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1 tiserum recognizes a 150-kDa protein from M. gallisepticum.
2 plasmal conjunctivitis, caused by Mycoplasma gallisepticum.
3  days after experimental inoculation with M. gallisepticum.
4 n direct contact of HD-11 cells with live M. gallisepticum.
5  the first genome-scale reconstruction of M. gallisepticum.
6 transmission potential, among isolates of M. gallisepticum.
7 of naive juveniles or the introduction on M. gallisepticum.
8 increased rapidly after reintroduction of M. gallisepticum.
9 date genomic variability among strains of M. gallisepticum.
10 ntical homolog in four strains of Mycoplasma gallisepticum.
11 GAA repeats in M9/pMGA gene expression in M. gallisepticum.
12 in the avian respiratory pathogen Mycoplasma gallisepticum.
13 omoterless lacZ gene and transformed into M. gallisepticum by using transposon Tn4001 as a vector.
14 te catabolism and all genes examined from M. gallisepticum, C. perfringens, and S. pneumoniae were un
15                                           M. gallisepticum cgMLST represents a standardized, accurate
16 ountries on 4 continents were typed using M. gallisepticum cgMLST.
17                      Southern analysis of M. gallisepticum chromosomal DNA confirmed that the phase-v
18 rchived isolates of the bacterium Mycoplasma gallisepticum collected during sequential emergence even
19                                   Mycoplasma gallisepticum colonizes the chicken respiratory mucosa a
20                            A total of 425 M. gallisepticum conserved genes (49.85% of M. gallisepticu
21                  For the transformants of M. gallisepticum containing the reporter with deletion of n
22 t, vaccination, and surveillance, Mycoplasma gallisepticum continues to cause significant morbidity,
23                 By injecting live Mycoplasma gallisepticum cultures into the ankle joint a typical my
24  that both GapA and CrmA are required for M. gallisepticum cytadherence and pathogenesis.
25  CrmA might play an essential role in the M. gallisepticum cytadherence process.
26 nt was used to probe a genomic library of M. gallisepticum DNA.
27 of the avian respiratory pathogen Mycoplasma gallisepticum encode a family of hemagglutinins that are
28 ic and include the lipoprotein of Mycoplasma gallisepticum, encoded by the MGA0674 gene.
29  isolates of the pathogen collected after M. gallisepticum established itself in western North Americ
30  proteins important for cytadherence, few M. gallisepticum factors or pathways contributing to host c
31            To effectively analyze Mycoplasma gallisepticum for virulence-associated determinants, the
32 ermits the comprehensive screening of the M. gallisepticum genome for the identification of novel vir
33  gallisepticum conserved genes (49.85% of M. gallisepticum genome) were selected as core genome targe
34  obtained in this manner to the annotated M. gallisepticum genome, the precise locations of transposo
35                             A total of 81 M. gallisepticum genomes from 5 countries on 4 continents w
36 f this scheme, a diverse collection of 37 M. gallisepticum genomes was used to identify cgMLST target
37 ovel insights into inter- and intrastrain M. gallisepticum genomic variability and the genetic basis
38 -kDa cytadhesin-like protein from Mycoplasma gallisepticum has been identified.
39                    House finch-associated M. gallisepticum (HFMG) spread rapidly and increased in vir
40                             MGA_1199, the M. gallisepticum homologue of the cytadherence-associated p
41  Mycoplasma pneumoniae, and MGA_0928, the M. gallisepticum homologue of the M. pneumoniae cytoskeleta
42 thought to be important for mechanisms of M. gallisepticum-host interaction, pathogenesis, and immune
43 e that glycerol metabolism is required by M. gallisepticum in a niche that we have yet to study.
44 lisepticum vaccines, and the detection of M. gallisepticum in game and free-flying song birds has str
45  disease manifestations characteristic of M. gallisepticum infection.
46  second hypothesis that reintroduction of M. gallisepticum into a multiage group of previously expose
47                                   Mycoplasma gallisepticum is a significant respiratory and reproduct
48                                   Mycoplasma gallisepticum is an etiologic agent of chronic respirato
49                                   Mycoplasma gallisepticum is an important pathogen of chickens and t
50 low) strain of the avian pathogen Mycoplasma gallisepticum is capable of producing H2O2 when grown in
51 se finches and that the reintroduction of M. gallisepticum is sufficient to cause a new outbreak, eve
52                                   Mycoplasma gallisepticum is the most virulent and economically impo
53                                   Mycoplasma gallisepticum is the primary etiologic agent of chronic
54 oducible method for differentiation among M. gallisepticum isolates.
55                                   Mycoplasma gallisepticum, known primarily as a respiratory pathogen
56 omparative transcriptomic analysis of the M. gallisepticum live attenuated vaccine strain F and the v
57 characteristics drive patterns of Mycoplasma gallisepticum (MG) infections in the house finch (Carpod
58 e conjunctival bacterial pathogen Mycoplasma gallisepticum (MG), to experimentally examine the impact
59                                    Global M. gallisepticum mutagenesis is currently limited to the us
60                An annual pulse of Mycoplasma gallisepticum-naive juveniles increasing the number of s
61  experimentally determined growth rate of M. gallisepticum of 0.244+/-0.03[Formula: see text].
62  M. synoviae, but not on the sialidase of M. gallisepticum or the sialidases or other enzymes essenti
63                        The reemergence of M. gallisepticum outbreaks among poultry, the increased use
64       Moreover, molecular epidemiology of M. gallisepticum outbreaks can be performed using RFLP and/
65 -kDa cell surface antigen (M9) of Mycoplasma gallisepticum PG31 that mediates antibody-induced agglut
66       This construct was transformed into M. gallisepticum PG31.
67 ptomic vlhA gene expression directly from M. gallisepticum populations present on tracheal mucosae du
68  in the natural host compared to virulent M. gallisepticum R(low).
69               These results indicate that M. gallisepticum regulates gene expression upon exposure to
70  the avian respiratory tract with Mycoplasma gallisepticum results in a profound inflammatory respons
71                                   Mycoplasma gallisepticum Rhigh, the attenuated, high-passage deriva
72 rom tracheas of 20 chickens infected with M. gallisepticum Rlow and 20 mock-infected animals at days
73    Fibronectin was found to be present in M. gallisepticum Rlow protein extracts by Western blotting
74 and the sequenced genomes compared to the M. gallisepticum Rlow reference genome.
75 leotide identity with the pMGA1.1 gene of M. gallisepticum S6.
76 rst description of a functional gene from M. gallisepticum showing homology to cytadhesin genes from
77  to determine the feasibility of using an M. gallisepticum-specific gene encoding a phase-variable pu
78 ions, and significantly higher numbers of M. gallisepticum-specific IgG- and IgA-secreting plasma/B c
79                                Currently, M. gallisepticum strain differentiation based on sequence a
80 mic sequence of the virulent, low-passage M. gallisepticum strain R (R(low)) has been reported, genom
81 n of the phenotypic expression of Mycoplasma gallisepticum strain R low (passage 15) to that of strai
82 ously demonstrated that avirulent Mycoplasma gallisepticum strain R(high) (passage 164) is lacking th
83 to investigate transcriptional changes in M. gallisepticum strain R(low) upon exposure to eukaryotic
84 n following challenge with the pathogenic M. gallisepticum strain R(low).
85  We have determined that virulent Mycoplasma gallisepticum strain Rlow is capable of binding the extr
86 locus sequence typing (cgMLST) scheme for M. gallisepticum strains and field isolates.
87 of cgMLST allowed differentiation between M. gallisepticum strains of the same outbreak.
88                                   Mycoplasma gallisepticum, the cause of chronic respiratory infectio
89                                   Mycoplasma gallisepticum, the primary etiologic agent of chronic re
90 of a widespread poultry pathogen, Mycoplasma gallisepticum, through a previously unknown host, the ho
91 l microsatellite of the bacterium Mycoplasma gallisepticum, through the collection and analysis of in
92 lts can initiate an epidemic and transmit M. gallisepticum to naive house finches and that the reintr
93 igned to target 5 sequences unique to the M. gallisepticum ts-11 strain: vlhA3.04a, vlhA3.04b, vlhA3.
94 5, and mg0359 was able to distinguish the M. gallisepticum ts-11 vaccine strain from field isolates.
95 othetical protein gene, mg0359, unique to M. gallisepticum ts-11 vaccine strain.
96             Analyses of the genome of the M. gallisepticum vaccine strain F revealed numerous differe
97  isolates, whole-genome sequencing of the M. gallisepticum vaccine strain ts-11 and several "ts-11-li
98                         A live attenuated M. gallisepticum vaccine strain, GT5, was previously shown
99 current approaches to quickly distinguish M. gallisepticum vaccine strains from field isolates.
100  among poultry, the increased use of live M. gallisepticum vaccines, and the detection of M. gallisep
101  an attenuated strain and plays a role in M. gallisepticum virulence.
102 omic variability and the genetic basis of M. gallisepticum virulence.
103 metabolic model for the bacterium Mycoplasma gallisepticum was created.
104 eproducible methods for comparisons among M. gallisepticum whole genomes.
105 recently reported that the interaction of M. gallisepticum with chicken tracheal epithelial cells (TE
106 he notion that the initial interaction of M. gallisepticum with host respiratory epithelial cells con

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