ライフサイエンスコーパス: gall

1 ractions in the development of fusiform rust gall.

2 alled tissues and in tissues surrounding the gall.

3 imilation and importation of carbon into the gall.

4 resulting in the formation of characteristic galls.

5 ions with forms that feed from leaf and root galls.

6 nfected with the ubc1 mutant did not produce galls.

7 increasing drought stress tolerance of crown galls.

8 ia, a physiological condition found in crown galls.

9 SAD6-OE-RNAi or by RNA interference in crown galls.

10 genes are related pathogens that cause crown gall and hairy root diseases, which result from integrat

11 thogens that cause different diseases, crown gall and hairy root.

12 acid by bacterial pathogens that cause crown gall and related diseases.

13 eginning with those factors that cause plant galls and continuing through carbohydrate metabolism to

14 we analyzed the suberin composition of crown galls and found a reduction in the amounts of long-chain

15 lower susceptibility to the RKNs and smaller galls and GCs.

16 Both GALLS and VirE2 contain nuclear localization sequences a

17 with the galactose-inducible promoters GALS, GALL, and GAL1, allowing for low, moderate, and high lev

18 Since Arabidopsis crown galls are covered by a suberin-containing periderm inste

19 ms that these arthropods use to induce plant galls are poorly understood.

20 the organism was recovered from an infected gall bladder (Aeromonas veronii biotype veronii).

21 confirmed the presence of perforation of the gall bladder and cholecysto-cutaneous fistula.

22 entery, pancreas, portal hepatis, bile duct, gall bladder and jejunum was recorded from the right tho

23 L. monocytogenes can replicate in the murine gall bladder and provide evidence that its replication t

24 Histologic examination of the resected gall bladder and stenotic ureteric segment showed CMV in

25 d in enhanced flux of FC from macrophages to gall bladder bile and feces in vivo.

26 bile of Cel(-/-) mice, and the mass of CE in gall bladder bile was elevated.

27 ake of the tracer in the kidneys, liver, and gall bladder but rapid clearance via the urine/bladder w

28 ere readily identified in the bile ducts and gall bladder by special stains and by in situ hybridizat

29 reased risk of liver cancer, bladder cancer, gall bladder cancer, malignant lymphoma, and lung cancer

30 ial cells, the epidermis and hair follicles, gall bladder epithelium, choroid plexus, and biliary epi

31 FGF19 regulates bile acid homeostasis and gall bladder filling; FGF19 binds only to FGF receptor 4

32 port of cleavage products results in intense gall bladder fluorescence.

33 Carcinoma of the gall bladder has a guarded prognosis with predominant si

34 Osseous metastasis in carcinoma of the gall bladder is rare and hence bone scintigraphy does no

35 xpressed in embryonic septum transversum and gall bladder mesenchyme.

36 velopmental defects in lung, intestinal, and gall bladder morphogenesis.

37 r stomach, respiratory tract, bile duct, and gall bladder of B6,129 CYP1A2-null and wild-type mice as

38 d spleen towards the left, and the liver and gall bladder on the right.

39 e animals, revealing strong signals from the gall bladder over a period of several days, in diseased

40 The ultrasound findings included gall bladder wall thickening in 66 patients (41.2%).

41 The present study suggests that increased gall bladder wall thickness, pleural effusion, ascites,

42 In addition, the gall bladder was absent and the extrahepatic bile duct c

43 istula with its opening in the fundus of the gall bladder was revealed.

44 describe two patients with carcinoma of the gall bladder with osteolytic metastasis (stage 4).

45 Parasites are present in the gut, gall bladder, and biliary tree, and biliary epithelial c

46 d from foregut endoderm such as lung, liver, gall bladder, and pancreas.

47 l for the development of the mouse pancreas, gall bladder, and the interhepatic bile ducts.

48 led metabolites accumulates in the liver and gall bladder, consistent with the known routes of excret

49 ed a more selective pattern of expression in gall bladder, intestine, brain, ovary, spleen, and thymu

50 ts are solubilized by bile released from the gall bladder, resulting in the formation of two product

51 L. monocytogenes may be carried in the human gall bladder.

52 ng mirror-image reversals of heart, gut, and gall bladder.

53 protein expressed by the human intestine and gall bladder.

54 scan or PET CT in cases of carcinoma of the gall bladder.

55 copy to rule out Veress needle injury to the gall bladder.

56 e canaliculi, and subsequently stored in the gall bladder.

57 onse: 11 with bile duct cancer and four with gall-bladder carcinoma.

58 Foxf1+/- gall bladders were significantly smaller and had severe

59 mutant for tumor formation, indicating that GALLS can substitute for VirE2.

60 sulting in the development of spindle-shaped galls (cankers) on branches or stems.

61 ategories (i.e. oak, grape seed, grape skin, gall, chestnut, quebracho, tea and acacia).

62 Unlike VirE2, GALLS contains a nucleoside triphosphate binding motif s

63 Unlike VirE2, GALLS contains ATP-binding and helicase motifs similar t

64 The presence of a gall cost a ramet an average of 1743 seeds, but the cost

65 Coral-dwelling gall crabs (Cryptochiridae) are obligate symbionts of st

66 , triggering morphogenetic changes to induce galls, de novo formed 'pseudo-organs' containing several

67 Our results show that gall development involved the amplification of existing

68 t whether miRNAs play roles in fusiform rust gall development, we cloned and identified 26 miRNAs fro

69 T plays an important role in correct GCs and gall development, where miRNA172 is modulated by auxins.

70 the lipid transfer protein AtLTPI-4 in crown gall development.

71 iral N gene response against bacterial crown gall disease and highlight the importance of achieving t

72 systemic acquired resistance (SAR) on crown gall disease caused by Agrobacterium tumefaciens.

73 um plants that are highly resistant to crown gall disease development.

74 ns is a soilborne pathogen that causes crown gall disease in many dicotyledonous plants by transfer o

75 8, the pathogenic bacteria that causes crown gall disease in plants, harbors one circular and one lin

76 m tumefaciens, the causative agent for crown gall disease of plants has proven a productive model for

77 imary virulence factor responsible for crown gall disease of plants.

78 Crown gall disease, caused by the soil bacterium Agrobacterium

79 its own DNA into host plants to cause Crown Gall disease.

80 method to produce plants resistant to crown gall disease.

81 e variation in their susceptibility to crown gall disease.

82 itro, how whitefly infestation affects crown gall disease.

83 Agrobacterium tumefaciens causes crown gall disease.

84 nt pathogen and the causative agent of crown gall disease.

85 pathogenic bacterium that induces the 'crown gall' disease in plants by transfer and integration of a

86 The gall-dwelling colonies of a social aphid species (Pemphi

87 e plant KRP6 transcription to the benefit of gall establishment.

88 In this issue of Immunity, Gall et al. characterize, in a murine model of autoimmun

89 In the second study, by Le Gall et al., the modulation of epitope immunodominance a

90 Using the protocol of Gall et al., we developed a robust methodology for ampli

91 es two proteins from one open reading frame: GALLS-FL and a protein comprised of the C-terminal domai

92 VirD2 interacted with GALLS-FL and localized inside the nucleus, where its pre

93 GALLS-FL and VirE2 contain nuclear localization signals

94 GALLS-FL tagged with yellow fluorescent protein localize

95 Unlike VirE2, full-length GALLS (GALLS-FL) contains ATP-binding and helicase motifs simil

96 s from a specialist herbivore, the goldenrod gall fly (Eurosta solidaginis).

97 e E,S-conophthorin produced by the goldenrod gall fly as the specific chemical component that elicits

98 cheal cells of the freeze-tolerant goldenrod gall fly, Eurosta solidaginis, chilling to 0 degrees C e

99 Gall formation also perturbed vascular development with

100 ance and differentiation, that a decrease in gall formation did not prevent pathogen development.

101 insect infestation on Agrobacterium induced gall formation has not been investigated.

102 we explored the cellular events that underly gall formation in Arabidopsis thaliana with the help of

103 r salicylic acid (SA) synthesis, compromised gall formation indicating an involvement of SA in whitef

104 This finding demonstrates that although gall formation is a typical symptom of the disease and i

105 s exhibited at least a two-fold reduction in gall formation on both stem and crown root.

106 t ultimately dominate and be responsible for gall formation.

107 ents responsible for arthropod-induced plant gall formation.

108 ion has in the study of insect-induced plant gall formation.

109 ells in a meristematic state was crucial for gall formation; disruption of the VC activity significan

110 Cross-sections through galls formed by feeding nematodes on rme1 roots were ide

111 Mostly all fusiform rust galls formed under field conditions are produced as a re

112 in ways that appear to directly benefit the gall former.

113 Across both gall-former species we find consistent differences in bo

114 A gall-forming aphid has an extended post-reproductive lif

115 Here we report the gall-forming aphid-like parasite phylloxera, Daktulospha

116 underlie the distributions of 10 species of gall-forming arthropods and their ability to adapt to ne

117 Gall-forming arthropods are highly specialized herbivore

118 ver, the lack of parallel genetic studies on gall-forming arthropods limits our ability to define the

119 Endoparasitism by gall-forming insects dramatically alters the plant pheno

120 ilarity strongly affect the distributions of gall-forming species, individually and as a community.

121 ed with wild-type cells that often developed galls from initially chlorotic tissue, plants infected w

122 Unlike VirE2, full-length GALLS (GALLS-FL) contains ATP-binding and helicase motif

123 ive gene repression events observed in early gall/GCs development are thought to be mediated by post-

124 ::GUS showed restricted promoter activity in galls/GCs that was regulated by auxins through auxin-res

125 Here, we show that the GALLS gene encodes two proteins from one open reading fr

126 of these domains abolish the ability of the GALLS gene to substitute for virE2.

127 The GALLS gene was essential for pathogenicity of A. rhizoge

128 ey transfer T strands efficiently due to the GALLS gene, which complements an A. tumefaciens virE2 mu

129 tion, and defense investment in favor of the galling habit.

130 oxidizes l-Gal to l-galactono-1,4-lactone (l-GalL), has been purified from pea seedlings and cloned f

131 r and histological features of these petiole galls have been preserved in exquisite detail, including

132 t occurrence of external foliage-feeding and galling in the terrestrial fossil record.

133 te plants remarkably decreased the number of galls in transformed hairy roots inoculated with RKN.

134 ctant of a closely associated herbivore, the gall-inducing fly Eurosta solidaginis, exhibit enhanced

135 f tall goldenrod (Solidago altissima) to the gall-inducing tephritid Eurosta solidaginis.

136 ic and kill E. solidaginis hatchlings before gall induction.

137 ow that the main colorant of historical iron gall ink (IGI) is an amorphous form of Fe(III) gallate.x

138 tilized TERS to identify indigo dye and iron gall ink in situ on Kinwashi paper.

139 In addition, TERS was used to identify iron gall ink on a historical document with handwritten text

140 mponents of a complex chemical mixture, iron gall ink, can be identified.

141 een growth and terpenoids manifested through galling insects supported the GDBH.

142 optera: Cynipidae) feed within inconspicuous galls inside the flowering stems of the prairie perennia

143 l-GalL is a proposed substrate for ascorbate biosynthesis

144 Because the only known function of l-GalL is ascorbate synthesis, these antisense plants prov

145 Notably, chewing, sucking and gall-making herbivores were more affected by top-down th

146 ; Diptera: Cecidomyiidae), a plant parasitic gall midge and a pest of wheat (Triticum spp.), with the

147 cally important gall midge species, the rice gall midge and the Hessian fly, with their host plants,

148 ibility gene for infestation of wheat by the gall midge M. destructor, commonly known as the Hessian

149 The interaction of a third gall midge species, the orange wheat blossom midge, with

150 e interactions of two economically important gall midge species, the rice gall midge and the Hessian

151 lly anchored genetic map is the first of any gall midge species.

152 Mayetiola destructor), the most investigated gall midge, was the first insect hypothesized to have a

153 netics underlying important aspects of these gall midge-grass interactions, a unique opportunity exis

154 Gall midges constitute an important group of plant-paras

155 Gall midges induce formation of host nutritive cells and

156 e discoveries suggest that the HF, and other gall midges, may be considered biotrophic, or hemibiotro

157 eat worldwide, and an emerging model for all gall midges, we investigated its antioxidant responses d

158 group, the Eriophyoidea, which includes the gall mites and comprises at least 3,500 Recent species,

159 Antiquity of the gall mites in much their extant form was unexpected, par

160 According to the Gall model, 37.4 breast cancers were expected in the mod

161 find consistent differences in body size and gall morphology associated with host plant use, as well

162 Agrobacterium tumefaciens-derived crown galls of Arabidopsis (Arabidopsis thaliana) contain elev

163 s, analysis of Meloidogyne incognita-induced galls of KRP6-overexpressing lines revealed a role for t

164 n rme1 roots were identical to sections from galls of susceptible tomato roots.

165 strain C58, highly expressed AtLTPI-4 Crown galls of the atltpI-4 loss-of-function mutant were much

166 Drosophila tracheal system, mutations in oak gall (okg) and conjoined (cnj) confer identical defects,

167 hly complex species- and generation-specific galls on oaks and other Fagaceae.

168 le from chrysopine, a newly discovered crown gall opine.

169 and expressed in plant cells, causing crown gall or hairy root disease.

170 but fewer nodules and nematode-induced root galls per plant, than control hairy roots.

171 The GALLS protein can complement an A. tumefaciens virE2 mut

172 Instead, these bacteria express the GALLS protein, which is essential for their virulence.

173 In plant cells, the GALLS proteins interacted with themselves, VirD2, and ea

174 On some hosts, both GALLS proteins were required to substitute for VirE2.

175 different botanical sources (oak, chestnut, gall, quebracho, tea, grape skin and grape seed) were co

176 Many are found in cryptic habitats such as galls, several widespread genera are surface feeders on

177 crescentic pit, circular-oval pit, or a true gall) shows that species within crab genera tend to inha

178 the VC activity significantly decreased the gall size.

179 e (Sternorrhyncha: Psyllidae), the commonest galling species associated with B. dracunculifolia, in 1

180 families was significantly repressed in the galled stem.

181 um stems but strongly differed from 50:50 in galled stems, with "+" and "-" enantiomers strongly domi

182 nes in enantiomeric ratios characteristic of galled stems.

183 ervation, cholecystitis and complications of gall stones such as pancreatitis, and ovarian diseases.

184 Despite their lack of similarity, GALLS substituted for VirE2.

185 The galls support species-rich, closed communities of inquil

186 n years ago) a larva of the Holometabola was galling the internal tissue of Psaronius tree-fern frond

187 f SAD6 with fatty acid desaturation in crown galls, the lipid pattern was analyzed of plants with con

188 ycolysis; and fermentation increased in leaf-gall tissues.

189 rns of these miRNAs and their targets in the galled tissues and in tissues surrounding the gall.

190 ny of these domains abolished the ability of GALLS to substitute for VirE2.

191 We assess the importance of gall traits in structuring oak cynipid communities and s

192 rimary metabolism, we also characterized the gall transcriptome to infer the level of global reconfig

193 or the synthesis of mannopine (MOP) by crown gall tumor cells, MocC is essential for the utilization

194 dies on the temperature sensitivity of crown gall tumor development on plants.

195 ntrol the infection process leading to crown gall tumor disease on susceptible plants.

196 erium Agrobacterium tumefaciens causes crown gall tumor formation in plants.

197 An early step in crown gall tumor formation involves the attachment of Agrobact

198 Isolate Rr 2-17, from a grapevine crown gall tumor, is a member of the Novosphingobium genus tha

199 segment of DNA to a host plant, generating a gall tumor.

200 a gene of the plasmid pSa can suppress crown gall tumorigenesis incited by Agrobacterium tumefaciens.

201 During the process of crown gall tumorigenesis, Agrobacterium tumefaciens transfers

202 cient, but the ecotype is deficient in crown gall tumorigenesis, transformation to kanamycin resistan

203 DNA or T-DNA) into plant cells during crown gall tumorigenesis.

204 th pJW323 and pTiA6, the initiation of crown gall tumors (i.e., T-DNA transfer) is greatly suppressed

205 host pathogens, the causative agent of crown gall tumors Agrobacterium tumefaciens and the parasitic

206 olved in the biosynthesis of opines in crown gall tumors are always matched by Ti plasmid genes confe

207 The formation of crown gall tumors by Agrobacterium tumefaciens requires that t

208 Agrobacterium tumefaciens induces crown gall tumors by transferring a piece of its tumor-inducin

209 ciens is a plant pathogen that incites crown gall tumors by transferring to and expressing a portion

210 by the conjugative opines produced by crown gall tumors induced on plants by the bacteria.

211 al, a subset of the opines produced by crown gall tumors initiated on plants by the pathogen, serves

212 Agrobacterium tumefaciens induces crown gall tumors on plants by transferring a nucleoprotein co

213 Agrobacterium tumefaciens causes crown gall tumors on various plants by delivering transferred

214 small carbon compounds produced by the crown gall tumors that are induced by the bacteria.

215 Arabidopsis (Arabidopsis thaliana) crown gall tumors, which develop upon infection with the virul

216 to octopine, a nutrient released from crown gall tumors.

217 f Agrobacterium tumefaciens that cause crown gall tumors.

218 to octopine, a nutrient released from crown gall tumors.

219 responsible for opine biosynthesis in crown gall tumors.

220 sible for biosynthesis of mannopine in crown gall tumors.

221 plant genome, ultimately resulting in crown gall tumour formation.

222 ium tumefaciens is well known to cause crown gall tumours at plant wound sites and to benefit from th

223 se to octopine, an opine released from crown gall tumours, and is also positively autoregulated by Tr

224 n which opines, substrates produced by crown gall tumours, induce a quorum-sensing system.

225 s called opines that are released from crown gall tumours.

226 ve been determined for ded1, trp3, rps4, and gall under a variety of growth conditions.

227 e levels of unsaturated fatty acids in crown galls under hypoxia and drought stress conditions.

228 thesis developed significantly smaller crown galls under normal, but not under high, relative humidit

229 The development of crown galls was not affected either in SAD6-OE or SAD6-OE-RNAi

230 Larvae of the gall wasp Antistrophus rufus Gillette (Hymenoptera: Cyni

231 Oak gall wasps (Hymenoptera: Cynipidae, Cynipini) are charac

232 thin stems in a complex prairie habitat, and gall wasps themselves apparently influence the plant to

233 ics of the interactions between host plants, gall wasps, and natural enemies.

234 ciated populations of two species of cynipid gall wasps, Belonocnema treatae and Disholcaspis quercus

235 adequate means do not exist to control crown gall, we created resistant plants by introducing transge

236 Leaf galling, which is negatively correlated with moisture to

237 In crown galls, which endogenously express AtLtpI-4, it is involv

238 IAA induced their activity in galls while PEO-IAA treatment and mutations in AuxRe mot

239 tors in my life, including my father and Joe Gall, who is my "Doktor Vater." In turn, as an establish

240 th to those of female eggs, yet emerged from galls with shorter pedicels than those of female wasps.