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1 ost common cause of clostridial myonecrosis (gas gangrene).
2 and mortality associated with C. perfringens gas gangrene.
3 pha -toxin (Cpa247-370) in a murine model of gas gangrene.
4 r pathologies ranging from food poisoning to gas gangrene.
5 ion with C. perfringens in a murine model of gas gangrene.
6 s been isolated from patients suffering from gas gangrene.
7 tive to radical amputation for patients with gas gangrene.
8 nimal diseases, including food poisoning and gas gangrene.
9 are two hallmarks of Clostridium perfringens gas gangrene.
10 ny of the localized and systemic features of gas gangrene.
11 llary leak characteristics of C. perfringens gas gangrene.
12 hich plays a key role in the pathogenesis of gas gangrene.
13 ange of diseases in humans, including lethal gas gangrene.
14 ponsible for extensive tissue destruction in gas gangrene.
15 Alpha-toxin is the key determinant in gas-gangrene.
19 ought to be important in the pathogenesis of gas gangrene and the lack of phagocytic cells at the sit
22 ectrum of disease, including food poisoning, gas gangrene (clostridial myonecrosis), enteritis necrot
23 e cause of several human diseases, including gas gangrene (clostridial myonecrosis), enteritis necrot
24 iotype A strains are the causative agents of gas-gangrene in man and are also implicated as etiologic
25 to localized and systemic manifestations of gas gangrene including enhanced vascular permeability, l
30 It is concluded that tissue destruction in gas gangrene is related to profound attenuation of blood
31 Clostridium perfringens strain ATCC 13124, a gas gangrene isolate and the species type strain, and th
32 , severe decompression sickness, clostridial gas gangrene, necrotizing fasciitis, and acute crush inj
33 ncluded that fulminant tissue destruction in gas gangrene results from profound attenuation of blood
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