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1 zures) and with the power change of the high-gamma rhythm.
2 excitatory/inhibitory equilibrium and on the gamma rhythm.
3 e associated with brain rhythms, notably the gamma rhythm.
4 emporal-parietal structures and expressed in gamma rhythms.
5 ty characteristic of assembly formation with gamma rhythms.
6 high frequency oscillations associated with gamma rhythms.
7 tial inhibitory neuron subtype necessary for gamma rhythms.
8 interneurons that contribute to hippocampal gamma rhythms.
9 rain) that apparently cannot be tolerated by gamma rhythms.
10 rated by as little as 400 microm, making the gamma rhythm a poor candidate for binding or communicati
11 theories of schizophrenia and that theta and gamma rhythm abnormalities are evident in schizophrenic
12 cal field potential, observable as theta and gamma rhythms and coupling between these rhythms, is pre
14 aspartate (NMDA) receptor antagonists alters gamma rhythms, and can induce cognitive as well as psych
20 ies are consistent with data suggesting that gamma rhythms are used for relatively local computations
22 ematical analysis, we study the breakdown of gamma rhythms as the driven ensembles become too small,
23 whose contrast varies across space generate gamma rhythms at significantly different frequencies in
24 est that robust nesting of hippocampal theta-gamma rhythms at the time of retrieval is a specific rec
25 t in a network with realistic heterogeneity, gamma rhythms based on the interaction of excitatory and
26 Primary visual cortex exhibits two types of gamma rhythm: broadband activity in the 30-90 Hz range a
27 s of several parts of the brain suggest that gamma rhythms can be generated by pools of excitatory ne
30 ndependently predicts that context-dependent gamma rhythms depend critically on SOM interneurons.
31 pocampal subfield CA1 and that slow and fast gamma rhythms differentially coordinate place cells duri
32 the emergence of the AS-related hippocampal gamma rhythm during the first postnatal week, as well as
33 und" gamma rhythm (resembling the persistent gamma rhythms evoked in vitro by cholinergic agonists) i
34 ations may expand the computational power of gamma rhythms for optimizing the synthesis and storage o
36 The results suggest the presence of a single gamma rhythm generator with a frequency range of 65-75 H
38 tant mice exhibit enhanced baseline cortical gamma rhythms, impaired gamma rhythm induction after opt
39 n: (i) spike times are entrained to a global Gamma rhythm (implying a consistent representation of th
41 arp stimulus-onset transient and a sustained gamma rhythm in local field potential recorded from the
42 e coincident expression of multiple types of gamma rhythm in sensory cortex suggests a mechanistic su
46 cover a previously unappreciated function of gamma rhythms in recruiting both neuronal and glial resp
47 The parameter range chosen is motivated by gamma rhythms, in which the gamma-aminobutyric acid type
48 ed baseline cortical gamma rhythms, impaired gamma rhythm induction after optogenetic drive of PV int
50 icularly strongly linked to cognition is the gamma rhythm: it is involved in attention, short- and lo
51 cordings in slice preparations we found that gamma rhythms lead to activity-dependent modification of
56 ed scopolamine on movement-induced theta and gamma rhythms recorded in the superficial layers of the
57 onous spontaneous activity to a "background" gamma rhythm (resembling the persistent gamma rhythms ev
59 isual stimuli that induce a strong, coherent gamma rhythm result in enhanced pairwise and higher-orde
61 gamma regimes can interact with a bottom-up gamma rhythm to provide regulation of signals between th
63 requires coupling of approximately 30-100Hz gamma rhythms to particular phases of the theta cycle.
65 of excitatory and inhibitory neurons in the gamma rhythm varies across local circuits and conditions
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