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1 cient synaptic inhibition upon activation of gamma-aminobutyric acid receptors.
2 also includes the metabotropic glutamate and gamma-aminobutyric acid receptors.
3 dopamine and muscarinic cholinergic, but not gamma-aminobutyric acid receptors.
4 by picrotoxin, an antagonist of the GABA(A) (gamma-aminobutyric acid) receptor.
5 entiate neuronal inhibition through GABA(A) (gamma-aminobutyric acid) receptors.
6 onal cell-surface antibodies, mainly against gamma-aminobutyric acid receptors (53% vs 11%; P < .001)
7 ethyl-d-aspartate receptor in 4 patients and gamma-aminobutyric acid receptor A in 1 patient of 111 p
10 piramate-a glutamate receptor antagonist and gamma-aminobutyric acid receptor agonist-would result in
11 he known subunit stoichiometry of the A-type gamma-aminobutyric acid receptor allowed us to create re
12 enhance inhibitory transmission mediated by gamma-aminobutyric acid receptors and have anticonvulsan
14 hreshold dose of picrotoxin, a use-dependent gamma-aminobutyric acid receptor antagonist, reduces cor
16 ter-operated ion channels, such as the GABA (gamma-aminobutyric acid) receptor, are important in fast
17 of reduced lysosomal degradation of RhoB in Gamma-aminobutyric acid receptor-associated protein (GAB
19 le-associated protein 1 light chain 3 (LC3), gamma-aminobutyric acid receptor-associated protein (GAB
22 by disinhibition because local antagonism of gamma-aminobutyric acid receptors blocked responses to C
23 etylcholine receptor for neonicotinoids, the gamma-aminobutyric acid receptor/chloride channel for po
24 and pharmacological evidence that ionotropic gamma-aminobutyric acid receptors contribute to fluid tr
25 +/- 9% enhancement) than it was at enhancing gamma-aminobutyric acid receptor current (3 +/- 3% enhan
26 n N-methyl-D-aspartate receptor currents and gamma-aminobutyric acid receptor currents within the sam
27 E cells juxtaposed next to cohorts of normal gamma-aminobutyric acid receptor expressing secretory ce
28 nscriptional control of the mammalian type A gamma-aminobutyric acid receptor (GABA(A)R) subunit gene
30 the Ca(2+)-sensing receptor (CaR) and type B gamma-aminobutyric acid receptor (GABA-B-R) from human e
34 cently become appreciated that activation of gamma-aminobutyric acid receptors (GABA-Rs) on ss-cells
35 oxication causes changes in the rodent brain gamma-aminobutyric acid receptor (GABAAR) subunit compos
37 stsynaptic potentials mediated by ionotropic gamma-aminobutyric acid receptors (GABAARs) and glycine
41 t rapid antidepressants cause a shift in the gamma-aminobutyric acid receptor (GABABR) signaling path
43 ange the type of excitability: a depolarized gamma-Aminobutyric acid receptor (GABAR) reversal potent
47 igated the role of regionally discrete GABA (gamma-aminobutyric acid) receptors in the sedative respo
48 n of unknown function isolated previously as gamma-aminobutyric acid receptor-interacting factor 1 (G
50 macologically distinct ionotropic receptors: gamma-aminobutyric acid receptors, levamisole-sensitive
51 The effect is caused by a decrease in type A gamma-aminobutyric acid receptor-mediated inhibition of
53 sed by cocaine-induced reduction of GABA(A) (gamma-aminobutyric acid) receptor-mediated inhibition of
55 ated the expression of functional ionotropic gamma-aminobutyric acid receptors on the apical plasma m
57 ministration model, we studied glutamate and gamma-aminobutyric acid receptor regulation in the synap
58 lar endothelial growth factor, integrin, and gamma-aminobutyric acid receptor signaling cascades, plu
59 s, and behavioral results suggest that local gamma-aminobutyric acid receptor signaling mediates the
61 ficant association with polymorphisms in the gamma-aminobutyric acid receptor subunit B3 gene (GABRB3
62 on chromosome 15 to an area surrounding the gamma-aminobutyric acid-receptor subunit genes, in AutD,
63 ved from studies of anesthetic resistance in gamma aminobutyric acid receptors, tandem pore potassium
64 inactivation of BLA by microinfusion of the gamma-aminobutyric acid receptor type A agonist muscimol
65 physiological studies have demonstrated that gamma-aminobutyric acid receptors type A (GABA(A)) media
66 steroids on neural transmission mediated by gamma-aminobutyric acid receptors within forebrain neuro
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