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1 typic changes, possibly due to mechanisms of functional compensation.
2 protein-protein interaction rather than from functional compensation.
5 nsurance within this ecosystem, that is, the functional compensation among microorganisms for the rea
6 ntain regulatory sites in chromatin and into functional compensation among nucleosome binding archite
7 prising findings were interpreted to reflect functional compensation among the BAX, BAK, and BOK prot
9 he stage of pre-TCR expression and alleviate functional compensation between E2A and HEB, we use a do
10 ese findings provide a biochemical basis for functional compensation between pRB family proteins.
12 D-type cyclin causes specific expression and functional compensation by another member of the family
13 c the early-onset of LCA, most likely due to functional compensation by closely related genes encodin
14 ction previously undetected as the result of functional compensation by de novo expression of Hes5 in
16 development in MyoD-null mice is not due to functional compensation by MyoD non-expressing lineages.
19 /EBPbeta targets in vivo has been limited by functional compensation by other C/EBP family proteins a
20 notype in PDZK1-deficient mice may be due to functional compensation by other PDZ domain-containing p
23 pigmentation in cht melanocytes results from functional compensation by the closely related Rab32.
24 oductive ability of male mice, likely due to functional compensation by the relatively higher express
25 as been provided that gene traffic may allow functional compensation during meiotic sex chromosome ac
26 ere is a significantly higher probability of functional compensation for a duplicate gene than for a
27 nism used during activity reduction provides functional compensation for gene KO in the cGKII KO hipp
28 daptive to the extent that it contributes to functional compensation for lesion-induced impairments.
29 in c-IAP1 and c-IAP2 expression may provide functional compensation for loss of XIAP during developm
30 restricting neuroendocrine cell fate despite functional compensation for Rb deficiency in other cell
31 pathway in the KO hippocampus that provides functional compensation for the LTP impairment observed
32 osal that absence of overt defects is due to functional compensation from a related homeodomain trans
33 ished in simple disruption analysis due to a functional compensation from the residual bHLH homodimer
34 ormance is equated, MCI patients demonstrate functional compensation in brain regions subserving sema
36 of functional networks that allows for rapid functional compensation in response to focal dysfunction
38 e experiments demonstrate the feasibility of functional compensation in vivo for the first time, whic
42 xpression patterns after duplication, making functional compensation less probable for ancient duplic
43 lve in vivo to highly virulent phenotypes by functional compensation of a detrimental amino acid subs
45 ere is little evidence supporting widespread functional compensation of divergently expressed duplica
46 elevated levels of LeETR4 mRNA, indicating a functional compensation of LeETR4 for reduced NR express
49 , termed redundancy, is thought to be due to functional compensation of the mutated sites by other el
50 in is essential for predicting the limits of functional compensation that can be achieved after vario
52 agic pathways, suggesting the possibility of functional compensation when one of them is compromised.
53 recombinant decorin and biglycan confirmed a functional compensation, with both having similar effect
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