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1 or rest pain, 37.2% for ulcer, and 38.2% for gangrene).
2 common cause of clostridial myonecrosis (gas gangrene).
3 itis about 40 days after onset of lower limb gangrene.
4 with C. perfringens in a murine model of gas gangrene.
5 l diseases, including food poisoning and gas gangrene.
6 plays a key role in the pathogenesis of gas gangrene.
7 of diseases in humans, including lethal gas gangrene.
8 ible for extensive tissue destruction in gas gangrene.
9 oronary disease, and amputation for ischemic gangrene.
10 gingival inflammation to grotesque orofacial gangrene.
11 mortality associated with C. perfringens gas gangrene.
12 -toxin (Cpa247-370) in a murine model of gas gangrene.
13 e absence of gangrene and 9.2% with PEH with gangrene.
14 thologies ranging from food poisoning to gas gangrene.
15 en isolated from patients suffering from gas gangrene.
16 to radical amputation for patients with gas gangrene.
17 emergency with a PEH causing obstruction or gangrene.
18 ular complications, including amputation and gangrene.
19 re is pain at rest, ischaemic ulceration, or gangrene.
20 ul pain-free limb with healing of ulcers and gangrene.
21 Alpha-toxin is the key determinant in gas-gangrene.
22 two hallmarks of Clostridium perfringens gas gangrene.
23 f the localized and systemic features of gas gangrene.
24 y leak characteristics of C. perfringens gas gangrene.
25 with resulting skin ischemia, necrosis, and gangrene.
26 l comorbidities, and incidence of PEH hernia gangrene.
32 ll had ischaemic ulcer in legs or minor skin gangrene and met haemodynamic criteria (ankle pressure <
35 t to be important in the pathogenesis of gas gangrene and the lack of phagocytic cells at the site of
39 gnosis of rest pain, ischemic ulceration, or gangrene between January 1, 2000, and September 30, 2002
41 um of disease, including food poisoning, gas gangrene (clostridial myonecrosis), enteritis necrotican
42 use of several human diseases, including gas gangrene (clostridial myonecrosis), enteritis necrotican
44 ptimal management of ischemic ulceration and gangrene in ESRD patients is quite controversial, and be
45 pe A strains are the causative agents of gas-gangrene in man and are also implicated as etiological a
46 localized and systemic manifestations of gas gangrene including enhanced vascular permeability, local
47 on of perfusion and a high incidence of limb gangrene, indicating that MMP-2 plays a critical role in
49 ODUCTION: Ritual circumcision complicated by gangrene is a leading cause of penile loss in young men
54 is concluded that tissue destruction in gas gangrene is related to profound attenuation of blood flo
55 tridium perfringens strain ATCC 13124, a gas gangrene isolate and the species type strain, and the en
56 sk for developing appendiceal perforation or gangrene may be determined, in part, by variation in the
57 (N=8128), rest pain (N=3056), and ulceration/gangrene (N=11,770) and Current Procedural Terminology c
58 vere decompression sickness, clostridial gas gangrene, necrotizing fasciitis, and acute crush injury.
59 e percentages of patients who presented with gangrene or ischemic ulceration rather than rest pain in
60 heral vascular (arterial claudication and/or gangrene or significant tissue loss and/or arterial thro
61 The primary indication for treatment was gangrene or ulceration in 42 patients (81%) and rest pai
65 bypass graft surgery, stroke, claudication, gangrene, or tissue loss and/or peripheral arterial thro
67 paraesophageal hernia causing obstruction or gangrene (PEH) and perforated peptic ulcer (PPU) was ana
71 ded that fulminant tissue destruction in gas gangrene results from profound attenuation of blood flow
75 cular insufficiency such as limb necrosis or gangrene were observed only in animals expressing solubl
76 the aorta may cause claudication and tissue gangrene, whereas aortitis may lead to aneurysm formatio
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