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1 cell, the large ganglion cell, and the large fusiform cell.
2 h can be readily assessed in recordings from fusiform cells.
3 trauma-induced elevation of activity of DCN fusiform cells.
4 gic axon terminals increased excitability of fusiform cells.
5 cells, which give rise to the pax6-negative fusiform cells.
6 ptic potentials (PSPs) in both cartwheel and fusiform cells.
7 n dorsal cochlear nucleus principal neurons, fusiform cells.
8 s have shown that in normal hearing animals, fusiform cell activity can be modulated by activation of
9 synapses on bushy cells (AN-BC synapses) and fusiform cells (AN-FC synapses) and PF synapses on FC (P
10 nucleus, labeled boutons were present in the fusiform cell and deep layers and appeared to contact fu
11 ed waveforms were found predominantly in the fusiform cell and deep layers, with smaller numbers foun
12 mably reflecting direct excitatory inputs to fusiform cells and an indirect inhibitory input to fusif
14 spontaneous firing of both regular (probably fusiform cells) and bursting neurons (probably cartwheel
15 we show that excitatory projection neurons (fusiform cells) and inhibitory stellate interneurons of
16 and inhibitory input on projection neurons (fusiform cells), and inhibitory interneurons (vertical a
17 ured cells, which acquired the appearance of fusiform cells, and it inhibited H(+)K(+)-adenosine trip
19 neuron, whereas the large ganglion and large fusiform cells are efferent neurons that convey electros
21 eveal that 5-HT exerts a potent influence on fusiform cells by altering their intrinsic properties, w
23 Dolichos biflorus lectin confirmed that the fusiform cells expressed markers of parietal cell differ
25 rm cells and an indirect inhibitory input to fusiform cells from the granule cell-cartwheel cell syst
27 rm cells, thick-smooth dendrite cells, small fusiform cells, granule cells, and primary afferent fibe
29 that also receive parallel fiber input, the fusiform cells, had intermediate properties with respect
31 , a reduction in KCNQ2/3 channel activity in fusiform cells in noise-exposed mice by 4 days after exp
32 stic cues are brought to smooth dendrites of fusiform cells in the deep layer by auditory nerve fiber
36 Longitudinal BrdU labeling suggests that the fusiform cells migrate to the ONL to replenish the pool
37 hese cell types are bushy cells, bipolar (or fusiform) cells, octopus cells, stellate cells, giant ce
44 taneous firing (hyperactivity) is induced in fusiform cells of the dorsal cochlear nucleus (DCN) foll
45 several higher auditory stations but not in fusiform cells of the dorsal cochlear nucleus (DCN), key
49 ir excitatory input from the auditory nerve; fusiform cells receive excitatory inputs from both the a
50 Optogenetically activated populations of fusiform cells reliably enhanced interneuron excitabilit
52 mples and were characterized by an elongated fusiform cell shape, abundant cytoplasmic rough endoplas
54 ell types revealed that eight distributed on fusiform cells, six on vertical cells and five on cartwh
55 ereas deep afterhyperpolarizations following fusiform cell spike trains potently inhibited stellate c
57 onstrate increased synchrony and bursting of fusiform cell spontaneous firing, which correlate with f
58 e of increased synchrony and bursting in DCN fusiform cells suggests that a neural code for phantom s
62 nitus, we recorded spontaneous activity from fusiform cells, the principle neurons of the DCN, in nor
63 ganglion cells, large ganglion cells, large fusiform cells, thick-smooth dendrite cells, small fusif
64 o suppress tinnitus-related hyperactivity of fusiform cells using the cholinergic agonist, carbachol.
67 haped waveforms probably are associated with fusiform cells, whereas M-shaped spikes likely originate
68 ree distinct cell types were identified: (1) fusiform cells with small, spindle-shaped somata and bip
69 postulated that response properties of aged fusiform cells would show a loss of inhibition, resembli
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