ライフサイエンスコーパス: gamma-aminobutyric acid

1 enrichment of glutamate, glutamine and GABA (gamma-aminobutyric acid).

2 gamma-Aminobutyric acid, 4-hydroxyproline, glycine, leuc

3 hat bilateral infusions into the RMTg of the gamma-aminobutyric acid A (GABAA) agonist, muscimol, ind

4 ed alpha cell dysfunction was due to reduced gamma-aminobutyric acid A receptor activation in pancrea

5 n the Nlrp3 inflammasome were independent of gamma-aminobutyric acid A receptor activation or N-methy

6 elective action on the omega1 subtype of the gamma-aminobutyric acid A receptor, zolpidem tartrate pr

7 pplied to awake NHP brain PET studies with a gamma-aminobutyric acid A-benzodiazepine receptor ligand

8 eptidyl-peptidase-like protein-6 (DPPX), and gamma-aminobutyric acid-A receptor (GABAAR).

9 in two neurotransmitter receptor genes, the gamma-Aminobutyric acid-A receptor delta and gamma-amino

10 iated protein-like 2), glycine receptor, and gamma-aminobutyric acid-A receptor.

11 thyl-4-isoxazolepropionic acid, glycine, and gamma-aminobutyric acid-A receptors), were prevalent in

12 gamma-Aminobutyric acid aminotransferase (GABA-AT) is a

13 od-brain barrier and inhibit the activity of gamma-aminobutyric acid aminotransferase (GABA-AT), the

14 al activator that regulates transcription of gamma-aminobutyric acid aminotransferase (GABA-AT; GabT)

15 GAD65 synthesizes gamma-aminobutyric acid, an important autocrine and para

16 idative stress, and is associated with GABA (gamma-aminobutyric acid, an inhibitory neurotransmitter)

17 orized by MOA: sodium channel blockers (SC), gamma-aminobutyric acid analogs (G), synaptic vesicle pr

18 with furosine (2-FM-lysine), 2-furoylmethyl-gamma-aminobutyric acid and 2-FM-arginine were detected.

19 es in some neurotransmitter systems, such as gamma-aminobutyric acid and glutamate, mainly in the cor

20 oride-dependent functional plasticity of the gamma-aminobutyric acid and glycinergic system by target

21 nd delivering neuroactive substances such as gamma-aminobutyric acid and serotonin, which act on the

22 elease the inhibitory neurotransmitter GABA (gamma-aminobutyric acid) and are inhibited by iSPNs and

23 as well as regulating release of glutamate, gamma-aminobutyric acid, and acetylcholine from presynap

24 tivated by l-glutamate (l-Glu), l-aspartate, gamma-aminobutyric acid, and acetylcholine, with l-Glu e

25 Interactions between the endocannabinoid, gamma-aminobutyric acid, and glutamate systems and their

26 biogenesis including synthesis of glutamate/gamma-aminobutyric acid as a potential transcriptional t

27 that the neurons also release glutamate and gamma-aminobutyric acid as cotransmitters, with striking

28 gamma-Aminobutyric acid-A receptor delta and gamma-aminobutyric acid B receptor subunit 1; their diff

29 Emerging evidence suggests that functional gamma-aminobutyric acid B receptors (GABABRs) are expres

30 -4-isoxazolepropionic acid receptor (AMPAR), gamma-aminobutyric acid-B receptor (GABABR), leucine-ric

31 ne receptors, particularly retinoic acid and gamma-aminobutyric acid-B2 receptor signalling, among no

32 Decreased expression of the gamma-aminobutyric acid binding site, a transmembrane do

33 BrdU(+) /GABA(+) (gamma-aminobutyric acid) cells were also found but no ne

34 s and the specific impairment of perisomatic gamma-aminobutyric acid circuits are hallmarks of the sc

35 per and lower limb was associated with lower gamma-aminobutyric acid concentration in the sensorimoto

36 Additionally, the reduced gamma-aminobutyric acid concentration may contribute to

37 n older adults to a likely decrease in GABA (Gamma Aminobutyric Acid) concentration in visual cortex,

38 n models were used to compare differences in gamma-aminobutyric acid concentrations between patients

39 microinjection to the NAcSh decreased local gamma-aminobutyric acid concentrations.

40 Here we report a high GABA (gamma-aminobutyric acid) content in reactive astrocytes

41 Fast glutamate and gamma-aminobutyric acid cotransmission convey discrete p

42 imaged the distribution of neurotransmitters-gamma-aminobutyric acid, dopamine and serotonin-with hig

43 during real-time-corrected three-dimensional gamma-aminobutyric acid-edited magnetic resonance (MR) s

44 h long-range projections that activate local gamma-aminobutyric acid-ergic (GABAergic) circuits.

45 With gamma-aminobutyric acid-ergic interneuron-specific NMDAR

46 a variety of neurotransmitters, such as the gamma-aminobutyric acid-ergic system, the study of prefr

47 large part by direction-selective release of gamma-aminobutyric acid from starburst amacrine cells on

48 major excitatory (glutamate) and inhibitory (gamma aminobutyric acid (GABA)) neurotransmitter circuit

49 Gamma aminobutyric acid (GABA), a neurotransmitter of th

50 Drosophila optic lobes that possibly release gamma aminobutyric acid (GABA), the major inhibitory neu

51 ants changes the number of dopamine (DA)- or gamma aminobutyric acid (GABA)-expressing neurons, with

52 Abnormalities in prefrontal gamma aminobutyric acid (GABA)ergic transmission, partic

53 The effects of gamma-aminobutyric acid (GABA) A receptor activation on

54 determine whether antecedent stimulation of gamma-aminobutyric acid (GABA) A receptors with the benz

55 gamma-Aminobutyric acid (GABA) administration can inhibi

56 Gamma-aminobutyric acid (GABA) and enkephalin tonically

57 atory amino acid neurotransmitter systems of gamma-aminobutyric acid (GABA) and glutamate, respective

58 Levels of gamma-aminobutyric acid (GABA) and glutamic acid decarbo

59 and the inhibitory transmitters include both gamma-aminobutyric acid (GABA) and glycine (GLY).

60 irtual" proton in the decarboxylated product gamma-aminobutyric acid (GABA) and is then exported via

61 levels of glutamate, inhibitory transmitters gamma-aminobutyric acid (GABA) and N-acetylaspartylgluta

62 acid decarboxylase, suggesting that they use gamma-aminobutyric acid (GABA) as their neurotransmitter

63 The deficit reflected weakened gamma-aminobutyric acid (GABA) circuits and compromised

64 find that pairing of acetylcholine (ACh) and gamma-aminobutyric acid (GABA) Comment: Please use the g

65 Gamma-aminobutyric acid (GABA) concentrations and GAD67

66 antihypertensive activity due to their large gamma-aminobutyric acid (GABA) content (6.8-10.6 mg/g) a

67 The inhibitory neurotransmitter gamma-aminobutyric acid (GABA) contributes to the networ

68 his enhanced vulnerability may be related to gamma-aminobutyric acid (GABA) deficits observed in schi

69 nide restored the impaired oxytocin-mediated gamma-aminobutyric acid (GABA) excitatory-inhibitory shi

70 lobe of the cockroach Periplaneta americana, gamma-aminobutyric acid (GABA) has been identified as th

71 istry with neuronal tract-tracing methods or gamma-aminobutyric acid (GABA) immunohistochemistry to b

72 scopy to map histamine (HA), FMRF-amide, and gamma-aminobutyric acid (GABA) immunoreactivity in the c

73 We also compared the distribution of ASP and gamma-aminobutyric acid (GABA) in brainstem neurons by u

74 Abnormally elevated levels of gamma-aminobutyric acid (GABA) in the medial prefrontal

75 rmal behaviors were consistent with abnormal gamma-aminobutyric acid (GABA) interneuron function, whi

76 d-aspartate receptor (NMDAR) hypofunction on gamma-aminobutyric acid (GABA) interneurons disinhibitin

77 Gamma-aminobutyric acid (GABA) is a major inhibitory neu

78 Gamma-aminobutyric acid (GABA) is a widely conserved sig

79 We discovered that gamma-aminobutyric acid (GABA) is co-released with its f

80 The main inhibitory neurotransmitter gamma-aminobutyric acid (GABA) is known to play a pivota

81 ted, the role of VGLUT3 in neurons releasing gamma-aminobutyric acid (GABA) is not settled.

82 gamma-Aminobutyric acid (GABA) is one of the major inhib

83 gamma-Aminobutyric acid (GABA) is the major inhibitory t

84 gamma-Aminobutyric acid (GABA) is the most abundant inhi

85 gamma-Aminobutyric acid (GABA) is the primary inhibitory

86 In contrast, KATPHI islets had low gamma-aminobutyric acid (GABA) levels and lacked (13)C i

87 Low-pressure plasma also increased gamma-aminobutyric acid (GABA) levels from approximately

88 I mice and at that time, postmortem striatal gamma-aminobutyric acid (GABA) levels were elevated and

89 nia, the density of cartridges detectable by gamma-aminobutyric acid (GABA) membrane transporter 1 im

90 Early-born gamma-aminobutyric acid (GABA) neurons (EBGNs) are major

91 R antagonists interfere with the function of gamma-aminobutyric acid (GABA) neurons and alter the bra

92 netic stimulation of mouse zona incerta (ZI) gamma-aminobutyric acid (GABA) neurons or their axonal p

93 at to show that optogenetic inhibition of LH gamma-aminobutyric acid (GABA) neurons restricted to cue

94 d c-fos expression in VTA 5-HT2CR expressing gamma-aminobutyric acid (GABA) neurons, but not 5-HT2CR

95 Aberrant glutamate and gamma-aminobutyric acid (GABA) neurotransmission contrib

96 Gamma-aminobutyric acid (GABA) neurotransmission in the

97 lease either the amino acid (AA) transmitter gamma-aminobutyric acid (GABA) or glutamate.

98 ablished that dopaminergic neurons often use gamma-aminobutyric acid (GABA) or glutamatergic cotransm

99 riments did not confirm colocalizations with gamma-aminobutyric acid (GABA) or the circadian coupling

100 ory responses to hypoglycemia, and increased gamma-aminobutyric acid (GABA) output contributes to cou

101 The non-protein amino acid, gamma-aminobutyric acid (GABA) rapidly accumulates in pl

102 ering neurites are also immunopositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA R

103 Nonselectively reducing intrastriatal gamma-aminobutyric acid (GABA) receptor-A inhibition syn

104 rprisingly, severe antagonists of ionotropic gamma-aminobutyric acid (GABA) receptors.

105 dent drinking, and basal and alcohol-induced gamma-aminobutyric acid (GABA) release in the central nu

106 Our results demonstrate that the gamma-aminobutyric acid (GABA) release is lower and opio

107 We found that local gamma-aminobutyric acid (GABA) release on dendrites of m

108 studies revealed a significant reduction in gamma-aminobutyric acid (GABA) release probability witho

109 oreover, in vitro, neurounina-1 also reduced gamma-aminobutyric acid (GABA) release, enhanced GABA(A)

110 se and nitrate stimulated PGC1alpha-mediated gamma-aminobutyric acid (GABA) secretion from muscle.

111 and exendin-4, a GLP-1 receptor agonist, on gamma-aminobutyric acid (GABA) signaling in hippocampal

112 Altered gamma-aminobutyric acid (GABA) signaling in the prefront

113 f one target, NKCC1, initiates the switch in gamma-aminobutyric acid (GABA) signaling, limits early s

114 c spinal cord to determine whether glutamate-gamma-aminobutyric acid (GABA) switching is cell autonom

115 Deficits in the gamma-aminobutyric acid (GABA) system have been reported

116 The gamma-aminobutyric acid (GABA) system in the nucleus acc

117 is associated with reduced concentrations of gamma-aminobutyric acid (GABA) that are normalized by an

118 additionally stained with antibodies against gamma-aminobutyric acid (GABA) to identify GABAergic int

119 lutamate signaling (P-value=7.22 x 10(-15)), gamma-aminobutyric acid (GABA) transport (P-value=1.36 x

120 f NL3 leads to a large increase in vesicular gamma-aminobutyric acid (GABA) transporter (vGAT) and gl

121 We saw that stimulation of vesicular gamma-aminobutyric acid (GABA) transporter (VGAT)-expres

122 AT-1, encoded by SLC6A1, is one of the major gamma-aminobutyric acid (GABA) transporters in the brain

123 synthesis of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA) using pyridoxal 5'-phosph

124 PFC) levels of glutamate+glutamine (Glx) and gamma-aminobutyric acid (GABA) were measured before, dur

125 d caffeic acids, as well as higher amount of gamma-aminobutyric acid (GABA) were observed in biodynam

126 opy to investigate in vivo concentrations of gamma-aminobutyric acid (GABA) within primary and second

127 important ion channels, gated by glutamate, gamma-aminobutyric acid (GABA), and acetylcholine, also

128 or inhibitory neurotransmitter of the brain, gamma-aminobutyric acid (GABA), can be released through

129 differentially methylated probes implicated gamma-aminobutyric acid (GABA), dopamine and serotonin n

130 ors for various neurotransmitters-glutamate, gamma-aminobutyric acid (GABA), dopamine, serotonin, ace

131 re predicted to form anion channels gated by gamma-aminobutyric acid (GABA), glutamate, histamine, or

132 the major sleep-promoting neurotransmitter, gamma-aminobutyric acid (GABA), in the GABA shunt genera

133 pathway of the inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), in this putative imbalan

134 Low levels of gamma-aminobutyric acid (GABA), one of two major neurotr

135 markers for the inhibitory neurotransmitter gamma-aminobutyric acid (GABA), particularly within the

136 Recently, it has been demonstrated that gamma-aminobutyric acid (GABA), the chief inhibitory neu

137 gamma-Aminobutyric acid (GABA), the major inhibitory neu

138 A considerable increase in gamma-aminobutyric acid (GABA), together with some other

139 germination time and temperature to maximise gamma-aminobutyric acid (GABA), total phenolics compound

140 fast-acting neurotransmitters glutamate and gamma-aminobutyric acid (GABA), two major neurotransmitt

141 sor of a potential lipophilic bioisostere of gamma-aminobutyric acid (GABA), various late-stage diver

142 The inhibitory neurotransmitter, gamma-aminobutyric acid (GABA), was then actively delive

143 spectrometry (MS)-based transport assays for gamma-aminobutyric acid (GABA), which is the major inhib

144 brospinal fluid (CSF)-induced enhancement of gamma-aminobutyric acid (GABA)-A receptor activity was f

145 yl-d-aspartate (NMDA) glutamate receptor and gamma-aminobutyric acid (GABA)-A receptor during progres

146 ated with a positive allosteric modulator of gamma-aminobutyric acid (GABA)-A receptors in cerebrospi

147 how a unique role of GABARAPs, in particular gamma-aminobutyric acid (GABA)-A-receptor-associated pro

148 t thalamus, a well-defined system containing gamma-aminobutyric acid (GABA)-ergic and glutamatergic n

149 mission is likely due to a reduced number of gamma-aminobutyric acid (GABA)-ergic boutons, which may

150 Gamma-aminobutyric acid (GABA)-ergic disturbances are ha

151 hat houses stress-sensitive, PVH-projecting, gamma-aminobutyric acid (GABA)-ergic neurons as represen

152 Dysfunction related to gamma-aminobutyric acid (GABA)-ergic neurotransmission i

153 The regulation of synaptic strength at gamma-aminobutyric acid (GABA)-ergic synapses is depende

154 This study describes the gamma-aminobutyric acid (GABA)-immunoreactive (GABA-ir)

155 Calretinin- and gamma-aminobutyric acid (GABA)-immunoreactive (IR) cells

156 dritic spines received inputs from symmetric gamma-aminobutyric acid (GABA)-immunoreactive terminals,

157 yl-D-aspartate (NMDA) excitation balanced by gamma-Aminobutyric acid (GABA)-mediated inhibition and l

158 ion have higher IFITM levels and deficits in gamma-aminobutyric acid (GABA)-related markers that are

159 At the circuit level, these gamma-aminobutyric acid (GABA)-releasing projections tar

160 vealed that CR-positive terminals lacked the gamma-aminobutyric acid (GABA)-synthesizing enzyme gluta

161 roduction of the inhibitory neurotransmitter gamma-aminobutyric acid (GABA).

162 alterations in regulation of the HPA axis by gamma-aminobutyric acid (GABA).

163 t, with slightly different sensitivities, on gamma-aminobutyric acid (GABA)A , alpha-amino-3-hydroxy-

164 Native gamma-aminobutyric acid (GABA)A receptors consisting of

165 gamma-Aminobutyric acid (GABA)A receptors were blocked b

166 ght to be due to internalization of synaptic gamma-aminobutyric acid (GABA)A receptors, and withdrawa

167 rons is modulated by intrinsic and extrinsic gamma-aminobutyric acid (GABA)ergic and glutamatergic af

168 nized to involve functional deficits in both gamma-aminobutyric acid (GABA)ergic and glutamatergic sy

169 ide exchange factor selectively localized to gamma-aminobutyric acid (GABA)ergic and glycinergic post

170 tamatergic cells and decreased the number of gamma-aminobutyric acid (GABA)ergic cells, whereas (-)Ba

171 Convergent findings indicate that cortical gamma-aminobutyric acid (GABA)ergic circuitry is altered

172 d orexinergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements involved wi

173 d orexinergic systems and is extended to the gamma-aminobutyric acid (GABA)ergic elements of these nu

174 ontains glutamatergic excitatory neurons and gamma-aminobutyric acid (GABA)ergic inhibitory interneur

175 the superior colliculus receives inhibitory gamma-aminobutyric acid (GABA)ergic input from the basal

176 gamma-Aminobutyric acid (GABA)ergic inputs are strategic

177 dial tegmental nucleus (RMTg), which contain gamma-aminobutyric acid (GABA)ergic neurons that mediate

178 cell types (cholinergic, glutamatergic, and gamma-aminobutyric acid (GABA)ergic neurons) across its

179 olinergic but also include a small number of gamma-aminobutyric acid (GABA)ergic neurons.

180 wn to potentiate glutamatergic and attenuate gamma-aminobutyric acid (GABA)ergic neurotransmission, a

181 l prefrontal cortex (mPFC) glutamatergic and gamma-aminobutyric acid (GABA)ergic receptors in control

182 cortex underlies the developmental shift in gamma-aminobutyric acid (GABA)ergic responses, whereas t

183 entified a form of long-term potentiation of gamma-aminobutyric acid (GABA)ergic synapses on these ne

184 embrane, and are also sites of clustering of gamma-aminobutyric acid (GABA)ergic synapses.

185 for cognitive alterations and damage to the gamma-aminobutyric acid (GABA)ergic system in the hippoc

186 hout the retina, the ratio of glycinergic to gamma-aminobutyric acid (GABA)ergic to amacrine cells re

187 synaptic connectivity and function of a few gamma-aminobutyric acid (GABA)ergic wide-field amacrine

188 s present solely in neurons, specifically in gamma-aminobutyric acid (GABA)ergic, glutamatergic, and

189 the wheel-running performances of wildtype (gamma-aminobutyric acid [GABA]-CB(1)(+)/(+)) and mutant

190 ivities between major neurochemical classes (gamma-aminobutyric acid [GABA]/glycine ACs > glycine ACs

191 f excitatory (glutamatergic) and inhibitory (gamma-aminobutyric acid [GABA]ergic) neurons of the pref

192 The gamma-aminobutyric acid (GABAA) receptor gamma2 subunit

193 associated with mutations in the inhibitory gamma-aminobutyric acid (GABAA) receptor gamma2 subunit

194 nanolone, a positive allosteric modulator of gamma-aminobutyric acid (GABAA) receptors, for the treat

195 izing synaptic inhibition mediated by type A gamma-aminobutyric acid (GABAA) receptors, which are Cl(

196 Ionotropic receptors of gamma-aminobutyric acid (GABAAR) regulate neuronal inhib

197 n with altered brain levels of glutamate and gamma-aminobutyric acid have been identified in both ani

198 this study, we examined the distribution of gamma-aminobutyric-acid;-immunoreactive (GABA-ir) neuron

199 The neuropil organization and the pattern of gamma-aminobutyric acid immunostaining of the medulla an

200 rticocortical axons and normal intracortical gamma-aminobutyric acid inhibition in contrast with what

201 Gamma-aminobutyric acid is the principle inhibitory neur

202 ficant differences in prefrontal or striatal gamma-aminobutyric acid level.

203 he aims of this study were to investigate if gamma-aminobutyric acid levels (i) are abnormal in patie

204 Specifically for each unit decrease in gamma-aminobutyric acid levels (in mM), there was a pred

205 Patients had significantly lower gamma-aminobutyric acid levels in the hippocampus (adjus

206 glutamate + glutamine, N-acetylaspartate, or gamma-aminobutyric acid levels in the left DLPFC.

207 c resonance spectroscopy at 3 T, to quantify gamma-aminobutyric acid levels in the prefrontal cortex,

208 This study suggests that reduced gamma-aminobutyric acid levels reflect pathological abno

209 gamma-aminobutyric acid-mediated (GABAergic) inhibition

210 en used to assess cortical glutamatergic and gamma-aminobutyric acid-mediated tone in adults with maj

211 The gamma-aminobutyric acid modulator propofol induces neuro

212 Mice lacking GIRK2 in gamma-aminobutyric acid neurons (GAD-Cre:Girk2(flox/flox

213 rons and a specific population of inhibitory gamma-aminobutyric acid neurons (i.e., parvalbumin-conta

214 RK-dependent signaling in dorsal hippocampal gamma-aminobutyric acid neurons but no evident behaviora

215 y exciting and inhibiting neighbouring GABA (gamma-aminobutyric acid) neurons in the ventral tegmenta

216 s in the pre- and postsynaptic components of gamma-aminobutyric acid neurotransmission and in the den

217 s study supports the idea that modulation of gamma-aminobutyric acid neurotransmission may be an impo

218 ines of evidence implicate the glutamate and gamma-aminobutyric acid neurotransmitter systems in the

219 discovered cyanobacterial TCA cycle (via the gamma-aminobutyric acid pathway or alpha-ketoglutarate d

220 oxication causes changes in the rodent brain gamma-aminobutyric acid receptor (GABAAR) subunit compos

221 Mechanisms controlling the metabotropic gamma-aminobutyric acid receptor (GABAB) cell surface st

222 t rapid antidepressants cause a shift in the gamma-aminobutyric acid receptor (GABABR) signaling path

223 ange the type of excitability: a depolarized gamma-Aminobutyric acid receptor (GABAR) reversal potent

224 ethyl-d-aspartate receptor in 4 patients and gamma-aminobutyric acid receptor A in 1 patient of 111 p

225 piramate-a glutamate receptor antagonist and gamma-aminobutyric acid receptor agonist-would result in

226 ministration model, we studied glutamate and gamma-aminobutyric acid receptor regulation in the synap

227 s, and behavioral results suggest that local gamma-aminobutyric acid receptor signaling mediates the

228 g amylin's effects on energy balance through gamma-aminobutyric acid receptor signaling.

229 The gamma-aminobutyric acid receptor-associated protein (GAB

230 of reduced lysosomal degradation of RhoB in Gamma-aminobutyric acid receptor-associated protein (GAB

231 le-associated protein 1 light chain 3 (LC3), gamma-aminobutyric acid receptor-associated protein (GAB

232 etylcholine receptor for neonicotinoids, the gamma-aminobutyric acid receptor/chloride channel for po

233 onal cell-surface antibodies, mainly against gamma-aminobutyric acid receptors (53% vs 11%; P < .001)

234 cently become appreciated that activation of gamma-aminobutyric acid receptors (GABA-Rs) on ss-cells

235 Type-A gamma-aminobutyric acid receptors (GABAARs) are the prin

236 The accumulation of gamma-aminobutyric acid receptors (GABAARs) at the appro

237 cient synaptic inhibition upon activation of gamma-aminobutyric acid receptors.

238 also includes the metabotropic glutamate and gamma-aminobutyric acid receptors.

239 Specifically, deficits in inhibitory gamma-aminobutyric acid regulating excitatory cell input

240 tor (CB1R); 3) binds to CB1R, which inhibits gamma-aminobutyric acid release from the cholecystokinin

241 in mouse brain, prevented ethanol-stimulated gamma-aminobutyric acid release in the central amygdala,

242 Elevated gamma-aminobutyric acid release under chronic stress is

243 arachidonoylglycerol-dependent inhibition of gamma-aminobutyric acid release without altering postsyn

244 ts that chronic stress increases presynaptic gamma-aminobutyric acid release.

245 ting cell types include hypocretin and GABA (gamma-aminobutyric-acid)-releasing neurons of the latera

246 inly result from the synchronous activity of gamma-aminobutyric acid-releasing cells.

247 Diverse gamma-aminobutyric acid-releasing interneurons regulate

248 events including the migration of GABAergic (gamma-aminobutyric-acid-releasing) neurons from ventral

249 Here we show that a GABAergic (gamma-aminobutyric-acid-releasing) pathway originating f

250 inhibition is mediated largely by GABAergic (gamma-aminobutyric acid-secreting) interneurons, a cell

251 Moreover, up-regulation of the gamma-aminobutyric acid shunt and alanine metabolism exp

252 succinate-semialdehyde dehydrogenase in the gamma-aminobutyric acid shunt pathway and an aconitase f

253 e, fermentation, alanine metabolism, and the gamma-aminobutyric acid shunt, while [(13)C]glutamate an

254 ce can be temporarily reverted by inhibiting gamma-aminobutyric acid signaling but not by a Ras-ERK b

255 This results in delayed gamma-aminobutyric acid switch and higher susceptibility

256 mental process, the excitatory-to-inhibitory gamma-aminobutyric acid switch; defects in this switch h

257 Immunohistochemistry for the gamma aminobutyric acid synthesizing enzyme glutamic aci

258 sential tremor may be neurodegenerative; low gamma aminobutyric acid tone seems to be a central featu

259 uits treated with melatonin exhibited higher gamma-aminobutyric acid transaminase (GABA-T) enzyme act

260 elevates the CG7433 protein, a mitochondrial gamma-aminobutyric acid transaminase (GABAT), and reduce

261 tion in prefrontal signaling pathways (e.g., gamma-aminobutyric acid transmission) and neural network

262 ansmission or pharmacological enhancement of gamma-aminobutyric acid transmission.

263 : inhibitory synaptic currents and vesicular gamma-aminobutyric acid transporter (vGAT) staining inte

264 Using vesicular gamma-aminobutyric acid transporter-channelrhodopsin 2-e

265 olarising synaptic inhibition through GABAA (gamma aminobutyric acid type A) and glycine receptors de

266 on [Cl(-)](i) is an important determinant of gamma-aminobutyric acid type A (GABA(A)) receptor (GABA(

267 Benzodiazepines modulate gamma-aminobutyric acid type A (GABA(A)) receptors throu

268 potential firing by local application of the gamma-aminobutyric acid type A (GABA-A) agonist muscimol

269 tly identified four de novo mutations in the gamma-aminobutyric acid type A (GABAA ) receptor beta3 s

270 We identified a cysteine substituted mutant gamma-aminobutyric acid type A (GABAA) receptor with uni

271 Proteostasis maintenance of gamma-aminobutyric acid type A (GABAA) receptors dictate

272 nt-neurosteroids at the same binding site on gamma-aminobutyric acid type A (GABAA) receptors was eva

273 OEt) displayed micromolar affinity for brain gamma-aminobutyric acid type A (GABAA) receptors.

274 The extrasynaptic alpha-5 gamma-aminobutyric acid type A receptor (alpha5-GABAAR)

275 ctive steroids are efficacious modulators of gamma-aminobutyric acid type A receptor (GABA(A)) recept

276 The gamma-aminobutyric acid type A receptor (GABAA-R) is a m

277 -RICS-deficient neurons show reduced surface gamma-aminobutyric acid type A receptor (GABAAR) levels

278 cotransporter expression and impaired spinal gamma-aminobutyric acid type A receptor function, indica

279 However, DCS-LTD persists despite either gamma-aminobutyric acid type A receptor or N-methyl-D-as

280 f genetic and biological evidence implicates gamma-aminobutyric acid type A receptor subunits on chro

281 contributor to the central disinhibition of gamma-aminobutyric acid type A receptor- and glycine rec

282 Alpha-5 gamma-aminobutyric acid type A receptors (alpha5-GABAARs

283 ttributed primarily to increased activity of gamma-aminobutyric acid type A receptors (GABA(A)Rs), an

284 oop 2 region of glycine receptors (GlyRs) or gamma-aminobutyric acid type A receptors (GABA(A)Rs), wh

285 oid type 1 receptor) receptors and modulates gamma-aminobutyric acid type A receptors (GABAAR).

286 cotinic acetylcholine receptors (nAChRs) and gamma-aminobutyric acid type A receptors (GABAARs) are m

287 ed that act as convulsants and inhibitors of gamma-aminobutyric acid type A receptors (GABAARs) rathe

288 l acts as a positive allosteric modulator of gamma-aminobutyric acid type A receptors (GABAARs), an i

289 Extrasynaptic gamma-aminobutyric acid type A receptors (GABAARs),which

290 nic inhibitory neurotransmission mediated by gamma-aminobutyric acid type A receptors (GABAARs).

291 g of the scaffolding protein Gephyrin and of gamma-aminobutyric acid type A receptors at inhibitory n

292 is well known for benzodiazepines acting at gamma-aminobutyric acid type A receptors.

293 ibution of synaptic acetylcholine and GABAA (gamma-aminobutyric acid type A) receptors into extrasyna

294 gamma-Aminobutyric acid type B (GABAB) receptor autoanti

295 Autoantibodies to the gamma-aminobutyric acid type B (GABAB) receptor have rec

296 The gamma-aminobutyric acid type B (GABAB) receptor undergoe

297 sustained neuronal inhibition is mediated by gamma-aminobutyric acid type B (GABAB) receptors, which

298 Ethanol, like rapid antidepressants, alters gamma-aminobutyric acid type B receptor (GABABR) express

299 gamma-Aminobutyric acid type B receptor autoimmunity des

300 The gamma-aminobutyric acid type B-receptor agonist lesogabe