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1 approximately 65 kDa that is neither CD4 nor galactocerebroside.
2 for the degradation of the myelin glycolipid galactocerebroside.
3 catalyzes the final step in the synthesis of galactocerebroside, a glycosphingolipid characteristical
4  rate constants shows that O1 and O4 bind to galactocerebroside and sulfated galactocerebroside, resp
5                 The myelin-associated lipids galactocerebroside and sulfatide were modestly reduced i
6                                              Galactocerebroside and sulfatide, major galactosphingoli
7 in which galactosphingolipids (in particular galactocerebroside and/or sulfatide) act as sensors and/
8                            Oligodendrocytes (galactocerebroside) and astrocytes (GFAP) had constituti
9 ich is a substrate for the deficient enzyme (galactocerebroside beta-galactosidase).
10 ion factor related, locus 2) NG2+, or GalC+ (galactocerebroside) cells.
11 el raft-like system consisting of nonhydroxy galactocerebroside, cholesterol, and dipalmitoylphosphat
12 dition of cholesterol, however, disrupts the galactocerebroside domains, resulting in a slight increa
13 revealed that loss of ERK2 resulted in fewer galactocerebroside-expressing mature oligodendrocytes in
14 roteins and an abundance of the galactolipid galactocerebroside (GalC) and its sulfated derivative su
15 embrane greatly enriched in the galactolipid galactocerebroside (GalC) and its sulfated derivative su
16 ath is greatly enriched in the galactolipids galactocerebroside (GalC) and sulfatide.
17 arbonic anhydrase isoenzyme II (CA-II) or to galactocerebroside (GalC) for visualization of oligodend
18 late proliferation and expression of surface galactocerebroside (galC) in cultured Schwann cells from
19 oliferation and an expression of the surface galactocerebroside (galC) in response to forskolin were
20 e from a GBS patient with antibodies against galactocerebroside (GalC), which cross-reacted with the
21 fatide); and RMAb, which is directed against galactocerebroside (GC).
22 ated with antibodies to CD4 (anti-T4A) or to galactocerebroside (GC).
23                                              Galactocerebrosides (GCs) represent a major class of gly
24 zing the degradation of its major substrate, galactocerebroside, in oligodendrocytes (OLs) and Schwan
25        Levels of ceramide, glucosylceramide, galactocerebroside, lactosylceramide, globotriaosylceram
26                                      Hydroxy-galactocerebrosides (mixed chain length, constituent of
27                        The dissolved hydroxy-galactocerebroside molecules reenter on monolayer expans
28  showing that anti-sulfatide O4 but not anti-galactocerebroside O1 blocks terminal differentiation, p
29                           Sulfatide, but not galactocerebroside or ceramide, strongly inhibited the n
30  and negatively charged liposomes containing galactocerebroside or complexed with lactosylated polyet
31 d beta-tubulin(III)-positive neurons; and 3) galactocerebroside-positive oligodendrocytes.
32 erentiated oligodendrocytes begin to express galactocerebroside recognized by O1 antibodies and subse
33 d O4 bind to galactocerebroside and sulfated galactocerebroside, respectively, with unusually small a
34 antibodies: MAbO4, which is directed against galactocerebroside sulfate (sulfatide); and RMAb, which
35     We showed previously that an antibody to galactocerebroside/sulfatide arrested terminal different
36     Galactose oxidase converts galactose and galactocerebroside to their corresponding aldehydes and
37  has been developed to measure galactose and galactocerebroside using galactose oxidase immobilized o
38         The linear response to galactose and galactocerebroside was to at least 300 microM.
39 verts sulfatide (sulfogalactocerebroside) to galactocerebroside, was measured, and its inhibition by
40 he minimal detection limits of galactose and galactocerebroside were 1 and 2 microM, respectively.

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