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1 is brought about by the action of the enzyme gamma-glutamyl transpeptidase.
2 d periplasmic fibers; it contains urease and gamma-glutamyl transpeptidase.
5 rase (39.9 +/- 28.6U/L vs 23.8 +/- 14.1U/L), gamma-glutamyl transpeptidase (34.3 +/- 16.6 vs 24.5 +/-
6 r demonstrated higher activity of the enzyme gamma-glutamyl transpeptidase, a membrane-bound enzyme i
7 : HR, 1.87; 95% CI, 1.10; 3.18; P = 0.021); gamma-glutamyl transpeptidase above the upper limit of n
9 the influence of Tat on glutathione levels, gamma-glutamyl transpeptidase activity, and the expressi
13 ids transfer, alkaline phosphatase activity, gamma-glutamyl-transpeptidase activity and physiological
14 ficant increases in the urinary excretion of gamma-glutamyl transpeptidase, alkaline phosphatase, lac
16 2-fold increases in the urinary excretion of gamma-glutamyl transpeptidase and alkaline phosphatase,
17 ductular structures, which are positive for gamma-glutamyl transpeptidase and CK-14 and CK-19 and do
18 ression of two cholangiocyte-specific genes (gamma-glutamyl transpeptidase and cytokeratin 19) was si
19 dex less than 30 kg/m(2), genotype 2, normal gamma-glutamyl transpeptidase and increased alanine amin
20 had lower concentrations of the liver enzyme gamma-glutamyl transpeptidase and lower scores on a meas
21 ine aminotransferases, alkaline phosphatase, gamma glutamyl transpeptidase, and homeostasis model ass
22 utilization, thermostable carboxypeptidase, gamma glutamyl-transpeptidase, and deblocking aminopepti
23 50 mg, significantly reduced levels of ALP, gamma-glutamyl transpeptidase, and alanine aminotransfer
24 concentrations of alanine aminotransferase, gamma-glutamyl transpeptidase, and alkaline phosphatase.
25 ed to the GSH usage (glutathione peroxidase, gamma-glutamyl transpeptidase, and glutathione S-transfe
26 d levels of catalase, glutathione reductase, gamma-glutamyl transpeptidase, and glutathione synthetas
27 pression of catalase, glutathione reductase, gamma-glutamyl transpeptidase, and glutathione synthetas
29 tathione S-transferase pi 7, and for luminal gamma-glutamyl transpeptidase, but they did not express
31 nary concentrations of alkaline phosphatase, gamma-glutamyl transpeptidase, cystatin C, neutrophil ge
33 t pump (BSEP) disease, and 4 others with low gamma-glutamyl transpeptidase disease (levels <100 U/L),
34 nositol-anchored proteins, and transmembrane gamma-glutamyl transpeptidase exhibited the expected api
35 after 5 days liver histology was normal, but gamma-glutamyl transpeptidase expression was observed, w
36 soglutathione (GSNO), and GSNO activation by gamma-glutamyl transpeptidase (gamma-GT) is required.
37 to glutamyl adducts that could be cleaved by gamma-glutamyl transpeptidase (gamma-GT), found predomin
42 at two H. pylori virulence determinants, the gamma-glutamyl transpeptidase GGT and the vacuolating cy
43 Two H. pylori persistence determinants, the gamma-glutamyl-transpeptidase GGT and the vacuolating cy
45 surrogate markers of NAFLD, such as elevated gamma glutamyl transpeptidase (GGT) and alanine aminotra
47 cury-glutathione complex by the ectoproteins gamma-glutamyl transpeptidase (GGT) and dipeptidase.
49 phosphatase, alanine transaminase (ALT), and gamma-glutamyl transpeptidase (GGT) as well as histologi
51 ly we identified an additional member of the gamma-glutamyl transpeptidase (GGT) family, gamma-glutam
52 nase (GGL), a newly identified member of the gamma-glutamyl transpeptidase (GGT) family, we generated
54 in either CagA, VacA, lipopolysaccharide, or gamma-glutamyl transpeptidase (GGT) implicated the latte
55 rone are capable of altering the activity of gamma-glutamyl transpeptidase (GGT) in human prostate ca
62 vated ALT, aspartate transaminase (AST), and gamma-glutamyl transpeptidase (GGT) levels (P <.01).
66 Histochemical evaluation, via staining for gamma-glutamyl transpeptidase (GGT), a marker for dyspla
67 (EHS) and H. pylori These include flagellin, gamma-glutamyl transpeptidase (ggt), collagenase, the se
68 they also expressed alpha-fetoprotein (AFP), gamma-glutamyl transpeptidase (GGT), cytokeratin 19 (CK-
69 SH is degraded by the sequential reaction of gamma-glutamyl transpeptidase (GGT), gamma-glutamyl cycl
70 Acivicin, which inhibits GSH breakdown by gamma-glutamyl transpeptidase (GGT), had no effect on th
72 ansferase (ALT), alkaline phosphatase (ALP), gamma-glutamyl transpeptidase (GGT), or total bilirubin.
73 d the metabolism of leukotriene C4 (LTC4) in gamma-glutamyl transpeptidase (GGT)-deficient mice and h
74 m of the cisplatin-glutathione complex via a gamma-glutamyl transpeptidase (GGT)-dependent pathway in
77 Furthermore, serial sections stained for gamma-glutamyl-transpeptidase (GGT, a marker of fetal he
79 ase [ALT], aspartate aminotransferase [AST], gamma-glutamyl transpeptidase [GGT], alkaline phosphatas
80 mma-glutamyl compounds have been identified: gamma-glutamyl transpeptidase (GGT1) and gamma-glutamyl
83 e proximal tubule under the direction of the gamma-glutamyl transpeptidase-I promoter were developed.
84 otransferase, aspartate aminotransferase, or gamma-glutamyl transpeptidase in donors with HGV infecti
86 ter surgery, urinary protein and creatinine, gamma-glutamyl transpeptidase, lactate dehydrogenase, hi
87 creened using a blood alcohol concentration, gamma glutamyl transpeptidase level, and short Michigan
88 linking induces greater phosphorylation, and gamma-glutamyl transpeptidase levels are reduced compare
89 ough RBC count, mean corpuscular volume, and gamma-glutamyl transpeptidase levels remained significan
91 nder, higher body mass index, triglycerides, gamma-glutamyl transpeptidase, maximum alanine aminotran
93 of alkaline phosphatase of 75.6%; P<0.0001; gamma-glutamyl transpeptidase of 117.9%, P<0.0001; bilir
94 including gamma-glutamylcysteine synthetase, gamma-glutamyl transpeptidase, or multidrug resistance p
95 .002 for ferritin) and fibrosis (P<.0001 for gamma-glutamyl transpeptidase, P=.01 for alkaline phosph
96 ll be a useful animal model for the study of gamma-glutamyl transpeptidase physiology and glutathione
97 mine; 2) loss of TA1 response to arginine in gamma-glutamyl transpeptidase-positive transformed and t
98 ubunits Dug1, Dug2, and Dug3) but not by the gamma-glutamyl-transpeptidase, raising the question of t
100 the presence of excess CapD, a B. anthracis gamma-glutamyl transpeptidase, the protective capsule is
101 lso believed to function consecutively after gamma-glutamyl transpeptidase to cleave cystinyl-bis-gly
102 bypasses the need for GSNO bioactivation by gamma-glutamyl transpeptidase to increase CFTR maturatio
103 is showed that pretreatment body mass index, gamma-glutamyl transpeptidase, triglyceride, IL-28B TT g
104 3-4 adverse events were increased levels of gamma-glutamyl transpeptidase (two [4%]), a reduction in
106 ts; mean corpuscular volume, SGOT, SGPT, and gamma-glutamyl transpeptidase were significantly higher.
108 bile duct-like structures were positive for gamma-glutamyl transpeptidase, which suggests that they
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