ライフサイエンスコーパス: gametic

1 responses or synthesis induces switching of gametic and nongametic cell identities and specialized n

2 nty on the phase of the double heterozygote, gametic and nongametic disequilibria need to be combined

3 taZ = (Vgam/V)S, where Vgam and V denote the gametic and total variances in the character and S desig

4 The recent insights into the balance of gametic and zygotic contributions to imprint specificati

5 Patterns of gametic and zygotic LDs indicate the absence of epistasi

6 e genes, Mtd1, is a candidate participant in gametic cell fusion; two others, Mta1 and Ezy2, are cand

7 ase soon after inception, implying that male gametic cells follow a simple S to M cycle.

8 sion and DNA methylation from vegetative and gametic cells of each mating type and from zygotes.

9 In gametic cells this fiber underlies the gametic mating st

10 hich contain translationally silent mRNAs in gametic cells.

11 tween Arabidopsis thaliana (2n = 2x = 10; n, gametic chromosome number; x, haploid chromosome number)

12 an drive sex chromosome evolution to include gametic competition and meiotic drive.

13 gifts, the molecular composition of the non-gametic components of male ejaculates and their interact

14 /W) + (B/W2), where C, W and B represent the gametic covariance of the character and fitness, the mea

15 or subclasses of genes associated with these gametic differentially methylated regions (gDMRs), namel

16 s program and switch off the plus program of gametic differentiation.

17 and linkage are also taken into account, the gametic disequilibria generated by the Bulmer and Hill-R

18 dy-Weinberg disequilibria at each locus, (2) gametic disequilibrium (including two alleles in the sam

19 lly unlinked SNP pairs that show exceptional gametic disequilibrium across the lab strains.

20 Here, we report finding substantial gametic disequilibrium among allozyme and complementary

21 imes with different degrees of epistasis and gametic disequilibrium are allowed.

22 that are associated with ordinal traits when gametic disequilibrium between a marker and trait loci e

23 er and breed information, in the presence of gametic disequilibrium between the marker locus (ML) and

24 report that AFF3 can specifically bind both gametic DMRs (gDMRs) and enhancers within imprinted loci

25 for establishment of methylation at maternal gametic DMRs.

26 ere is no environmental contribution and the gametic effects are additive or the character is fitness

27 mitted via independent iterative-somatic and gametic epigenetic mechanisms across multiple generation

28 he majority of alleles at these loci were in gametic equilibrium.

29 Transcriptome analysis revealed a rewired gametic expression program for Volvox MT genes relative

30 r generating maximum likelihood estimates of gametic frequencies from multiallelic genotypic data is

31 neration autotetraploid Pacific oysters from gametic frequencies.

32 tes, enabling the identification of specific gametic functions, and their contributions to zygote and

33 slocate from the cytosol to the nucleus upon gametic fusion, initiating zygote development.

34 ring development, prior to the initiation of gametic fusion.

35 processes are typically sex-specific, e.g., gametic/gametophytic competition typically occurs among

36 We identified 361 gametic genes with mating type-specific expression patte

37 ypothesize that the exposure of the maternal gametic genome to exogenous gonadotropins during the end

38 n humans, so this modification cannot be the gametic imprint for the PWS/AS region in humans.

39 H3 Lys9 methylation is a candidate maternal gametic imprint for this region, and we show how changes

40 We hypothesize that the gametic imprint may be set, at least in part, by the tra

41 n of H19 has been proposed to constitute the gametic imprint, which controls the reciprocal allelic e

42 Methylation may be inherited as the gametic imprinting mark or may be acquired postfertiliza

43 The evidence suggests that gametic imprints are essential for normal development, a

44 was affected, at least in part, by a lack of gametic imprints, as judged by DNA methylation and expre

45 on established during gametogenesis, such as gametic imprints, cannot be restored after nuclear trans

46 Wolbachia is closely associated with gametic incompatibility but also interacts with Drosophi

47 h after vegetative cells were transferred to gametic induction medium, the 122-kDa form was detected

48 The concept of gametic isolation has its origins in the 1937 edition of

49 Gametic isolation is generally subsumed under either pre

50 surprisingly more effective through the male gametic line.

51 Unlike gametic linkage disequilibrium defined for a random-mati

52 ten observed in a population with the use of gametic linkage disequilibrium.

53 shment and maintenance of the sperm-specific gametic mark.

54 Although there has been much focus on gametic marking of ICRs as the point of imprint specific

55 In gametic cells this fiber underlies the gametic mating structure and makes contact with it.

56 proteins displayed on the plasma membrane of gametic mating structures.

57 stis, sperm and fetal oocytes demonstrates a gametic methylation imprint with unmethylated paternal g

58 with respect to H19, but without loss of the gametic methylation imprint.

59 In the mouse this DMR is a gametic methylation mark.

60 The inheritance of gametic methylation patterns is a critical event in the

61 I investigated gametic nonreduction and neopolyploid formation in natur

62 or mt(-) carried genes expressed only in the gametic or zygotic phases of the life cycle.

63 show that genetic hierarchies of somatic and gametic parasitism following fusion can be replicated by

64 In addition, gametic phase disequilibrium among loci was investigated

65 htly linked (rf = 0.0016) and exhibit strong gametic phase disequilibrium in introduced populations i

66 heterozygosity signals a departure from the gametic phase equilibrium: We describe the specific form

67 le, rapid method for empirically determining gametic phase for double and triple heterozygotes.

68 refore, retard the rate of decay of linkage (gametic phase) disequilibrium (LD) among mutations.

69 ndicate very low rates of exchange, all four gametic phases were observed both at the tip of the X an

70 dopsis are competent to differentiate into a gametic precursor and that the function of AGP18 is impo

71 hromosome bivalent pairing configuration and gametic recombination to discern different mechanisms of

72 allelic variation is always maintained under gametic selection alone, but with any fertility or viabi

73 ference" among loci, and the consequences of gametic selection for the joint distribution of inbreedi

74 ent in each of the two marker intervals; (2) gametic selection operative prior to fertilization; and

75 any combination of fertility, viability, and gametic selection through meiotic drive.

76 Gametic selection was not a source of bias in dominance

77 and (4) dominance estimation is unbiased by gametic selection.

78 ent of embryonic heterozygosity, ploidy, and gametic syngamy but is directly correlated with the embr

79 the genetic identity of various somatic and gametic tissues within vascularly fused Botryllus schlos

80 These changes underlie the transition from gametic to embryonic chromatin and are thought to facili

81 of RNA have restricted in-depth analysis of gametic transcriptomes in flowering plants.

82 the fusion of egg and sperm involves the two gametic transcriptomes.

83 Somatic/gametic transmission alters DNA methylation at enhancers

84 elimination of these organelles, except for gametic transport in some phylogenetic groups.

85 that showed extreme associations (only three gametic types) among conspecific alleles.

86 In the latter case, C is the gametic variance in fitness.

87 somatic appearance may mask the winner of a gametic war.