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1 tor, monocyte chemoattractant protein 1, and gamma interferon.
2 e I interferons (IFN-I), interleukin 12, and gamma interferon.
3 reas C4 synthesis occurred on treatment with gamma interferon.
4 by 28 dpi except interleukin 12 (IL-12) and gamma interferon.
5 ved coma scores, as well as higher levels of gamma interferon.
6 in vitro when chlamydiae are incubated with gamma interferon.
7 se and cationic amino acid transporter 2 via gamma interferon.
8 MCP-1, MCP-2, MCP-3, and monokine induced by gamma-interferon.
9 n 17 and tumor necrosis factor-alpha but not gamma-interferon.
10 ursting during transcriptional activation by gamma-interferon.
11 induction of CD25, CD69, interleukin-2, and gamma-interferon.
14 ecruitment to a region containing a putative gamma-interferon activation sequence (GAS) element at -4
16 t CTLs failed to upregulate FasL and produce gamma interferon after engagement with target cells and
17 ad elevated splenic CD4(+) T cells producing gamma interferon and interleukin-17A, indicating that PN
18 boratory HIV database (1,300 peptides) using gamma interferon and interleukin-2 (IFN-gamma/IL-2) Fluo
19 e, suggesting Th1 polarization, and produced gamma interferon and other cytokines after reactivation
23 L)-17A secretion by Th17 cells, 2) inhibited gamma-interferon and tumor necrosis factor alpha secreti
25 tein 2, RANTES, tumor necrosis factor alpha, gamma interferon, and interleukin-10 were upregulated in
26 L10 [gamma interferon-inducible protein 10], gamma interferon, and lambda interferon) in association
27 ach of these strains was also used to infect gamma interferon- and lipopolysaccharide-activated murin
29 cing T cells that antagonized the release of gamma-interferon by spontaneously arising GAD65 autoimmu
30 actic protein-1/CCL2 and monokine induced by gamma interferon/CXCL9 in the hypotensive and normotensi
31 WT, interleukin-4-deficient (IL-4(-/-)), and gamma interferon-deficient (IFN-gamma(-/-)) C57BL/6 mice
33 inal wash antibodies and an antigen-specific gamma interferon-dominated Th1-biased T cell response.
35 eptides from rDEN2Delta30 and used them in a gamma interferon enzyme-linked immunosorbent spot assay
37 etermined via a newly established guinea pig gamma interferon enzyme-linked immunosorbent spot assay.
39 ear cells from these donors were screened in gamma interferon enzyme-linked immunosorbent spot assays
40 tibility complex (MHC) tetramer staining and gamma interferon enzyme-linked immunospot (ELISPOT) assa
41 or cytokines/chemokines (IL-2, IL-12, IL-17, gamma interferon, granulocyte-macrophage colony-stimulat
42 Resistance to Toxoplasma is dependent on gamma interferon (IFN-gamma) activation of both hematopo
44 Affymetrix microarray analysis revealed that gamma interferon (IFN-gamma) and 16 antiviral interferon
45 hat Brucella-specific CD8(+) T cells express gamma interferon (IFN-gamma) and can transition to long-
48 function, defined by increased production of gamma interferon (IFN-gamma) and granzyme B and expressi
49 CNS CD4 T cells was associated with reduced gamma interferon (IFN-gamma) and granzyme B expression b
50 e show that activated CD8(+) T cells express gamma interferon (IFN-gamma) and granzymes but that gran
51 splenic T cells secreted significantly more gamma interferon (IFN-gamma) and had significantly more
54 elper-1 (Th1), Th2, and Th17 cells, elevated gamma interferon (IFN-gamma) and interleukin (IL)-17 pro
55 enged with HHV-6 preparations indicated that gamma interferon (IFN-gamma) and interleukin-10 (IL-10)
58 nity to acute infection is the production of gamma interferon (IFN-gamma) and interleukin-12 (IL-12),
59 xpression of inflammatory markers, including gamma interferon (IFN-gamma) and interleukin-12p40 (IL-1
60 e to that provoked by LVS but with increased gamma interferon (IFN-gamma) and interleukin-17A (IL-17A
63 associated with NK cells and DCs, including gamma interferon (IFN-gamma) and RANTES, were increased
65 tumor necrosis factor alpha (TNF-alpha), and gamma interferon (IFN-gamma) and the memory markers CD27
66 ting a cytokine storm by their production of gamma interferon (IFN-gamma) and tumor necrosis factor a
67 ociated with increased production of mucosal gamma interferon (IFN-gamma) and tumor necrosis factor a
68 sion is induced by proinflammatory cytokines gamma interferon (IFN-gamma) and tumor necrosis factor a
69 YopE69-77-specific CD8(+) T cells producing gamma interferon (IFN-gamma) and tumor necrosis factor a
70 Th1, Th2, and Th17 cytokines; however, early gamma interferon (IFN-gamma) and tumor necrosis factor a
72 dged by low cytotoxic function and decreased gamma interferon (IFN-gamma) and tumor necrosis factor a
73 ic CD8(+) T lymphocytes capable of producing gamma interferon (IFN-gamma) and tumor necrosis factor a
75 ory CD8(+) T cells that were able to secrete gamma interferon (IFN-gamma) and tumor necrosis factor a
76 tion of interleukin-17A (IL-17A), IL-22, and gamma interferon (IFN-gamma) as well as the antimicrobia
77 mice were also completely unable to produce gamma interferon (IFN-gamma) at any time points followin
78 ty of NK cells to proliferate and to produce gamma interferon (IFN-gamma) but positively associated w
79 monstrated that the functional production of gamma interferon (IFN-gamma) by antigen-specific CD4(+)
81 to amplified T cell activation, higher serum gamma interferon (IFN-gamma) concentrations, enhanced in
83 s in vivo, wild-type mice were injected with gamma interferon (IFN-gamma) DNA or colony-stimulating f
86 on in BALB/c mice, whereas neutralization of gamma interferon (IFN-gamma) enhanced protection in C57B
87 ested for responses against HERV peptides in gamma interferon (IFN-gamma) enzyme immunospot (ELISPOT)
88 saic immunogens induced significantly higher gamma interferon (IFN-gamma) enzyme-linked immunosorbent
89 m HIV-1 Gag and Env were used as antigens in gamma interferon (IFN-gamma) enzyme-linked immunosorbent
90 y enzyme-linked immunosorbent assay (ELISA), gamma interferon (IFN-gamma) enzyme-linked immunosorbent
91 e transcriptases (RT), were identified using gamma interferon (IFN-gamma) enzyme-linked immunosorbent
92 e-specific T-cell activation was measured by gamma interferon (IFN-gamma) enzyme-linked immunosorbent
93 ial epitope targeting rates, as confirmed by gamma interferon (IFN-gamma) enzyme-linked immunosorbent
94 ith primary HIV-1 infection were screened by gamma interferon (IFN-gamma) enzyme-linked immunospot (E
95 topes targeted, measured directly ex vivo by gamma interferon (IFN-gamma) enzyme-linked immunospot (E
96 +) T cell responses defined by full-proteome gamma interferon (IFN-gamma) enzyme-linked immunospot (E
97 22 did not worsen abscesses but did increase gamma interferon (IFN-gamma) expression at these sites.
98 osuppressive anthrax lethal toxin impairs NK gamma interferon (IFN-gamma) expression, but neither let
99 mice show the importance of both T cells and gamma interferon (IFN-gamma) for survival of a measles v
100 ation of STAg led to increased production of gamma interferon (IFN-gamma) from natural killer (NK) ce
105 vo approaches reveals that the expression of gamma interferon (IFN-gamma) is epigenetically silenced
108 restingly, IgA(-/-) mice had lower pulmonary gamma interferon (IFN-gamma) levels and decreased number
111 of the biofilm, even when prestimulated with gamma interferon (IFN-gamma) or TNF-alpha or cocultured
112 eukin-17A/F (IL-17A/F) and CXCL10 but not of gamma interferon (IFN-gamma) or tumor necrosis factor (T
113 KT) cells from coexposed mice expressed less gamma interferon (IFN-gamma) per cell than did those fro
116 rease P. falciparum-specific in vitro recall gamma interferon (IFN-gamma) production after CHMI, and
118 T-bet, but not its tyrosine mutant, rescues gamma interferon (IFN-gamma) production and inhibits Th2
119 s of cognate antigen-specific T cell clones: gamma interferon (IFN-gamma) production and mobilization
120 reatment with anti-CXCL10 antibodies reduced gamma interferon (IFN-gamma) production and Th17 cell in
122 (MHCII) and CD80 on CD11c-positive cells and gamma interferon (IFN-gamma) production by NK cells afte
123 iters and increased numbers of and increased gamma interferon (IFN-gamma) production by several diffe
124 servation of CD4(+) T cells and an increased gamma interferon (IFN-gamma) production from stimulated
125 ureus and greatly promoted proliferation and gamma interferon (IFN-gamma) production in CD4 and CD8 T
126 ost potent combinations capable of eliciting gamma interferon (IFN-gamma) production in NK cells.
127 evident, while decreased mitogen-stimulated gamma interferon (IFN-gamma) production suggested immuno
128 ciency prevents proper T cell activation and gamma interferon (IFN-gamma) production, which are criti
129 role in CD4+ Th1 lineage differentiation and gamma interferon (IFN-gamma) protein expression by CD4+
130 IL-10)/tumor necrosis factor alpha and IL-12/gamma interferon (IFN-gamma) ratios, and higher antibody
131 vants correlated better with enhanced T-cell gamma interferon (IFN-gamma) recall responses rather tha
133 ssing target cells, including stimulation of gamma interferon (IFN-gamma) release, specific target ce
134 enes might mediate the greater virulence and gamma interferon (IFN-gamma) resistance of C. muridarum
135 tion inflammatory syndrome (IRIS), we tested gamma interferon (IFN-gamma) response by enzyme-linked i
136 mune response to malaria by initiating a CD4 gamma interferon (IFN-gamma) response early in a blood-s
138 n in rhesus macaques, SC-Ad generated higher gamma interferon (IFN-gamma) responses and higher transg
139 berculosis (TB) vaccine candidates, elicited gamma interferon (IFN-gamma) responses from both TB and
140 /-) mice display organ burdens and pulmonary gamma interferon (IFN-gamma) responses similar to those
142 pitope and found individuals who had ex vivo gamma interferon (IFN-gamma) responses to the peptide ep
145 of HIV-1-specific CD4 T cell proliferation, gamma interferon (IFN-gamma) secretion, and, to a lesser
146 ion of MDMs by lipopolysaccharide (LPS) plus gamma interferon (IFN-gamma) stimulation caused no effec
149 esponse to viral antigen, while secretion of gamma interferon (IFN-gamma) was more limited in compari
151 er of CD4 T cells and CD8 T cells expressing gamma interferon (IFN-gamma) were observed in IL-17A(-/-
152 hese activated effector CD8 T cells produced gamma interferon (IFN-gamma) when stimulated with dengue
153 activated T cells with the respective CD4(+) gamma interferon (IFN-gamma)(+), CD4(+) interleukin-5 (I
154 This was further validated by depleting gamma interferon (IFN-gamma), a cytokine known to contro
155 pon MAIT cell selection by MR1, secretion of gamma interferon (IFN-gamma), and an innate interleukin
156 tion, secretion of the inflammatory cytokine gamma interferon (IFN-gamma), and host defense against t
157 chose TLR7, transcription factor p65 (RelA), gamma interferon (IFN-gamma), and IFN-gamma-inducible pr
159 ression of mRNAs for CD3delta, CD4, CD8beta, gamma interferon (IFN-gamma), and inducible nitric oxide
161 ent on CD8(+) T cells, involves perforin and gamma interferon (IFN-gamma), and is correlated with the
162 tumor necrosis factor alpha (TNF-alpha) and gamma interferon (IFN-gamma), and previous studies have
163 lammatory cytokines IL-22, IL-17a, IL-1beta, gamma interferon (IFN-gamma), and TNF-alpha, as well as
164 in interleukin 1beta (IL-1beta), IL-6, IL-8, gamma interferon (IFN-gamma), and tumor necrosis factor
165 simultaneously produce interleukin-2 (IL-2), gamma interferon (IFN-gamma), and tumor necrosis factor
166 mory phenotype and usually expressed CD107a, gamma interferon (IFN-gamma), and tumor necrosis factor
167 ing tumor necrosis factor alpha (TNF-alpha), gamma interferon (IFN-gamma), CCL2, CCL3, and interleuki
168 reased production of interleukin 17 (IL-17), gamma interferon (IFN-gamma), CCL4, and granulocyte-macr
169 train of C. neoformans engineered to produce gamma interferon (IFN-gamma), denoted H99gamma, demonstr
170 production of interleukin 15 (IL-15), IL-18, gamma interferon (IFN-gamma), granulocyte colony-stimula
171 6), tumor necrosis factor alpha (TNF-alpha), gamma interferon (IFN-gamma), IL-1beta, and inducible ni
172 PD-L1 blockade leads to balanced increase in gamma interferon (IFN-gamma), IL-2, and IL-13 secretion,
173 myristate acetate and ionomycin, we examined gamma interferon (IFN-gamma), IL-4, IL-5, and IL-17 prod
174 oduced significantly higher amounts of IL-2, gamma interferon (IFN-gamma), IL-4, IL-6, and IL-17A in
176 er, we also observed increased production of gamma interferon (IFN-gamma), IL-5, IL-9, IL-17, and IL-
177 ing tumor necrosis factor alpha (TNF-alpha), gamma interferon (IFN-gamma), IL-6, and IL-1beta were hi
180 wed significantly lower expression levels of gamma interferon (IFN-gamma), interleukin-6 (IL-6), and
181 by DH82 cells (P < 0.01), while secretion of gamma interferon (IFN-gamma), interleukin-6 (IL-6), inte
182 of CD4(+) Th1 cells and NKT cells producing gamma interferon (IFN-gamma), it increased the ratio of
184 iver, infected TLR4(-/-) mice showed reduced gamma interferon (IFN-gamma), tumor necrosis factor (TNF
185 0057(pYA5199) produced significant levels of gamma interferon (IFN-gamma), tumor necrosis factor alph
186 igher in DeltaTgPL1-infected mice, including gamma interferon (IFN-gamma), tumor necrosis factor alph
187 the DLNs soon after immunization, including gamma interferon (IFN-gamma), tumor necrosis factor alph
188 1 response, and specifically, high levels of gamma interferon (IFN-gamma), tumor necrosis factor alph
190 R. typhi(GFPuv)-infected BALB/c mice elicits gamma interferon (IFN-gamma), tumor necrosis factor alph
191 mokine (C-X-C motif) ligand 10 (CXCL10), and gamma interferon (IFN-gamma), was also significantly upr
192 by treatment with E2, but the proportion of gamma interferon (IFN-gamma)- and tumor necrosis factor
193 ween cytokine-producing T cell subsets with, gamma interferon (IFN-gamma)- and tumor necrosis factor
194 vium subsp. paratuberculosis survival inside gamma interferon (IFN-gamma)-activated bovine macrophage
196 lex that promoted translational silencing in gamma interferon (IFN-gamma)-activated myeloid cells.
197 11c(high) dendritic cell populations and the gamma interferon (IFN-gamma)-dependent CD4(+) T cell res
200 ranscription factor CEBP-beta, regulates the gamma interferon (IFN-gamma)-induced expression of Dapk1
202 nfection is dependent on the activity of the gamma interferon (IFN-gamma)-inducible nitric oxide synt
203 ls are the primary effector cells regulating gamma interferon (IFN-gamma)-mediated host resistance.
204 to be perforin (Prf)-mediated cell lysis and gamma interferon (IFN-gamma)-mediated induction of an an
205 umenting a cell-autonomous role for IRF-1 in gamma interferon (IFN-gamma)-mediated inhibition of MNV
206 otein FopC, which is required for evasion of gamma interferon (IFN-gamma)-mediated signaling, is able
207 4-depleted mice also increased the number of gamma interferon (IFN-gamma)-positive CD8(+) central mem
208 ve demonstrated that antigen-specific CD8(+) gamma interferon (IFN-gamma)-positive T-cell responses a
210 ted killing, and decreases the proportion of gamma interferon (IFN-gamma)-producing NK cells that had
211 in response to C. rodentium was dependent on gamma interferon (IFN-gamma)-producing NK1.1(+) cells an
214 and production of ESAT-6- or CFP-10-specific gamma interferon (IFN-gamma)-secreting and tumor necrosi
215 of activated CD8(+) T cells and Gag-specific gamma interferon (IFN-gamma)-secreting CD8(+) cells, low
216 In addition, the frequency and number of gamma interferon (IFN-gamma)-secreting effector memory (
217 lper type 1 isotype-switched antibodies, and gamma interferon (IFN-gamma)-secreting T cell responses.
218 The inflammatory reaction is orchestrated by gamma interferon (IFN-gamma)-secreting Th1 cells, and re
231 ressing genes involved in cellular immunity (gamma interferon [IFN-gamma] and the IFN-gamma-inducible
232 (interleukin-17 [IL-17] and IL-6) and liver (gamma interferon [IFN-gamma] and tumor necrosis factor a
233 terleukin 2 [IL-2], IL-4, IL-10, IL-17A, and gamma interferon [IFN-gamma]) and rendered T cells refra
234 ed substantially higher levels of antiviral (gamma interferon [IFN-gamma], 10-kDa gamma interferon-in
235 r alpha [TNF-alpha], interleukin-12 [IL-12], gamma interferon [IFN-gamma], and IL-6), chemokines (mon
236 flammatory Th1 (interlukin-1beta [Il-1beta], gamma interferon [Ifn-gamma], and tumor necrosis factor-
237 expressed latency-specific genes (e.g., the gamma interferon [IFN-gamma], Cxcl9, and Ccl5 genes) are
238 cell-mediated immune cytokine and chemokine (gamma interferon [IFN-gamma], interleukin 10 [IL-10], IL
239 udies with protection from clinical malaria (gamma interferon [IFN-gamma], interleukin-10 [IL-10], an
240 espite having no impact on mucosal cytokine (gamma interferon [IFN-gamma], tumor necrosis factor [TNF
241 iently produced multiple effector cytokines (gamma interferon [IFN-gamma], tumor necrosis factor alph
242 f systemic and TB antigen-stimulated type 1 (gamma interferon [IFN-gamma], tumor necrosis factor alph
243 evels of degranulation, cytokine expression (gamma interferon [IFN-gamma], tumor necrosis factor alph
244 ulating levels of proinflammatory cytokines (gamma interferon [IFN-gamma], tumor necrosis factor alph
245 whereas recombinant viruses expressing IL-4, gamma interferon, IL-12p35, IL-12p40, or IL-12p70 do not
246 of natural killer (NK) cells, which produce gamma interferon in an IFN-I-dependent fashion, and is i
250 increased the production of IL-6 and CXCL10/gamma interferon-induced protein 10, which are implicate
251 iviral (gamma interferon [IFN-gamma], 10-kDa gamma interferon-induced protein [IP-10]) and proinflamm
252 ors (interferon regulatory factor 7, CXCL10 [gamma interferon-inducible protein 10], gamma interferon
253 g as a nuclear pattern recognition receptor, gamma interferon-inducible protein 16 (IFI16) colocalize
258 H2-M molecular chaperone, the proteasome and gamma-interferon-inducible lysosomal thiol reductase rev
259 ng the AIM2 (absent in myeloma 2) and IFI16 (gamma-interferon-inducible protein 16) inflammasomes.
260 PAN RNA expression decreased expression of gamma interferon, interleukin-18, alpha interferon 16, a
262 Mphis incubated with lipopolysaccharide and gamma interferon (LPS/IFN-gamma) and with interleukin-4
263 HCV-specific CD8 T-cell activation (CD107a, gamma interferon, macrophage inflammatory protein 1beta,
264 4, also known as LIGHT), monokine induced by gamma interferon (MIG), and granzyme B (P <0.00001).
266 [GRO-alpha], IP-10, and monokine induced by gamma interferon [MIG]), were upregulated in Campylobact
267 ors in BBB disruption, the administration of gamma interferon-neutralizing antibody ameliorated the e
268 ls were found in FI-RSV immune mice, whereas gamma interferon-positive (IFN-gamma(+)) and TNF-alpha(+
269 gher among Th17 cells than among IL-17(-) or gamma interferon-positive (IFN-gamma(+)) cells, even upo
271 er the first MVA-CMDR boost, vaccine-induced gamma interferon-positive (IFN-gamma(+)) Gag-specific T-
272 tion correlated with high numbers of CD4(+), gamma interferon-positive (IFN-gamma(+)), tumor necrosis
273 crosis factor alpha-positive [TNF-alpha(+)], gamma interferon-positive [IFN-gamma(+)]) CD4(+) effecto
276 rs of activated CD4+ T cells in all tissues, gamma interferon-producing (IFN-gamma+) CD8+ T cells in
277 okine production, leukocyte infiltrates, and gamma interferon-producing CD4(+) T cells, as well as an
278 ally, there was a marked reduction of CD4(+) gamma interferon-producing effector T cells responsible
279 e the function of CD8(+) T cells in terms of gamma interferon production or prevent their apoptosis t
280 cells exhibited high functional avidity for gamma interferon production, whereas VP3(173-181)-specif
281 crophage markers, and increased the level of gamma interferon production, while it decreased the leve
282 NLuc infection of AG129 mice (alpha/beta and gamma interferon receptor deficient) showed rapid spread
283 imilar disease development in alpha/beta and gamma interferon receptor knockout mice, including neuro
284 However, the trpB mutant remains virulent in gamma interferon receptor-deficient (IFN-gammaR(-/-)) mi
285 ly reported that mice lacking alpha/beta and gamma interferon receptors (IFN-alpha/betaR and -gammaR)
286 ly reported that mice lacking alpha/beta and gamma interferon receptors permit high levels of DENV re
287 acid hybridization probe, a M. tuberculosis gamma interferon release assay, and is closely related t
288 s have correlated the results of interferon (gamma interferon) release assays (IGRAs) with known mark
290 were screened for the ability to stimulate a gamma interferon response in vitro using whole blood fro
291 and TssM produced high-titer IgG and robust gamma interferon-secreting T cell responses against the
292 of Af5517-aspirated mice displayed decreased gamma interferon secretion and increased interleukin-4 t
293 ptibility within tumor necrosis factor alpha/gamma interferon-stimulated macrophages in a STAT3/6-ind
294 g regimen resulted in significantly elevated gamma interferon T cell responses and high-avidity antig
296 and CAAT mutant promoters were activated by gamma interferon, the Sp1 and Inr mutants were repressed
297 larly, increased levels of STAT-1 induced by gamma interferon treatment inhibited HPV genome amplific
298 levels of granzyme B, granzyme K, perforin, gamma interferon, tumor necrosis factor alpha, and inter
299 ointestinal tract, but a slight reduction of gamma interferon was observed in the ceca of mice infect
300 roteins, intracellular cytokine staining for gamma interferon was utilized to identify novel RSV-spec
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