ライフサイエンスコーパス: gait

1 ance Error Scoring System [mBESS] and tandem gait).

2 ents with and 8 patients without freezing of gait.

3 n's disease and primary progressive freezing gait.

4 ntly improved recovery of motor function and gait.

5 pinal and sensory afferent signals adjusting gait.

6 ntral and dorsal striatum caused freezing-of-gait.

7 EMG patterns and hindlimb kinematics during gait.

8 sitive myoclonus, and difficulty with tandem gait.

9 f walking showed step-to-step variability of gait.

10 ements motivated by our previous findings in gait.

11 generators define interlimb coordination and gait.

12 ng able to walk on land with a tetrapod-like gait.

13 greater response to GPi DBS with respect to gait.

14 roblem for the swimming speed and locomotion gait.

15 kable given the apparent optimality of human gait.

16 d a wide-based, spastic, and/or stiff-legged gait.

17 of the brain regions involved in freezing of gait.

18 with increases of voluntary control of human gait.

19 on underpins variable locomotor movements or gaits.

20 nections ensure fore-hind alternation in all gaits.

21 r role of passive-elastic mechanics in these gaits.

22 kg in grip strength, -0.05 meters/second in gait, 0.03 seconds in chair stands, and -0.16 Short Phys

23 ), lower gait speed (1.15 per 0.1-m/s slower gait [1.06-1.24]), lower gray matter volume (0.72 per 1-

24 als with cerebral palsy often exhibit crouch gait, a debilitating and inefficient walking pattern mar

25 Evaluation of gait ability and intramuscular coherence was made twice

26 tion found in patients displayed progressive gait abnormalities and vision loss, in addition to bioch

27 neurodegenerative disorder characterized by gait abnormalities, ataxia, dysarthria, dystonia, vertic

28 lectual disability with or without seizures, gait abnormalities, problems of social behavior, and oth

29 isability (ID), severe speech impairment and gait abnormalities.

30 sfunctions such as weakness of hind-limb and gait abnormality in an age-dependent manner.

31 on and obstacle avoidance requires effective gait adaptation in response to sensory cues.

32 Understanding brain dynamics supporting gait adaptation is crucial for understanding motor defic

33 ory cortical networks that are active during gait adaptation to shifts in the tempo of an auditory pa

34 s of beta band oscillations interplay during gait adaptation, however, has not been established.

35 s of beta band activity modulation accompany gait adaptations: one likely serving movement initiation

36 were either blind in one eye, dependent on a gait aid or both.

37 comorbidity, including increased reliance on gait aids (adjusted odds ratio, 1.9; 95% CI, 1.4-2.6); n

38 ing prosthesis mass, and allowing asymmetric gaits all decrease human metabolic rate for a given spee

39 andering that paralleled previously reported gait alterations in Chn1KO/KO and Epha4KO/KO adult mice.

40 CatWalk gait analysis also supported that antinociceptive effect

41 Clinical gait analysis incorporating three-dimensional motion ana

42 is shape is known to differ between sex, and gait analysis is performed in populations with wide rang

43 ion Technology, Wageningen, The Netherlands) gait analysis was significantly impaired in propofol-sed

44 The results of knee gait analysis were comparable with normal population dat

45 Through kinematic gait analysis, we found a relationship between K/L score

46 ch helps in maintaining motor tone, posture, gait and also coordinates skilled voluntary movements in

47 of RTT-associated behavioural, sensorimotor, gait and autonomic (respiratory and cardiac) phenotypes

48 a brief cognitive examination (Mini-Cog) and gait and balance assessment (Timed Up and Go test).

49 ergone comprehensive risk factor assessment, gait and balance assessment as well as brain MRI.

50 Gait and balance disorders are major problems that contr

51 sease (SVD) or a combination thereof explain gait and balance function in the elderly.

52 Gait and balance impairment is highly prevalent in older

53 The Gait and Brain Study is an ongoing prospective cohort st

54 Gait and cognition have been related to mortality in pop

55 ying cholinergic deficit that contributes to gait and cognitive dysfunction in these patients.

56 ut the origins of humans' economical walking gait and endurance running capabilities remain unclear.

57 nder-like diagonal-couplets lateral sequence gait and has evolved a robust pelvic girdle that shares

58 n infarcts were associated with slower timed gait and lower volume of supratentorial white matter.

59 ntexts.Biomechanical understanding of animal gait and maneuverability has primarily been limited to s

60 cific lineage with functional restoration of gait and mechanical properties.

61 otor impairment, resting tremor and abnormal gait and posture, phenotypes reminiscent of Parkinson's

62 le exoskeletons for the management of crouch gait and provide insights into their future implementati

63 erintensity was associated with slower timed gait and worse performance on DSST, and lower volumes of

64 e, patients exhibit resting tremor, unstable gait, and impaired balance, which may be associated with

65 layed impaired motor coordination, disturbed gait, and profound cognitive disability.

66 icated in the pathophysiology of freezing of gait, and suppressing them may form a key strategy in de

67 INTERPRETATION: Lesions causing freezing of gait are located within a common functional network char

68 brain MRI and simple tests of cognition and gait as part of a substudy (PURE-MIND).

69 visits including neurologic, cognitive, and gait assessments.

70 n; however, the fundamental features of this gait ataxia have not been effectively isolated.

71 Gait ataxia was a common feature.

72 man presented for evaluation of progressive gait ataxia with a superimposed spastic paraparesis.

73 by neurogenic muscular atrophy, progressive gait ataxia with tremor, cerebellar vermis atrophy, and

74 Clinical symptoms of gait ataxia, diplopia, cognitive impairment, and facial

75 The second presented at 3.5 years with gait ataxia, dysarthria, gross motor regression, hypoton

76 r the presentation of daytime somnolence and gait ataxia.

77 A rehabilitation nursing programme included gait, balance, functional training, strengthening, flexi

78 ht alternation of neural activity, switching gaits between the left-right alternating walking-like ac

79 Footprints preserve direct records of gait biomechanics and behavior but they have been rare i

80 In patients without freezing of gait, both bicycling and walking led to a suppression of

81 e knee extensor moment present during crouch gait by a mean of 35% in early stance and 76% in late st

82 (0 = impaired to 12 = robust), consisting of gait, chair stands, and balance tests.

83 oscillations accompanied by speed-dependent gait changes from walk to trot and to gallop and bound.

84 imed chair stand, physical function battery, gait characteristics (speed; step number, rate, and leng

85 tory RORbeta IN function result in an ataxic gait characterized by exaggerated flexion movements and

86 Gait, cognition, and imaging markers of SVD are associat

87 was specific to lesions causing freezing of gait compared to lesions causing other movement disorder

88 have substantially different affinities for GAIT complex binding.

89 s for their interaction with the RNA-binding GAIT complex.

90 for electrophysiological studies of cortical gait control and its disorders.

91 Dual-task gait cost was defined as the percentage change between s

92 g by baseline cognition except for dual-task gait cost when dichotomized.

93 CI, 0.99-11.71; P = .05)while high dual-task gait cost while counting backward (HR, 3.79; 95% CI, 1.5

94 e- and dual-task gait velocity and dual-task gait costs were the independent variables.

95 We analyze entire gait curves by means of a new functional F test with com

96 k and synchronizing target visibility to the gait cycle, we empirically validated two predictions der

97 ves of the muscles of the leg throughout the gait cycle.

98 o test hypotheses for specific phases of the gait cycle.

99 We apply this to gait data analysis using repeated-measurements data from

100 insufficient information from 3-dimensional gait data, rendering clinical interpretation of impaired

101 lish a brain-spine interface that alleviated gait deficits after a spinal cord injury in non-human pr

102 Quadrupeds express different gaits depending on speed of locomotion.

103 As a result, the expression of each gait depends on (1) left-right interactions within the s

104 n is suggested as a potential determinant of gait deterioration, but this has not been explored in a

105 gressive decline in his daily routine due to gait difficulties in the past year.

106 type and tremor and postural instability and gait difficulty (PIGD) scores.

107 remor dominant (TD) and postural instability/gait difficulty (PIGD) subtype patients during a motor t

108 tment response, and postural instability and gait difficulty motor PD subtype in linear regression an

109 planning surgical treatments in people with gait disability.

110 ive neurostimulation in stroke patients with gait disability.

111 al disorder and the postural instability and gait disorder phenotype.

112 o be a main player in the pathophysiology of gait disorders seen in Parkinson's disease.

113 ar, the pedunculopontine nucleus, to address gait disorders that respond poorly to levodopa and conve

114 pid and severe disease progression; onset of gait disorders was 3 times more rapid (p < 0.0001) and t

115 ric disorders (six events in four patients), gait disturbance (one event), elevated liver aminotransf

116 categorised as psychiatric disorders and as gait disturbance were assessed as unlikely to be related

117 translate into the split hand presentation, gait disturbance, split leg syndrome and bulbar symptoma

118 ers with the hallmark of progressive spastic gait disturbance.

119 lvement in addition to cognitive changes and gait disturbances from meningoencephalitis.

120 om severity, language impairments, seizures, gait disturbances, intelligence quotient (IQ) and adapti

121 cent assertions that the six determinants of gait do not serve to improve walking economy.

122 Z-score), while secondary outcomes included gait domains (Rhythm, Variability, Phases, Pace, Base of

123 s motor performance (open-field behavior and gait dynamics), corrects repetitive clasping behavior, a

124 n association with changes to body shape and gait dynamics.

125 Gait dysfunction can lead to an increased variability of

126 odeling and experimental studies of bouncing gait (e.g., walking, running, hopping) identified muscle

127 rses with the ability to perform comfortable gaits (e.g. ambling or pacing), so-called 'gaited' horse

128 erent positions within the overall conserved GAIT element structure contribute to differential affini

129 Thus, heterogeneity of GAIT elements may provide hierarchical fine-tuning of th

130 terferon-activated inhibitor of translation (GAIT)-elements relies on the conserved RNA folding motif

131 et dates back 6-7 Ma, reconstructing hominin gait evolution is complicated by a sparse fossil record

132 The model reproduces speed-dependent gait expression in intact and genetically transformed mi

133 it transitions in intact mice and changes in gait expression in mutants lacking certain types of comm

134 uggests explanations for the speed-dependent gait expression observed in vivo in intact mice and in m

135 d anxiety disorders), hypotonia, broad-based gait, facial dysmorphisms, and periods of fever and vomi

136 These results imply that the motor's gait follows a rotary hand-over-hand mechanism.

137 ationship between K/L score and 3D kinematic gait for patients.

138 D predicts a non-tremor-predominant subtype, gait freezing, and an aggressive clinical course.

139 duals (5-19 years) with mild-moderate crouch gait from cerebral palsy (GMFCS I-II) completed the stud

140 tion can lead to an increased variability of gait from one step to another, raising the likelihood of

141 r speed accompanied by sequential changes of gaits from walk to trot and to gallop and bound.

142 and differences between quadruped and biped gait have made this comparison challenging.

143 = 0.89, 95% confidence interval 0.83-0.95), gait (hazard ratio = 0.72, 95% confidence interval 0.62-

144 rldwide with an especially high frequency in gaited horses and breeds used for harness racing [2].

145 e gaits (e.g. ambling or pacing), so-called 'gaited' horses, have been highly valued by humans, espec

146 n stride length between normal and dual-task gait (ie, dual-task interference) was more pronounced in

147 right-handed man presented with progressive gait imbalance over 6 years.

148 requency stimulation of the MLR (MLR-HFS) on gait impairment in a rodent stroke model.

149 ems to be the major driving force for SVD on gait impairment in healthy elderly subjects.

150 shopping, and meals), history of falling or gait impairment, and depressive symptoms (2-item Patient

151 aspects of motor behavior in vivo, including gait impairments.

152 Measures of spontaneous movement and gait in an open-field test declined as expected in contr

153 the FBXO41 gene results in a severely ataxic gait in mice, which show delayed neuronal migration of g

154 can affect interlimb coordination and change gait independent of speed.

155 tions, inclusion of EMG signals and temporal gait information reduced classification error across amb

156 These episodes typically occur during gait initiation or turning.

157 onance was correlated to the behavior during gait initiation outside the scanner.

158 dex both normal and abnormal preparation for gait initiation within an fMRI experiment.

159 activation of hindlimb motor cortex preceded gait initiation.

160 After 1 month, the patient developed gait instability and cognitive decline rapidly evolving

161 ning produced acute effects that ameliorated gait instability in PD.

162 asibility of neural decoding for both users' gait intent and continuous kinematics.

163 Freezing of gait is a disabling symptom in Parkinson disease and rel

164 Freezing of gait is a poorly understood symptom of Parkinson disease

165 Gait is an important health indicator and poor gait is s

166 Dual-task gait is associated with progression to dementia in patie

167 it is an important health indicator and poor gait is strongly associated with disability and risk of

168 terminants of gait proposed that the goal of gait is to minimize vertical displacement of the body's

169 decreased body size, grip weakness, abnormal gait, joint laxity, and early-onset osteoarthritis.

170 Gait kinematics were recorded by 3D video analysis durin

171 Subject's gait was recorded in a gait laboratory, walking normally, with 5 degrees and 10

172 , 0.53-1.29]; effect size, 0.68), and tandem gait (mean [SD] time, 22.2 [8.3] vs 14.0 [3.7] seconds;

173 d experimental results confirm that multiple gaits, mimicking either symplectic or antiplectic metach

174 respect to the body is used to initiate the gait modification.

175 resent a novel method to study the effect of gait modifications and lateral wedge insoles (LWIs) on t

176 the EMG activity was equally constant during gait modifications and reaching.

177 to the control of visually guided locomotor gait modifications by constructing an estimation of obje

178 e locomotion in the neonatal mouse or change gait, motor coordination, or grip strength in adult mice

179 ic locomotor region (MLR) is known to elicit gait movements, this area might be a promising target fo

180 a-SNAP hypomorph, hydrocephalus with hopping gait, Napa(hyh/hyh) mice harbor significant defects in C

181 ing (12 months) with a multi-stage BMI-based gait neurorehabilitation paradigm aimed at restoring loc

182 en-Lawrence (K/L) score and the 3D kinematic gait of patients with medial KOA remains unclear.

183 apparatus that are coincident with specific gaits of flagellar actuation, suggesting that it is a co

184 ssociation of FT4 concentrations with Global gait (p = 0.2).

185 We found that when the value of the gait parameter was conspicuously greater (smaller) than

186 However, the velocity dependence of gait parameters and differences between quadruped and bi

187 tified the symmetry of short-term control of gait parameters and observed the significant dominance o

188 In mice, we represented spatial and temporal gait parameters as a function of velocity and establishe

189 Gait parameters in healthy human subjects followed simil

190 We determined the gait parameters of 20 healthy subjects with right-foot p

191 ficant changes in several dynamic and static gait parameters resulting in overall reduced gait veloci

192 determines average values of spatio-temporal gait parameters such as step duration, step length or st

193 ng speed and improvements in several dynamic gait parameters were detected by the CatWalk system.

194 freezing and worsened gait, whereas specific gait parameters were mildly improved by stimulation of p

195 ll walking that drives people to learn a new gait pattern using sensory prediction errors detected by

196 these neural circuits determines the speed, gait pattern, and direction of movement, so the specific

197 ted the association of thyroid function with gait patterns in 2645 participants from the Rotterdam St

198 d self-report measures of physical function, gait patterns, and falls between women cancer survivors

199 r thyroid function was associated with worse gait patterns.

200 and walking economy without imposing altered gait patterns.

201 isease and its consequences on cognitive and gait performance and brain atrophy are manifest in some

202 not carry load, their perceived exertion and gait performance did not significantly change with contr

203 Gait performance is affected by neurodegeneration in agi

204 p at 4 weeks: progressive motor neuron loss, gait problems, muscle denervation and atrophy, paralysis

205 The six determinants of gait proposed that the goal of gait is to minimize verti

206 contribute to differential affinities of the GAIT protein complex towards the elements.

207 cognitive function and objective measures of gait, quantification of HTLV-1 proviral load in peripher

208 dantrolene treatment significantly improved gait, reduced LC3-II levels, improved mitochondrial ATP

209 e motor cortex during motor control studies, gait rehabilitation, and locomotor neuroprosthetic devel

210 ng the neural responses that are aberrant in gait-related neurological disorders (e.g. cerebral palsy

211 agnostic confirmation on variable cutoffs of gait response to bedside fluid-drainage testing.

212 the speed-dependent expression of different gaits results from speed-dependent changes in the intera

213 A method for capturing gait signatures in neurological conditions that allows c

214 hifted from baseline, implicating changes in gait signatures, but with marked differences between mod

215 volume, and small-vessel disease but not on gait speed (0.85 vs 0.92 m/s, P = .01) or proportion of

216 1.05 per 1-year increase [1.01-1.08]), lower gait speed (1.15 per 0.1-m/s slower gait [1.06-1.24]), l

217 monary function had significantly slower 4-m gait speed (all but one p < 0.05).

218 re significantly associated with the 4-meter gait speed (rs928874, p = 5.61 x 10(-8); rs1788355, p =

219 sociation between continuous and categorical gait speed and 30-day all-cause mortality before and aft

220 the global and regional associations between gait speed and Abeta in the whole sample and the CN subs

221 Several regional associations between gait speed and PiB uptake withstood relevant adjustments

222 Gait speed can be used to refine estimates of operative

223 Gait speed changes were consistent with reported changes

224 Each 0.2-m/s decrease in gait speed corresponded to an 11% increase in 30-day mor

225 of SVD-related morphologic brain changes on gait speed in addition to age, sex and hypertension inde

226 To examine the association between Abeta and gait speed in elderly individuals without dementia and t

227 ther amyloid-beta (Abeta) is associated with gait speed in elderly individuals without dementia and w

228 influence the association between Abeta and gait speed in elderly individuals without dementia.

229 l Abeta deposition is associated with slower gait speed in elderly individuals without dementia; howe

230 al significant and independent predictors of gait speed in the regression model.

231 The 4-m gait speed is a reliable, valid, and responsive measure

232 Gait speed is an independent predictor of adverse outcom

233 Gait speed is independently associated with 30-day morta

234 The estimated 4-m gait speed minimal important difference was 0.03-0.06 m/

235 mpound B (PiB) positron emission tomography, gait speed over 4.57 m (15 ft), and cognition on the Min

236 isk of mortality and the surgical procedure, gait speed remained independently predictive of operativ

237 ong the physical domains of pre-frailty, low gait speed seems to be the best predictor of future CVD.

238 Furthermore, 4-m gait speed significantly predicted future hospitalizatio

239 Compared with patients in the fastest gait speed tertile (>1.00 m/s), operative mortality was

240 ions for clinical studies evaluating the 4-m gait speed test in acute respiratory distress syndrome s

241 The 5-m gait speed test was performed in 15171 patients aged 60

242 Addition of gait speed to the Society of Thoracic Surgeons predicted

243 The overall median gait speed was 0.63 m/s (25th-75th percentile, 0.47-0.79

244 Gait speed was also predictive of the composite outcome

245 Four-meter gait speed was assessed at 6- and 12-month follow-up (AR

246 We assessed the association of 5-m gait speed with outcomes in a cohort of 8039 patients wh

247 ores do not include frailty assessments (eg, gait speed), which are of particular importance for pati

248 es, pacemaker use, atrial fibrillation, slow gait speed, and nonfemoral access were significantly ass

249 tcomes (four-step stair climbing time, usual gait speed, and time to rise five times from a chair wit

250 Gait speed, cognitive index, conventional MRI markers of

251 d including age, sex, vascular risk factors, gait speed, cognitive index, MRI, and diffusion measures

252 res (>80th percentile) were more impaired by gait speed, difficulty with Instrumental Activities of D

253 climbing test, 6-min walking distance, fast gait speed, hand grip strength, and isometric leg extens

254 prediction of mortality, an older age, lower gait speed, lower gray matter volume, and greater global

255 ntegrating a measure of frailty, such as 5-m gait speed, to better capture the heterogeneity of the o

256 ex, and hypertension significantly explained gait speed.

257 strumental activity of daily living, or slow gait speed.

258 morbidity (OR, 1.03 per 0.1-m/s decrease in gait speed; 95% CI, 1.00-1.05).

259 mortality (OR, 1.11 per 0.1-m/s decrease in gait speed; 95% CI, 1.07-1.16).

260 le lowlanders walked on a treadmill at seven gait speeds (0.67-1.83 m s(-1)) on a level gradient unde

261 ification of frail patients with the slowest gait speeds facilitates preprocedural evaluation and ant

262 Rivastigmine can improve gait stability and might reduce the frequency of falls.

263 produce short-term aftereffects of increased gait stability once the cables were removed.

264 ffects to improve compensatory reactions and gait stability.

265 The primary outcome was Global gait (standardized Z-score), while secondary outcomes in

266 on are associated with alterations in Global gait, Tandem, Base of support and velocity.

267 A dual-task gait test evaluating the cognitive-motor interface may p

268 To determine whether a dual-task gait test is associated with incident dementia in MCI.

269 Dual-task gait testing is easy to administer and may be used by cl

270 thyroidism were associated with worse Global gait than euthyroidism (beta = -0.61; CI = -1.03, -0.18;

271 rain lesions causing acute onset freezing of gait to identify regions causally involved in symptom ge

272 rain, humans must use vision to tailor their gait to the upcoming ground surface without interfering

273 PN connections contribute to speed-dependent gait transition from walk, to trot, and then to gallop a

274 peds, including mice, demonstrate sequential gait transitions from walk to trot and to gallop and bou

275 experimental data, including speed-dependent gait transitions in intact mice and changes in gait expr

276 r economy, bipedal jerboa (family Dipodidae) gait transitions likely enhance maneuverability.

277 Unlike the speed-regulated gait use of cursorial animals to enhance locomotor econo

278 esterase inhibitor rivastigmine would reduce gait variability.

279 centage change between single- and dual-task gait velocities: ([single-task gait velocity - dual-task

280 Slow single-task gait velocity (<0.8 m/second) was not associated with pr

281 executive functioning (r = 0.54, P = 0.001), gait velocity (r = 0.41, P = 0.02), but not memory.

282 and dual-task gait velocities: ([single-task gait velocity - dual-task gait velocity]/ single-task ga

283 n outcome measure, and single- and dual-task gait velocity and dual-task gait costs were the independ

284 Gait velocity was recorded under single-task and 3 separ

285 city - dual-task gait velocity]/ single-task gait velocity) x 100.

286 gait parameters resulting in overall reduced gait velocity.

287 ies: ([single-task gait velocity - dual-task gait velocity]/ single-task gait velocity) x 100.

288 The diagonal-couplet lateral sequence gait was accomplished by rotation of the pectoral and pe

289 Gait was assessed by electronic walkway.

290 Gait was assessed using clinical scoring of the beam-wal

291 Subject's gait was recorded in a gait laboratory, walking normally

292 Gait was slower among CIPN+, with those women taking sig

293 Fourteen cases of lesion-induced freezing of gait were identified from the literature, and lesions we

294 ion Test (DSST), and Timed Up and Go test of gait were seen with each age decade from the 40s to the

295 sed severe episodes of freezing and worsened gait, whereas specific gait parameters were mildly impro

296 ncluded subjects deliberately changing their gait, which could bias the endpoint measures.

297 l conditions that allows comparison of human gait with animal models would be of great value in trans

298 she experienced a more rapid decline in her gait with parkinsonism, visual difficulties with restric

299 imulating hormone), FT4 (free thyroxine) and gait, without known thyroid disease or dementia.

300 tion between global PiB retention and slower gait withstood adjustment for covariates (beta = -0.068,