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1 overall, HR = 0.98, 95% C.I. = 0.62-1.55 for gallbladder).
2 n and (250.6-278.5) x10(-5) mm(2)/sec in the gallbladder.
3  in the canalicular network, bile ducts, and gallbladder.
4 f the extrahepatic biliary tract, except the gallbladder.
5 extrusion, for Salmonella maintenance in the gallbladder.
6 estinal peptide-a myorelaxant factor for the gallbladder.
7 ecapitulate a transport function of, primary gallbladder.
8 mount preparations from guinea pig and mouse gallbladder.
9 d vacuole in epithelial cells of the gut and gallbladder.
10 ation and defective contraction of the fetal gallbladder.
11 ues, including livers > mesonephros > guts > gallbladder.
12 mation, colonization, and persistence in the gallbladder.
13 s to persist as asymptomatic biofilms in the gallbladder.
14 is followed by carcinoma of the pancreas and gallbladder.
15 the intra- or extrahepatic bile ducts or the gallbladder.
16 neoplastic lesion or LOH was found in normal gallbladders.
17  ectopic cystic duct-like epithelia in their gallbladders.
18 on gallstones from Escherichia coli infected gallbladders.
19   Estimated radiation doses were highest for gallbladder (0.27 mSv/MBq), upper large intestine (0.19
20 atio [HR] 1.62, 99% CI 1.56-1.69; p<0.0001), gallbladder (1.31, 1.12-1.52; p<0.0001), kidney (1.25, 1
21 ase in BMI and endometrial (1.59, p<0.0001), gallbladder (1.59, p=0.04), oesophageal adenocarcinoma (
22 ded 411 cases with biliary tract cancer (237 gallbladder, 127 extrahepatic bile duct, and 47 ampulla
23 tic distributions included lungs (100%), the gallbladder (49%) if present, and locations involving he
24 pertension (18.2%) and empyema or perforated gallbladder (6.1%).
25 to 2014 was queried for patients with pT2/T3 gallbladder adenocarcinoma who underwent resection.
26                               The Sox17(+/-) gallbladder also showed a drastic reduction in sonic hed
27 re were 94 abdominal or pelvic surgeries (36 gallbladder): among 2,762 comparators there were 134 abd
28 mission when bile flow is diverted through a gallbladder anastomosis to jejunum, ileum or duodenum (s
29  min, followed by an anterior image over the gallbladder and abdomen.
30 l malrotation, midline liver with left-sided gallbladder and an interrupted inferior vena cava with a
31              There was no difference between gallbladder and bile duct tumors (P = .68).
32                           Only tumors of the gallbladder and bile ducts were assessed.
33 oma (HCC), intrahepatic bile duct (IBD), and gallbladder and biliary tract cancers outside of the liv
34 sophageal; stomach; colon and rectum; liver; gallbladder and biliary; pancreatic; larynx; tracheal, b
35 as to isolate stem cells from an adult mouse gallbladder and determine whether they were unique, comp
36 ity from non-Hodgkin lymphoma (P < .001) and gallbladder and extrahepatic bile duct cancer (P = .01)
37                          We compared primary gallbladder and IHBD cells by flow cytometry and found p
38 ound to be abundantly expressed in the human gallbladder and in the common bile duct, with only minor
39 ntestine and during chronic infection of the gallbladder and inform the basis for development of ther
40 bladder (P = 0.009), and a lower AUC for the gallbladder and intestines (P = 0.001).
41 increased AUC for liver, and a lower AUC for gallbladder and intestines (P = 0.042, 0.034, and 0.001,
42 time-activity curves of the liver and of the gallbladder and intestines were obtained and correlated
43 obtained and no activity was detected in the gallbladder and intestines.
44 terica serovar Typhi occurs in the bile-rich gallbladder and is frequently associated with the presen
45  we observed labeled epithelial cells in the gallbladder and lower intestines, as well as Microfold c
46 nd intestines, and to a lesser extent in the gallbladder and pancreatic duct.
47 ella enterica serovar Typhi can colonize the gallbladder and persist in an asymptomatic carrier state
48 expressed in mucosal epithelial cells of the gallbladder and small intestine.
49 cally important for E. coli to thrive in the gallbladder and upper intestinal tract, where high bile
50                 PK uptake was highest in the gallbladder and urinary bladder, followed by the liver,
51 my is an important tool for use in difficult gallbladders and achieves morbidity rates comparable to
52 d to cholic acid or bile, and in vivo in the gallbladders and livers of infected mice.
53 were detected in intestine, pancreas, liver, gallbladder, and cystic duct.
54 utaneously administered (54)Mn in the liver, gallbladder, and gastrointestinal tract of the KO mice,
55                Rates of prespecified muscle, gallbladder, and hepatic adverse effects and cancer were
56 m) and digestive accessory organs (pancreas, gallbladder, and liver).
57 s of whole-body PET data in 12 patients, the gallbladder appeared to be the dose-limiting organ, with
58                       Adenocarcinomas of the gallbladder are uncommon, aggressive tumors with poor su
59     The primary neuroendocrine tumors of the gallbladder are very rare, representing 0.2% of all neur
60 lous communication between cystic artery and gallbladder as the cause, a simultaneous cholecystoduode
61 ive dose of less than 0.30 mSv/MBq, with the gallbladder as the critical organ; the human target dose
62 (n = 7), periportal lymph nodes (n = 1), and gallbladder bed (n = 1).
63     We have performed a detailed analysis of gallbladder bile acid and lipid metabolism in Tgr5(-/-)
64                                              Gallbladder bile acid composition changed from predomina
65                                  Analysis of gallbladder bile acid composition showed marked increase
66                        Serum biochemistries, gallbladder bile acids, and liver sections were examined
67                    Cftr-/- mice had abnormal gallbladder bile and duodenal acidity, and overexpressed
68                         Interestingly, human gallbladder bile contains high levels of FGF19 (21.9 +/-
69                                              Gallbladder bile sterol levels were analyzed in choleste
70 al imbalance of lipids and bile salts in the gallbladder bile.
71 isorder due to biochemical imbalances in the gallbladder bile.
72  esophagus, colorectal, liver, pancreas, and gallbladder/bile duct cancers) in 69,310 nonsmoking and
73                                              Gallbladder biles and gallstones were examined by micros
74                             Lipid changes in gallbladder biles of gallstone patients vs overweight su
75  and hepatic levels of bile acids after BDL, gallbladder bilirubin and urinary bile acid concentratio
76                       We studied whether the gallbladder can modulate the microaggregates of choleste
77 ary cancer (cholangiocarcinoma) (63 months), gallbladder cancer (47 months), metastatic colorectal ca
78 ausal association of gallbladder stones with gallbladder cancer (GBC) is not yet well established.
79  metastases on outcome after R0 resection of gallbladder cancer (GBCA).
80 ture (HR 2.082, P=0.008), obstruction due to gallbladder cancer (HR 31.029, P=0.012) and only partial
81        Preoperative predictors of incidental gallbladder cancer (iGBC) have been poorly defined despi
82 ciated with substantial variation in risk of gallbladder cancer (sibling relative risk 3.15 [95% CI 1
83 ese imaging studies also serve to screen for gallbladder cancer and HCC.
84  IV stricture, biliary obstruction caused by gallbladder cancer and when only partial liver drainage
85 for re-resection for incidentally discovered gallbladder cancer appears to be between 4 and 8 weeks a
86          The discovery cohort comprised 1042 gallbladder cancer cases and 1709 controls and the repli
87 ols and the replication cohort contained 428 gallbladder cancer cases and 420 controls.
88 n study (GWAS), we did a genome-wide scan of gallbladder cancer cases and hospital visitor controls,
89 125b-5p up-regulation promoted cell death in gallbladder cancer cells in the presence of cisplatin.
90 ockdown of miR-125b-5p reduced cell death in gallbladder cancer cells treated with cisplatin.
91                          Although the OR for gallbladder cancer could not be calculated, the OR for t
92 using data from 4,180 patients with resected gallbladder cancer diagnosed from 1988 to 2003 from the
93 years with microscopically confirmed primary gallbladder cancer diagnosed or treated at Tata Memorial
94                                              Gallbladder cancer is highly lethal, with notable differ
95 ough adjuvant chemoradiotherapy for resected gallbladder cancer may improve survival for some patient
96  the net survival benefit of adjuvant RT for gallbladder cancer patients based on specific tumor and
97 tion as a new biomarker for the prognosis of gallbladder cancer patients.
98 he benefit of adjuvant radiotherapy (RT) for gallbladder cancer remains controversial because most pu
99                                              Gallbladder cancer represents the most common malignancy
100 ement from the addition of adjuvant RT after gallbladder cancer resection.
101 eport of common genetic variation conferring gallbladder cancer risk at genome-wide significance.
102 ion (1987-2007) were analyzed; patients with gallbladder cancer were excluded.
103                       Patients with resected gallbladder cancer were selected from the Surveillance,
104 esection for metastatic colorectal cancer or gallbladder cancer were usually considered cured.
105 l samples, miR-125b-5p was down-regulated in gallbladder cancer whereas Bcl2 was up-regulated and the
106 of 207 patients with incidentally discovered gallbladder cancer who underwent reoperation and had ava
107                         Of 449 patients with gallbladder cancer, 207 cases (46%) were discovered inci
108 lence of seropositivity was higher: 100% for gallbladder cancer, 97% of extrahepatic bile duct cancer
109 Although gallstones are recognized causes of gallbladder cancer, associations with other cancers of t
110  increased risk of cholangiocarcinoma (CCA), gallbladder cancer, hepatocellular carcinoma (HCC), and
111 fy common genetic susceptibility alleles for gallbladder cancer.
112 chemoradiotherapy for patients with resected gallbladder cancer.
113 olipid transporter genes in the pathology of gallbladder cancer.
114 , whereas VEGF variants were associated with gallbladder cancer.
115 hich mediates the function of miR-125b-5p in gallbladder cancer.
116 zes advances in understanding the biology of gallbladder cancer.
117 es to identify miR-125b-5p down-regulated in gallbladder cancer.
118 of Bcl2 is correlated with poor prognosis in gallbladder cancer.
119 select patients with incidentally discovered gallbladder cancer.
120 ant epithelioid hemangioendothelioma: n = 1, gallbladder cancer: n = 1 or metastatic disease [colorec
121 d extended/radical cholecystectomy for T2/T3 gallbladder cancer; however, many tumors are discovered
122 perihilar and distal cholangiocarcinomas and gallbladder cancers in Japanese patients.
123  considered in the differential diagnosis of gallbladder cancers.
124                        Bile was collected by gallbladder cannulation, and liver samples were analyzed
125                                              Gallbladder carcinoma (GBC) is frequent in the Indian su
126                             Individuals with gallbladder carcinoma (GBC), the most aggressive maligna
127 in extrahepatic cholangiocarcinoma (EHCC) or gallbladder carcinoma (GBCA) is unknown.
128  cholelithiasis may indicate XGC rather than gallbladder carcinoma.
129            Polypoidal thickening was seen in gallbladder carcinoma.
130      Lymphadenopathy was seen in 1 case with gallbladder carcinoma.
131  extrahepatic cholangiocarcinomas as well as gallbladder carcinomas, are a genetically diverse collec
132 ra- and extrahepatic cholangiocarcinomas and gallbladder carcinomas, are a genetically diverse collec
133  frequently altered gene in a series of nine gallbladder carcinomas.
134 ts for preventing asymptomatic serovar Typhi gallbladder carriage.
135 s showed that the expanded EpCAM(+) CD49f(+) gallbladder cells and IHBD cells exhibit differences rel
136  bile acid induction of hepatic Abcg5/g8 and gallbladder cholesterol content, suggesting a role of FX
137 functional interplay between LXR and LDLR in gallbladder cholesterol crystallization and possibly cho
138 ) sensitized mice to lithogenic diet-induced gallbladder cholesterol crystallization, which was assoc
139 formation on cholesterol gallstones promotes gallbladder colonization and maintenance of the carrier
140 duced biofilms on gallstone surfaces promote gallbladder colonization and maintenance of the carrier
141 s occur in humans and mice, which facilitate gallbladder colonization and shedding.
142 evels of importance in biofilm formation and gallbladder colonization and that the ECM diminishes dis
143 ith cholecystolithiasis and patients without gallbladder concretions.
144 ded duodenum filled with fluid and a dilated gallbladder containing dilute bile with high bicarbonate
145                  Ultrasound demonstrated the gallbladder containing multiple stones, with wall thicke
146 d cholescintigraphy is performed to quantify gallbladder contraction and emptying.
147 t-prandial variations in gallbladder volume; gallbladder contraction in response to cholecystokinin-8
148 ibution of embryonic cholecystitis and fetal gallbladder contraction in the early pathogenesis of con
149  of the unique short duration and reversible gallbladder contraction observed in vivo upon administra
150                                The defective gallbladder contraction positively correlated with the s
151 lar carcinogenesis of adenocarcinomas of the gallbladder, coupled with the availability of novel mole
152                                              Gallbladder cystic mucosal hyperplasia was commonly foun
153      The concept that motor disorders of the gallbladder, cystic duct and sphincter of Oddi can cause
154 latation as long as they involve presence of gallbladder deposits and tumors.
155 ancer death (5 more per 10 000 woman-years), gallbladder disease (20 more per 10 000 woman-years), de
156  dementia (22 more cases [95% CI, 4 to 53]), gallbladder disease (21 more cases [95% CI, 10 to 34]),
157 antly increased, per 10000 person-years, for gallbladder disease (30 more cases [95% CI, 16 to 48]),
158  thrombosis (7 more per 10 000 woman-years), gallbladder disease (33 more per 10 000 woman-years), an
159                                              Gallbladder disease (GBD) is a highly prevalent conditio
160 irrhosis (P(interaction) = 0.02) but not for gallbladder disease (P(interaction) = 0.4).
161 ancers in the cholecystectomy cohort and the gallbladder disease cohort compared with a control cohor
162 asing alcohol consumption, while the risk of gallbladder disease decreased (P(trend) < 0.0001 for eac
163             In analyses for association with gallbladder disease or gallstones, top bilirubin SNPs in
164 1996-2005), incidence rates of cirrhosis and gallbladder disease were 1.3 per 1,000 persons (n = 2,10
165 smoking on incidences of liver cirrhosis and gallbladder disease were examined in a prospective study
166 eta-analyses (human height, QTc interval and gallbladder disease); all previous reported association
167                                          For gallbladder disease, alcohol reduces risk and smoking re
168 ased risk for stroke, thromboembolic events, gallbladder disease, and urinary incontinence.
169 olism, dementia (in women aged >/=65 years), gallbladder disease, and urinary incontinence; benefits
170 ding acute cholecystitis, chronic acalculous gallbladder disease, high-grade and partial biliary obst
171  Mongolia has an extremely high incidence of gallbladder disease.
172  are, on occasion, initially misdiagnosed as gallbladder disease.
173  cirrhosis and 1.29 (95% CI: 1.22, 1.37) for gallbladder disease.
174  cirrhosis and 0.59 (95% CI: 0.55, 0.64) for gallbladder disease.
175 ed to patients undergoing surgery for benign gallbladder disease.
176  cholecystectomy in patients with Functional Gallbladder Disorder (FGBD) and the relevance of sphinct
177 iary atresia, as it loses its bile ducts and gallbladder during metamorphosis.
178 nt of variation [CV]) for calculation of the gallbladder ejection fraction (GBEF) in healthy subjects
179 active intestinal peptide and had defects in gallbladder emptying and in levels of secondary BAs, but
180                                              Gallbladder emptying in response to cholecystokinin octa
181 han healthy subjects and nutrient-stimulated gallbladder emptying is impaired with "gallbladder pares
182 tr-/- and CftrDeltaF508 mice have defects in gallbladder emptying that disrupt enterohepatic circulat
183                                              Gallbladder emptying was similar in the 3 groups.
184 ion, hormone concentrations, and gastric and gallbladder emptying were measured several times over 4
185 n of a laparoscopic cholecystectomy due to a gallbladder empyema.
186 ased risk of cancers of the liver, pancreas, gallbladder, endometrium, stomach, kidney, brain (benign
187 ditional deletion of Sox17 in the primordial gallbladder epithelia but not in fetal liver hepatoblast
188 lls were evident on the surface of the mouse gallbladder epithelia up to 21 days postinfection.
189 sted the hypothesis that well-differentiated gallbladder epithelial cells (GBECs) are capable of engr
190 a was able to adhere to and invade polarized gallbladder epithelial cells apically in the absence and
191 here to and form extensive bacterial foci on gallbladder epithelial cells as early as 12 h postinocul
192 othesize that in addition to gallstones, the gallbladder epithelium aids in the establishment/mainten
193 d carriage showed invasion and damage of the gallbladder epithelium and lamina propria up to 2 months
194 work, we present evidence of the role of the gallbladder epithelium in chronic carriage by a mechanis
195 tence and/or bacterial aggregation in/on the gallbladder epithelium with luminal cell extrusion, for
196                                          The gallbladder excretes cytotoxic bile acids into the duode
197                                              Gallbladder explants secrete 500 times more FGF19 than F
198     The anatomopathologic examination of her gallbladder following cholecystectomy showed signs of ch
199 iver where it replicates robustly and in the gallbladder, from which it can return to the intestine a
200 leum G1 lesions, 1 pancreas G2 lesion, and 1 gallbladder G3 lesion.
201                                              Gallbladder had the highest radiation exposure (21 muSv/
202        Fistula between the cystic artery and gallbladder has been commonly reported to occur after la
203 e were 134 abdominal or pelvic surgeries (37 gallbladder) (hazard ratio HR] = 0.70, 95% confidence in
204 mall intestine in 16 (36%), and the liver or gallbladder in 13 (30%).
205 eter less than 6 mm and those with an intact gallbladder in whom the cystic duct would be overlapped
206 pplication of bradykinin every 10-min on the gallbladder induced consistent pressor reflex responses.
207 reoperative severity grade and the extent of gallbladder inflammation on histopathology.
208  .02) and the presence of a gallstone in the gallbladder infundibulum (78% and 22% for conversion and
209 itis) and the presence of a gallstone in the gallbladder infundibulum are associated with conversion
210 e and bacterial survival in vivo and reduced gallbladder interleukin-10 (IL-10) levels.
211 nsport of ingested fluorescent lipids to the gallbladder is blocked.
212 ) epithelial cell subpopulation from primary gallbladder is enriched in colony-forming cells, compare
213 a or intervention, between cystic artery and gallbladder is rare with very few reports in literature.
214     On the basis of whole-body PET data, the gallbladder is the dose-limiting organ, with an average
215 fication of resident stem cells in the mouse gallbladder is, to date, unexplored.
216 alignancies, including cancers of the colon, gallbladder, kidney, and pancreas, its role in prostate
217 causality, 41% of uterine and 10% or more of gallbladder, kidney, liver, and colon cancers could be a
218 the bladder, cervix, endometrium, esophagus, gallbladder, kidney, liver, and lung.
219 he bladder, breast, colon/rectum, esophagus, gallbladder, kidney, liver, lung, skin (melanoma-only),
220 ients were scheduled for cholecystectomy for gallbladder lithiasis.
221 on of Diseases, Ninth Revision, diagnosis of gallbladder malignancy who underwent a laparoscopic chol
222                                          The gallbladder may have a role in regulating cholesterol ho
223  Stomach Meridian of Foot-Yangming (SMFY) or Gallbladder Meridian of Foot-Shaoyang (GMFS) in healthy
224 biliary cholesterol secretion, and protected gallbladder motility function by desaturating bile in mi
225 astrectomy on hunger, satiation, gastric and gallbladder motility, and gastrointestinal hormone respo
226 y and transcript for GPBAR1 were detected in gallbladder muscularis.
227 opathological examination revealed a primary gallbladder neuroendocrine tumor.
228 le cannot be safely identified in "difficult gallbladders." OBJECTIVE: To conduct a systematic review
229                             Emergence in the gallbladder occurred later (P = 0.009, highest dose), an
230  serum whereas bile was decreased within the gallbladder of Cic-L(-/-) mice.
231 iated with the presence of gallstones in the gallbladder of infected carriers upon which Salmonella c
232                                          The gallbladders of Cftr-/- mice were enlarged and had defec
233 rs, was lower in the ileum but higher in the gallbladders of Cftr-/- mice, compared with wild-type mi
234 ate colonization and persistent infection in gallbladders of humans and mouse models of chronic carri
235 he surfaces of cholesterol gallstones in the gallbladders of mice and human carriers.
236 ellent and no activity was identified in the gallbladder or intestine.
237 t transformation in predisposed mice, murine gallbladder organoids, and fibroblasts, with TP53 mutati
238  emergence time of (99m)Tc-mebrofenin in the gallbladder (P = 0.009), and a lower AUC for the gallbla
239 ions of the upper abdomen, ADC values of the gallbladder, pancreas, spleen, and kidney may be compara
240 bdominal regions (left and right liver lobe, gallbladder, pancreas, spleen, and renal cortex and medu
241 lated gallbladder emptying is impaired with "gallbladder paresis" occurring in approximately 20%.
242 ution in applying this technique to inflamed gallbladder pathology.
243 n be used as a decision aid to predict which gallbladder patients may benefit from adjuvant CRT.
244 rvival in vivo without altering the examined gallbladder pro- or anti-inflammatory cytokines.
245       Dosimetry calculations showed that the gallbladder received the highest dose (229.50 muGy/MBq [
246 ximately 17 muSv/MBq (62 mrem/mCi), with the gallbladder receiving the highest dose of 190 muSv/MBq.
247 s related to participation in the programme (gallbladder removal and ruptured Achilles tendon).
248 en cholecystectomy as the primary method for gallbladder removal countrywide in 2011.
249 usness due to drugs for pre-existing angina, gallbladder removal, hospital admission with suspected h
250  often used to diagnose dysfunction (dynamic gallbladder scintigraphy and sphincter manometry) is con
251 e thought to contribute to the disruption of gallbladder smooth muscle (GBSM) function that occurs in
252                                   Of the 350 gallbladder specimens from gallstone patients, hyperplas
253              Histological examination of the gallbladder specimens was done for preneoplastic lesions
254 ans receiving highest absorbed dose were the gallbladder, spleen, stomach, liver, kidneys, and bladde
255  In addition, the relationship between adult gallbladder stem cells and intrahepatic bile duct (IHBD)
256  the hypothesis that biofilms on cholesterol gallbladder stones facilitate typhoid carriage in mice a
257                        Causal association of gallbladder stones with gallbladder cancer (GBC) is not
258 chronic, asymptomatic infection of the human gallbladder, suggesting that this bacterium utilizes nov
259 e the RR was 0.99 (95% C.I. = 0.56-1.77) for gallbladder surgery.
260 or pelvic surgery nor the specific subset of gallbladder surgery.
261 lly ill patients have larger, thicker-walled gallbladders than healthy subjects and nutrient-stimulat
262  fecal shedding and enhanced colonization of gallbladder tissue and bile.
263 hed response rates of adenocarcinomas of the gallbladder to chemotherapy are less than 30% and no sur
264 PEBDs), 11 ileal exclusions (IEs), and seven gallbladder-to-colon (GBC) diversions.
265 ation Survey (20-74 years old) who underwent gallbladder ultrasonography from 1988 to 1994.
266  destruction, focal biliary cirrhosis, micro-gallbladder, vas deferens loss, airway disease, and meco
267                                        After gallbladder visualization had been confirmed at 60 min,
268 y ill patients (21%), there was no change in gallbladder volume in response to nutrient, and overall
269 at (LE3), different dynamics of postprandial gallbladder volume were induced (P </= 0.001).
270 potent GLP-1 secretagogue, has low effect on gallbladder volume, and improves glucose homeostasis in
271 ects of lipid emulsions on gastric emptying, gallbladder volume, and triglyceride absorption are depe
272                     There were no changes in gallbladder volume.
273  scintigraphy or post-prandial variations in gallbladder volume; gallbladder contraction in response
274               In the critically ill, fasting gallbladder volumes (critically ill, 61 mL [36-100 mL] v
275                                              Gallbladder volumes were measured with the use of real-t
276 ferences in GLP-1 and PYY concentrations and gallbladder volumes were observed after the capsaicin in
277 ed activities (in h) were the liver (0.117), gallbladder wall (0.046), and small intestine (0.033), a
278 , averaged over both men and women, were the gallbladder wall (0.59 +/- 0.44), small intestine (0.12
279 highest dose exposure (in muGy/MBq) were the gallbladder wall (111 +/- 60) > liver (21 +/- 7), kidney
280 ing bioengineered tissue can reconstruct the gallbladder wall and repair the biliary epithelium follo
281                                 Doses to the gallbladder wall and spleen may limit the amount of perm
282                               The absence of gallbladder wall enhancement (58% and 40% for conversion
283                               The absence of gallbladder wall enhancement (associated with the presen
284    On preoperative CT images, the absence of gallbladder wall enhancement was associated with the pre
285                                          The gallbladder wall received the highest radiation dose in
286          The absorbed doses to the liver and gallbladder wall were slightly but significantly higher
287 der wall, 0.258 mGy/MBq (0.955 rad/mCi), and gallbladder wall, 0.193 mGy/MBq (0.716 rad/mCi).
288  the upper large intestine, small intestine, gallbladder wall, and lower large intestines were 0.082,
289 stic stranding, wall thickness, THAD, fat in gallbladder wall, cholelithiasis, infiltration, biliary
290 ons in order to search for thickening of the gallbladder wall, pleural effusion, and ascites.
291 The organ receiving the highest dose was the gallbladder wall, with an average of 2.81 x 10(-2) mSv.M
292 blingual gland, spleen, pancreas, liver, and gallbladder was observed.
293              Three-dimensional images of the gallbladder were acquired at 30-minute intervals from -3
294           During surgery, 23/39 (59%) of the gallbladders were found to contain biliary stones or slu
295                              By 2013, 62% of gallbladders were removed laparoscopically countrywide a
296  is a procedure that removes portions of the gallbladder when structures of the Calot triangle cannot
297 pithelial cell population from primary mouse gallbladder with stem cell characteristics and found it
298 n vivo model of rotavirus infection of mouse gallbladder with UK x RRV reassortants to study the gene
299                   The critical organ was the gallbladder, with an average radiation-absorbed dose of
300 observed in the kidneys, bladder, liver, and gallbladder, with mean standardized uptake values at 1 h

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