ライフサイエンスコーパス: gait ataxia

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1 r the presentation of daytime somnolence and gait ataxia.

2 ent experienced exacerbation of pre-existing gait ataxia.

3 racterized by failure to thrive, tremor, and gait ataxia.

4 nding that is consistent with their profound gait ataxia.

5 les are lost, mutant mice exhibit increasing gait ataxia accompanied by spike broadening and decelera

6 TAS include progressive intention tremor and gait ataxia, accompanied by characteristic white matter

7 odegenerative diseases involving progressive gait ataxia and cerebellar atrophy.

8 matic neurological phenotype presenting as a gait ataxia and culminating in premature death.

9 Gait ataxia and lower limb dysaesthesiae were the most f

10 ination was abnormal in three, with limb and gait ataxia and mild spastic paraparesis.

11 All had gait ataxia and most had limb ataxia.

12 ing presented 4 weeks prior with dysarthria, gait ataxia, and bilateral upper extremity weakness.

13 characterized by progressive action tremor, gait ataxia, and cognitive decline.

14 ut also more broadly for adults with tremor, gait ataxia, and parkinsonism who are seen in movement-d

15 xhibit significant motor impairments such as gait ataxia, associated with multiple neuropathological

16 motor problems such as intention tremor and gait ataxia, cognitive decline and psychiatric problems

17 Clinical symptoms of gait ataxia, diplopia, cognitive impairment, and facial

18 taxia that leads to moderate disability with gait ataxia, dysarthria, dysmetria, mild oculomotor abno

19 The second presented at 3.5 years with gait ataxia, dysarthria, gross motor regression, hypoton

20 ice lacking just Kcnc3 is hypermetria, while gait ataxia emerges when additionally Kcnc1 alleles are

21 Those with more severe gait ataxia had longer disease duration.

22 n; however, the fundamental features of this gait ataxia have not been effectively isolated.

23 evident in 75%, lower limb ataxia in 90% and gait ataxia in 100% of patients.

24 combination of reported intention tremor and gait ataxia in male carriers (17%, 38%, 47%, and 75% [lo

25 gr3 were generated by gene targeting and had gait ataxia, increased frequency of perinatal mortality,

26 Friedreich's ataxia and FXTAS carriers (e.g. gait ataxia, loss of coordination) are consistent with a

27 Egr3-/- mice develop gait ataxia, scoliosis, resting tremors, and ptosis, sug

28 s, we asked whether patients with cerebellar gait ataxia showed abnormal responses of otolithic vesti

29 ative disorders characterized by progressive gait ataxia, upper limb incoordination, and dysarthria.

30 Gait ataxia was a common feature.

31 4; P =.003) of combined intention tremor and gait ataxia when compared with male controls.

32 man presented for evaluation of progressive gait ataxia with a superimposed spastic paraparesis.

33 by neurogenic muscular atrophy, progressive gait ataxia with tremor, cerebellar vermis atrophy, and

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