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1 ly little is known about the biology of this fungal pathogen.
2 crease in susceptibility to a root-infecting fungal pathogen.
3 timize host immune responses toward a deadly fungal pathogen.
4 CNS infection attributable to a bacterial or fungal pathogen.
5 t genome-scale co-functional network for any fungal pathogen.
6 may have been uniquely leveraged by a common fungal pathogen.
7 y interfere with their ability to clear this fungal pathogen.
8 ting direct contact between NK cells and the fungal pathogen.
9 he virulent form of this thermally dimorphic fungal pathogen.
10 lls in the host response to an opportunistic fungal pathogen.
11 ded shelf life and are less susceptible to a fungal pathogen.
12 ut the global molecular epidemiology of this fungal pathogen.
13 ization in a generalist but not a specialist fungal pathogen.
14 spensable for the virulence of a major human fungal pathogen.
15 al target in Candida albicans, a major human fungal pathogen.
16 ommensal bacteria, pathogenic bacteria and a fungal pathogen.
17 died, although it is key in the clearance of fungal pathogens.
18 y an important role in plant defense against fungal pathogens.
19 ility to contain and eliminate bacterial and fungal pathogens.
20 recognition and promotes the elimination of fungal pathogens.
21 strategies to reduce the negative impacts of fungal pathogens.
22 n plant growth promotion and control of crop fungal pathogens.
23 ulates the inflammatory cytokine response to fungal pathogens.
24 gning drugs that can specifically target the fungal pathogens.
25 defense against extracellular bacterial and fungal pathogens.
26 otential relevance for infections with other fungal pathogens.
27 first line of defense against bacterial and fungal pathogens.
28 pounds against a target found in three major fungal pathogens.
29 resistance to lethal challenge with multiple fungal pathogens.
30 them have been functionally characterized in fungal pathogens.
31 networks of basidiomycetous fungi and human fungal pathogens.
32 se against a broad spectrum of bacterial and fungal pathogens.
33 ly been associated with the host response to fungal pathogens.
34 ecific CD4 T cell responses against multiple fungal pathogens.
35 n as a live-cell imaging probe for different fungal pathogens.
36 ular filaments underpin virulence of diverse fungal pathogens.
37 essential in shaping the immune response to fungal pathogens.
38 ity of yeast forms of the dimorphic systemic fungal pathogens.
39 genesis, a key virulence trait of many human fungal pathogens.
40 ed (hemi)biotrophic filamentous oomycete and fungal pathogens.
41 plays an important role in immunity against fungal pathogens.
42 unctional polymorphism in immune response to fungal pathogens.
43 host defenses against bacterial, viral, and fungal pathogens.
44 in vitro activity against a wide variety of fungal pathogens.
45 pectin acetylation in plant defense against fungal pathogens.
46 ls, Gram positive and negative bacteria, and fungal pathogens.
47 cascade in regulating plant defense against fungal pathogens.
48 e investigated hemibiotrophic and biotrophic fungal pathogens.
49 applicable to other important intracellular fungal pathogens.
50 tifungal agent with activity against diverse fungal pathogens.
51 and susceptibility to numerous bacterial and fungal pathogens.
52 C. albicans and is readily extended to other fungal pathogens.
53 nomenon has not been extensively studied for fungal pathogens.
54 response that is necessary for clearing many fungal pathogens.
55 ns had lower relative abundances of putative fungal pathogens.
56 ogical cutoff values for clinically relevant fungal pathogens.
58 front-line defense of numerous bacterial and fungal pathogens against H2O2-induced oxidative damage f
59 play role in mammalian host defense against fungal pathogens, although the molecular details remain
60 Cryptococcus neoformans is an opportunistic fungal pathogen and a leading cause of fungal-infection-
61 IFI outbreaks are caused by many different fungal pathogens and are associated with numerous settin
63 tance - due to removal during gut passage of fungal pathogens and chemical attractants to granivores.
65 e of plants against bacterial, oomycete, and fungal pathogens and has a unique mode of action and str
66 ted for the rapid detection of low-abundance fungal pathogens and identification of the infecting pat
69 ted against bacterial polysaccharides engage fungal pathogens and promote their clearance in vivo and
70 ired to inhibit the growth of five different fungal pathogens and rootworm larvae (Diabrotica balteat
72 ed aphids challenged with a heat-inactivated fungal pathogen, and found that immune costs are limited
74 t resistance to different insect herbivores, fungal pathogens, and a parasitic plant, suggesting that
75 ionships with their fungal crop, specialized fungal pathogens, and bacteria that provide chemical def
76 elocation, occupation, or immunosuppression; fungal pathogens appearing in geographical areas in whic
77 a albicans, the most commonly isolated human fungal pathogen, are controlled by transcriptional and p
78 da species, the most commonly isolated human fungal pathogen, are frequently associated with biofilms
79 honuclear neutrophils (PMN) to bacterial and fungal pathogens as well as to model inflammatory stimul
83 f NK cells has been demonstrated against the fungal pathogens Aspergillus fumigatus and Cryptococcus
88 d- and warm-adapted amphibian species to the fungal pathogen Batrachochytrium dendrobatidis (Bd) usin
89 bian species suffering extirpations from the fungal pathogen Batrachochytrium dendrobatidis (Bd), we
90 ous factors, including pesticide use and the fungal pathogen Batrachochytrium dendrobatidis (Bd).
91 ence against pathogens, including the deadly fungal pathogen Batrachochytrium dendrobatidis (Bd).
92 , distribution and transmission of the model fungal pathogen Batrachochytrium dendrobatidis (Bd, caus
93 reen their animals for two amphibian chytrid fungal pathogens Batrachochytrium dendrobatidis (Bd) and
95 virulence trait shared by many bacterial and fungal pathogens, blocking microbial iron acquisition by
96 s required for defense responses against the fungal pathogen Botrytis cinerea, and thus we conclude t
98 F1.2) and for resistance to the necrotrophic fungal pathogens Botrytis cinerea and Alternaria brassic
100 anisms and the Cu homeostasis machinery that fungal pathogens bring into play to succeed in establish
101 us neoformans is a ubiquitous, opportunistic fungal pathogen but the cell signaling pathways that dri
102 major target of clinical drugs for managing fungal pathogens, but some of the CYP51 key features imp
105 etween primary human dendritic cells and the fungal pathogens C. albicans, C. parapsilosis and the en
106 s a common strategy used by successful human fungal pathogens, C. albicans provokes recognition by ho
108 dentified that protein effectors secreted by fungal pathogens can spread between host cells via PD.
110 activation of the Oma1 ortholog in the human fungal pathogen Candida albicans also alters TOR signali
111 n O- and N-linked mannan biosynthesis in the fungal pathogen Candida albicans and highlights new insi
114 oly-beta-peptides) that are toxic toward the fungal pathogen Candida albicans but exert little effect
118 Here we show that the hyphae of the human fungal pathogen Candida albicans continue to extend thro
119 hogenesis and responses to host cells in the fungal pathogen Candida albicans Eukaryotic Target of Ra
122 orphological transition of the opportunistic fungal pathogen Candida albicans from budding to hyphal
123 ompromised host, invasive infection with the fungal pathogen Candida albicans is associated with high
124 y a transcriptional regulator from the human fungal pathogen Candida albicans that binds DNA specific
125 Here we define circuitry that enables the fungal pathogen Candida albicans to couple cell cycle dy
126 pertoire that enable the human opportunistic fungal pathogen Candida albicans to proliferate in two d
127 ing N-acetylglucosamine (GlcNAc), induce the fungal pathogen Candida albicans to switch from budding
130 ucture of the kinase domain of Trl1 from the fungal pathogen Candida albicans with GDP and Mg2+ in th
132 py, and modeling analysis of the polymorphic fungal pathogen Candida albicans, which contains one of
143 nhibitory activity against the opportunistic fungal pathogens Candida albicans and Cryptococcus neofo
146 , we describe the recognition of an emerging fungal pathogen, Candida glabrata, by the human NK cytot
147 ubtelomeric regions in the most common human fungal pathogen: Candida albicans In this organism, the
148 Aspergillus terreus is an airborne human fungal pathogen causing life-threatening invasive asperg
149 ida auris is an emerging multidrug-resistant fungal pathogen causing nosocomial and invasive infectio
151 dida albicans is among the most common human fungal pathogens, causing a broad range of infections, i
152 lings after wounding or inoculation with the fungal pathogen Ceratocystis polonica, which is vectored
154 oth proteases are inhibited by Avr2 from the fungal pathogen Cladosporium fulvum, but only Rcr3 acts
155 anum lycopersicum) mediate resistance to the fungal pathogens Cladosporium fulvum and Verticillium da
156 istone deacetylase inhibitor produced by the fungal pathogen Cochliobolus carbonum race 1, promotes v
157 aria X ananassa) fruits interacting with the fungal pathogen Colletotrichum acutatum was actualized o
159 called core chromosomes, the genomes of many fungal pathogens comprise distinct unstable chromosomes
160 e of Candida albicans, the predominant human fungal pathogen, contains two paralogous TAF12 genes, Ca
164 mia, P5CDH is essential for virulence of the fungal pathogen Cryptococcus neoformans, and bacterial P
165 ens, including the major human opportunistic fungal pathogen Cryptococcus neoformans, remains unknown
171 t pulmonary infection with the opportunistic fungal pathogen Cryptococcus neoformans; however, the ro
172 mers that are highly active against a second fungal pathogen, Cryptococcus neoformans, and moderately
173 onal methods, current trends and advances in fungal pathogen detection with an emphasis on biosensors
178 ction with Cryptococcus neoformans, a common fungal pathogen, follows deposition of yeast spores in t
179 ntimicrobial-resistant bacterial, viral, and fungal pathogens for which diminishing treatment options
181 heat and barley, predominantly caused by the fungal pathogen Fusarium pseudograminearum, is a disease
182 s and examined their influence on two native fungal pathogens, Fusarium brachygibbosum U4 and Alterna
184 thermore, rpt1-1 exhibited resistance to the fungal pathogen Golovinomyces cichoracearum, but not to
191 e, while maintaining orthologs of most known fungal pathogen-host interaction proteins, stress respon
192 raxinus excelsior is being devastated by the fungal pathogen Hymenoscyphus fraxineus, which causes as
193 apoplastic effectors from a wheat-infecting fungal pathogen in a taxonomically distant nonhost plant
198 d with defense responses against insects and fungal pathogens in Pinus species, increasing current kn
201 endophytes, as well as pure Taxol, suppress fungal pathogens including wood-decaying fungi (WDF) [8-
203 ction is caused by respiratory bacterial and fungal pathogens, including Pseudomonas aeruginosa, Haem
204 using BODIPY-cPAF26 for wash-free imaging of fungal pathogens, including real-time visualization of A
205 ; however, its role in host immunity against fungal pathogens, including the major human opportunisti
211 determining the antifungal susceptibility of fungal pathogens is central to the clinical management o
214 bicans is also a predominantly opportunistic fungal pathogen, leading to disease manifestations such
215 is, free-living infectious stages typical of fungal pathogens lengthen the timespan of transmission.
218 atural enemies such as insect herbivores and fungal pathogens maintain high diversity by elevating mo
219 -peroxidase, MakatG1, in the locust-specific fungal pathogen, Metarhizium acridum, functions as a ROS
222 hila melanogaster, during infection with the fungal pathogen, Metarhizium robertsii, and the conseque
224 d plant gene confers partial resistance to a fungal pathogen not by preventing initial infection but
232 ity to Botrytis cinerea, a major postharvest fungal pathogen of tomato, is conferred by specific flav
235 hat just like in pen3, NHR to the nonadapted fungal pathogens Phakopsora pachyrhizi and Blumeria gram
236 stemic stress responses to the opportunistic fungal pathogen Pneumocystis in lymphocyte-deficient mic
242 Aspergillus fumigatus, a ubiquitous human fungal pathogen, produces asexual spores (conidia), whic
243 se caused by Emmonsia sp., a novel dimorphic fungal pathogen recently described in South Africa.
245 anscription factors, and human opportunistic fungal pathogens require SREBP activation for virulence.
246 ection with Verticillium dahliae (a vascular fungal pathogen responsible for devastating wilt disease
250 ntry rates by Colletotrichum higginsianum, a fungal pathogen showing direct penetration of leaf epide
252 suppression), the incidence of opportunistic fungal pathogens such as Candida albicans has increased.
254 ed by a large number of viral, bacterial, or fungal pathogens, such as respiratory tract infections,
256 (Bgh), is an obligate biotrophic ascomycete fungal pathogen that can grow and reproduce only on livi
258 Candida albicans is an opportunistic human fungal pathogen that causes a variety of diseases, rangi
259 Cryptococcus neoformans is an opportunistic fungal pathogen that causes approximately 625,000 deaths
260 Cryptococcus neoformans is an encapsulated fungal pathogen that causes cryptococcosis, which is a m
263 cillium dahliae is a destructive, soil-borne fungal pathogen that causes vascular wilt disease in man
266 us neoformans is a facultative intracellular fungal pathogen that has a polysaccharide capsule and ca
267 agent of gray mold disease, is an aggressive fungal pathogen that infects more than 200 plant species
268 Cryptococcus neoformans is an opportunistic fungal pathogen that initiates infection following inhal
269 Aspergillus fumigatus is an opportunistic fungal pathogen that invades pulmonary epithelial cells
270 Cryptococcus neoformans is an opportunistic fungal pathogen that is inhaled into the lungs and can l
271 Rhodotorula is an emerging opportunistic fungal pathogen that is rarely reported to cause endocar
272 agent of cryptococcosis, is an opportunistic fungal pathogen that kills over 200,000 individuals annu
274 Aspergillus fumigatus is the opportunistic fungal pathogen that predominantly affects the immunocom
275 agent of cryptococcosis, is an opportunistic fungal pathogen that primarily affects AIDS patients and
276 ergillus fumigatus is an opportunistic human fungal pathogen that sheds galactosaminogalactan (GG) in
278 ans and Candida tropicalis are opportunistic fungal pathogens that can transition between white and o
279 f unrelated bacterial, viral, protozoan, and fungal pathogens that cause skin lesions, as well as oth
281 vum) are clearly involved in defense against fungal pathogens, the function of border-like cells rema
284 -only SODs are virulence factors for certain fungal pathogens; thus this unique active site may be a
289 ent points of vulnerability as bacterial and fungal pathogens utilise this natural opening as an entr
290 henotypes on their hosts (protection against fungal pathogens vs. parasitoid wasps) and symbionts wit
291 5693 of Aspergillus fumigatus , an important fungal pathogen, was grown on three wheat grain substrat
293 creased susceptibility to both bacterial and fungal pathogens, whereas plants with reduced TOR signal
295 Cryptococcus neoformans (Cn) is a deadly fungal pathogen whose intracellular lifestyle is importa
298 pathogen induced VPE enhancing resistance to fungal pathogens with cell death phenomenon under transi
300 Candida albicans is the most common human fungal pathogen, yet is a normal commensal resident of t
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