ライフサイエンスコーパス: gallate

1 he abundance of EGC, theanine and afzelechin gallate.

2 related with epicatechin and epicatechin-3-O-gallate.

3 hin and epicatechin, but not epicatechin-3-O-gallate.

4 industry, BHT, alpha-tocopherol, and dodecyl gallate.

5 constant, followed by (-)-epigallocatechin-3-gallate.

6 redox-active cofactors such as ascorbate and gallate.

7 ls, epigallocatechin gallate and epicatechin gallate.

8 the antioxidants ascorbic acid and n-propyl gallate.

9 some EGCG epimerized to (+)-gallocatechin-3-gallate.

10 ed similar amounts of (-)-epigallocatechin-3-gallate.

11 ld be totally inhibited by 200 microm propyl gallate.

12 eviously to be better surfactants than alkyl gallates.

13 In addition, about 130 genes in the large gallate 1-beta-glucosyltransferase (GGT) superfamily wer

14 he green tea extract, (-)-epigallocatechin-3-gallate [(2R,3R)-5,7-dihydroxy-2-(3,4,5-trihydroxyphenyl

15 tea-derived flavanol, (-)-epigallocatechin 3-gallate [(2R,3R)-5,7-dihydroxy-2-(3,4,5-trihydroxyphenyl

16 pigallocatechin (2) and epigallocatechin-3-O-gallate (3).

17 nalysis using luteolin (12) and (-)-catechin gallate (37) as model compounds revealed that FabG was i

18 abundant being gallic acid, (-)-epicatechin-gallate, 4-p-coumaroylquinic acid, quercetin-3-O-galacto

19 uced higher decrease in (-)-epigallocatechin gallate (71% and 79%) and quercetin-3-glucoside (29% and

20 (1475-2,070 mug/g extracts) and epicatechin gallate (885-1,603 mug/g extracts); while all-trans-lute

21 We recently discovered that ethyl gallate, a nonflavonoid phenolic antioxidant found in fo

22 n tea extracts containing epigallocatechin 3-gallate, a potent DYRK1A inhibitor, to adult mBACtgDyrk1

23 Epigallocatechin-3-gallate, a potent Mnb/Dyrk1A inhibitor in vitro, apparen

24 recently showed that (-)-epigallocatechin-3-gallate, a tea polyphenol, not only inhibits L-DOPA meth

25 istration of dexamethasone, epigallocatechin gallate, alpha-phenyl-N-tert-butyl nitrone, and ebselen

26 Catechins [(-)-epigallocatechin gallate and (-)-epicatechin gallate)] were most efficien

27 -)-epicatechin gallate, (-)-epigallocatechin gallate and (-)-epigallocatechin protected at lowest con

28 high binding energies for (-)-epicatechin-3-gallate and (-)-epigallocatechin-3-gallate for the bindi

29 ddition of the antioxidants epigallocatechin gallate and alpha-lipoic acid reduces polyglutamine aggr

30 lavan-3-ols, most of which are gallocatechin gallate and catechin.

31 the green tea polyphenols, epigallocatechin gallate and epicatechin gallate.

32 verages were incubated with epigallocatechin gallate and green tea extract at 62 or 37 degrees C for

33 Interestingly, whereas epigallocatechin gallate and heparin prevent membrane damage as judged by

34 a oligomers is inhibited by epigallocatechin gallate and increased by the A2V mutation, previously re

35 2,3-trihydroxybenzene moiety, such as methyl gallate and myricetin.

36 We found that in both protocols, ethyl gallate and norepinephrine improved mean arterial pressu

37 pplementation of combined epigallocatechin-3-gallate and resveratrol (EGCG+RES) increased energy expe

38 by two amyloid inhibitors (epigallocatechin gallate and scyllo-inositol) that are currently in clini

39 he green tea polyphenol (-)-epigallocatechin gallate and the red wine extract resveratrol both remode

40 , (+)-catechin, gallocatechin, gallocatechin gallate and theasinensin B, and lower levels of (-)-epic

41 , (+)-catechin, gallocatechin, gallocatechin gallate and theasinensin B, and lower levels of (-)-epic

42 ively inhibited by 500 microm or 1 mm propyl gallate and to a lesser extent by 5 mm aminooxyacetate,

43 ely (p<0.05) correlated with (-)-epicatechin gallate and total phenolic contents.

44 nti-peroxide activity of gallic acid, methyl gallate, and alpha-tocopherol in a bulk Kilka fish oil a

45 ( approximately 100%), for Trolox, n-propyl gallate, and ascorbic acid the extent of geminate recomb

46 ecially epigallocatechin galate, epicatechin gallate, and epicatechin) than tea infused at 80 degrees

47 n hazelnut shells were catechin, epicatechin gallate, and gallic acid, as quantified by high performa

48 including (+)-catechin, (-)-epigallocatechin gallate, and green tea extract were added to washed MFGs

49 (TFdiG), theaflavin-3'-gallate, theaflavin-3-gallate, and theaflavin inhibited PL with IC50 of 1.9, 4

50 aflavin, theaflavin-3-gallate, theaflavin-3'-gallate, and theaflavin-3,3'-digallate (TFdiG), were com

51 ration containing 65% (-)-epigallocatechin-3-gallate] and caffeine on 4-(methylnitrosamino)-1-(3-pyri

52 nd of (-)-epicatechin and (-)-epicatechin-3O-gallate as extension units (positive correlation).

53 We identified (-)-epigallocatechin 3-O:-gallate, as a potent inhibitor of religation by mammalia

54 tannins [procyanidin B2, procyanidin B2 3'-O-gallate (B2g) and procyanidin trimer (catechin-4-8-catec

55 ent- and bitter-tasting (-)-epigallocatechin gallate, bitter-tasting caffeine, and the umami-tasting

56 r the major tea component epigallocatechin-3-gallate blocked expression of the hyper-, but not hypoph

57 ts reveal that polyphenols (epigallocatechin gallate, bromophenol blue, and resveratrol) and glycosam

58 ctivity of gallic acid and a series of alkyl gallates (C4-C18) and glycosylated alkyl gallates (C4-C1

59 kyl gallates (C4-C18) and glycosylated alkyl gallates (C4-C18) on fish oil-in-water emulsions was stu

60 Medium-size alkyl gallates (C6-C12) were the best antioxidants.

61 compounds, such as gallic acid, epicatechin gallate, catechin, epicatechin and isoquercitrin, were i

62 chin (C), (-)-epicatechin (EC), (-)-catechin gallate (CG), and (-)-epicatechin gallate (ECG) in grape

63 n (C), methyl gallate (MG), and catechin-3-O-gallate (CG).

64 more active (IC50 = 31 microM) than catechin gallate (CG, IC50 = 53 microM) or epicatechin gallate (E

65 uclear perfluoroarylborate, -aluminate, and -gallate cocatalysts/activators B(C6F5)3 (3), B(o-C6F5C6F

66 The gallate degradation pathway ofPseudomonas putidaKT2440 r

67 l)-cyclopropenium tetrakis(pentafluorophenyl)gallate (DPMNCPGa) (Chart 1) have been investigated in a

68 othiocyanates; G) and (-)-epigallocatechin-3-gallate (E) were investigated in colon (LoVo and CaCo-2)

69 (EC), epigallocatechin (EGC), epicatechin 3-gallate (ECG) and epigallocatechin 3-gallate (EGCG) and

70 (-)-Epicatechin-3-gallate (ECG) functioned similar to EGCG by completely b

71 )-catechin gallate (CG), and (-)-epicatechin gallate (ECG) in grape products were successfully quanti

72 gallocatechin gallate (EGCG) and epicatechin gallate (ECG)) inhibit GDH in vitro and that EGCG blocks

73 zodioxol-5-amine (MSNBA) and (-)-epicatechin-gallate (ECG).

74 allate (CG, IC50 = 53 microM) or epicatechin gallate (ECG, IC50 = 76 microM) against the colorectal a

75 n C, (-)-epicatechin EC, (-)-epicatechin-3-O-gallate, ECG] and oligomers [B1, B2, B3, B4 dimers and t

76 Epigallocatechin-3-gallate (EGCG) accounts for almost 50% of the total cate

77 in, (-)-epicatechin, or (-)-epigallocatechin gallate (EGCG) added as chain breakers.

78 on conferred by sugars to epigallocatechin-3-gallate (EGCG) against deterioration.

79 , ACS1, and the antioxidant epigallocatechin gallate (EGCG) all stimulated GSH levels and significant

80 llocatechin (EGC) and (-)-epigallocatechin-3-gallate (EGCG) and butylated hydroxytoluene (BHT) correl

81 echin 3-gallate (ECG) and epigallocatechin 3-gallate (EGCG) and caffeine in 29 commercial green tea s

82 ituents of green tea, (-)-epigallocatechin-3-gallate (EGCG) and caffeine, on intestinal tumorigenesis

83 its aggregation, such as epigallocatechin-3-gallate (EGCG) and dopamine (DA).

84 polyphenols from green tea (epigallocatechin gallate (EGCG) and epicatechin gallate (ECG)) inhibit GD

85 the four compounds tested, epigallocatechin gallate (EGCG) and epicatechin gallate were found to inh

86 tituents, in particular (-)-epigallocatechin gallate (EGCG) and other polyphenols with 1,2-benzenedio

87 the polyphenolic compounds epigallocatechin gallate (EGCG) and silibinin bind to specific conformers

88 tea catechins such as (-)epigallocatechin-3-gallate (EGCG) are known to improve energy metabolism at

89 -unexplored property of (-)-epigallocatechin gallate (EGCG) as a chiral solvating agent for enantiodi

90 Here, we identify (-)-epigallocatechin-3-gallate (EGCG) as a new inhibitor of hepatitis C virus (

91 The STAT1 inhibitor epigallocatechin gallate (EGCG) attenuated STAT1 phosphorylation in cispl

92 est green tea-derived (-)-epigallocatechin-3-gallate (EGCG) can attenuate neuronal damage mediated by

93 an ingredient of green tea, epigallocatechin gallate (EGCG) could attenuate oxidative stress-induced

94 Green tea polyphenol, epigallocatechin-3-gallate (EGCG) differentially regulates the cellular gro

95 (-)-Epigallocatechin gallate (EGCG) effectively reduces the cytotoxicity of t

96 green tea polyphenol (-)-epigallocatechin-3-gallate (EGCG) exerts a beneficial role on reducing brai

97 It was reported that epigallocatechin-3-gallate (EGCG) exhibits anti-inflammatory properties, bu

98 hat prostate tumor specific epigallocatechin-gallate (EGCg) functionalized radioactive gold nanoparti

99 -Gly and the polyphenol (-)-epigallocatechin gallate (EGCG) has been determined using time-averaged n

100 ea-derived polyphenol (-)-epigallocatechin-3-gallate (EGCG) has been extensively studied for its anti

101 The flavonoid epigallocatechin gallate (EGCG) has previously been shown to redirect the

102 Green tea constituent (-) epigallocatechin-3-gallate (EGCG) has shown remarkable cancer-preventive an

103 biological effects of (-)-epigallocatechin-3-gallate (EGCG) have been extensively investigated in cel

104 etinamide (4-HPR) and (-)-epigallocatechin-3-gallate (EGCG) in altering expression of oncogenic micro

105 The polyphenol epigallocatechin-3-gallate (EGCG) in combination with doxorubicin (Dox) exh

106 evaluate the efficacy of epigallocatechin-3-gallate (EGCG) in down-regulating Mcl-1 expression and i

107 tive botanicals including epigallocatechin-3-gallate (EGCG) in green tea polyphenols (GTPs) and sulfo

108 ated green tea polyphenol epigallocatechin-3-gallate (EGCG) in polylactic acid-polyethylene glycol na

109 n of green tea catechin and epigallocatechin gallate (EGCG) in soy lecithin liposomes was examined at

110 techins epicatechin (EC) or epigallocatechin gallate (EGCG) inhibited formation of highly reactive in

111 the green tea polyphenol epigallocatechin-3 gallate (EGCG) inhibits growth and transformed phenotype

112 tive green tea polyphenol epigallocatechin-3-gallate (EGCG) inhibits growth in soft agar of breast ca

113 the green tea polyphenol epigallocatechin 3-gallate (EGCG) inhibits growth of NF639 Her-2/neu-driven

114 Epigallocatechin gallate (EGCg) is a major form of tea catechin and has a

115 Epigallocatechin gallate (EGCG) is a major polyphenol in green tea that h

116 that the commonly used (-)-epigallocatechin gallate (EGCG) is a much less efficient amyloid inhibito

117 Epigallocatechin gallate (EGCG) is a powerful antioxidant and commonly us

118 (-)-Epigallocatechin-3-gallate (EGCG) is a well-known chemoprevention factor.

119 (-)-Epigallocatechin-3-gallate (EGCG) is an important bioactive constituent of

120 ated that the GT polyphenol epigallocatechin gallate (EGCG) is capable of antagonizing AhR-mediated g

121 gents like the polyphenol epigallocatechin-3-gallate (EGCG) is emerging as an experimental secondary

122 The green tea catechin epigallocatechin-3-gallate (EGCG) is generally considered to be the biologi

123 Epigallocatechin gallate (EGCG) is often described as the most potently c

124 (-)-Epigallocatechin gallate (EGCG) is one of the major green tea catechins t

125 Epigallocatechin gallate (EGCG) is the major active polyphenol in green t

126 Epigallocatechin gallate (EGCG) is the major component of green tea extra

127 Epigallocatechin-3-gallate (EGCG) is the major polyphenol in green tea.

128 Epigallocatechin-3-gallate (EGCG) is the most biologically potent polypheno

129 The green tea component epigallocatechin-3-gallate (EGCG) may be beneficial in autoimmune diseases;

130 TE) supplementation high in epigallocatechin gallate (EGCG) on blood lipids in healthy postmenopausal

131 effect of the polyphenol epigallocatechin 3-gallate (EGCG) on hCT fibrillation was also investigated

132 lyphenols, the effects of epigallocatechin-3 gallate (EGCG) on Her-2/neu-overexpressing breast cancer

133 the green tea polyphenol epigallocatechin-3 gallate (EGCG) on mammary tumor cells were assessed.

134 esveratrol, curcumin, and epigallocatechin-3-gallate (EGCG) on the aggregation of Abeta(17-36) peptid

135 yphenolic amyloid inhibitor epigallocatechin gallate (EGCG) on the aggregation pathway.

136 s were incubated with (-)-epigallocatechin-3-gallate (EGCG) or theaflavin-3,3'-digallate (TFdiG) (20

137 400muM) and influence (-)-epigallocatechin-3-gallate (EGCG) oxidation (400muM) in Tween- or sodium do

138 olyphenol constituent (-)-epigallocatechin-3-gallate (EGCG) possess potent neuroprotective activity i

139 green tea shoot (GL) while epigallocatechin gallate (EGCG) recorded higher levels in GL.

140 In contrast, epigallocatechin-3-gallate (EGCG) signals ECs via the 67 kDa laminin-recept

141 th a standardized dose of epigallocatechin-3-gallate (EGCG) was administered using the standard phase

142 Of the various GrTPs, (-)-epigallocatechin-3-gallate (EGCG) was the most potent inhibitor.

143 f the catechins, especially epigallocatechin gallate (EGCG) were found in green teas.

144 mponent in green tea, (-)-epigallocatechin-3-gallate (EGCG)'s potential benefits to human health have

145 yphenols (catechins), (-)-epigallocatechin-3-gallate (EGCG), (-)-epigallocatechin, (-)-epicatechin-3-

146 vestigated the effects of epigallocatechin-3-gallate (EGCG), a bioactive green tea polyphenol, on EGF

147 Epigallocatechin gallate (EGCG), a bioactive polyphenol in green tea, mim

148 (-)-Epigallocatechin-3-gallate (EGCG), a catechin found in green tea, has been

149 ine whether the antioxidant epigallocatechin gallate (EGCG), a catechin-base flavonoid derived from g

150 was to provide support for epigallocatechin gallate (EGCG), a component of green tea, to be consider

151 nd in vivo treatment with Epigallocatechin-3-gallate (EGCG), a Dyrk1a inhibitor, modulated trisomic N

152 uded three phenolics: (-)-epigallocatechin-3-gallate (EGCG), a flavanoid polyphenol found in green te

153 orted previously that (-)-epigallocatechin-3-gallate (EGCG), a green tea polyphenol antioxidant, stim

154 we examined the effect of epigallocatechin-3-gallate (EGCG), a green tea polyphenol, on IL-1beta-indu

155 entified in this study that epigallocatechin gallate (EGCG), a green tea-derived catechin, acts as a

156 we studied the impact of epigallocatechin-3-gallate (EGCG), a known receptor tyrosine kinase (RTK) i

157 Epigallocatechin-3-gallate (EGCG), a main catechin of green tea, has been s

158 (-)-Epigallocatechin gallate (EGCG), a major biologically active constituent

159 ed that the antioxidant (-)-epigallocatechin gallate (EGCG), a major component in green tea extracts,

160 (-)-Epigallocatechin-3-gallate (EGCG), a major component of green tea, protects

161 t studies have shown that epigallocatechin-3-gallate (EGCG), a major constituent of green tea extract

162 Epigallocatechin gallate (EGCg), a major form of tea catechins, has a var

163 Epigallocatechin gallate (EGCG), a major form of tea catechins, possesses

164 (-)-Epigallocatechin-3-O-gallate (EGCG), a major ingredient of green tea, has bee

165 (-)-Epigallocatechin-3-gallate (EGCG), a major polyphenol component of green te

166 Epigallocatechin-3-gallate (EGCG), a polyphenol extracted from green tea, i

167 are mainly attributed to epigallocatechin-3-gallate (EGCG), a polyphenolic compound from the group o

168 Treatment with epigallocatechin-3-gallate (EGCG), a polyphenolic compound of green tea, re

169 ermine to what extent (-)-epigallocatechin-3-gallate (EGCG), a widely used dietary supplement, modula

170 We found that epigallocatechin-3-gallate (EGCG), an anti-inflammatory compound found in g

171 s, and the polyphenol (-)-epigallocatechin-3-gallate (EGCG), and has been implicated in a number of d

172 f a model polyphenol, (-)-epigallocatechin-3-gallate (EGCG), and matrix pH (2-7) on the net anti-/pro

173 cysteine (NAC), curcumin, epigallocatechin-3 gallate (EGCG), and p38 inhibitor SB203580 were added to

174 of curcumin, caffeine, (-)-epigallocatechin gallate (EGCG), and tea in animal models of carcinogenes

175 ajor green tea catechin, epigallo-catechin 3-gallate (EGCG), and the major dietary protein and allerg

176 r green tea catechin, (-)-epigallocatechin-3-gallate (EGCG), but effects were observed only at microm

177 green tea polyphenol, (-)-epigallocatechin 3-gallate (EGCG), exerts potent neuroprotective actions in

178 hibitor, green tea flavonol epigallocatechin-gallate (EGCG), from gestation to adulthood suppressed 3

179 id, caffeine, curcumin, (-)-epigallocatechin gallate (EGCG), gallic acid, propyl gallate, resveratrol

180 nin, baicalein, curcumin, epigallocatechin 3-gallate (EGCG), genistein, quercetin, and resveratrol bo

181 tea extracts, catechin (-)-Epigallocatechin gallate (EGCg), has been reported to be biologically act

182 tuent of green tea, (-)-epigallocatechin-3-O-gallate (EGCG), has been shown to have cancer-preventive

183 nd the green tea flavonoid, epigallocatechin gallate (EGCG), is reported to have glucose-lowering eff

184 molecules, FK506 and (-)-epigallocatechin-3-gallate (EGCG), known to inhibit alphaSYN fibril formati

185 d its major catechin, (-)-epigallocatechin-3-gallate (EGCG), on collagen homeostasis in keloid fibrob

186 yphenolic constituent (-)-epigallocatechin-3-gallate (EGCG), possesses remarkable cancer chemoprevent

187 polyphenols, such as (-)-epigallocatechin-3-gallate (EGCG), potently and specifically inhibit the ch

188 their active ingredient, epigallocatechin-3-gallate (EGCG), protects cells from subsequent OGD/R-ind

189 gallocatechin (EGC) and (-)-epigallocatechin gallate (EGCG), respectively.

190 gallocatechin (EGC) and (-)-epigallocatechin gallate (EGCG), respectively.

191 Epigallocatechin gallate (EGCg), the main antimicrobial tea catechin, has

192 we evaluated the effect of epigallocatechin gallate (EGCG), the main antioxidant derived from green

193 It was hypothesized that epigallocatechin-gallate (EGCG), the main catechin present in green tea,

194 jection (20 mg/kg) of (-)-epigallocatechin-3-gallate (EGCG), the main polyphenolic constituent of gre

195 Here, we report that (-)-epigallocatechin-3-gallate (EGCG), the main polyphenolic constituent of gre

196 orted previously that (-)-epigallocatechin-3-gallate (EGCG), the major and most active component in g

197 Epigallocatechin-3-gallate (EGCG), the major constituent of green tea, poss

198 Epigallocatechin 3-gallate (EGCG), the major polyphenol found in green tea,

199 We report herein that (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol from green tea, can

200 Epigallocatechin gallate (EGCG), the major polyphenol in green tea and th

201 he effect of a pure form of epigallocatechin gallate (EGCG), the major polyphenol in green tea, in mi

202 we provide evidence that epigallocatechin-3-gallate (EGCG), the major polyphenol in green tea, is a

203 opical application of (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol of green tea, preve

204 studies indicate that (-)-epigallocatechin-3-gallate (EGCG), the major polyphenol present in green te

205 t for the first time that epigallocatechin-3-gallate (EGCG), the major polyphenolic constituent of gr

206 Epigallocatechin-3-gallate (EGCG), the major tea polyphenolic compound, is

207 Epigallocatechin-3-gallate (EGCG), the most abundant GTP, displays antiprol

208 (-)-Epigallocatechin-3-gallate (EGCG), the principal polyphenol in green tea, h

209 Epigallocatechin-3-gallate (EGCG), the principal polyphenol isolated from g

210 Epigallocatechin-3-gallate (EGCG), the principal polyphenol present in gree

211 tea flavan-3-ol substrates, epigallocatechin gallate (EGCG), was employed.

212 polyphenols, curcumin and epigallocatechin-3-gallate (EGCG), with that by the endogenous substrate he

213 described polyphenol, (-)-epigallocatechin-3-gallate (EGCG).

214 isothiocyanate (BITC), and epigallocatechin gallate (EGCG).

215 yphenolic constituent (-) epigallocatechin-3-gallate (EGCG).

216 ng site of SULT1A1 bound to epigallocatechin gallate (EGCG).

217 n, epicatechin, and (-)-epigallocatechin-3-O-gallate (EGCG)] and bioflavonoids (quercetin, fisetin, a

218 feine (6.2 micromol) or (-)-epigallocatechin gallate (EGCG; 6.5 micromol) once a day 5 days a week fo

219 tamin E analog trolox and epigallocatechin-3-gallate, enhanced hypoxia-induced caspase-3 activation a

220 5-MTHF) in combination with epigallocatechin-gallate-enriched extract (EGCGe) and epigallocatechin-en

221 CG), (-)-epigallocatechin, (-)-epicatechin-3-gallate, (-)-epicatechin, and their epimers, and black t

222 erythrocyte hemolysis while (-)-epicatechin gallate, (-)-epigallocatechin gallate and (-)-epigalloca

223 degrees C: catechins [(-)-epigallocatechin-3-gallate, (-)-epigallocatechin, (-)-epicatechin-3-gallate

224 Gallate ester derivatives of the novel C4- and C8-substi

225 Catechins and their gallate esters are a class of polyphenolic compounds.

226 -4 and -5 and report here that the catechin gallate esters found in green tea potently inhibit the a

227 Catechin gallate esters therefore provide selective inhibition of

228 ate, (-)-epigallocatechin, (-)-epicatechin-3-gallate], flavones (kaempferol, kaempferol-3-glucoside,

229 atechin-3-gallate and (-)-epigallocatechin-3-gallate for the binding site on BSA near Trp213.

230 ated with 20 micromol/L (-)-epigallocatechin gallate (green tea) was restored back to wild-type level

231 and polyphenols (vanillin, epigallocatechin gallate, green tea extract, and protocatechualdehyde) at

232 Using other polyphenols, we showed that the gallate group was essential for inhibition, and antioxid

233 oil system decreased in the order of methyl gallate>gallic acid>alpha-tocopherol.

234 The addition of Trolox, BHA, and propyl gallate had no significant effect on furan formation fro

235 Similarly, (-)-epicatechin-3-gallate had the highest effect on the Circular Dichroic

236 , where an alternative structure for Fe(III) gallate has been commonly cited.

237 Recently, (-)-epigallocatechin gallate has been found to be a potent inhibitor of the p

238 Epigallocatechin gallate has been shown to be an inhibitor of the proteal

239 enols, such as catechin and epigallocatechin gallate, have been used to augment the action of traditi

240 al agent (IC50=29.5 muM), followed by methyl gallate (IC50=38.0 muM, log P=-0.23) and alpha-tocophero

241 sing the DYRK1A inhibitor epigallocatechin-3-gallate improved Ts65Dn skeletal phenotypes.

242 age capacity in vivo Proanthocyanidin trimer gallate in particular modified lipid desaturation in C.

243 as smaller than that of (-)-epigallocatechin gallate in Phase III clinical trials and about one order

244 g the major contribution of epigallocatechin gallate in the peroxyl radical scavenging of green tea e

245 dant capacity than their corresponding alkyl gallates, in emulsions prepared with lecithin or Tween-2

246 the green tea polyphenol epigallocatechin-3 gallate, induced ERalpha.

247 nd, to a lesser degree, (-)-epigallocatechin gallate inhibited cyclooxygenase (Cox)-2 gene expression

248 green tea polyphenol, (-)-epigellocathecin-3-gallate, is also inhibited by apigenin, suggesting that

249 ere dose dependent, whereas epigallocatechin gallate levels did not accumulate nor appear dose relate

250 -galloyl catechin (GC), catechin (C), methyl gallate (MG), and catechin-3-O-gallate (CG).

251 in the presence of gallic acid (GA), methyl gallate (MG), MG/GA combinations (75:25, 50:50, and 25:7

252 Proanthocyanidin trimer gallate modulates lipid deposition and fatty acid desatu

253 eptide, a tetraethylene glycol spacer, and a gallate moiety was designed, synthesized, and characteri

254 ds: rifampicin, curcumin, epigallocatechin-3-gallate, myricetin, and scyllo-inositol, in cells expres

255 aptoethanol (beta-ME), Trolox (TX), n-propyl gallate (n-PG), and ascorbic acid (AA).

256 para-nitrobenzoic acid (pNBA), and n-propyl gallate (nPG)--on bilayer properties using a gramicidin

257 ative, oligomerized (-)-epigallocatechin-3-O-gallate (OEGCG) as a carrier for oral lycopene delivery.

258 ors NADH and NADPH and is sensitive to octyl gallate (Ogal), a plastidial terminal oxidase inhibitor.

259 e green tea catechin, (-)-epigallocatechin-3-gallate or (-)-EGCG, has been shown to act as a proteaso

260 te treatment protocol, we administered ethyl gallate or norepinephrine after a sustained approximatel

261 the infusion and randomly administered ethyl gallate or norepinephrine in respective groups.

262 Contrary, epigallocatechin gallate, pelargonidin and catechin, with less impressive

263 Literature mentions propyl gallate (PG) as a non-toxic synthetic antioxidant that c

264 ity of gallic acid (GA) and its ester propyl gallate (PG) in the presence of UV-A light against Esche

265 pure OPC dimer, trimer, tetramer, and their gallates (pOPCs).

266 the phenolic compounds catechin, epicatechin gallate, procyanidin B1, rutin, gallic acid, caffeic aci

267 of l-ascorbic acid, was inhibited by lauryl gallate, propyl gallate, protocatechuic acid ethyl ester

268 cid, was inhibited by lauryl gallate, propyl gallate, protocatechuic acid ethyl ester, and salicylhyd

269 1 and B2, catechin, epicatechin, epicatechin gallate, quercetin 3-beta-d-glucoside, delfinidin 3-gluc

270 Coumaroylquinic acid, epicatechin gallate, quercetin, and six other phenolics were identif

271 ularly in the late treatment protocol, ethyl gallate resulted in a lower heart rate, a lower troponin

272 Exposure to trimer gallate resulted in the transcriptional down-regulation

273 2 by the green tea compound epigallocatechin gallate results in an increase in [Ca(2+)](ER) due to in

274 catechin gallate (EGCG), gallic acid, propyl gallate, resveratrol, and alpha-tocopherol) were investi

275 Among these polyphenols, (-)-epicatechin-3-gallate showed the highest Stern-Volmer modified quenchi

276 llagic acid, limonin, oleanolic acid, propyl gallate, sinapic acid and ursolic acid demonstrated sign

277 In the emulsion system, methyl gallate still behaved better than gallic acid, but the h

278 3 pOPCs (dimer gallate, trimer and/or trimer gallate) suggested the absence of synergistic potential.

279 polyphenols, tannic acid (TA), theaflavin-3'-gallate (TF2B) and theaflavin-3,3'-digallate (TF3) exhib

280 st that phenolic antioxidants, such as ethyl gallate, that inhibit hydrogen peroxide signaling, may r

281 utrient screen, EGCG ((-)-epigallocatechin 3-gallate), the major phytochemical in green tea, emerged

282 The fate of (-)-epicatechin-3-O-gallate, the main flavan-3-ol in green tea, is unclear b

283 aboratory have shown that epigallocatechin-3-gallate, the major polyphenol present in green tea, inhi

284 ck tea polyphenols, theaflavin, theaflavin-3-gallate, theaflavin-3'-gallate, and theaflavin-3,3'-diga

285 flavin-3,3'-digallate (TFdiG), theaflavin-3'-gallate, theaflavin-3-gallate, and theaflavin inhibited

286 nylcyclopropenium tetrakis(pentafluorophenyl)gallate (TPCPGa) and diphenyl-(2-methoxy-1-naphthyl)-cyc

287 combination exposure of 2 or 3 pOPCs (dimer gallate, trimer and/or trimer gallate) suggested the abs

288 Four alkyl gallate triphenylphosphonium lipophilic cations were syn

289 nt-based compounds (hippuric acid and propyl gallate) using cell- and membrane-based transport inhibi

290 hemodynamic and biochemical effects of ethyl gallate vs. those of the commonly used vasopressor, nore

291 stem, the natural product epigallocatechin 3-gallate was found to block Wnt signaling, independent of

292 ssays (MTT) we found that C8-propyl-catechin gallate was more active (IC50 = 31 microM) than catechin

293 gallocatechin gallate (EGCG) and epicatechin gallate were found to inhibit GDH with nanomolar ED(50)

294 he thermosensitive compound epigallocatechin gallate were recovered without significant losses.

295 Glucosyl alkyl gallates were shown previously to be better surfactants

296 efficacy of CPC separation, three glucoside gallates were subsequently isolated by HPLC chromatograp

297 epigallocatechin gallate and (-)-epicatechin gallate)] were most efficient in the inhibition of AAPH-

298 acid, vanillic acid, and epigallocatechin-3-gallate, were effective inhibitors of the activity of ca

299 ehavior of the antioxidant activity of alkyl gallates when increasing alkyl chain length was observed

300 nt of glioma cells with (-)-epigallocatechin gallate, which targets the ATP-binding domain of GRP78 a

301 ll ink (IGI) is an amorphous form of Fe(III) gallate.xH2O (x = approximately 1.5-3.2).