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1 minase, alkaline phosphatase, bilirubin, and gamma-glutamyltransferase).
2 atory measure of alcohol consumption (plasma gamma-glutamyltransferase).
3 occurring in >3% of patients) were increased gamma-glutamyltransferase (14 [10%] patients in the idal
4 d with -7.94 (-11.1, -4.8) IU/L; P < 0.001], gamma-glutamyltransferase [-15.08 (-15.5, -14.7) compare
6 resolved, as evidenced by lower median (IQR) gamma-glutamyltransferase [54 U/L (41, 103 U/L) compared
7 trast, elevated alanine aminotransferase and gamma glutamyltransferase activities were associated wit
8 oups had significant reductions in levels of gamma-glutamyltransferase and alanine aminotransferase a
9 also associated with lower plasma levels of gamma-glutamyltransferase and alkaline phosphatase than
10 er previously purified T. denticola enzymes, gamma-glutamyltransferase and cystalysin, CGase mediates
13 athione and cysteine through upregulation of gamma-glutamyltransferases and transcriptional repressio
14 ds, high-sensitivity C-reactive protein, and gamma-glutamyltransferase) and novel (adiponectin, E-sel
15 5), alanine aminotransferase (p < or =.001), gamma-glutamyltransferase, and bilirubin (p < or =.05) (
17 nsitivity C-reactive protein, triglycerides, gamma-glutamyltransferase, and fatty liver, whereas fast
18 minotransferase; aspartate aminotransferase; gamma-glutamyltransferase; and, in women, C-reactive pro
19 n for AST (coefficient of variation, 13.9%), gamma-glutamyltransferase (coefficient of variation, 13.
20 of 113 in the chemotherapy group), increased gamma glutamyltransferase concentration (24 [21%] vs one
21 sting energy expenditure increased and serum gamma-glutamyltransferase concentrations decreased in bo
23 roposed to occur via a three enzyme pathway, gamma-glutamyltransferase, cysteinylglycinase (CGase), a
24 proteoliposomes have been prepared in which gamma-glutamyltransferase (EC 2.3.2.2) was functionally
25 ntribution of glutamyl transpeptidase (GGT) (gamma-glutamyltransferase [EC 2.2.2]) to Helicobacter py
26 ions in one patient and a transitory grade 3 gamma-glutamyltransferase elevation in another patient.
28 markers, such as liver markers (reflected by gamma-glutamyltransferase, fetuin-A, and sex hormone-bin
29 erase (AST), alanine aminotransferase (ALT), gamma glutamyltransferase (gammaGT), lactate dehydrogena
30 ferase (ALT), alkaline phosphatase (ALP) and gamma glutamyltransferase (GGT), on diabetes and cardiov
31 ted serum alanine aminotransferase (ALT) and gamma-glutamyltransferase (GGT) activities are markers o
33 exons generates unique noncoding 5'-ends for gamma-glutamyltransferase (GGT) cDNAs in several species
34 lved in the strong dose-response relation of gamma-glutamyltransferase (GGT) concentration with incid
35 (ALT), aspartate aminotransferase (AST), and gamma-glutamyltransferase (GGT) elevation was defined as
38 lecting liver fat accumulation [reflected by gamma-glutamyltransferase (GGT), alanine transaminase (A
39 formed values of alanine transaminase (ALT), gamma-glutamyltransferase (GGT), and direct bilirubin on
40 ow-density lipoprotein cholesterol, glucose, gamma-glutamyltransferase (GGT), and hsCRP, as compared
41 ansferase, aspartate aminotransferase (AST), gamma-glutamyltransferase (GGT), and total bilirubin, wh
42 (NAFLD) alanine aminotransferase (ALT), and gamma-glutamyltransferase (GGT), with 3 separate measure
44 The enzyme responsible for this catalysis (gamma-glutamyltransferase [GGT]; EC 2.3.2.2) was purifie
46 colorimetric method to measure GS-catalyzed gamma-glutamyltransferase (GT) activity in rat L6 skelet
47 ith long-acting pasireotide monotherapy were gamma-glutamyltransferase increased (four [10%] of 41 pa
48 ferase level (-10.9 vs -36.2 u/L; P = .009), gamma-glutamyltransferase level (-9.4 vs -41.2 u/L; P =
49 % confidence interval [CI], 1.004-1.031) and gamma-glutamyltransferase level (OR, 1.016 per U/L; 95%
50 phatase level less than or equal to 240 U/L, gamma-glutamyltransferase level less than or equal to 13
51 ange, 5-45 U/L [0.08-0.75 mukat/L]), a serum gamma-glutamyltransferase level of 210 U/L (3.50 mukat/L
53 aminotransferase, alkaline phosphatase, and gamma-glutamyltransferase levels and the decrease in cal
54 In alcohol-dependent participants, 24-month gamma-glutamyltransferase levels were lower in TEL than
55 ruritus, intrahepatic cholestasis, low serum gamma-glutamyltransferase levels, and characteristic "By
56 nia (two [8%] in each cohort), and increased gamma-glutamyltransferase (one [4%] in cohort 1, three [
57 ing toxicities included hematuria, increased gamma-glutamyltransferase or ALT, insomnia, and nausea/v
58 abnormal concentrations of the liver enzymes gamma-glutamyltransferase (OR per 1-SD increase in BPA c
59 er biopsy specimens from 3 children with low gamma-glutamyltransferase PFIC before and after PEBD.
60 orrelated with liver enzymes, in particular, gamma-glutamyltransferase (R = 0.70), independent of bod
61 ression) in arachidonate 15-lipoxygenase and gamma-glutamyltransferase transcripts were demonstrated.
62 s, while accounting for measurement error in gamma-glutamyltransferase, using data collected in the C
63 .7 IU/L to 25.1 +/- 20 IU/L); mean levels of gamma-glutamyltransferases were reduced from 51 IU/L bef
64 ic alanine or aspartate aminotransferase, or gamma-glutamyltransferase, without increased bilirubin,
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