ライフサイエンスコーパス: gamma-radiation

1 nate and acquired immune responses following gamma radiation.

2 oietic progenitor cells from lethal doses of gamma radiation.

3 ure the apoptotic response of individuals to gamma radiation.

4 cient mice were more sensitive to total body gamma radiation.

5 ein (GFP)-Rad51 foci following DNA damage by gamma radiation.

6 ir ability to increase p53 after exposure to gamma radiation.

7 effects of UV and the cytostatic effects of gamma radiation.

8 rans cultures recover from acute exposure to gamma radiation.

9 us stimuli and reduce sensitivity of mice to gamma radiation.

10 tion lesions is almost twice as great as for gamma radiation.

11 uscles lacking satellite cell activity after gamma radiation.

12 e (Thr-68) and cannot be activated following gamma radiation.

13 sponse to DNA-damaging agents such as UV and gamma radiation.

14 p53 is activated and its levels increased by gamma radiation.

15 of DNA strand breaks caused by adriamycin or gamma radiation.

16 th trk A or when these cells were exposed to gamma radiation.

17 of phosphorylation at this site after UV and gamma radiation.

18 mechanisms activate p53 after DNA damage by gamma radiation.

19 responses induced by 2.5, 5.0, and 7.5 Gy of gamma radiation.

20 chemical dosimetry for low dose detection of gamma radiation.

21 so higher after (56)Fe radiation relative to gamma radiation.

22 nergy protons from solar flares and not from gamma radiation.

23 iderable amounts of hydrogen when exposed to gamma-radiation.

24 IEC-6 intestinal epithelial cells following gamma-radiation.

25 s demonstrated in IEC-6 cells following 4 Gy gamma-radiation.

26 followed by gradual capsular stripping with gamma-radiation.

27 ts with TNFalpha, vinblastine, etoposide and gamma-radiation.

28 to apoptosis induced by TNFalpha, TRAIL and gamma-radiation.

29 y 100-fold more efficient in killing HC than gamma-radiation.

30 and UV-B light but only mildly sensitive to gamma-radiation.

31 e was compared with that induced by external gamma-radiation.

32 rosarcoma HT1080 cells following exposure to gamma-radiation.

33 15 months for patients with ISR treated with gamma-radiation.

34 stimuli, including cisplatin, etoposide, and gamma-radiation.

35 ive MRK attenuates the G(2) arrest caused by gamma-radiation.

36 lls from normal individuals upon exposure to gamma-radiation.

37 ls than in wild type cells after exposure to gamma-radiation.

38 m, mainly driven by effects at high doses of gamma-radiation.

39 e response effect and optimal time points of gamma-radiation.

40 greater sensitivity than wild type cells to gamma-radiation.

41 5C in the nucleus after exposure of cells to gamma-radiation.

42 ses point sources of 137Cs that emit 662-keV gamma-radiation.

43 cific reduction in clonogenic survival after gamma-radiation.

44 with doses of 0 to 18 Gy of either beta- or gamma-radiation.

45 ese cell lines to either alkylating agent or gamma-radiation.

46 ure of ML1 cells to etoposide, adriamycin or gamma-radiation.

47 duction of nuclear apoptosis by etoposide or gamma-radiation.

48 he hPer2/hp53 complex even when treated with gamma-radiation.

49 perties of this food were examined following gamma-radiation.

50 Cockayne Syndrome (CS) fibroblasts following gamma-radiation.

51 y correlated with the exposure dose of X- or gamma-radiations.

52 The effects of different doses of gamma radiation (0-20kGy) on the color and lipid oxidati

53 hat irradiation with relatively low doses of gamma-radiation (0.2Gy and 1Gy) does not lead to loss of

54 ation and compared the response with low LET gamma-radiation ((137)Cs; 0.5 Gy/min; 2 Gy).

55 ch more robust transcriptional response than gamma-radiation (2000 cGy) when evaluated 2 h after the

56 emia was induced using omeprazole, following gamma-radiation, 5-fluorouracil, and dextran sulphate so

57 How this bacterium can grow under chronic gamma radiation [50 grays (Gy) per hour] or recover from

58 Gamma radiation (8 Gy) inhibited DNA synthesis and progr

59 In contrast, 2,500 cGy gamma radiation, a dose that inactivates >5 log10 of T c

60 e-strand breaks (DSBs) caused by exposure to gamma radiation across archaea, bacteria, and eukaryotes

61 NQO1(-/-) mice exposed to gamma-radiation also showed lymphoma tissues (32%) and l

62 ex2-altered cells were not hypersensitive to gamma-radiation, an agent that causes DSBs throughout th

63 ose) or 1.0 Gy (moderate dose) of whole-body gamma radiation and allowed to develop for 16 weeks.

64 rotein is induced 15- to 30-fold in cells by gamma radiation and chemical mutagens but not by UV trea

65 ed in the phosphorylation of BRCA1 following gamma radiation and hydroxyurea treatment.

66 No strong link has been established between gamma radiation and its effect on mast cell survival and

67 the Gamma Forest were radiated with 1.8 kGy gamma radiation and survival microbial community analyze

68 er replication stresses such as UV exposure, gamma radiation and treatment with hydroxyurea.

69 (ARF)) showed normal downstream responses to gamma radiation and underwent cell cycle arrest.

70 These results indicate that gamma radiation and UV-C activate NF-kappaB through two

71 RP are viable but have severe sensitivity to gamma-radiation and alkylating agents.

72 Higher tail moments of gamma-radiation and benzo(a)pyrene diol epoxide-induced

73 In general, effects of combinations of gamma-radiation and Cd seem to be antagonistic at lower

74 ells displayed a deregulated p53 response to gamma-radiation and decreased regulation of downstream t

75 derived from BS donors are resistant to both gamma-radiation and doxorubicin-induced cell killing, an

76 mage, resulting in sensitization of cells to gamma-radiation and doxorubicin.

77 However, gamma-radiation and Fe(2+)-EDTA produced different propo

78 se in DNA, but the large differences between gamma-radiation and Fe(2+)-EDTA suggest that factors oth

79 two hydroxyl radical-mediated DNA oxidants, gamma-radiation and Fe(2+)-EDTA, produced nucleoside 5'-

80 Furthermore, both gamma-radiation and Fe2+-EDTA/H2O2 showed relatively mod

81 al-induced guanine oxidation associated with gamma-radiation and Fe2+-EDTA/H2O2.

82 thod was applied to plasmid DNA treated with gamma-radiation and peroxynitrite.

83 o the p53 mutation and are hypersensitive to gamma-radiation and reactive oxygen species due to the K

84 Cells were exposed to 5Gy gamma-radiation and repair followed for up to 60 minutes

85 MKP-1 was induced by gamma-radiation and repressed radiation-induced pro-apop

86 Both gamma-radiation and RIT caused cell death via an apoptot

87 ransfectants was preferentially inhibited by gamma-radiation and specific classes of apoptosis induce

88 mice were eliminated with sublethal doses of gamma-radiation and then reconstituted with syngeneic BM

89 and Histoplasma capsulatum (HC) to external gamma-radiation and to the organism-specific mAbs 18B7 a

90 hyperactivate ATM, ATR, and caspase-2 after gamma-radiation and trigger a caspase-2-dependent apopto

91 utilized: pharmacological, radiosurgery with gamma radiation, and external beam radiation.

92 for bleomycin, benzo[a]pyrene diol epoxide, gamma-radiation, and 4-nitroquinoline-1-oxide sensitivit

93 ere cultured for 90 hours, exposed to 1.0 Gy gamma-radiation, and harvested at 3 hours after gamma-ra

94 -deoxyribose oxidation products generated by gamma-radiation are similar for purified DNA and cells.

95 that plant seeds treated with high doses of gamma radiation arrest development as seedlings, the cau

96 odurans strain became almost as sensitive to gamma radiation as the ddrA knockout strain.

97 f commercial cranberry syrup irradiated with gamma radiation at a rate of 5 kGy and stored for 6 mont

98 s failed to undergo apoptosis in response to gamma radiation at both 28 and 37 degrees C.

99 c DNA than wild type cells after exposure to gamma-radiation at doses of 2 or 5 Gy.

100 After exposure to gamma radiation, bacteria isolated from the site with in

101 w that mouse embryonic stem cells exposed to gamma-radiation bear the effects of the insult for weeks

102 pressors of the uvh1 mutant's sensitivity to gamma radiation but do not affect the susceptibility of

103 53BP1 foci formation is not restricted to gamma-radiation but is also detected in response to UV r

104 eta mutant prevented NF-kappaB activation by gamma radiation, but not by UV-C.

105 e were susceptible to induction of tumors by gamma-radiation, but most tumors retained and expressed

106 Phosphorylation of Nbs mediated by gamma-radiation, but not that induced by hydroxyurea or

107 e of eight TCCs with mutant p53 responded to gamma radiation by elevation of p53, p21(WAF1), or mdm2

108 Bcl-2 can protect endothelial cells against gamma-radiation by a cytochrome c-independent signaling

109 ated in intestinal epithelia following 14 Gy gamma-radiation by Western blotting and immunohistochemi

110 Gamma-radiation can cause apoptosis, the process of prog

111 Same doses of gamma-radiation caused dramatic hair loss in wild-type m

112 reduced 53BP1 foci formation in response to gamma-radiation compared with cells expressing wild-type

113 n tumor latency, spectrum or frequency after gamma-radiation, compared to their control counterparts.

114 Gamma radiation did not affect the thermal properties of

115 tor tolerance induction and the selection of gamma radiation dose is critical for potential clinical

116 The total gamma-radiation dose accumulated by the fruits during th

117 00 MeV/nucleon) and results were compared to gamma radiation doses of 2 or 5 Gy, which are equitoxic

118 rial community was exposed to four different gamma radiation doses ranging from 0.46 to 3.96 kGy to t

119 ns, more than 90% of the OTA was degraded by gamma-radiation doses >/=2.5kGy, and a 2-fold reduction

120 HCl3, CH2Cl2) to enable instant detection of gamma radiation down to the 0.01 Gy level.

121 deficient fibroblasts were hypersensitive to gamma radiation, doxorubicin, and hydrogen peroxide and

122 en at the same position; and (3) both UV and gamma-radiation efficiently induce LOH at doses of radia

123 an be labeled with 99mTc, a widely available gamma-radiation-emitting radionuclide, for intravenous i

124 es of same food component absorbed different gamma radiation energy though exposed to same radiation

125 Exposure of mice and HL-60 cells to gamma radiation enhanced the levels of NQO2, which led t

126 We observed that cells exposed to gamma radiation exhibited increased levels of L1 retrotr

127 r endothelial cells (HDMEC), when exposed to gamma-radiation, exhibited a time-dependent activation o

128 tivity during the D. radiodurans response to gamma radiation exposure are unknown.

129 vels of cyclin D1, we infer that relative to gamma radiation exposure to (56)Fe radiation induced mar

130 Intriguingly, exposure of NQO1(-/-) mice to gamma-radiation failed to induce C/EBPalpha and Pu.1, as

131 serovar Typhimurium, were exposed to 25 kGy gamma radiation for complete sterilization.

132 millisievert/year (mSv/y)], larger levels of gamma radiation for the island of Rongelap (mean = 19.8

133 We find low levels of gamma radiation for the settled island of Enewetak [mean

134 First, what is the lowest dose of x- or gamma-radiation for which good evidence exists of increa

135 revealed a consistent pattern; Fe2+-EDTA and gamma-radiation generated MDA but not base propenals or

136 hnia magna, is affected by acute exposure of gamma radiation (GR) in combination with the polycyclic

137 untreated allogeneic splenic leukocytes; (3) gamma radiation group mice received gamma irradiated (2,

138 se rates were lower than those in the active gamma-radiation group and similar to those in the placeb

139 Ionising gamma radiation has been shown to reduce recurrence of r

140 Ionizing gamma radiation has several therapeutic indications incl

141 imited range of beta-particles compared with gamma-radiation, however, opens the question of whether

142 defective repair for both nonspecific DSBs (gamma-radiation hypersensitivity and genomic instability

143 hermore, these mutants are unable to reverse gamma-radiation hypersensitivity of BRCA1-null human bre

144 Twenty-five patients were treated with gamma radiation in a dose of 15 Gy, and 26 were treated

145 inococcus radiodurans following treatment by gamma radiation in an environment lacking nutrients.

146 Ps with or without PEGylation into mice; the gamma radiation in blood specimens and dissected organs

147 ): 1) to evaluate the actual distribution of gamma radiation in human in-stent restenosis (ISR) lesio

148 We examined the effects of intravascular gamma radiation in patients with in-stent restenosis of

149 ave shown the effectiveness of both beta and gamma radiation in preventing recurrent restenosis in pa

150 The core circadian genes are induced by gamma radiation in wild-type mice but not in mPer2 mutan

151 sence of Puma, HSCs were highly resistant to gamma-radiation in a cell autonomous manner.

152 of investigating the effects of exposure to gamma-radiation in a multistressor context.

153 umor latency, progression and lifespan after gamma-radiation in Atm heterozygous mice compared with t

154 obility and growth, responded to exposure to gamma-radiation in combination with the heavy metal cadm

155 ypt survival was also demonstrated following gamma-radiation in FVB/N mice rendered hypergastrinemic

156 ubstrate used was calf-thymus DNA exposed to gamma-radiation in N2O-saturated aqueous solution.

157 arious inter-strand cross-linking agents and gamma-radiation in vitro.

158 mycin C, methyl methanesulfonate, and UV and gamma-radiation, indicating that mammalian pol zeta help

159 diodurans are exposed to a 5000-Gray dose of gamma radiation, individual cells suffer massive DNA dam

160 vating enzyme, suggesting that both UV-C and gamma radiation induce degradation of IkappaBs by means

161 embryo fibroblast cell line (CREF cells) to gamma radiation induced simultaneous expression of PCNA

162 of these, NORF5/HUG1 (hydroxyurea and UV and gamma radiation induced), is induced by DNA damage, and

163 Gamma-radiation induced a BLM-regulated pathway that sel

164 Gamma-radiation induced browning inhibition in minimally

165 Ultraviolet-C and gamma-radiation induced JNK activation in both LNCaP and

166 gamma-radiation induced the death of P. falciparum in a

167 ytes from patients with RA were resistant to gamma radiation-induced apoptosis, a process known to be

168 Gamma radiation-induced clustered DNA damage containing

169 uman and murine mast cells were resistant to gamma radiation-induced cytotoxicity and, importantly, t

170 This contributed to the development of gamma radiation-induced myeloproliferative disease in NQ

171 Disruption of the NQO2 gene in mice leads to gamma radiation-induced myeloproliferative diseases.

172 helial cells expressing Bcl-2 also inhibited gamma-radiation-induced activation of p38 MAPK and p53 a

173 The median gamma-radiation-induced and benzo(a)pyrene diol epoxide-

174 LR9 engagement on murine CD4 T cells reduces gamma-radiation-induced apoptosis as judged by decreased

175 int kinase 1 (Chk1) is sufficient to restore gamma-radiation-induced apoptosis in p53 mutant zebrafis

176 n that LNCaP cells are entirely resistant to gamma-radiation-induced apoptosis, but can be sensitized

177 ryonic fibroblasts and osteosarcoma cells to gamma-radiation-induced apoptosis, with an increase in s

178 ion of pRb2/p130 increased the percentage of gamma-radiation-induced apoptotic cells from 27 to 47%.

179 ersion of virulence, and molecular basis for gamma-radiation-induced cell death in malaria parasites.

180 suggest a role for pRb2/p130 in glioblastoma gamma-radiation-induced cell death, indicating that the

181 inoblastoma family member pRb2/p130 enhances gamma-radiation-induced cell death.

182 pression levels in these cell lines; and (c) gamma-radiation-induced chromatid breaks were counted as

183 embryonic fibroblasts from MMP-9-/- mice on gamma-radiation-induced damage of DNA.

184 reaks (DSBs) are formed during processing of gamma-radiation-induced DNA clustered damage sites.

185 reventing centrosome amplification following gamma-radiation-induced DNA damage and does so by transc

186 In response to gamma-radiation-induced DNA damage, organisms either act

187 important determinant of cell fate following gamma-radiation-induced DNA damage.

188 cated in cellular senescence and response to gamma-radiation-induced DNA damage.

189 Next, gamma-radiation-induced G2 delay and apoptosis were test

190 We found a dose-response relationship for gamma-radiation-induced G2 delay and apoptosis.

191 Therefore, we concluded that gamma-radiation-induced G2 delay, apoptosis, p53 increas

192 ontrols, we investigated the relationship of gamma-radiation-induced G2-M arrest and lung cancer risk

193 ion and hospital controls), a lower level of gamma-radiation-induced G2-M arrest was associated with

194 gamma-Radiation-induced G2-M arrest was measured as the

195 The mean percentage of gamma-radiation-induced G2-M arrest was significantly lo

196 cond, wortmannin treatment strongly inhibits gamma-radiation-induced hyperphosphorylation and foci fo

197 The median gamma-radiation-induced increases of cells in the S and

198 Also observed were joint effects between gamma-radiation-induced increases of S and G(2) phase fr

199 Furthermore, gamma-radiation-induced JNK activation was suppressed by

200 In addition, exposure of cells to gamma-radiation induces MRK activity.

201 lly expressed, wild-type BRCA1 decreased the gamma radiation (IR) sensitivity and increased the effic

202 Using a gamma radiation (IR)-induced replication stress T-cell l

203 The use of gamma radiation is a technique for preserving food that

204 This study demonstrates that chronic LDR gamma radiation is genotoxic in an exposure scenario rea

205 mune cells, susceptibility of lymphocytes to gamma radiation is well known.

206 Although pathogen inactivation by gamma-radiation is an attractive approach for whole-orga

207 tients with in-stent restenosis treated with gamma-radiation is well tolerated and associated with a

208 Correspondingly, we found that gamma radiation led to activation of IKK, the protein ki

209 posure of mice and HL-60 cells to 3 Grays of gamma-radiation led to increased NQO1 that stabilized C/

210 suggested that exposure of NQO1(-/-) mice to gamma-radiation led to myeloproliferative disease.

211 Distributions of gamma radiation levels are provided, and hot spots are d

212 External gamma radiation levels on Bikini Island significantly ex

213 We report measurements of background gamma radiation levels on six islands in the northern Ma

214 eed this standard (P = <<0.01), and external gamma radiation levels on the other islands are below th

215 ans, supporting the hypothesis that even LDR gamma radiation may induce cancer.

216 Bcl-2 level and decreased etoposide- and UV/gamma radiation-mediated DNA fragmentation.

217 (N-methyl-N'-nitro-N-nitrosoguanidine), and gamma radiation, none of which resulted in a decrease in

218 gamma Radiation of oligodendrocytes also activated a sim

219 tion (TLR) and angiographic restenosis after gamma radiation of restenotic lesions.

220 a high-fat diet, exposed to low-dose (60)Co gamma-radiation of 25 mGy at 2 mo of age, and evaluated

221 Moreover, LC-MS/MS results showed that gamma-radiation of d(GT) under anaerobic condition yield

222 detailed sequence analysis of the effects of gamma radiation on an entire human chromosome, which giv

223 A damage in vivo, we compared the effects of gamma radiation on DNA synthesis on whole-body sections

224 here is little information on the effects of gamma radiation on mast cells, which are important in bo

225 8 mrem/y = 0.198 mSv/y), and relatively high gamma radiation on the island of Bikini (mean = 184 mrem

226 ygous mice were exposed to 7.5 Gy of (137)Cs-gamma radiation on their right sides, and Aprt-deficient

227 ascular ultrasound to evaluate the effect of gamma-radiation on recurrent in-stent restenosis.

228 splenic leukocytes treated with (2,500 cGy) gamma radiation or 150 micromol/L S-59 and 2.1 J/cm2 UVA

229 synthesis was inhibited 24 h after UV light, gamma radiation or DNA cross-linking by cisplatin in hum

230 Treatment of scid/scid mice with gamma radiation or N-ethyl-N-nitrosourea resulted in app

231 ed levels of p53 protein made in response to gamma radiation or the treatment of cells with etoposide

232 Activation of IKKbeta by gamma radiation or tumor necrosis factor-alpha led to in

233 gh-level ULBP1 expression was not induced by gamma radiation or UV radiation.

234 either a 700 cGy dose of Cobalt-60 ((60)Co) gamma-radiation or 600 cGy, 250 kVp x-irradiation.

235 e significantly more resistant to killing by gamma-radiation or 9- amino camptothecin than were cells

236 response to treatment with dexamethasone or gamma-radiation or in response to anti-CD3/anti-CD28 sti

237 PI and nontoxic doses of the ROS generators, gamma-radiation or t-butyl-hydroperoxide, attenuated the

238 M) at DNA damage sites in cells treated with gamma-radiation or the radiomimetic drug neocarzinostati

239 eated cells and in cells treated with either gamma-radiation or ultraviolet (UV) radiation.

240 ltured cells to survive after treatment with gamma-radiation or with the topoisomerase-I inhibitor to

241 lls exposed to low doses of alpha radiation, gamma radiation, or chemical mutagens in the presence an

242 Four days following 14 Gy gamma-radiation, or 2 injections of 400 mg/kg 5-fluorour

243 o densely ionizing 350 MeV/amu Si-particles, gamma-radiation, or sham-irradiated and transplanted 3 d

244 ependent (cold shock, serum deprivation, and gamma-radiation) pathways in TIMP-1-negative BL lines.

245 60Co gamma-radiation produces a 1.8-fold increase in the yiel

246 link between Ub protein ligase activity and gamma-radiation protection function of BRCA1, and provid

247 Intracoronary gamma-radiation reduces recurrent in-stent restenosis (I

248 Intracoronary gamma-radiation reduces recurrent in-stent restenosis.

249 itizes radioresistant LNCaP and PC3 cells to gamma radiation, regardless of the status of p53.

250 cal sensor systems for low dose detection of gamma radiation remains highly desired for medical radia

251 Precise detection of low-dose X- and gamma-radiations remains a challenge and is particularly

252 th cytokine-independent survival and partial gamma-radiation resistance.

253 and apoptotic index were 10 h and 48 h after gamma-radiation, respectively.

254 Exposure of these tumor-bearing mice to gamma radiation resulted in p53 protein accumulation and

255 Exposure of NQO1(-/-) mice to gamma-radiation resulted in reduced apoptosis in granulo

256 10 microM HA14-1 for 1 h followed by 1-6 Gy gamma radiation, resulted in a highly synergistic (combi

257 Gamma-radiation results in cell cycle arrest and apoptos

258 Parallel studies with gamma-radiation revealed levels of MDA similar to those

259 e 1 null (NQO1(-/-)) mice exposed to 3 Gy of gamma-radiation showed an increase in neutrophils, bone

260 These data suggest that MRK may mediate gamma-radiation signaling leading to cell cycle arrest a

261 N and HC proved to be extremely resistant to gamma-radiation such that significant killing was observ

262 r, many fungi manifest extreme resistance to gamma-radiation, such that the doses of several thousand

263 These cells are hypersensitive to gamma-radiation (suggesting defective recombinational re

264 hypersensitive to the cytostatic effects of gamma radiation, suggesting that NHEJ is indeed a critic

265 ensitive to the growth-inhibitory effects of gamma radiation, suggesting that this repair pathway is

266 e to survive for a long term after high-dose gamma-radiation that normally would pose 100% lethality

267 When exposed to gamma radiation, the halogenated solvents decompose into

268 esistant to the growth inhibitory effects of gamma radiation, the sog1 mutation affects the proper de

269 th the cytotoxic drug 5-fluorouracil or with gamma-radiation, the bcl-w-null animals exhibited substa

270 gamma-Radiation therapy can effectively prevent recurren

271 The results of our study support the use of gamma-radiation therapy for the treatment of in-stent re

272 Intracoronary gamma-radiation therapy reduces recurrent in-stent reste

273 We studied the effects of intracoronary gamma-radiation therapy versus placebo on the clinical a

274 For gamma-radiation, there was a 0.99 correlation between th

275 After gamma radiation, these mice show a marked increase in tu

276 ious studies have shown the effectiveness of gamma-radiation to prevent recurrent restenosis, even in

277 5FU and gamma-radiation treated bax-null mice surprisingly showe

278 and thrombosis rates were compared with the gamma-radiation-treated (n=125) and the placebo patients

279 res minus the percentage of mitotic cells in gamma-radiation-treated cultures from the same subject.

280 ma-radiation, and harvested at 3 hours after gamma-radiation treatment.

281 st after transforming growth factor-beta and gamma-radiation treatment.

282 h ISR treated with ICRT who were enrolled in gamma radiation trials.

283 Here we show that DNA damage induced by gamma-radiation triggers the phosphorylation of nuclear

284 s specifically phosphorylated in response to gamma-radiation, ultraviolet light and exposure to hydro

285 Intracoronary gamma-radiation used as adjunct therapy for patients wit

286 ent stent implantation and were treated with gamma-radiation using 192Ir.

287 hese data demonstrates that genes induced by gamma radiation, UV radiation, and the zinc-induced p53

288 The cellular p53 response to gamma radiation was also assessed by immunoblotting.

289 samples were collected from mice exposed to gamma radiation was analyzed.

290 s stabilization of TAp63gamma in response to gamma radiation was significantly decreased in the absen

291 In food matrices, the elimination of OTA by gamma-radiation was found more difficult, as radiation d

292 ypt survival in INS-GAS mice following 14 Gy gamma-radiation was inhibited by administration of a CCK

293 y alterations, their downstream responses to gamma radiation were studied in vitro.

294 tly more efficient in killing CN and HC than gamma-radiation when based on the mean absorbed dose to

295 lease histamine in response to high doses of gamma radiation, whereas other reports suggest that mast

296 However, gamma radiation, which also stabilizes p53, did not resu

297 ion of human breast and lung cancer cells to gamma radiation, which was further enhanced by Rad001.

298 early clinical benefits after intracoronary gamma radiation with 192Ir seem durable at 5-year clinic

299 cer because it simultaneously emits beta and gamma radiations with proper energy and half-life; there

300 y randomized to receive either intracoronary gamma-radiation with (192)Ir (15 Gy) or placebo.