1 ed 1 (G1) for acute appendicitis, 2 (G2) for gangrenous acute appendicitis, 3 (G3) for perforation or

2 ancement was associated with the presence of gangrenous acute cholecystitis (sensitivity, 73%).

3 enhancement (associated with the presence of gangrenous acute cholecystitis) and the presence of a ga

4 pendicitis, and 0.64 (95% CI: 0.50-0.80) for gangrenous appendicitis.

5 omy for acute or complicated (perforated and gangrenous) appendicitis had similar complication rates,

6                       Ventral hernias with a gangrenous bowel were less likely to undergo laparoscopi

7  died of septic complications secondary to a gangrenous gallbladder diagnosed 1 day after the procedu

8                      The patient developed a gangrenous liver and died before she could undergo retra

9 nic bleeding and intestinal damage including gangrenous mucosal necrosis, phenotypes also evident in

10      MyD88(-/-) mice suffer from bacteremia, gangrenous mucosal necrosis, severe colitis, and death f

11 , ears, fingers, and toes that progressed to gangrenous necrosis.

12 ased on simple (non-perforated) and complex (gangrenous or perforated) inflammation, although many pa

13 ppendicitis (uncomplicated vs. perforated or gangrenous), plasma and peritoneal cytokine concentratio

14 (SA, acute) or complicated appendicitis (CA, gangrenous/ruptured)).