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1 of the cytoplasmic cavity in the presence of galactoside.
2 the chromogenic substrate o-nitrophenyl-beta-galactoside.
3 ined on induction with isopropyl thio-beta-D-galactoside.
4 , thiodigalactoside, and m-nitrophenyl-alpha-galactoside.
5 side was rapidly metabolized to peonidin-3-O-galactoside.
6 affinity for melibiose or nitrophenyl-alpha-galactoside.
7 te -1 to enable the binding of various alpha-galactosides.
8 FV was also affected in the presence of beta-galactosides.
9 modification blocks galectin binding to beta-galactosides.
10 ndary carbon sources such as alpha- and beta-galactosides.
11 recognition domain that interacts with beta-galactosides.
12 naling requires binding to cell surface beta-galactosides.
13 ratios despite intense selection on the pure galactosides.
14 ns that are selectively favored on different galactosides.
15 the utilization of raffinose and other alpha-galactosides.
16 hich is induced on exposure to galactose and galactosides.
17 transports sucrose but not lactose or other galactosides.
18 strates dissimilar to either glucuronides or galactosides.
19 ls to catalyze the formation of flavonol 3-O-galactosides.
20 ility of PllA as a probe for detecting alpha-galactosides.
21 anomerization to obtain good yields of alpha-galactosides.
22 yptophan, Wessely-Moser isomers apigenin-6-C-galactoside-8-C-arabinoside & apigenin-6-C-arabinoside-8
23 its three major lipids, acylated cholesteryl galactoside (ACGal), monogalactosyl diacyglycerol (MGalD
28 beta-1,3-glucosyltransferase (B3GLCT), beta-galactoside alpha-2,3-sialyltransferase 5 (ST3GAL5), and
29 study we identified high expression of beta-galactoside alpha-2,3-sialyltransferase, ST3GAL6, in MM
31 ether inactivation of the UDP-galactose:beta-galactoside-alpha1-3-galactosyltransferase (alpha1,3GT)
34 ation of the Fc fragment is mediated by beta-galactoside alpha2,6-sialyltransferase 1 (ST6Gal-1), act
36 prisingly, however, binding of high-affinity galactoside analogues is severely compromised in the mut
37 Here we show that DDAOG, a conjugate of beta-galactoside and 7-hydroxy-9H-(1,3-dichloro-9,9-dimethyla
38 ns, catalyzes the coupled translocation of a galactoside and a H(+) across the cytoplasmic membrane o
40 ns, catalyzes the coupled translocation of a galactoside and an H(+) across the Escherichia coli memb
44 n, respectively) together with quercetin-3-O-galactoside and quercetin-3-O-glucoside (15 and 140mugg(
46 in family defined by their affinity for beta-galactosides and by their conserved carbohydrate recogni
50 C-arabinoside & apigenin-6-C-arabinoside-8-C-galactoside, and 9,12,13-trihydroxy-trans-10-octadecenoi
52 noninducing species, orthonitrophenyl-beta-D-galactoside; and (3) transform an inducible switch to on
53 sion strain as a biosensor to determine that galactosides are released from the seeds of several diff
54 way, and could be involved in the sorting of galactoside-bearing glycoconjugates, since it was found
55 ha-L-arabinosides, beta-D-glucosides, beta-D-galactosides, beta-L-xylosides, beta-D-arabinosides), si
56 pK for binding is approximately 10.5); (iii) galactoside binding and dissociation, not microH+, are t
57 imately, these structural changes facilitate galactoside binding and may be involved in the storage o
59 driving forces for alternating access; (iv) galactoside binding involves induced fit, causing transi
63 en-Friedenreich antigen (T antigen) and beta-galactoside binding lectins (galectins) have been implic
64 s performed with a model ligand on both beta-galactoside binding lectins showed additional interactio
66 ase (PI3K) and Ras is suppressed by the beta-galactoside binding protein (betaGBP) molecule, a cytoki
68 Galectin-3 (Gal-3), a member of the beta-galactoside binding protein family containing the NWGR a
69 ced cellular expression of the secreted beta-galactoside binding protein Galectin 1-like 2 (Drgal1-L2
70 Alterations in the production of the beta-galactoside binding protein galectin-3 and of MUC2 intes
76 plasmic loop IV/V and in the vicinity of the galactoside binding site at the interface of helices IV,
78 conformational changes in LacY initiated by galactoside binding were monitored in real time by Trp q
80 e residues have been shown to participate in galactoside binding: Cys148 hydrophobically interacts wi
86 Galectin-3 is a member of a family of beta-galactoside-binding animal lectins expressed abundantly
91 ative trait locus for the transcript soluble galactoside-binding lectin 9 (LGALS9) that is in linkage
96 Galectin-3 (Gal-3) is a member of the beta-galactoside-binding lectin family and plays an important
97 we show that galectin-12, a member of a beta-galactoside-binding lectin family preferentially express
98 Galectin-1 is a member of the conserved beta-galactoside-binding lectin family that binds galactoside
103 Several lines of evidence implicate the beta-galactoside-binding lectin galectin-3 in development and
104 e hypothesized that human galectin-3, a beta-galactoside-binding lectin involved in immune regulation
105 es exhibited increased affinity for the beta-galactoside-binding lectin RCA-I in the presence of AZT,
109 show that ablation of galectin-3 (Gal-3), a galactoside-binding lectin, accelerates high-fat diet-in
112 e show that expression of galectin-3, a beta-galactoside-binding lectin, is up-regulated in a mouse m
114 -1 (Gal-1), an evolutionarily conserved beta-galactoside-binding lectin, plays essential roles in the
115 rminal sugar, the expression profile of beta-galactoside-binding lectins (galectins) in MDA-MB-435 ce
116 Galectin-1 is a member of a family of beta-galactoside-binding lectins that are soluble adhesion mo
118 he prototype galectin family, which are beta-galactoside-binding lectins, exhibit subunit-specific al
120 tin (Gal)-3, a M(r) 31000 member of the beta-galactoside-binding protein family, is a multifunctional
143 an galectin-1 (dGal-1), a small dimeric beta-galactoside-binding protein, induces phosphatidylserine
149 Galectin-3 (gal-3), a member of the beta-galactoside-binding proteins family, was identified as a
150 a member of a growing family of animal beta-galactoside-binding proteins shown to be involved in cel
152 Galectins are a family of mammalian beta-galactoside-binding proteins that positively and negativ
153 er of the galectin family consisting of beta-galactoside-binding proteins with conserved carbohydrate
155 roteoliposomes reveals dramatic increases in galactoside-binding rates induced by interaction with th
157 Tumor-derived galectin-1 (Gal-1), a beta-galactoside-binding S-type lectin, has been shown to enc
158 d were very poor inhibitors of the canonical galactoside-binding site for the tested galectins, with
159 own inhibitors of galectin-3 target its beta-galactoside-binding site in the carbohydrate recognition
160 r inhibition of the evolutionarily conserved galactoside-binding site of galectins has not been demon
164 we transferred the Gal-1-KO mutation (lectin galactoside-binding soluble 1(-/-) ) from the B6 strain
166 cking Gal-1-mediated angiogenesis or lectin, galactoside-binding, soluble, 1 deficiency results in a
168 del was built for the three considered alpha-galactosides both in the seed and in the soaking water,
169 greatly reduced or no growth on sucrose and galactosides but did not affect growth on monosaccharide
170 ntially incapable of oxidizing galactose and galactosides, but instead efficiently catalyse the oxida
173 ntaneous hydrolysis of the 2,4-dinitrophenyl galactosides, confirming earlier studies on the role pla
178 drate-binding protein with affinity for beta-galactoside-containing glycoconjugates, is upregulated u
180 basis for PllA's high specificity for alpha-galactoside-containing ligands, and we show that PllA ca
182 ide, delphinidin-3-O-glucoside, cyanidin-3-O-galactoside, cyanidin-3-O-glucoside, cyanidin-3-O-arabin
183 that delphinidin-3-glucoside, delphinidin-3-galactoside, delphinidin-3-arabinoside and petunidin-3-a
185 of cultured malignant T cells induced a beta-galactoside-dependent inhibition of normal T-cell prolif
186 diate that undergoes alternating access; (v) galactoside dissociates, releasing the energy of binding
187 produced coupled reaction-diffusion of alpha-galactosides during the soaking-cooking process with a g
189 nize a wide variety of glycans with terminal galactosides for conferring epithelial cell adhesion.
191 mutant hardly catalyzes transport, but binds galactosides from either side of the membrane with the s
193 black carrot pomace with cyanidin-3-xyloside-galactoside-glucoside-ferrulic acid (C3XGGF, 60.85-74.22
195 c membrane protein, catalyzes stoichiometric galactoside/H(+) symport by an alternating access mechan
203 -3-O-rhamnogalactoside, rutin, quercetin-3-O-galactoside (hyperoside), quercetin-3-O-glucoside (querc
205 ivers with 5-bromo-4-chloro-3-indolyl beta-d-galactoside in any of the 18 recipient mice analyzed wer
207 concluded that indole-3-acetyl-myo-inositol galactoside in the endosperm supplies about 2 picomoles
209 eriments examining microbial growth on alpha-galactosides in seed wash suggested that alpha-galactosi
213 alectin-3, an animal lectin recognizing beta-galactosides, in regulating dendritic cell motility both
214 ceable residue was mutated individually, and galactoside-induced opening or closing of periplasmic or
216 competitive inhibitor isopropyl thio-beta-D-galactoside (IPTG) requires the mutant enzyme to adopt i
217 olecules suggest that the O6 hydroxyl on the galactoside is essential for establishing a water-mediat
218 er exclusion"), and with lactose permease, a galactoside is required for unphosphorylated IIA(Glc) bi
219 rate-binding proteins with affinity for beta-galactosides, is a key modulator of diverse cell functio
220 a special lectin with high affinity to beta-galactosides, is implicated in protection against ischem
221 lthough the P. squamosus lectin binds beta-D-galactosides, it has an extended carbohydrate-combining
222 e to form an alpha 1-3 link with beta-linked galactosides; it is part of a family of homologous retai
223 ricin glycome is enriched with terminal beta-galactosides, known binding partners for a family of mul
225 mber of the galectin family of secreted beta-galactoside lectins containing a conserved carbohydrate
228 resents five copies of m-nitrophenyl-alpha-D-galactoside (MNPG) rather than five copies of beta-D-gal
229 ntal conditions, benzochlorin without a beta-galactoside moiety or the related glucose conjugate did
230 l-04 binds alpha-(1,6)-linked glucosides and galactosides of varying size, linkage, and monosaccharid
231 ed us to visualize the localized presence of galactosides on and around roots in unsterilized soil, a
236 de, pelargonidin-3-O-glucoside, peonidin-3-O-galactoside, peonidin-3-O-glucoside, cyanidin-3-O-xylosi
240 otes efflux and prevents accumulation of the galactoside, probably by converting the proton symporter
241 ilization during competition for mixtures of galactosides produces frequency-dependent selection at l
242 ned in the extracts from quince, quercetin-3-galactoside (Q-Ga), quercetin-3-rutinoside (Q-Ru), querc
243 de, quercetin 3-O-rutinoside, quercetin 3'-O-galactoside, quercetin 3'-O-glucoside, quercetin 3'-O-rh
244 constituents gallic acid, rutin, quercetin-3-galactoside, quercetin-3-glucoside, myricetin, quercetin
246 phism is protected only in a narrow range of galactoside ratios despite intense selection on the pure
248 lectin-binding data indicate a possible beta-galactoside-recognized protein specificity of the galact
249 ncubated with lactose (known to bind to beta-galactoside-recognized proteins) prior to the addition o
250 chloro-9,9-dimethylacridin-2-on-7-yl) beta-D-galactoside, reducing the inhibitor concentration to K(i
252 galactoside-binding lectin family that binds galactoside residues on cell surface glycoconjugates.
254 eferentially recognizes alpha-2,6 sialylated galactosides showed strong binding reactivity with undif
255 ns needed for utilization of alpha- and beta-galactosides, slowed growth on diverse carbon sources, a
257 ch control attachment of a galalpha1-4gal di-galactoside structure (lic2A and lgtC phase-on) or an al
258 n vitro to catalyze the hydrolysis of a beta-galactoside substrate 500 times more efficiently (k(cat)
259 lactose transfer from UDP-Gal to beta-linked galactosides, such as lactose, and in the absence of an
266 mbrane; (ii) LacY must be protonated to bind galactoside (the pK for binding is approximately 10.5);
268 ng sialic acid to the nonreducing end of the galactosides through a sialyltransferase-catalyzed enzym
269 ctins are a family of lectins that bind beta-galactosides through their conserved carbohydrate recogn
270 bolizable galactose analogue thiomethyl-beta-galactoside (TMG) by a permease-catalyzed sugar:H(+) sym
273 lacF lacR double mutant (lacF encodes a beta-galactoside transport protein) grown in medium containin
275 ould be transported by the overproduced beta-galactoside transporters and cause the induction of alph
276 model in which lacR mutants overproduce beta-galactoside transporters, thereby overwhelming the induc
278 an efficient route for the synthesis of beta-galactosides using a bacterial beta-4-galactosyltransfer
279 lactosides in seed wash suggested that alpha-galactoside utilization could play an important role in
280 t cause the constitutive expression of alpha-galactoside utilization genes but caused the aberrant ex
282 ransporters and cause the induction of alpha-galactoside utilization genes in the presence of both su
285 a narrow periplasmic opening and an occluded galactoside was obtained, confirming many observations a
288 , cyanidin-3-O-arabinoside, and cyanidin-3-O-galactoside were the predominant anthocyanins characteri
290 yzes the coupled stoichiometric symport of a galactoside with a cation (either Na(+), Li(+), or H(+))
291 a coli catalyzes stoichiometric symport of a galactoside with an H(+), using a mechanism in which sug
292 found in this study was m-nitrophenyl alpha-galactoside with an IC50 of 0.6 (2) mM in the LT ELISA a
294 galactose from UDP-galactose to beta-linked galactosides with retention of its alpha configuration.
296 r depectinisation, two more ACNs (cyanidin-3-galactoside-xyloside and cyanidin-3-galactoside-xyloside
297 cid as the major ACN, followed by cyanidin-3-galactoside-xyloside-glucoside-coumaric acid, and cyanid
299 anidin-3-galactoside-xyloside and cyanidin-3-galactoside-xyloside-glucoside-sinapic acid) were also i
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