ライフサイエンスコーパス: gamma-linolenic acid

1 hidonic acid, oleic acid, linolenic acid, or gamma-linolenic acid).

2 aenoic acid, arachidonic acid, CLA:9c11t and gamma linolenic acid.

3 c acid alone or in combination with 5 mole % gamma-linolenic acid.

4 gamma-Linolenic acid (18:3n-6) was positively associated

5 epidermal 15-lipoxygenase transforms dihomo-gamma-linolenic acid (20:3n-6) to 15-hydroxyeicosatrieno

6 eic acid (16:1n-7; P = 2.8 x 10(-7)), dihomo-gamma-linolenic acid (20:3n-6; P = 2.3 x 10(-4)), the ra

7 Gamma-linolenic acid, a precursor of AA, also inhibited

8 tic acid (SA/PA) and arachidonic acid/dihomo-gamma-linolenic acid (AA/DGLA) ratios were associated wi

9 Dihomo-gamma-linolenic acid also inhibited fatty acid biosynthe

10 c acid and eicosapentaenoic acid, along with gamma-linolenic acid and antioxidants, may modulate syst

11 al diet containing eicosapentaenoic acid and gamma-linolenic acid and elevated antioxidants (EPA+GLA;

12 with prebiotics and black currant seed oil (gamma-linolenic acid and omega-3 combination) was effect

13 in an increase in the accumulation of dihomo-gamma-linolenic acid and, subsequently, decrease the pro

14 enteral supplementation of n-3 fatty acids, gamma-linolenic acid, and antioxidants compared with an

15 enteral supplementation of n-3 fatty acids, gamma-linolenic acid, and antioxidants did not improve t

16 Eicosapentaenoic acid, gamma-linolenic acid, and arachidonic acid decreased in

17 eic acid (LA), gamma-linolenic acid, di-homo-gamma-linolenic acid, and arachidonic acid to the omega3

18 Circulating gamma-linolenic acid, dihomo-gamma-linolenic acid, and arachidonic acid were not sign

19 dietary PUFA), gamma-linolenic acid, dihomo-gamma-linolenic acid, and arachidonic acid, with total a

20 nd cell death in 8-10 h while linoleic acid, gamma-linolenic acid, and docosapentaenoic also strongly

21 rmation as an iPF(2) metabolite, analysis of gamma-linolenic acid autooxidation products and the comp

22 tized to necrotic death from AA and the PUFA gamma-linolenic acid, but not from saturated or monounsa

23 he Delta5-fatty acid desaturation of di-homo-gamma-linolenic acid (C20:3 Delta8,11,14).

24 Dietary supplementation with di-homo-gamma-linolenic acid could be a reasonable interventiona

25 atory fatty acids, eicosapentaenoic acid and gamma-linolenic acid (derived from fish oil and borage o

26 The results showed that the ratio of dihomo-gamma-linolenic acid (DGLA) to deoxycholic acid (DCA) sp

27 habditis elegans, dietary exposure to dihomo-gamma-linolenic acid (DGLA), an omega-6 fatty acid, caus

28 ctivity, resulting in accumulation of dihomo-gamma-linolenic acid (DGLA), which displaces arachidonic

29 induced by dietary supplementation of dihomo-gamma-linolenic acid (DGLA, 20:3n-6) in the roundworm Ca

30 imrose oil elevates concentrations of dihomo-gamma-linolenic acid (DGLA; 20:3n-6), which results in t

31 8; 95% CI: 0.98, 1.68; P = 0.021) and dihomo-gamma-linolenic acid (DGLA; HR: 1.38; 95% CI: 1.04, 1.84

32 Incorporation of dihomo-gamma-linolenic acid (DHGLA), the metabolite of GLA, int

33 t here the x-ray crystal structure of dihomo-gamma-linolenic acid (DHLA) in the cyclooxygenase site o

34 to those seen previously with AA and dihomo-gamma-linolenic acid (DHLA).

35 their ability to convert linoleic acid (LA), gamma-linolenic acid, di-homo-gamma-linolenic acid, and

36 entaenoic acid or eicosapentaenoic acid with gamma-linolenic acid diet compared with ratios from rats

37 es from rats given the eicosapentaenoic with gamma-linolenic acid diet released 35% or 24% more prost

38 igher after feeding the eicosapentaenoic and gamma-linolenic acid diet vs. the linoleic acid diet.

39 Circulating gamma-linolenic acid, dihomo-gamma-linolenic acid, and a

40 ding linoleic acid (the major dietary PUFA), gamma-linolenic acid, dihomo-gamma-linolenic acid, and a

41 enoic acid-enriched or eicosapentaenoic with gamma-linolenic acid-enriched diet rapidly modulated the

42 ning eicosapentaenoic acid from fish oil and gamma-linolenic acid from borage oil have been designed

43 of the TE domain activity by the PUFA dihomo-gamma-linolenic acid; gamma- and alpha-linolenic acids,

44 an enzyme(s) specific for the elongation of gamma-linolenic acid (GLA) (18:3n-6), a cDNA expression

45 ecursors for LC-PUFA - stearidonic (SDA) and gamma-linolenic acid (GLA) - from Echium plantagineum (E

46 d containing eicosapentaenoic acid (EPA) and gamma-linolenic acid (GLA) foster formation of less infl

47 Gamma-linolenic acid (GLA), a nutritionally important fa

48 nutritionally desirable constituents such as gamma-linolenic acid (GLA), alpha-linolenic acid (ALA) a

49 of linolenic acid (i.e., Delta(6,9,12) 18:3, gamma-linolenic acid (GLA), and Delta(9,12,15) 18:3, alp

50 The n6 fatty acids linoleic acid (LA), gamma-linolenic acid (GLA), dihomo-GLA, arachidonic acid

51 ant positive associations were observed with gamma-linolenic acid (GLA), dihomo-GLA, docosatetraenoic

52 Beech seeds oil contains 4.2% of gamma-linolenic acid (GLA).

53 gests reduced conversion of linoleic acid to gamma-linolenic acid (GLA).

54 f this work was to establish the richness in gamma-linolenic acid (GLA, 18:3n6) and stearidonic acid

55 es of eicosapentaenoic acid (EPA; fish oil), gamma-linolenic acid (GLA; borage oil) (EPA+GLA), and an

56 risk and those with higher concentrations of gamma-linolenic acid (GLA; HR: 1.28; 95% CI: 0.98, 1.68;

57 he desaturated and elongated intermediate of gamma-linolenic acid, increased with fish and borage oil

58 of total dietary linolenic acid (alpha- and gamma-linolenic acid) intake.

59 of normal skin epidermis to desaturate LA to gamma-linolenic acid, it is transformed by epidermal 15-

60 i powders have shown to contain fatty acids, gamma-linolenic acid, linoleic acid, palmitic acid, and

61 pectively), while associations of DGAT2 with gamma-linolenic acid (log10(Bayes Factor) = 6.16) and of

62 resent in freeze-thawed plasma suggests that gamma-linolenic acid may also be an important source of

63 Cerotic and di-homo-gamma-linolenic acids may serve as markers of disease an

64 As direct incubation with gamma-linolenic acid or arachidonic acid also attenuated

65 eic acid (LA) or other downstream PUFAs like gamma-linolenic acid or arachidonic acid alter the trans

66 ated by supplementing the nematode diet with gamma-linolenic acid or C20 PUFAs of either the n6 or th

67 d (OR: 0.21; P for trend = 0.03), and dihomo-gamma-linolenic acid (OR: 0.24; P for trend = 0.03); the

68 urated fatty acids and low concentrations of gamma-linolenic acid, palmitic acid, and long-chain mono

69 gamma-Linolenic acid reduced severity of AD.

70 Reaction of PFI with dihomo-gamma-linolenic acid resulted in the development of two

71 studies showed that eicosapentaenoic acid or gamma-linolenic acid supplementation of animals exposed

72 Dihomo-gamma-linolenic acid, the desaturated and elongated inte

73 Dihomo-gamma-linolenic acid, the desaturated and elongated inte

74 The ratio of dihomo-gamma-linolenic acid to deoxycholic acid species is a po

75 Plasma di-homo-gamma-linolenic acid was independently associated with t

76 he desaturated and elongated intermediate of gamma-linolenic acid, was increased with 20% fish and 20

77 er and levels of eicosapentaenoic and dihomo-gamma-linolenic acids were higher after feeding the eico

78 entrations, and specifically those of dihomo-gamma-linolenic acid, were associated with a higher chil

79 Associations of di-homo-gamma-linolenic acid with the presence of cirrhosis and

80 pentaenoic acid alone or in combination with gamma-linolenic acid would modulate alveolar macrophage

81 tory fatty acids, eicosapentaenoic acid, and gamma-linolenic acid would reduce the intrapulmonary syn