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1 c states that have an increased incidence of gastric carcinoids.
2 was to determine whether SRS could localize gastric carcinoids.
3 the upper abdomen on SRS may be caused by a gastric carcinoid and not a pancreatic endocrine tumor.
4 ciencies (including B12 and hypomagnesemia), gastric carcinoid and rebound hyperacidity are biologica
6 the connection between hypergastrinemia and gastric carcinoids, and most importantly, improving upon
12 mys species exhibits a genetic propensity to gastric carcinoid formation that can be accelerated by a
13 three patients who underwent splenectomy for gastric carcinoid, gastric adenocarcinoma and cancer of
14 neoplasia type 1 (MEN-1) gene was studied in gastric carcinoids of patients with MEN-1 and chronic at
15 ypersecretion, acute interstitial nephritis, gastric carcinoid tumor, cardiovascular risk with clopid
17 s the understanding of the biologic basis of gastric carcinoid tumors increases, the treatment will l
18 nd clinical arenas, the optimal treatment of gastric carcinoid tumors is still a matter of debate.
20 ed with an increased risk of gastric polyps, gastric carcinoid tumors, and possibly adenocarcinomas.
21 cusses the current biologic understanding of gastric carcinoid tumors, including the role of hypergas
28 asive method that can identify patients with gastric carcinoids with a reasonable sensitivity and a h
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