ライフサイエンスコーパス: fusidic acid

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1 cter oralis susceptible to metronidazole and fusidic acid.

2 sttranslocational state using the antibiotic fusidic acid.

3 usB bound staphylococcal EF-G, the target of fusidic acid.

4 agents, such as bile acids, novobiocin, and fusidic acid.

5 osome complex in a manner similar to that of fusidic acid.

6 erhydrophenanthrene, an ABC ring analogue of fusidic acid 1.

7 The activity of fusidic acid against 778 isolates of Staphylococcus aure

8 e co-crystal structure of HSA complexed with fusidic acid, an antibiotic that competitively displaces

9 In contrast, fusidic acid and a GTP analog that fix EF-G to the ribos

10 stance will likely limit the clinical use of fusidic acid and bacitracin and, possibly, rifaximin if

11 tetracycline, chloramphenicol, erythromycin, fusidic acid and spectinomycin, do not induce the confor

12 -dependent release occurs in the presence of fusidic acid and viomycin.

13 -hydrolyzable GTP analogue GDPNP or GTP plus fusidic acid causes an increase in the efficiency of ene

14 Fusidic acid (CEM-102) is an established antistaphylococ

15 ility testing reagents (including Etest) for fusidic acid (CEM-102) performed at an excellent level o

16 have visualized EF-G in a ribosome-EF-G-GDP-fusidic acid complex.

17 dic acid, suggesting that EF-G stabilized by fusidic acid does not represent the natural post-termina

18 DB, and the proposed mechanism of action of fusidic acid, evidence is presented in support of the Gr

19 Fusidic acid (FA) has been used for decades for bone inf

20 Resistance to the antibiotic fusidic acid (FA) in the human pathogen Staphylococcus a

21 omes and to elucidate the mechanism by which fusidic acid (FA) inhibits multiple-turnover EF-G.GTPase

22 of cholesterol and steroid-based antibiotic fusidic acid (FA) on the behavior of lipid bilayers usin

23 We observed a dramatic increase in fusidic acid (FA) resistance, and clonal expansion of FA

24 The antibiotic fusidic acid (FA) targets elongation factor G (EF-G) and

25 Fusidic acid (FA), an antibiotic used against pathogenic

26 coccal translation system from inhibition by fusidic acid in a specific and dose-dependent fashion.

27 d pUB101 capable of conferring resistance to fusidic acid in S. aureus.

28 The antibiotic fusidic acid increases the population of the 0.6 state,

29 nt splitting, while the inhibitory effect of fusidic acid on the splitting was nominal.

30 The primary mechanism of fusidic acid resistance in clinical strains of Staphyloc

31 cture of the complex formed between the FusB fusidic acid resistance protein and the drug target (EF-

32 shedding light on the molecular mechanism of fusidic acid resistance.

33 tion assays and the frequency of mutation to fusidic acid resistance.

34 erved increases in the number of spontaneous fusidic acid-resistant mutants of five- and sevenfold in

35 ma-imidoguanosine 5'-triphosphate (GDPNP) or fusidic acid reveal conformational changes occurring dur

36 xacin, vancomycin, trimethoprim, gentamicin, fusidic acid, rifampin, and mupirocin) performed by the

37 sults were compared with those obtained with fusidic acid, showing that both drugs act in a different

38 did not bind to EF-G-ribosome complexes with fusidic acid, suggesting that EF-G stabilized by fusidic

39 Fusidic acid traps EF-G in a conformation intermediate b

40 but significantly (p < 0.001); resistance to fusidic acid was stable (p > 0.05); resistance to tetrac

41 in provided no protection from inhibition by fusidic acid when added to an in vitro E. coli translati

42 On the other hand, sordarin or fusidic acid, when applied together with eEF2/GTP, speci

43 wo proteins differ in their sensitivities to fusidic acid, with the latter activity inhibited by the

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