in 55 cystic fibrosis patients witho

39 IgG4 responses to recombinant A. fumigatus allergens were detected in all patients, but n

40 t include immune responses to recombinant A. fumigatus allergens.

41 not limited to the classically described A. fumigatus allergens.

42 ngi such as Candida albicans and Aspergillus fumigatus also form biofilms during infection.

43 rick test response positivity to Aspergillus fumigatus, Alternaria alternata, or Cladosporium herbaru

44 IgE sensitization to Aspergillus fumigatus and a positive sputum fungal culture result ar

45 ted in the development of azole-resistant A. fumigatus and address the therapeutic options available.

46 us nidulans, Aspergillus oryzae, Aspergillus fumigatus and Aspergillus niger.

47 have a limited ability to migrate toward A. fumigatus and block the growth of A. fumigatus conidia (

48 Trafficking of TLR9 to A. fumigatus and C. albicans phagosomes requires Dectin-1 r

49 with the human pathogenic fungi Aspergillus fumigatus and Candida albicans induces a distinct subset

50 on of innate immune responses to Aspergillus fumigatus and Candida albicans.

51 , AIM2 and NLRP3, recognize intracellular A. fumigatus and collectively induce protective immune resp

52 the first survey of glucose epimerases in A. fumigatus and contributes to our understanding of the ro

53 L-ficolin, has been observed to recognize A. fumigatus and could participate in fungal defense.

54 ted against the fungal pathogens Aspergillus fumigatus and Cryptococcus neoformans, little was known

55 st commonly by Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, result in more de

56 lturally derived azole-resistant Aspergillus fumigatus and emerging threats such as multidrug resista

57 a-(1,3)-glucan polysaccharide of Aspergillus fumigatus and ensuing CD4+ T-cell polarization are poorl

58 e sensitized and challenged with Aspergillus fumigatus and evaluated ex vivo in tissue culture.

59 d calcium-mediated signalling in Aspergillus fumigatus and found that calcium chelation severely impe

60 ns of the class II and class V myosins in A. fumigatus and found that while the class II myosin (myoB

61 cnn1b-Tg and wild-type mice with Aspergillus fumigatus and house dust mite allergen and compared the

62 pulmonary aspergillosis but sensitized to A. fumigatus and in nine patients with allergic broncho-pul

63 s the dominant UDP-glucose 4-epimerase in A. fumigatus and is essential for normal growth in galactos

64 primary immune responses directed against A. fumigatus and novel therapeutic strategy for IPA.

65 In Aspergillus fumigatus and other fungal pathogens, the transcription

66 standing of host-pathogen interactions in A. fumigatus and patients with cystic fibrosis and the ongo

67 e domains from these proteins (Sph3h from A. fumigatus and PelAh from P. aeruginosa) were found to de

68 d by the opportunistic pathogens Aspergillus fumigatus and Pseudomonas aeruginosa, respectively.

69 typic bronchiole and cultures of Aspergillus fumigatus and Pseudomonas aeruginosa.

70 usceptible to aeroallergens from Aspergillus fumigatus and the house dust mite, resulting in an asthm

71 y result from the interaction of Aspergillus fumigatus and the immune system of its human host.

72 These cells recognize naturally processed A. fumigatus and the multispecific T-cell lines, directed a

73 Sensitisation to Aspergillus fumigatus and/or Penicillium chrysogenum was associated

74 eveloped invasive aspergillosis (Aspergillus fumigatus) and mucormycosis (Lichtheimia corymbifera) mu

75 fungi including Candida species, Aspergillus fumigatus, and Cryptococcus neoformans.

76 ung disease caused by the fungus Aspergillus fumigatus, and is a leading cause of invasive fungal inf

77 domonas aeruginosa, Haemophilus, Aspergillus fumigatus, and nontuberculous mycobacteria.

78 for TLR9 trafficking to beta-1,3 glucan-, A. fumigatus-, and C. albicans-containing phagosomes.

79 In conclusion, irrespective of the A. fumigatus antigen, the T-cell immune response in patient

80 f Alternaria alternata allergen, Aspergillus fumigatus antigens, house dust mite and endotoxin antige

81 produced IFN-gamma after stimulation with A. fumigatus antigens.

82 ons caused by triazole-resistant Aspergillus fumigatus are associated with a higher probability of tr

83 infections with fungal pathogen Aspergillus fumigatus are associated with caspofungin prophylaxis.

84 All patients with IgE sensitisation to A. fumigatus are at risk of lung damage irrespective of whe

85 luding Asp f 5 and Asp f 13 from Aspergillus fumigatus are thought to be important for initiation and

86 th and virulence of the pathogen Aspergillus fumigatus are unknown.

87 s brasiliensis, and occasionally Aspergillus fumigatus, are primary pulmonary pathogens of otherwise

88 usively via association with properdin on A. fumigatus as validated by detection of C3b deposition an

89 utcome of human macrophage infection with A. fumigatus, as well as the impact of calcineurin inhibito

90 from the human pathogenic fungus Aspergillus fumigatus, Aspergillus fumigatus tetramycovirus-1 (AfuTm

91 Accurate and prompt identification of A fumigatus-associated clinical status might allow early a

92 e showed that deletion of asp f3 rendered A. fumigatus avirulent in a mouse model of pulmonary asperg

93 d, necrosis-dependent lateral transfer of A. fumigatus between macrophages as an important host strat

94 e observed frequent cell-cell transfer of A. fumigatus between macrophages, which assists subsequent

95 ment of A. fumigatus with Sph3h disrupted A. fumigatus biofilms with an EC50 of 0.4 nM.

96 PelAh treatment also disrupted preformed A. fumigatus biofilms with EC50 values similar to those obt

97 tolerant filamentous fungi, in particular A. fumigatus but not total IgE, is associated with fixed ai

98 erwise naive mice in response to Aspergillus fumigatus, but not ovalbumin sensitization and challenge

99 c outcome of an interaction with Aspergillus fumigatus by influencing triacetylfusarinine production.

100 d conidial uptake and enhanced killing of A. fumigatus by MDMs and neutrophils.

101 Thus, A. fumigatus CalA is an invasin that interacts with integri

102 opportunistic pathogenic fungus Aspergillus fumigatus, called aspergillosis.

103 Aspergillus fumigatus can exploit the hypoxic microenvironment in th

104 eptible individuals, exposure to Aspergillus fumigatus can lead to the development of atopic lung dis

105 Three isolates (Fusarium solani, Aspergillus fumigatus, Candida albicans) recovered from patients wit

106 lallyl tryptophan synthase) from Aspergillus fumigatus catalyze C(4)- and C(7)-prenylation of the ind

107 Aspergillus fumigatus causes chronic cavitary pulmonary aspergillosi

108 The A. fumigatus cell wall is a complex network of polysacchari

109 al or biological (germination) removal of A. fumigatus cell wall melanin.

110 n CalA is expressed on the surface of the A. fumigatus cell wall, where it mediates invasion of epith

111 Acute A. fumigatus challenge in normal mice induced the recruitme

112 al activity, and lung fungal clearance in A. fumigatus-challenged mice.

113 d lung inflammation in naive and Aspergillus fumigatus-challenged wild-type and Rgs5(-/-) mice.

114 ncy in eosinophils, demonstrated impaired A. fumigatus clearance and evidence of germinating organism

115 Neutrophil/A fumigatus coculture, flow cytometry, and video microscop

116 common fungus in asthmatics was Aspergillus fumigatus complex and this taxon accounted for the incre

117 cs and the most common fungus is Aspergillus fumigatus complex.

118 l wall of the filamentous fungus Aspergillus fumigatus comprises polysaccharides.

119 ward A. fumigatus and block the growth of A. fumigatus conidia (proportion with growth blocked, 69%).

120 ng myeloid cell responses against inhaled A. fumigatus conidia and demonstrates a benefit for systemi

121 , are involved in the killing of Aspergillus fumigatus conidia and hyphae, using neutrophils from pat

122 9, a GBSIb type-specific mAb, reacts with A. fumigatus conidia and hyphae.

123 alpha-(1,3)-Glucan was isolated from A. fumigatus conidia and mycelia cell wall.

124 s and killing of L-ficolin-opsonized live A. fumigatus conidia by flow cytometry and microscopy.

125 tin in uptake or killing of intracellular A. fumigatus conidia either in vitro or in a murine model o

126 After phagocytosis, A. fumigatus conidia rapidly escaped from DCs, whereas A. t

127 The capacity of A. fumigatus conidia to activate platelets is at least part

128 Furthermore, while A. fumigatus conidia triggered expression of DC activation

129 In this model, Aspergillus fumigatus conidia were administered into the lungs of ne

130 Specifically, germinating A. fumigatus conidia were associated with Clec7a and were p

131 were protected from antifungals, whereas A. fumigatus conidia were efficiently cleared.

132 with apoptosis-like features in Aspergillus fumigatus conidia, the most prevalent human mold pathoge

133 ubchronic inhalation exposure to Aspergillus fumigatus conidia.

134 d survival after pulmonary challenge with A. fumigatus conidia.

135 e by facilitating proper formation of the A. fumigatus conidial surface.

136 The cell wall of Aspergillus fumigatus contains two galactose-containing polysacchari

137 ures displayed high overall similarity of A. fumigatus CYP51B to CYP51 orthologs from other biologica

138 We expressed, purified, and characterized A. fumigatus CYP51B, including determination of its substra

139 Through in vitro studies, we found that A. fumigatus DeltaacuK single and DeltaacuK DeltaacuM doubl

140 A. fumigatus DeltasakA and DeltampkC were virulent in mouse

141 bolites differentially modulated Aspergillus fumigatus development, shifting from weak vegetative gro

142 Homology modeling of Aspergillus fumigatus DHODH has identified a predicted binding mode

143 ort a fluorogenic probe to image Aspergillus fumigatus directly in human pulmonary tissue.

144 the SeptiFast assay in detecting Aspergillus fumigatus DNA in whole blood samples from 38 critically

145 of the species and resistance assays for A. fumigatus DNA were 10 and >/=75 genomes/sample, respecti

146 A. fumigatus DNAemia was associated with poor outcome.

147 ion by the human-pathogenic mold Aspergillus fumigatus during invasive growth.

148 fumigatus during pathogenesis, a prerequisite to underst

149 Macrophage phagocytosis of A. fumigatus enabled control of 90% of fungal germination.

150 o acid sequence identity with an Aspergillus fumigatus entity.

151 or eosinophils in the lung response after A. fumigatus exposure.

152 ife-threatening condition associated with A. fumigatus exposure.

153 e examined the effect of topical Aspergillus fumigatus extract exposure in wild-type and Serpinb3a-nu

154 In an Aspergillus fumigatus extract-induced inflammation model, IL-10-prod

155 important contributor to the clearance of A. fumigatus from the lung.

156 The A. fumigatus genome contains three genes encoding putative

157 provements for A. nidulans, A. oryzae and A. fumigatus genomes based on recently available RNA-Seq da

158 a-glucan surface exposure during Aspergillus fumigatus germination activates an Atg5-dependent autoph

159 Lately, increased azole resistance in A. fumigatus has become a significant challenge in effectiv

160 Azole resistance in Aspergillus fumigatus has emerged as a global health problem.

161 The genome of Aspergillus fumigatus has four arsM genes encoding ArsMs with only t

162 A. fumigatus has two CYP51 genes, CYP51A and CYP51B, which

163 st of the variants that we uncover within A. fumigatus have been previously hypothesized to contribut

164 ns, Cryptococcus neoformans, and Aspergillus fumigatus have transitioned from a rare curiosity to a l

165 nfection caused predominantly by Aspergillus fumigatus, have increased due to the growing number of i

166 ed increased phosphorylation of SakA, the A. fumigatus Hog1 homologue.

167 Moreover, substituting the A. fumigatus hsp90 promoter with 2 artificial promoters (po

168 A. fumigatus hyphae also contain surface structures that in

169 Treatment of A. fumigatus hyphae with either Sph3h or PelAh significantl

170 e an impaired ability to inhibit Aspergillus fumigatus hyphal growth in vitro and in infected corneas

171 y demonstrated enhanced lung clearance of A. fumigatus IL-33 functioned as a negative regulator of mu

172 mily member IL-33 in lung defense against A. fumigatus IL-33 was detected in the naive lung, which fu

173 which further increased after exposure to A. fumigatus in a dectin-1-independent manner.

174 t assessing the activity of neutrophils on A fumigatus in allogeneic HSCT recipients at different pos

175 Targeting protease activity of Aspergillus fumigatus in conditions such as SAFS or ABPA may have be

176 amples to perform multi-photon imaging of A. fumigatus in ex vivo human tissue.

177 Colonization by Aspergillus fumigatus in patients with cystic fibrosis (CF) can caus

178 Persistent isolation of A fumigatus in sputum is a significant risk factor for A f

179 e against the opportunistic mold Aspergillus fumigatus In this study, we investigated the IL-1 family

180 were distinctive volatile metabolites of A. fumigatus in vitro, distinguishing it from other pathoge

181 that the peroxiredoxin Asp f3 of Aspergillus fumigatus inactivates ROS.

182 involved in innate resistance to Aspergillus fumigatus, including complement activation or promotion

183 ivo increases in fungal surface chitin in A. fumigatus induced by caspofungin that was associated wit

184 to suppress house dust mite- and Aspergillus fumigatus-induced allergic inflammation in murine models

185 and goblet cell metaplasia in an Aspergillus fumigatus-induced asthma model.

186 cted A549 pulmonary epithelial cells from A. fumigatus-induced cell damage for up to 24 h.

187 A. fumigatus induces NET formation; however, NETs did not c

188 Our study demonstrates that Aspergillus fumigatus induces regulatory T-cells with a TH17-like ph

189 alveolar lavage fluid (BALF) and lungs of A. fumigatus-infected chronic granulomatous disease (CGD),

190 tibody (mAb), JF5, to neutrophil-depleted A. fumigatus-infected mice allowed specific localization of

191 y contributed to the host defense against A. fumigatus infection and to endotoxemia.

192 Aspergillus fumigatus infection produces ligands that could activate

193 g invasive C. albicans infection, but not A. fumigatus infection.

194 ystem that orchestrates responses against A. fumigatus infection.

195 esistance with limited immunopathology in A. fumigatus infection.

196 mount poorly protective Th2 responses to A. fumigatus infection.

197 role of neutrophils in protection against A. fumigatus infections, we developed an in vitro assay in

198 nt from that employed to control Aspergillus fumigatus infections.

199 to enhance protective immune responses to A. fumigatus infections.

200 (IA) resulting from infection by Aspergillus fumigatus is a leading cause of death in immunosuppresse

201 PI lipid remodelling and PerA function in A. fumigatus is a promising research direction to uncover a

202 Aspergillus fumigatus is a sporulating fungus found ubiquitously in

203 Azole resistance in Aspergillus fumigatus is an increasing problem.

204 Aspergillus fumigatus is an opportunistic fungal pathogen that invad

205 Aspergillus fumigatus is an opportunistic human fungal pathogen that

206 Aspergillus fumigatus is an opportunistic human pathogen responsible

207 ion against fungal infections by Aspergillus fumigatus is essential but not fully understood.

208 While A. fumigatus is reliant upon deficiencies in the host to fa

209 TR46/Y121F/T289A azole-resistant Aspergillus fumigatus is reported.

210 attenuated virulence of melanin-deficient A. fumigatus is restored in Atg5-deficient macrophages and

211 Aspergillus fumigatus is the causative agent of allergic broncho-pul

212 n B endoperoxidase (FtmOx1) from Aspergillus fumigatus is the first reported alpha-ketoglutarate-depe

213 Among these Aspergillus species, A. fumigatus is the most frequent agent of invasive aspergi

214 Aspergillus fumigatus is the most virulent species within the Asperg

215 Aspergillus fumigatus is the opportunistic fungal pathogen that pred

216 Neosartorya fumigata (Aspergillus fumigatus) is the most common cause of invasive aspergil

217 gillin (1), a meroterpenoid from Aspergillus fumigatus, is known for its antiangiogenic activity due

218 r of the human pathogenic fungus Aspergillus fumigatus, is the prototype of epipoly(thiodioxopiperazi

219 d TR46 Y121F T289A mutations in confirmed A. fumigatus isolates collected in institutions in the Unit

220 Heterogeneity among Aspergillus fumigatus isolates results in unique virulence potential

221 Third, primary environmental A. fumigatus isolates that rapidly germinate under airway c

222 s as well as azole resistance in Aspergillus fumigatus Its performance has been validated on bronchoa

223 tion; however, NETs did not contribute to A. fumigatus killing.

224 novel probe for noninvasive detection of A. fumigatus lung infection based on antibody-guided positr

225 omonas aeruginosa and the fungus Aspergillus fumigatus M-CSF treatment during engraftment or after in

226 There is evidence that some anti-A. fumigatus mAbs can provide protection against IA.

227 Exposure to the mold Aspergillus fumigatus may result in allergic bronchopulmonary asperg

228 The A. fumigatus morphological shift correlated with the produc

229 ily directed against metabolically active A. fumigatus morphotypes and is stronger against membrane p

230 es and a structural model of the Aspergillus fumigatus mtTyrRS showed that the overall topology of th

231 bin masks relevant structures, because an A. fumigatus mutant lacking the hydrophobin protein induced

232 and BALB/c mice were exposed to Aspergillus fumigatus, O3, or both (3 ppm for 2 hours).

233 o-severe asthma who were IgE sensitized to A fumigatus on either the rate of severe exacerbations, qu

234 ous activation of conidiation in Aspergillus fumigatus or Aspergillus flavus.

235 le-resistant disease are due to resistant A. fumigatus originating from the environment.

236 atural killer (NK) cells against Aspergillus fumigatus over 5 time points and compared the values to

237 Soluble CL-12 could recognize Aspergillus fumigatus partially through the carbohydrate-recognition

238 esistance assay was performed on Aspergillus fumigatus PCR-positive samples when a sufficient fungal

239 Our data support the hypothesis that A. fumigatus phenotypic variation significantly contributes

240 n in vivo mouse disease model of aspergillus fumigatus pneumonia.

241 epicutaneous sensitization with Aspergillus fumigatus protein extract.

242 The A. fumigatus proteins Crf1, Gel1, and Pmp20 induced strong

243 amined the response to different Aspergillus fumigatus proteins in healthy individuals and patients a

244 Further analysis of 15 selected single A. fumigatus proteins revealed a highly diverse reactivity

245 ity and gene expression to categorize the A. fumigatus putative in vivo secretome.

246 ey are activated by contact with Aspergillus fumigatus; putative consequences include antifungal defe

247 y aspergillosis is one of the most severe A. fumigatus-related diseases due to possible evolution tow

248 Aspergillus fumigatus remains the most common species in all pulmona

249 90) is an essential chaperone in Aspergillus fumigatus representing an attractive antifungal target.

250 he fungal opportunistic pathogen Aspergillus fumigatus Sch9 homologue (SchA).

251 Aspergillus fumigatus-secreted allergen proteases, Asp f 5 and Asp f

252 lays in vivo efficacy against a strain of A. fumigatus sensitive to the azole class of antifungals an

253 tients with cystic fibrosis (CF) can cause A fumigatus sensitization and/or allergic bronchopulmonary

254 We sought to identify A fumigatus sensitization in patients with CF by using the

255 rgic broncho-pulmonary aspergillosis from A. fumigatus sensitization with good negative and positive

256 in sputum is a significant risk factor for A fumigatus sensitization.

257 BAT was used to identify A fumigatus sensitization.

258 Itraconazole treatment did not affect A fumigatus sensitization.

259 th CF and ABPA when compared with those in A fumigatus-sensitized and nonsensitized patients with CF

260 pulmonary function and body mass index in A fumigatus-sensitized but not nonsensitized patients with

261 The BAT discriminates A fumigatus-sensitized from nonsensitized patients with CF

262 IgE, but not IgG, levels are increased in A fumigatus-sensitized patients with CF and ABPA when comp

263 ents with CF into 3 groups: nonsensitized, A fumigatus-sensitized, and ABPA.

264 Aspergillus fumigatus siderophore (SidA), a member of class B flavin

265 sms affect the function of representative A. fumigatus SM gene clusters, such as those involved in th

266 or the direct ex vivo characterization of A. fumigatus-specific CD4(+) T cells for rapid identificati

267 A. fumigatus-specific conventional T cell responses are cou

268 Serologic (total and A fumigatus-specific IgE), pulmonary function, and body ma

269 Total and A fumigatus-specific IgE, but not IgG, levels are increase

270 Administration of a [(64)Cu]DOTA-labeled A. fumigatus-specific monoclonal antibody (mAb), JF5, to ne

271 owing repeated inhalation of dry Aspergillus fumigatus spores aerosolized at concentrations potential

272 Aspergillus fumigatus spores were delivered to the lungs of naive BA

273 Germinating A. fumigatus spores were observed in lungs along with persi

274 Levels of the A fumigatus-stimulated basophil activation marker CD203c i

275 corticosteroid-treated mice infected with A. fumigatus, suggesting an effect of cortisone on bronchia

276 ic fungus Aspergillus fumigatus, Aspergillus fumigatus tetramycovirus-1 (AfuTmV-1), which reveals sev

277 atients infected with the fungus Aspergillus fumigatus, Th1 responses are considered protective, whil

278 umber of bronchial/bronchiolar spores for A. fumigatus than L. corymbifera.

279 ken together our data demonstrate that in A. fumigatus the regulatory hierarchy governing alkaline to

280 In Aspergillus fumigatus, the conidial surface contains dihydroxynaphth

281 o volatile metabolite profile of Aspergillus fumigatus, the most common cause of IA, and other pathog

282 We used Aspergillus fumigatus to induce EoE in TRAIL-sufficient (wild-type)

283 a novel stage-specific susceptibility of A. fumigatus to zinc and manganese chelation by neutrophil-

284 mplex network of interactions amongst the A. fumigatus TOR, SakA and SchA pathways.

285 ed from respiratory specimens as Aspergillus fumigatus using colonial and microscopic morphology.

286 were exposed via nose-only inhalation to A. fumigatus viable conidia, heat-inactivated conidia (HIC)

287 that methionine synthase is essential for A. fumigatus virulence, defining the biosynthetic route of

288 and the natamycin MIC90 against Aspergillus fumigatus was 4-fold higher in our study.

289 ion to animal dander, pollen, or Aspergillus fumigatus was associated with asthma.

290 Aspergillus fumigatus was the most commonly identified species.

291 nteractions between human neutrophils and A. fumigatus were observed in real time, at single-cell res

292 nses, and killing capacity of PMN against A. fumigatus were significantly decreased in all patients o

293 a, Streptococcus pneumoniae, and Aspergillus fumigatus when mice were heavily engrafted with leukemia

294 inst the widely distributed mold Aspergillus fumigatus, which is a major threat for immunocompromised

295 Therefore, in A. fumigatus, while AcuK and AcuM likely function as part o

296 hmatic patients who were IgE sensitized to A fumigatus with a history of at least 2 severe exacerbati

297 ulbs were positive for triazole-resistant A. fumigatus with CYP51A mutations.

298 Treatment of A. fumigatus with Sph3h disrupted A. fumigatus biofilms wit

299 ereas healthy people can inhale spores of A. fumigatus without developing disease, neutropenic patien

300 We also found that an A. fumigatus zafA mutant, which demonstrates deficient zinc