ライフサイエンスコーパス: galactose

1 lly important hexoses (glucose, mannose, and galactose).

2 p11 as CcrG, Campylobacter ChemoReceptor for Galactose.

3 responsible for lowering its affinity toward galactose.

4 ct of galectins comparable with that of free galactose.

5 show that Tlp11 specifically interacts with galactose.

6 e + UDP-glucose to glucose-1-phosphate + UDP-galactose.

7 ain decorated with terminal alpha-1,6-linked galactose.

8 l aldehydes from D-glucose, D-mannose, and D-galactose.

9 galacturonic acid followed by arabinose and galactose.

10 grown in mixtures of glucose (preferred) and galactose.

11 cid, S. oralis bound exposed beta-1,4-linked galactose.

12 this ingredient, due to a higher presence of galactose.

13 from 93% for lactose to 98% for glucose and galactose.

14 nd the presence of truncated glycans lacking galactose.

15 e 1-phosphate, and GDP-glucose when grown on galactose.

16 rminus can be substituted by an alpha-linked galactose.

17 patterns for fructose, mannose, glucose, and galactose.

18 t this did not generalize to Na-saccharin or galactose.

19 D-galactose (2 muM), galactitol (11 muM) and galactose 1-phosphate (0.1 mM), (corresponding to plasma

20 lethal disease caused by the dysfunction of galactose 1-phosphate uridylyltransferase (GALT).

21 y-dependent growth phenotype and accumulated galactose 1-phosphate, glucose 1-phosphate, and GDP-gluc

22 alactose and its metabolites, galactitol and galactose 1-phosphate, on oocyte quality as well as embr

23 5 A) and complexed with glucose (1.25 A) and galactose (1.8 A).

24 ate uridyltransferase (GALT), which converts galactose-1-phosphate + UDP-glucose to glucose-1-phospha

25 Galactosemia I results from loss of galactose-1-phosphate uridyltransferase (GALT), which co

26 UDP-galactose from uridine triphosphate and galactose-1-phosphate.

27 okinase (GALK), phosphorylating galactose to galactose-1-phosphate.

28 s cells (CCs), were exposed for 4 hours to D-galactose (2 muM), galactitol (11 muM) and galactose 1-p

29 e typically less reactive and selective than galactose 3,4-diols.

30 teral chains at C-2 as well as at C-6 of the galactose 3-O residues; mono-O-substituted galactoses we

31 st4, a gene implicated in human PE, encoding Galactose-3-O-sulfotransferase 4.

32 The polysaccharide rich in arabinose and galactose (39-54%) and mannoproteins (38-55%) were the m

33 alactosemia III results from the loss of UDP-galactose 4'-epimerase (GALE), which interconverts UDP-g

34 6.97%), glucose (88.90%, 89.31%, 87.68%) and galactose (5.34%, 5.17%, 5.35%).

35 Galactose (58.9-91.2%, w/w) was the main monosaccharide

36 tive-site structure vis-a-vis the archetypal galactose 6-oxidase from Fusarium graminearum.

37 Despite a long history of study, the galactose 6-oxidase/glyoxal oxidase family of mononuclea

38 switch to the secondary carbon source (e.g., galactose), a paradigm known as the Monod model.

39 mal studies have shown that a high intake of galactose, a breakdown product of lactose, increases ova

40 cells are at high density in the presence of galactose, a main sugar of the human nasopharynx, a high

41 he classical model expects (i.e., cannot use galactose above a glucose threshold) has a fitness disad

42 animals were tested for GI transit time and galactose absorption, and fecal weight and fat content w

43 NPC1-deficient ldl-D cells supplemented with galactose accumulated more cholesterol than those in whi

44 The galactose affinity of the RIP-II proteins enabled their

45 Peanut and galactose alpha-1,3-galactose (alpha-gal) are characteri

46 n-human glycan epitopes, galactose-alpha-1,3-galactose (alpha-gal) and Neu5Gc-alpha-2-6-galactose (Ne

47 E antibodies directed at galactose-alpha-1,3-galactose (alpha-Gal) are associated with a novel form o

48 Peanut and galactose alpha-1,3-galactose (alpha-gal) are characterized by high- or very

49 IgG to galactose-alpha-1,3-galactose (alpha-gal) are highly abundant natural antibo

50 ibodies (Ab) specific to galactose-alpha-1,3-galactose (alpha-gal) are responsible for a delayed form

51 Galactose-alpha-1,3-galactose (alpha-Gal) is a mammalian carbohydrate with s

52 inding glycan allergen galactose-alpha-(1,3)-galactose (alpha-Gal) is associated with IgE-mediated de

53 ific to the carbohydrate galactose-alpha-1,3-galactose (alpha-gal) is known to induce delayed anaphyl

54 tain the disaccharide galactosyl-alpha-(1,3)-galactose (alpha-Gal).

55 oligosaccharide epitope, galactose-alpha-1,3-galactose (alpha-gal).

56 other hand, the IgE-binding glycan allergen galactose-alpha-(1,3)-galactose (alpha-Gal) is associate

57 Two non-human glycan epitopes, galactose-alpha-1,3-galactose (alpha-gal) and Neu5Gc-alp

58 Serum IgE antibodies directed at galactose-alpha-1,3-galactose (alpha-Gal) are associated

59 IgG to galactose-alpha-1,3-galactose (alpha-gal) are highly abu

60 IgE antibodies (Ab) specific to galactose-alpha-1,3-galactose (alpha-gal) are responsibl

61 Galactose-alpha-1,3-galactose (alpha-Gal) is a mammalian

62 f IgE molecules specific to the carbohydrate galactose-alpha-1,3-galactose (alpha-gal) is known to in

63 onse to a mammalian oligosaccharide epitope, galactose-alpha-1,3-galactose (alpha-gal).

64 ral hours in patients with IgE to alpha-gal (galactose-alpha-1,3-galactose) have been reported.

65 The carbohydrate epitope galactose-alpha-1,3-galactose, located on the Fab region

66 Muscle biopsies were stained for galactose-alpha1,3-galactose, immunoglobulin M, immunogl

67 of Helicobacter pylori and the corresponding galactose analogue in 66-78% overall yields from free su

68 rolines (Hyps) are substituted with an alpha-galactose and 1-5 beta- or alpha-linked arabinofuranoses

69 A to the FBS resulted in a release of 2.8 mM galactose and 4.3 mM N-acetylneuraminic acid; these suga

70 which is in turn substituted with a terminal galactose and a second xylose residue.

71 oral cancer cells contain the terminal alpha-galactose and are more diverse with higher fucosylation

72 Pectin contained relatively high amounts of galactose and considerable beta-galactosidase (beta-Gal)

73 so possess transporters that allow growth on galactose and fructose.

74 ynthesizes unusual N-glycans with a range of galactose and fucose modifications on the Man2-3GlcNAc2

75 AlcOx are essentially incapable of oxidizing galactose and galactosides, but instead efficiently cata

76 to the hydrolysis of lactose, high levels of galactose and glucose were found together with galactool

77 Arabinose, xylose, mannose, galactose and glucose were the main sugar constituents o

78 YHB1 mRNA is stabilized in galactose and high culture density, indicating inactivat

79 the quantitative response of these genes to galactose and in the position of these genes in the over

80 Our results suggested that D-galactose and its metabolites disturbed the spindle stru

81 Here, we evaluate the effect of D-galactose and its metabolites, galactitol and galactose

82 both extracts with predominance of glucose, galactose and mannose with no uronic acids detection; Fl

83 eptococcus thermophilus and higher levels of galactose and phenylalanine.

84 ctrometry indicated reduced incorporation of galactose and sialic acid, as seen in other Golgi homeos

85 deletion of uge5 and uge3 blocked growth on galactose and synthesis of both Galf and galactosaminoga

86 an SRRP is required to bind beta-1,4-linked galactose and the first time that one of these adhesins

87 6-anhydro-alpha-L-galactopyranose, sulphated galactose and the gelling agent agar, with the sulphate

88 is of the 1,2-cis-glycosidic linkage between galactose and the linker (spacer) molecule and final pur

89 of commercial enzyme modified the Arabinose/Galactose and the Rhamnose/Galacturonic acid ratios in C

90 5, Uge3 activity is sufficient for growth on galactose and the synthesis of galactosaminogalactan con

91 4'-epimerase (GALE), which interconverts UDP-galactose and UDP-glucose, as well as UDP-N-acetylgalact

92 Uge3 can mediate production of both UDP-galactose and UDP-N-acetylgalactosamine (GalNAc) and is

93 ion 10R was composed of rhamnose, arabinose, galactose and uronic acid in 2.8:65.8:28.5:3M ratio, res

94 ion 50R was composed of rhamnose, arabinose, galactose and uronic acid in 4.3:56.2:37.4:2M ratio, res

95 d of arabinose, rhamnose, glucose, fructose, galactose and xylose.

96 ere observed between fermentable (glucose or galactose) and nonfermentable (glycerol) carbon sources

97 g/100g for lactose, 0.14 and 0.27mg/100g for galactose, and 0.16 and 0.26mg/100g for glucose.

98 r oligosaccharides (N-acetylneuraminic acid, galactose, and 6'-sialyllactose), linkage-specific siali

99 eromultivalent oligomers presenting mannose, galactose, and glucose residues.

100 , d-glucose, l-allose, d-allose, d-gulose, d-galactose, and l-mannose are delineated, and for all eig

101 Complexes of GlyA1 with glucose, galactose, and xylose allowed picturing the catalytic po

102 shifting hPSC-CMs from glucose-containing to galactose- and fatty acid-containing medium promotes the

103 sugars mannosamine-6-phosphate, sialic acid, galactose- and glucose-derived from hydrolysis of mixtur

104 ous rehydration and when feeding a glucose-, galactose-, and lactose-free formula.

105 of nine determined monosaccharides (fucose, galactose, arabinose, glucose, rhamnose, xylose, mannose

106 Fucose, galactose, arabinose, glucose, sucrose, rhamnose, xylose

107 hydro-heptitols derived from D-mannose and D-galactose are enantiomeric and are useful linkers for th

108 By-products such as glucose and galactose are generated.

109 of the metabolic transition from glucose to galactose are responsible for the variability in galacto

110 es when glucose or raffinose was replaced by galactose as the carbon source.

111 for the chemotactic response of C. jejuni to galactose, as shown using wild type, allelic inactivatio

112 The Fml adhesin FmlH binds galactose beta1-3 N-acetylgalactosamine found in core-1

113 n motif is located in close proximity to the galactose binding footprint on AAV9 capsids and postulat

114 by which Fap1 contributes to beta-1,4-linked galactose binding remains to be defined; however, bindin

115 two subunits, a ribotoxic A chain (RTA) and galactose-binding B chain (RTB).

116 eal that the crystallographically identified galactose-binding site in vSGLT is located in a more ext

117 lyses suggest the existence of an additional galactose-binding site in vSGLT that aligns to the S1 si

118 tensively studied and importance of terminal galactose, bisecting GlcNAc and core fucose has been rea

119 idine methyl ester linker, and an acetylated galactose bonded to one of the aromatic nitrogen atoms o

120 ctosaminogalactan containing lower levels of galactose but not the synthesis of Galf.

121 sferase with catalytic activity towards beta-galactose but rather a beta-1,4-glucuronyltransferase, d

122 s cerevisiae is activated by the presence of galactose but repressed by glucose.

123 epitope targets such as glucose, fructose or galactose by forming ternary complexes with high-epitope

124 Catabolite repression of galactose by glucose is one of the best-studied eukaryot

125 Catabolism of galactose by Streptococcus pneumoniae alters the microbe

126 ng growth arrest, likely due to overly rapid galactose catabolism and metabolic overload.

127 Effective hepatic blood flow (EHBF) by galactose clearance, wet-dry weights, cytokines, histopa

128 two strong hydrogen bonds between ppGBP and galactose compared with glucose may be responsible for l

129 ture compared with unliganded form and ppGBP-galactose complex.

130 blood flow and arterial and liver vein blood galactose concentrations at increasing galactose infusio

131 ions is modulated in a well-defined range of galactose concentrations, correlating with a dynamic cha

132 PTT:galactose conjugates exhibited similar thermal stability

133 media, is essential in phosphate-depleted or galactose-containing media.

134 strated that these cells are able to grow on galactose-containing medium but not on other fermentable

135 l wall of Aspergillus fumigatus contains two galactose-containing polysaccharides, galactomannan and

136 alectins because the polysaccharides contain galactose-containing side chains that might bind this cl

137 Galactose content of extracted polysaccharides can be mo

138 ere more efficiently bioactivated than their galactose counterparts.

139 relevant underivatized hexoses, d-glucose, d-galactose, d-mannose, and d-fructose, using only mass sp

140 In summary, IL-6 and IL-4 accentuated galactose deficiency of IgA1 via coordinated modulation

141 lesser degree, IL-4 significantly increased galactose deficiency of IgA1; changes in IgA1 O-glycosyl

142 IgG galactose deficiency was correlated with the severity of

143 t serum IgG-Fc glycosylation ,: particularly galactose deficiency, was higher in patients with CHB an

144 B cells, it failed to reduce serum levels of galactose-deficient IgA1 and antigalactose-deficient IgA

145 by pathogenic immune complexes consisting of galactose-deficient IgA1 bound by antiglycan antibodies.

146 lactose-deficient IgA1 or antibodies against galactose-deficient IgA1 did not change.

147 Serum levels of galactose-deficient IgA1 or antibodies against galactose

148 Galactose-deficient polymeric IgA1 alone, but not M4, in

149 A-binding M4 protein binds preferentially to galactose-deficient polymeric IgA1 and that these protei

150 nique loop regions, recognizing 6-O-sulfated galactose dictates tight specificity distinct from other

151 ited Gal1 produced during previous growth in galactose directly interferes with Gal80 repression to p

152 nt changes in plasticity between glucose and galactose distributed throughout the promoter, suggestin

153 and D' contain an intact gene encoding a UDP-galactose epimerase (galE1) and a truncated remnant (gal

154 demonstrated for H-type I and II; alpha(1,3)-galactose epitopes were prepared, and the pentasaccharid

155 No differences of galactose expression or deposition of immunoglobulin M a

156 ccharomyces cerevisiae adapting to growth in galactose for up to 8 generations.

157 sponse in PTDH3 activity between glucose and galactose from becoming larger.

158 ophosphorylase (UGP) alternatively makes UDP-galactose from uridine triphosphate and galactose-1-phos

159 isotope labeled carbon sources like glucose, galactose, fructose, and naphthalene.

160 tions of each of seven saccharides (glucose, galactose, fructose, sucrose, trehalose, raffinose, and

161 titotal ATG, but also antigalactose-alpha1-3-galactose (Gal) and anti-Neu5Gc antibodies, 2 xenocarboh

162 s specific for pig xenoantigens, alpha-(1,3)-galactose (GAL) and N-glycolylneuraminic acid (Neu5Gc),

163 Promoters of seven galactose (GAL) metabolic genes from S. cerevisiae, when

164 rization of six bifunctional UDP-l-Rha/UDP-d-galactose (Gal) transporters (URGTs).

165 Mannose (Man)- and galactose (Gal)-conjugated silica nanoparticles (SNPs) w

166 patients with chronic periodontitis contains galactose (Gal)-deficient IgG.

167 Marmosets maintained on 30% galactose (gal)-rich diet for 2 years were monitored for

168 Following previous growth in galactose, GAL gene transcriptional memory confers a str

169 Glucose- (glc-) and galactose- (gal-) PAS 10-mer structures are synthesized

170 ohydrate links to hydroxyproline through the galactose (galactosylation).

171 ctose are responsible for the variability in galactose gene activation.

172 ted determines the timing and variability of galactose gene activation.

173 The neutral polysaccharides consist of galactose, glucose and mannose whereas the acidic polysa

174 g from cultures grown in minimal media using galactose, glucose, or raffinose as the carbon source.

175 ly perturbed the components of the canonical galactose/glucose signaling pathways and found that thes

176 Finally, providing specific nutrients (galactose/glucose) to MuSCs directly controlled their fa

177 gene induction occurs at a constant external galactose:glucose ratio across a wide range of sugar con

178 s with IgE to alpha-gal (galactose-alpha-1,3-galactose) have been reported.

179 unique positioning of the 3-O-sulfated beta-galactose headgroup.

180 biopsies were stained for galactose-alpha1,3-galactose, immunoglobulin M, immunoglobulin G, complemen

181 ption and that mutant WTA lacked appreciable galactose in all except one mutant - which retained a le

182 Dietary supplementation with galactose in six patients resulted in changes suggestive

183 igher content of (methyl)glucuronic acid and galactose in tension wood than in normal wood.

184 al(166)-Glu(170) of FaeGad bind the terminal galactose in the lactosyl unit and provide affinity and

185 y determined the hepatic removal kinetics of galactose, including hepatic intrinsic clearance of gala

186 een the HAS1-TDA1 alleles specifically under galactose induction and saturated growth.

187 During galactose induction, GAL10 sense transcription occurs in

188 y a mutant promoter with delayed response to galactose induction, we found that the two reporters phy

189 blood galactose concentrations at increasing galactose infusions.

190 The three fap2 mutants failed to show galactose-inhibitable coaggregation with Porphyromonas g

191 that SV2A is able to transport extracellular galactose inside the cells.

192 cose is absent from the nasopharynx, whereas galactose is abundant.

193 o)Fuc2NAc4N(alpha1-6)GlcNAc(beta1--> , where galactose is linked to the hydroxyglutarate moiety of Fu

194 DPG), kaempferol and UDPG, quercetin and UDP-galactose, isoliquiritigenin and UDPG, and luteolin and

195 re inhibitory for biofilm formation, whereas galactose, lactose, and low concentrations of sialic aci

196 e, glucose-1-phosphate, glucose-6-phosphate, galactose, lactose, and sucrose--at low mM concentration

197 Supplementation with galactose leads to biochemical improvement in indexes of

198 otein synthesis, and ricin B can bind to the galactose ligand on the cell membrane of host cells.

199 The carbohydrate epitope galactose-alpha-1,3-galactose, located on the Fab region of cetuximab, was i

200 Therefore, glucose-galactose-malabsorption was assumed.

201 ccharides (N-acetylneuraminic acid (Neu5Ac), galactose, mannose, and fucose) and significantly (p < 0

202 patient-derived fibroblasts in glucose-free galactose medium revealed a respiratory chain defect in

203 apitulated the delay phenotype in 1% glucose-galactose medium, and most had partial effects when test

204 delay when tested individually in 1% glucose-galactose medium.

205 In this system, it is believed that galactose metabolic (GAL) genes are induced only when gl

206 pon depletion of glucose, the genes encoding galactose metabolic proteins will activate.

207 itute the majority of transcriptional CCR of galactose metabolism operons.

208 Saccharomyces species, delayed commitment to galactose metabolism until glucose was exhausted.

209 ly, we find that Gal1p, an enzyme needed for galactose metabolism, accumulates more quickly if glucos

210 reparing" for the transition from glucose to galactose metabolism.

211 pared to the unmodified control strands, the galactose-modified oligonucleotides in general, and the

212 Tripodal RNA structure complexed with galactose-modified PEI could generate effective RNAi-med

213 Galactose-modified thymidine, LNA-T, and 2'-amino-LNA-T

214 s, including uniform responses (d-lactose, d-galactose, N-acetylglucosamine, N-acetylneuraminic acid)

215 Fecal anti-galactose/N-acetylgalactosamine lectin immunoglobulin A

216 Using the galactose network as a model, here we show how the regul

217 3-galactose (alpha-gal) and Neu5Gc-alpha-2-6-galactose (Neu5Gc) have been shown to be antigenic when

218 val of the terminal N-acetylgalactosamine or galactose of A- or B-antigens, respectively, yields univ

219 id residues from host glycoproteins, exposed galactose on the surface of septal epithelial cells, the

220 solubilized glycosyltransferases that attach galactose or sialic acid.

221 f this promoter in media containing glucose, galactose, or glycerol as a carbon source.

222 A galactose oxidase (GAOx) biosensor, based on the immobil

223 Galactose oxidase (GO) is a copper-dependent enzyme that

224 s alcohol oxidation mediated by Cu/TEMPO and galactose oxidase.

225 the posttranscriptional repression of GDP-l-galactose phosphorylase (GGP), a major control enzyme in

226 foliar AsA level by 20-30%, and KO of GDP-L-galactose phosphorylase (OsGGP) by 80%, while KO of myo-

227 ns and polysaccharides rich in arabinose and galactose (PRAG) were poor foam formers but good foam st

228 t only Polysaccharides Rich in Arabinose and Galactose (PRAGs) were considered in the final fitted ML

229 e, N-acetyl-D-galactosamine, D-glucose and D-galactose, present on the cell surface.

230 onse of GAL genes to mixtures of glucose and galactose rather than by induction kinetics.

231 potentially interfere with the high affinity galactose-recognition element that plays a critical role

232 cells consume glucose before galactose, the galactose regulatory pathway is activated in a fraction

233 d uptake in hepatocytes as a result of their galactose residues and can disrupt endosomes efficiently

234 We find that galactose residues do not participate in the binding to

235 Mannose and galactose residues in the oligosaccharide fraction are p

236 thesized by CELLULOSE SYNTHASE-LIKE A2, with galactose residues in vivo.

237 rate recognition domains, and the GlcNAc and galactose residues make additional interactions in a wid

238 s is consistently enhanced by the absence of galactose residues per se or the lack of terminal sialyl

239 cross-link surface glycoproteins by binding galactose residues that are normally hidden below termin

240 inopyranose (d-Arap) caps the LPG side-chain galactose residues, blocking interaction with the midgut

241 me responsible for addition of the final two galactose residues, in alpha-linkages to the Skp1 core t

242 position of arabinose or the O-6 position of galactose residues.

243 expressing terminal N-acetylgalactosamine or galactose residues.

244 ive chains that are masked by acid-cleavable galactose residues.

245 se, and five distinct partially O-methylated galactose residues.

246 nting mannose or combinations of mannose and galactose residues.

247 1, whose reducing end sugars are glucose and galactose, respectively.

248 ding to plasma concentrations in patients on galactose-restricted diet) and compared to controls.

249 rroirs wines, and it modified the (Arabinose+Galactose)/Rhamnose ratio in Canada Judio, Albatana and

250 icant parameter affecting linearly yield and galactose/rhamnose contents.

251 tured in 1% glucose medium supplemented with galactose, Saccharomyces cerevisiae, but not S. bayanus

252 Radiolabeled galactose saturation binding experiments indicate that,

253 Galactose seems to make the weakest and allose the stron

254 vestigate the structural determinants of UDP-galactose selectivity.

255 ination of some F. nucleatum strains is also galactose sensitive, suggesting that a single galactose-

256 (called coadherences or coaggregations) are galactose sensitive.

257 alactose sensitive, suggesting that a single galactose-sensitive adhesin might mediate the interactio

258 In order to identify the fusobacterial galactose-sensitive adhesin, a system for transposon mut

259 Our results suggest that Fap2 is a galactose-sensitive hemagglutinin and adhesin that is li

260 Galactose-sensitive interactions are also involved in th

261 Fibroblasts supplemented with galactose showed restoration of protein glycosylation an

262 ed to 59 samples of Grana Padano PDO cheese: galactose showed the highest concentration and variabili

263 I backbone and two of them from the branched galactose-sialic acid disaccharide contained in this seq

264 sulphate group was at the C6 position of the galactose skeleton.

265 a novel class of pathognomonic marker due to galactose stress in affected neonates.

266 ions of two pairs of ancient paralogs of the GALactose sugar utilization network in two yeast species

267 Oral galactose supplementation is a treatment option and resu

268 In the absence of galactose supplementation, NPC1-deficient ldl-D cells al

269 se studies, BDCA-2 binds to IgG, which bears galactose-terminated glycans that are not commonly found

270 chromatographic material was modified with a galactose-terminated substituent and packed into miniatu

271 en reported that BDCA-2 binds selectively to galactose-terminated, biantennary N-linked glycans.

272 yces cerevisiae cells consume glucose before galactose, the galactose regulatory pathway is activated

273 of a higher affinity of galactokinase toward galactose, the lumped constant (LC) for (18)F-FDGal was

274 in both species-the GAL genes are induced by galactose-there are major differences in both the quanti

275 ed "-Omics" analyses showed that addition of galactose to culture medium improves total oxidative cap

276 oss of galactokinase (GALK), phosphorylating galactose to galactose-1-phosphate.

277 UGT8 uses UDP galactose to galactosidate ceramide, a key step in the s

278 code the enzymes needed for cells to convert galactose to glucose.

279 , and a fitness detriment during the glucose-galactose transition but a benefit when glucose is in ex

280 The newly identified galactose transport capability of SV2A may have an impor

281 In cardiomyocytes, galactose (transported through SGLT1) did not activate N

282 an acetyl-CoA transporter (pfact) and a UDP-galactose transporter (pfugt).

283 UDP-galactose transporter (UGT; SLC35A2) and UDP-N-acetylglu

284 The structure of the sodium/galactose transporter (vSGLT), a solute-sodium symporter

285 The macrophage galactose-type lectin (MGL) is a C-type lectin that bind

286 study we examined the function of macrophage galactose-type lectin-1 (MGL1), a mammalian CLR, in pneu

287 non-hydrolyzable donor analog UDP-phosphono-galactose (UDP-C-Gal).

288 be either mono-functional, synthesising UDP-galactose (UDP-Gal), or bi-functional, synthesising UDP-

289 ns corresponding to the externally available galactose units (20%).

290 Most importantly, direct measurement of galactose uptake in the same strain verified that SV2A i

291 These strains induce galactose utilization (GAL) genes hours before glucose e

292 For example, the galactose utilization network in Saccharomyces cerevisia

293 se, including hepatic intrinsic clearance of galactose (V(max)/K(m)) from measurements of hepatic blo

294 GC-FID results proved that galactose was the dominant sugar in the extracted polysa

295 filtrate and in all second fractions, where galactose was the major component.

296 se, xylose, and glucose, whereas mannose and galactose were present in small amounts.

297 e galactose 3-O residues; mono-O-substituted galactoses were not detected.

298 um containing 2.5% glucose supplemented with galactose, wild-type S. cerevisiae repressed GAL gene ex

299 ly composed of galacturonic acid, arabinose, galactose, xylose and glucose.

300 as a mass of 40 to 50 kDa and is composed of galactose, xylose, and five distinct partially O-methyla