ライフサイエンスコーパス: gasdermin

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1 gous to all three mouse gasdermins and human gasdermin.

2 e we show that both mice harbor mutations in gasdermin 3 (Gsdm3), a gene of unknown function.

3 21 (zona pellucida binding protein 2 [ZPBP2]-gasdermin A [GSDMA]).

4 peptide highly homologous to all three mouse gasdermins and human gasdermin.

5 Immunohistochemical analysis revealed that gasdermins are expressed specifically in cells at advanc

6 ona pellucida binding protein 2 (ZPBP2), and gasdermin B (GSDMB) and is correlated with high IgE leve

7 Gasdermin B (GSDMB) on chromosome 17q21 demonstrates a s

8 e located at 17q11.2-q12, and rs8069176 near gasdermin B (GSDMB; P = 1.88 x 10(-8)) at 17q12-q21.

9 consistently implicated the ORM1-like 3 and gasdermin B (ORMDL3-GSDMB), IL33, IL-1 receptor-like 1 a

10 between asthma risk allele, rs7216389-T and Gasdermin-B (GSDMB) in placenta (r2=27%) versus lung (r2

11 The exact function of human gasdermin-B (GSDMB), which regulates differentiation and

12 When activated, they cleave mouse and human gasdermin D (GSDMD) after Asp276 and Asp275, respectivel

13 tosis field, beginning with the discovery of Gasdermin D (GSDMD) as a substrate of caspase-1 and casp

14 ein, we report that the pyroptosis regulator gasdermin D (GSDMD) was necessary for IL-1beta secretion

15 , we report that EV71 induces degradation of gasdermin D (GSDMD), an essential component of pyroptosi

16 Gasdermin D (GSDMD), the pore-forming caspase-1 substrat

17 inflammasome activation induces a caspase-1/gasdermin D (Gsdmd)-dependent lytic cell death called py

18 te the maturation of interleukin (Il)-18 and gasdermin D (Gsdmd)-induced pyroptosis.

19 Our data identify gasdermin D as a critical target of caspase-11 and a key

20 ivation of the pore-forming effector protein gasdermin D by inflammatory caspases.

21 Here we show that gasdermin D is essential for caspase-11-dependent pyropt

22 During pyroptosis, GSDMD (gasdermin D), the pore-forming effector protein, is clea

23 Mechanistically, caspase-11 cleaves gasdermin D, and the resulting amino-terminal fragment p

24 Moreover, caspase-4, gasdermin D, interferon-beta, and cGAS levels were eleva

25 AS)-dependent interferon-beta production and gasdermin D-dependent interleukin-18 secretion.

26 but did not absolutely require Caspase-1 or Gasdermin D.

27 Gasdermin-D (GsdmD) is a critical mediator of innate imm

28 omain-like protein (MLKL) in necroptosis and gasdermin-D in pyroptosis] were recently discovered, bri

29 Inflammatory caspases cleave gasdermin-D in the interdomain linker but not GSDMB.

30 f the recently described pyroptotic effector gasdermin-D.

31 ing and release of IL-1beta independently of gasdermin-D.

32 To investigate the role of the gasdermin gene family an antiserum was raised to a pepti

33 discuss the implications for the rest of the gasdermin (GSDM) family, which are emerging as mediators

34 This expression pattern suggests a role for gasdermins in differentiation of the epidermis and its a

35 The N- and C-terminal domains of all gasdermins possess lipid-binding and regulatory activiti

36 akin gene, which encodes a new member of the gasdermin protein family.

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