ライフサイエンスコーパス: gastric biopsy

1 pathologic analysis of random post-treatment gastric biopsies.

2 cytoplasm of all bacteria examined in human gastric biopsies.

3 se and HspB within bacteria present in human gastric biopsies.

4 ence of mucosal dysplasia is noted on random gastric biopsy and may serve as a histologic marker in t

5 Stool, gastric biopsy, and serum samples were collected from 22

6 d transmission electron microscopy of serial gastric biopsies at 0, 3, 5, 8, and 12 months.

7 ssue samples were obtained during sequential gastric biopsies beginning at 3 weeks postinoculation an

8 EPIYA motifs were detected in total DNA from gastric biopsies by PCR.

9 ls of NOD1, CXCL8, IRF1, and CXCL10 in human gastric biopsies displaying severe gastritis, when compa

10 es, high-magnification endoscopy with random gastric biopsies, endoscopic ultrasonography, CT, and PE

11 e United States who underwent esophageal and gastric biopsies from 2008 through 2010.

12 D74 expression was increased dramatically on gastric biopsies from H. pylori-positive patients and ga

13 rDNA PCR fragment was amplified from 90% of gastric biopsies from histological H. pylori positive pa

14 released by reductive beta-elimination from gastric biopsies from rhesus monkeys.

15 upper gastrointestinal endoscopy with random gastric biopsies, high-magnification endoscopy with rand

16 e was confirmed in 992 H. pylori isolates in gastric biopsy material from infected patients.

17 scopy due to various non-emergent causes had gastric biopsies obtained at three adjacent sites.

18 otype using a molecular approach directly on gastric biopsies of dyspeptic patients attending consecu

19 tions using a molecular approach directly on gastric biopsies of dyspeptic patients consecutively att

20 We performed endoscopy with gastric biopsy on 15 healthy pet cats that were rigorous

21 y, VZV DNA and proteins were not detected in gastric biopsies or saliva.

22 isk MEN-1 patients, were correlated with the gastric biopsy results.

23 Gastric biopsies revealed chronic active gastritis with

24 shed by detection of CMV inclusion bodies in gastric biopsy samples and by hybridization with a CMV p

25 ed mucin oligosaccharides were released from gastric biopsy samples by beta-elimination and profiled

26 We studied gastric biopsy samples from 78,985 unique patients.

27 Similar studies were performed on gastric biopsy samples from H. pylori-infected and uninf

28 Gastric biopsy samples from patients infected with VacA(

29 Gastric biopsy samples were collected from patients with

30 ctive gastritis and intestinal metaplasia in gastric biopsy samples were inversely associated with Ba

31 as assessed by both histology and culture of gastric biopsy samples.

32 lserine receptor BAI1 was expressed in human gastric biopsy specimens and gastric phagocytes.

33 Gastric biopsy specimens and peripheral blood samples we

34 These results were then confirmed with gastric biopsy specimens and saliva from patients with c

35 heir ability to reliably detect H. pylori in gastric biopsy specimens and salivary samples.

36 Genomic DNA preparations from these gastric biopsy specimens and the corresponding H. pylori

37 tative culture and histologic examination of gastric biopsy specimens from 29 H. pylori-infected dysp

38 Mapped gastric biopsy specimens from 378 H. pylori-positive sub

39 ogen Helicobacter pylori, was quantitated in gastric biopsy specimens from 41 H. pylori-infected pati

40 of disease, from gastritis to carcinoma, and gastric biopsy specimens from Colombian and Honduran coh

41 e in epithelial cell p27(kip1) expression in gastric biopsy specimens from H. pylori-infected patient

42 To test this, we analyzed gastric biopsy specimens from H. pylori-positive and -ne

43 hogenesis, eosinophils have been detected in gastric biopsy specimens from patients with AIG.

44 iption polymerase chain reaction analysis of gastric biopsy specimens from patients with and without

45 Gastric biopsy specimens from the stomach antrum and fun

46 The patterns from 13 of the 19 gastric biopsy specimens matched those of the H. pylori

47 Nineteen gastric biopsy specimens obtained from patients undergoi

48 Gastric biopsy specimens of 68 peptic ulcer disease (PUD

49 cross- sectional study was performed on 800 gastric biopsy specimens of cows, sheep, goats and human

50 immunohistochemistry on mononuclear cells in gastric biopsy specimens of infected but not uninfected

51 IL-18 levels in the gastric biopsy specimens showed similar patterns to thos

52 oarray screening of H. pylori-infected human gastric biopsy specimens to identify candidate genes inv

53 DNA from gastric biopsy specimens was analyzed similarly for comp

54 cter pylori using DNA isolated from infected gastric biopsy specimens was approximately equal to geno

55 that IL-17C expression in H. pylori-infected gastric biopsy specimens was predominantly localized to

56 RNA from 41 H. pylori-positive gastric biopsy specimens was reverse transcribed to cDNA

57 Clinical data and gastric biopsy specimens were collected from 9 consecuti

58 Gastric biopsy specimens were collected from patients wh

59 One hundred twenty-six urease-negative gastric biopsy specimens were evaluated for the presence

60 Gastric biopsy specimens were examined by immunohistoche

61 -1), KATO III cells, and cells isolated from gastric biopsy specimens were infected with H pylori.

62 med before, during, and after treatment, and gastric biopsy specimens were obtained for quantitative

63 distinguish H. pylori strains directly from gastric biopsy specimens without culture of the organism

64 icobacter species present in urease-negative gastric biopsy specimens, 16S rDNA amplicons were cloned

65 anscript profiles and histologic features of gastric biopsy specimens, as well as blood eosinophil co

66 In human gastric biopsy specimens, the vacA i1 allele was strongl

67 rformed in H. pylori-infected and uninfected gastric biopsy specimens.

68 approximately 4.5-fold in H. pylori-infected gastric biopsy specimens.

69 ICC numbers were determined from gastric biopsy specimens.

70 sensitivities and specificities of >90% with gastric biopsy specimens.

71 concordance by culture and histology) coded gastric biopsy specimens.

72 isolates with known cagA status and in human gastric biopsy specimens.

73 ifferent patterns, respectively, from the 19 gastric biopsy specimens.

74 d for directly typing H. pylori strains from gastric biopsy specimens.

75 H. pylori isolates obtained from endoscopic gastric biopsy was determined by using agar dilution.

76 Gastric biopsies were collected for quantitative culture

77 Full thickness gastric biopsies were obtained laparoscopically from two

78 Six standard gastric biopsies were taken.

79 Patients with and without H pylori on gastric biopsy were compared, and odds of esophageal eos

80 s and freshly isolated epithelial cells from gastric biopsies with specific antibodies.

81 d of their H pylori infection as assessed by gastric biopsy, with elimination of gastritis; median an