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1 ntral nervous system (CNS) damage, promoting functional recovery.
2 compensation in perilesion cortex, enabling functional recovery.
3 trol of corticospinal connections to promote functional recovery.
4 prolonged hemorrhage, and impaired forelimb functional recovery.
5 ons that regulates motor behavior capable of functional recovery.
6 ings were noted in analyses of survival with functional recovery.
7 l repair cell, neuronal death and failure of functional recovery.
8 tive neurogenesis and accelerates vestibular functional recovery.
9 e to their distal counterparts contribute to functional recovery.
10 ition following spinal cord injury and limit functional recovery.
11 formation of new synaptic contacts to enable functional recovery.
12 and synaptic connections that drive greater functional recovery.
13 ction and an improved in vitro post-ischemic functional recovery.
14 ently accompanied by a degree of spontaneous functional recovery.
15 roke-induced signal for axonal sprouting and functional recovery.
16 tive program to enable axon regeneration and functional recovery.
17 ure myocytes may have contributed to delayed functional recovery.
18 tiated into GFAP+ astrocytes, and diminished functional recovery.
19 rategies that improve nerve regeneration and functional recovery.
20 -macular traction was associated with a full functional recovery.
21 l cord injury (SCI) is a major impediment to functional recovery.
22 onal program to enable axon regeneration and functional recovery.
23 brain tissue damage, and improves the animal functional recovery.
24 drugs) receptor hM4di abrogated spontaneous functional recovery.
25 and bipolar disorder (BD) impedes patients' functional recovery.
26 h acute ischemic stroke to improve patients' functional recovery.
27 n regaining tissue homeostasis and promoting functional recovery.
28 y neuromuscular junctions and enhanced motor functional recovery.
29 e failed to demonstrate long-term benefit on functional recovery.
30 g cells that surround hair cells, leading to functional recovery.
31 n regeneration, cutaneous reinnervation, and functional recovery.
32 ng mechanisms that promote kidney repair and functional recovery.
33 ional primary motor cortex (iM1) can promote functional recovery.
34 ndrocytes from progenitor cells and promotes functional recovery.
35 esis, thereby accelerating muscle repair and functional recovery.
36 stem (CNS) to regenerate causes very limited functional recovery.
37 eneration, circuit formation, and eventually functional recovery.
38 st-like cells did not reveal histological or functional recovery.
39 hemicord is one mechanism thought to mediate functional recovery.
40 esion site is pivotal for axon re-growth and functional recovery.
41 ession alters the pain threshold and impairs functional recovery.
42 lted in a concomitant increase in postinjury functional recovery.
43 rct size, attenuated apoptosis, and improved functional recovery.
44 t poststroke cortical plasticity and thereby functional recovery.
45 ptoms that are linked to poorer physical and functional recovery.
46 esulting in cumulative brain injury and poor functional recovery.
47 trimental and resulted in delayed repair and functional recovery.
48 SVZ manipulation is associated with DAergic functional recovery.
49 ation of the eCSC abolishes regeneration and functional recovery.
50 atment, which may contribute to the observed functional recovery.
51 e energy predicts late viability, defined by functional recovery.
52 rganization after SCI that may contribute to functional recovery.
53 score (mNSS) test was performed to evaluate functional recovery.
54 ors and other inflammatory responses prevent functional recovery.
55 which aim to enhance tissue regeneration and functional recovery.
56 uggesting a limited role for this pathway in functional recovery.
57 yelin synthesis to enhance myelin repair and functional recovery.
58 anges when considering therapies targeted at functional recovery.
59 s have suggested that amantadine may promote functional recovery.
60 on of BDNF signaling pathways may facilitate functional recovery.
61 rinsic power of myelin plasticity to promote functional recovery.
62 ing altered astrocyte responses and impaired functional recovery.
63 treatment can produce persistent effects on functional recovery.
64 that cell-based interventions may influence functional recovery.
65 ion of individual MAPKs allowed only partial functional recovery.
66 ssessments of muscle tissue regeneration and functional recovery.
67 obal I/R ex vivo and showed improved cardiac functional recovery.
68 but little is known about those who achieve functional recovery.
69 y lacks effective therapy enabling long-term functional recovery.
70 vomarkedly accelerated axon regeneration and functional recovery.
71 5%) were observed for up to 1 year to assess functional recovery.
72 L and OL progenitor replacement, and chronic functional recovery.
73 s of survival to discharge and survival with functional recovery.
74 vived to discharge, and 7176 (7.4%) achieved functional recovery.
75 dendritic plasticity and to induce long-term functional recovery.
76 s, promotes circuit restoration and improves functional recovery.
77 ly distinct subsets of neurons, resulting in functional recovery.
78 n functional connectivity is associated with functional recovery 1 year after cardiac arrest (CA).
79 rodent spinal cord, yet they support delayed functional recovery, a finding of great importance in pl
81 c syndrome that diminishes the potential for functional recovery after a transcatheter aortic valve r
82 ncy enhances cardiomyocyte regeneration with functional recovery after adult myocardial infarction as
85 the enhancement of both axonal regrowth and functional recovery after central nervous system injury
86 uced the risk of death and increased cardiac functional recovery after chronic myocardial ischemia.
93 at aggravates secondary injury and restrains functional recovery after experimental spinal cord injur
94 e ABC treatment promotes neuroanatomical and functional recovery after focal ischaemic stroke in an e
96 rated from skin or nerve promotes repair and functional recovery after incomplete cervical crush inju
99 p normally, but show enhanced anatomical and functional recovery after mid-thoracic dorsal hemisectio
100 ed the role of MDMs in long-term spontaneous functional recovery after middle cerebral artery occlusi
101 bl-deficient mice demonstrated a more robust functional recovery after myocardial ischemia/reperfusio
103 t 24-month-old mice exhibit an impairment of functional recovery after nerve injury compared to 2-mon
105 harmacological approach for the promotion of functional recovery after nerve injury.In vitroandin viv
107 oned mouse optic nerves (MONs) showed better functional recovery after OGD than the non-preconditione
111 vel function of HDAC3 inhibitor in promoting functional recovery after SCI by dampening inflammatory
119 ow that aged mice (18-19 months) had reduced functional recovery after spinal cord injury (SCI) assoc
124 of late gadolinium-enhancement in predicting functional recovery after ST-segment-elevation myocardia
127 GDF10 produced axonal sprouting and enhanced functional recovery after stroke; knocking down GDF10 bl
129 jured motor circuit plasticity in supporting functional recovery after trauma, and support a focus of
130 e of propofol on endogenous neurogenesis and functional recovery after traumatic brain injury, rats w
131 r neurogenesis and significantly accelerates functional recovery after unilateral vestibular injury.
133 e than nonhemorrhagic infarcts, with lack of functional recovery and adverse LV left ventricular remo
134 (plasma troponin I, myocardial lactate) and functional recovery and between myocardial tissue and pl
137 otic polyneuropathy is characterized by poor functional recovery and impaired nerve regenerative resp
138 ted efficacy from targeting this pathway for functional recovery and neural repair after spinal cord
139 enous Nogo receptor antagonist, in promoting functional recovery and neural repair after spinal cord
140 rophic factor (BDNF) can modulate vestibular functional recovery and neurogenesis in mammals, in this
142 mediates S129 phosphorylation) showed better functional recovery and smaller infarcts when subjected
143 vo resulted in an improvement in ventricular functional recovery and the prevention of myocardial rem
145 of sudden cardiac death and likelihood of LV functional recovery, and has significant potential to gu
146 hemia time are important for long-term renal functional recovery, and hypothermia should be considere
147 urviving networks appears to be critical for functional recovery, and this may be promoted with speci
149 although specific sensory reinnervation and functional recovery are usually worse for large myelinat
151 mate the dynamic probability of survival and functional recovery as a function of resuscitation effor
152 oliposomal treatment for 3 consecutive days, functional recovery as indicated by improved neurologic
153 e promoted axonal regeneration, which led to functional recovery as measured by sustained gait improv
154 epithelia, was coupled with a corresponding functional recovery, as seen in the suprathreshold ampli
155 Moreover, iM1 neuronal stimulations promoted functional recovery, as stimulated stroke mice showed fa
157 air regulating the rate of remyelination and functional recovery at early phases following injury.
159 ssociated with decreased mortality or better functional recovery but being underweight predicted unfa
160 ervention, persisting hyperglycemia prevents functional recovery but promotes beta-cell mass increase
161 I had similar predictive accuracy to SEE for functional recovery but was not assessable in 25% of pat
163 nnections, and RhoA-ROCK inhibition enhances functional recovery by blocking this detrimental effect.
164 ty-modifying molecular agents may facilitate functional recovery by selectively enhancing existing ne
165 ence that maximal circuit reorganization and functional recovery can be achieved by combining molecul
168 smaller than 10 mm in rodents, nearly normal functional recovery can be achieved; for longer gaps, ho
169 have resulted in promising, yet insufficient functional recovery compared to the clinical standard of
170 eived SCs from either source showed improved functional recovery compared with media- or fibroblast-t
171 ersed these structural defects, suggesting a functional recovery confirmed by electrophysiological re
173 ion at acute imaging and odds ratio (OR) for functional recovery decreased with increasing SEE, altho
174 ation in rates of survival and survival with functional recovery (defined as Cerebral Performance Cat
175 nsplants in vivo, we show that the extent of functional recovery depends on the age of the nerve graf
178 greatly reduced and there was no significant functional recovery even in Ryk conditional knockout mic
179 se of optical coherence tomography (SD-OCT), functional recovery evidenced by multifocal-electroretin
180 M residuals, we constructed the FRESH score (Functional Recovery Expected after Subarachnoid Hemorrha
183 ity, since it undergoes near full growth and functional recovery following acute depletion of granule
184 to achieve significant anatomical repair and functional recovery following CNS injury by manipulating
185 e mice, IL-4-deficient animals had decreased functional recovery following CNS injury; however, trans
189 Similarly, sEH(-/-) mice showed impaired functional recovery following hindlimb ischemia, which w
194 t mammalian heart is incapable of meaningful functional recovery following substantial cardiomyocyte
196 es were labeled by the biosensor, suggesting functional recovery from apoptotic caspase activation.
202 efficacy of daidzein on neuroprotection and functional recovery in a clinically relevant mouse model
203 nd PPARgamma agonist administration improved functional recovery in a clinically relevant mouse strok
204 These connections support proprioceptive functional recovery in a conditioning lesion paradigm, a
205 y prostacyclin triggers axonal sprouting and functional recovery in a mouse model of inflammatory spi
206 lays their molecular maturation, and impedes functional recovery in a mouse model of spinal cord inju
207 ce cardiac regeneration and left ventricular functional recovery in a swine model of chronic ischemic
208 oproteinase inhibitor, on cardiac injury and functional recovery in a swine model of neonatal hypoxia
209 stered intravenously induced morphologic and functional recovery in AKI, the Drosha-knockdown counter
210 fter stroke plays a crucial role in limiting functional recovery in an experimental model of diabetes
212 uman heart failure, and restoration produces functional recovery in animal models and in failing huma
213 ministration is neuroprotective and promotes functional recovery in animal models of adult spinal cor
214 hile early mobilization is safe and enhances functional recovery in critically ill adults, rehabilita
217 istration promotes beta-cell replication and functional recovery in human islets following implantati
219 d the potential for molecular, cellular, and functional recovery in mice from the severe disruption o
220 ing the repair phase was reported to enhance functional recovery in mice suggesting that GABA plays a
221 cardiac energy production and post-ischemic functional recovery in neonatal rabbit hearts subjected
222 cline attenuates cardiac injury and improves functional recovery in newborn piglets with hypoxia-reox
223 injuries are often followed by considerable functional recovery in patients and animal models, large
224 s system (CNS) axons is a major obstacle for functional recovery in patients suffering neurological d
225 arginase significantly improved postischemic functional recovery in rat hearts if administered in who
226 ved precursors (SKP-SCs) promoted repair and functional recovery in rats with thoracic contusions.
227 and 18 months and to compare structural and functional recovery in regions that received MSC injecti
229 as a novel, translatable strategy to promote functional recovery in stroke patients without adverse a
235 to identify the pathways driving spontaneous functional recovery in wild-type and plasticity-sensitiz
236 fusion with carnosine promoted post-ischemic functional recovery in WT but not in AR-null mouse heart
237 motion more or less unchanged, but abolished functional recovery, indicating that dI3 interneurons ar
238 ted benefit of the BDNFVal66Met carriers for functional recovery, involving structural and molecular
239 on improves coordinated locomotion, and this functional recovery is accompanied by preservation of my
241 nds on acute infarct size, whereas long-term functional recovery is an important outcome in patients.
244 TBI care, outcomes-based metrics, including functional recovery, may be more accurate than current p
245 th the early compensatory mechanisms and the functional recovery mechanisms, with reduced aromatic L-
247 propriate rehabilitation, it brought about a functional recovery of abnormally wired neuronal network
248 rd below the level of injury and facilitated functional recovery of both locomotor and urinary system
253 of CD200L expression by CNS-resident cells, functional recovery of mice following SCI was impaired.
256 r development for neuroprotection as well as functional recovery of patients with multiple sclerosis.
258 ed whether VEGF could promote anatomical and functional recovery of peripheral nerves after injury us
261 kidney-pancreas transplant by evaluating the functional recovery of the graft and biochemical markers
263 ondrial uncoupling protein 3 (UCP3) improves functional recovery of the rodent heart during reperfusi
264 f contralesional hemispheric compensation to functional recovery of the upper extremity after a unila
265 This post-lesion treatment paradigm improved functional recovery on elevated plus maze and Morris wat
268 utcome, but there are little data on whether functional recovery post-stroke varies among hospitals.
269 dence interval, 1.32-1.46) and survival with functional recovery (range, 0.8%-21.0%; median odds rati
270 dependent or dead 3 months postacute stroke; functional recovery rates varied considerably among hosp
272 brain after stroke underlies the incomplete functional recovery seen in patients and that boosting h
273 ntly acceleratedin vivoaxon regeneration and functional recovery similar to GSK3alpha(S/A)/beta(S/A)
274 ted neuroinflammation after ICH and promotes functional recovery, suggesting that TGF-beta1 may be a
275 rticipants with delirium demonstrated lesser functional recovery than their counterparts without deli
276 c stroke contribute to long-term spontaneous functional recovery through inflammation-resolving activ
279 highest TCR affinity, correlating with full functional recovery upon PD-1 ligand 1 (PD-L1) blockade.
283 This effect was ER-alpha mediated, because functional recovery was blocked with an ER-alpha antagon
285 g the Confusion Assessment Method, and their functional recovery was followed for 18 months thereafte
288 n an attempt to identify agents that promote functional recovery, we discovered that an FDA-approved
289 processes are coordinated over the course of functional recovery, we tracked receptive field reorgani
290 peri-infarct area, infarct size, and animal functional recovery were assessed at 1, 2, and 3 weeks a
291 s and LV dysfunction, mortality rates and LV functional recovery were comparable between valve replac
292 s sprouting of intact CST axons and promotes functional recovery when applied soon after injury.
293 pathway that could be manipulated to improve functional recovery when combined with rehabilitation pr
294 gen-glucose deprivation, they show a reduced functional recovery when returned to oxygen-glucose but
295 mpared to young rats, adult rats had delayed functional recovery, whereas the aged rats were deficien
296 reshold of 2.8 W m(-2) s(-2)x10(5) predicted functional recovery with sensitivity and specificity of
298 mpairment were strongly associated with poor functional recovery within 6 months, whereas higher body
299 d starting 4 h postinjury, LM11A-31 promotes functional recovery without causing any toxicity or incr
300 at high risk for death or severely impaired functional recovery without offering patients and their
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