ライフサイエンスコーパス: gastric carcinoma

1 , B16F10 melanoma, WT-GBM glioma and MKN45-P gastric carcinoma.

2 comparison of SBA with colorectal cancer and gastric carcinoma.

3 osa-associated lymphoid tissue lymphoma, and gastric carcinoma.

4 e instability (MSI) is observed in 13-44% of gastric carcinoma.

5 vely in patients with potentially resectable gastric carcinoma.

6 d specific marker for the MSI-H phenotype in gastric carcinoma.

7 rapy in patients with potentially resectable gastric carcinoma.

8 es hMLH1 or hMSH2 have not been described in gastric carcinoma.

9 rt of the standard adjuvant chemotherapy for gastric carcinoma.

10 ually exclusive diseases, duodenal ulcer and gastric carcinoma.

11 r many years, particularly for patients with gastric carcinoma.

12 geal carcinoma (NPC), Hodgkin's disease, and gastric carcinoma.

13 causing serious disease such as lymphoma and gastric carcinoma.

14 oma kindred with synchronous primary diffuse gastric carcinoma.

15 or hepatitis B virus but not in EBV-positive gastric carcinoma.

16 CD44/CD24 was associated with recurrence of gastric carcinoma.

17 the upper gastrointestinal tract, including gastric carcinoma.

18 ic diseases including gastritis, ulcers, and gastric carcinoma.

19 d have been linked to atrophic gastritis and gastric carcinoma.

20 polymerase beta variant was identified in a gastric carcinoma.

21 ether H. pylori infected individuals develop gastric carcinoma.

22 e to intestinal metaplasia and in one model, gastric carcinoma.

23 ty, and survival in patients with resectable gastric carcinoma.

24 with Helicobacter felis infection to induce gastric carcinoma.

25 has been linked to peptic ulcer disease and gastric carcinoma.

26 markers for better diagnosis and therapy of gastric carcinoma.

27 reases the risk for peptic ulcer disease and gastric carcinoma.

28 associated with a greater risk of intestinal gastric carcinoma.

29 dence interval [CI], 1.3-5.6) for intestinal gastric carcinoma.

30 17, and ADAM 20 transcripts were present in gastric carcinoma.

31 ine activity was achieved against N-87 human gastric carcinoma.

32 egation, which was further observed in human gastric carcinomas.

33 ome patients with lymphomas and renal, colon gastric carcinomas.

34 her by mutation or deletion in a majority of gastric carcinomas.

35 lelic loss on our panel of xenografted human gastric carcinomas.

36 an cancers including breast, lung, colon and gastric carcinomas.

37 vation of Smad4 was found in our study of 35 gastric carcinomas.

38 normal and tumor tissue DNA from 60 primary gastric carcinomas.

39 types of malignancies, including 10% of all gastric carcinomas.

40 DC73 has been reported in breast, renal, and gastric carcinomas.

41 ias in the stomach and esophagus and in some gastric carcinomas.

42 eported in esophageal, breast, prostate, and gastric carcinomas.

43 pharyngeal carcinomas (NPC) and a portion of gastric carcinomas.

44 enectomy) in patients affected with operable gastric carcinoma?

45 nts with esophageal cancer, 53 patients with gastric carcinoma (13 MSI-H and 44 MSI-negative), or ten

46 re were 53 patients enrolled onto the study (gastric carcinoma, 31; pancreatic carcinoma, 22).

47 e tracks of RIZ were found in 19 (48%) of 40 gastric carcinomas, 6 (33%) of 18 endometrial carcinomas

48 EBV tumor cell lines (Burkitt's lymphoma and gastric carcinoma), activation of viral gene expression

49 ate prediction of survival for patients with gastric carcinoma after neoadjuvant chemotherapy followe

50 uced 15- to 25-fold transcription from Zp in gastric carcinoma AGS cells.

51 pressed in nearly half of the EBV-associated gastric carcinomas analyzed.

52 sult in the prevention or early diagnosis of gastric carcinoma and also help in the identification of

53 oung age, such as diffuse (signet ring cell) gastric carcinoma and lobular breast carcinoma.

54 new tool for the study and visualization of gastric carcinoma and may also be of use in other cancer

55 lear cells (PBMC), or with a patient-derived gastric carcinoma and PBMCs from the same patient, we fo

56 acter pylori is known to be a major cause of gastric carcinoma and peptic ulceration.

57 ne, gastrointestinal, and nervous systems in gastric carcinomas and is implicated in heart failure an

58 ays a critical role in peptic ulcer disease, gastric carcinoma, and gastric lymphoma.

59 and is associated with peptic ulcer disease, gastric carcinoma, and gastric lymphoma.

60 of peptic ulcer disease, chronic gastritis, gastric carcinoma, and gastric mucosa-associated lymphoi

61 approximately 50% of patients with localized gastric carcinoma, and more than 60% die of cancer follo

62 al malignancies nasopharyngeal carcinoma and gastric carcinoma, and these data indicate that LMP2A af

63 growth potential of other B-cell lymphoma-, gastric carcinoma-, and nasopharyngeal carcinoma-derived

64 etics, phase I studies, and activity against gastric carcinoma are described in detail.

65 Pernicious anemia and gastric carcinoma are serious sequelae of autoimmune gas

66 Human gastric carcinomas are among the most treatment-refracto

67 These results indicate that few gastric carcinomas are driven solely by MET and VEGFR2,

68 sed for early detection and visualization of gastric carcinoma based on its altered redox metabolism,

69 ve gastric cancers greatly differ from other gastric carcinomas based on sex, anatomic subsite, and s

70 curs not only in gastric IM of patients with gastric carcinoma, but also in IM of cancer-free individ

71 DC promoter-luciferase constructs in a human gastric carcinoma cell line (AGS-B) that expresses the h

72 )-mediated signal transduction pathways in a gastric carcinoma cell line (KATO III).

73 in Burkitt's lymphoma cell line Ramos and in gastric carcinoma cell line HSC-39 and partially enhance

74 that endogenous c-KIT expression in a human gastric carcinoma cell line is also reduced on treatment

75 , whereas overexpression of CIITA in a human gastric carcinoma cell line, AGS, resulted in decreased

76 inocyte cell line, HaCaT, but did in a human gastric carcinoma cell line, HSC-39.

77 We utilized the highly sensitive gastric carcinoma cell line, SNU638, and two related MET

78 of nasopharyngeal carcinoma cell lines and a gastric carcinoma cell line.

79 activities against the Met dependent GTL-16 gastric carcinoma cell line.

80 of human leukemia cells and human colon and gastric carcinoma cell lines (IC(50) values as low as 1

81 iRNA) expression in several EBV-infected AGS gastric carcinoma cell lines was determined.

82 s been shown to be highly expressed in human gastric carcinoma cell lines, and our results raise the

83 nt signaling activation in a series of human gastric carcinoma cell lines.

84 e tumor necrosis factor alpha (TNF-alpha) in gastric carcinoma cells (AGS).

85 Here we show that AGS gastric carcinoma cells containing the lytic form of EBV

86 to kill EBV-positive, but not EBV-negative, gastric carcinoma cells in vitro.

87 illing of EBV-positive, versus EBV-negative, gastric carcinoma cells in vitro.

88 ared factors which regulate Zp and Rp in AGS gastric carcinoma cells that support a remarkably high l

89 The CDX2 gene in MKN45 gastric carcinoma cells was disrupted using targeted hom

90 Furthermore, in EBV-infected gastric carcinoma cells, the BZLF1-positive cells had an

91 ression and virus production in EBV-infected gastric carcinoma cells.

92 ownregulation in EBV-transformed B cells and gastric carcinoma cells.

93 ric chromosomal arm in our xenografted human gastric carcinomas demonstrated significant loss of hete

94 elopment of gastric carcinoma, we examined 8 gastric carcinoma-derived cell lines and 32 primary aden

95 gastric carcinoma (PGC, n = 131) and distal gastric carcinoma (DGC, n = 307) in consecutive 438 EGCs

96 EBV-positive gastric carcinoma does not require immune suppression, a

97 ibited the growth of human tumour cells AGS (gastric carcinoma), DU-145 and LNCaP (prostate carcinoma

98 he R258A mutant and a mutant associated with gastric carcinomas, E295K, provide molecular insight int

99 formation on this deletion in EBV-associated gastric carcinoma (EBVaGC) is limited.

100 Early gastric carcinoma (EGC) in Chinese patients remains poor

101 We examined 117 gastric carcinomas for MSI and observed instability at o

102 n's lymphoma, and 37 cases of EBV-associated gastric carcinoma (GC) from the United States, Brazil, a

103 The association of gastric carcinoma (GC) with EBV was examined by EBV-enco

104 c strategy targeting hypoxic cancer cells in gastric carcinoma (GC).

105 ssues (31 non-neoplastic stomach (NS) and 25 gastric carcinomas (GC)) were studied.

106 The etiology of MSI in gastric carcinoma has not been clearly defined.

107 Patients with local-regional gastric carcinoma have a low rate of curative resection

108 s been causally associated with cancer; some gastric carcinomas have a monoclonal EBV genome in every

109 Thus, Smad4 inactivation was noted in our gastric carcinomas; however, this event was rare.

110 ars consistent with the greater incidence of gastric carcinoma in men.

111 sophageal junction in comparison with distal gastric carcinomas in Brazilian patients.

112 Most gastric carcinomas in humans have been associated with c

113 cag+) and are associated with ulceration and gastric carcinoma induce greater cytokine production tha

114 Hereditary diffuse gastric carcinoma is an autosomal dominant cancer syndro

115 tward transcripts (BART) viral transcript in gastric carcinoma is in fact a functional viral long non

116 Gastric carcinoma is the fourth most common cause of can

117 Gastric carcinoma is the second most common cause of can

118 However, the proportion of EBV-positive gastric carcinomas is uncertain, and the etiologic signi

119 sing in a CDH1-associated hereditary diffuse gastric carcinoma kindred with synchronous primary diffu

120 lso enhances R-induced lytic infections in a gastric carcinoma line (AGS) carrying the R-KO virus, al

121 One exception was that infection of AGS, a gastric carcinoma line, resulted in maintenance of EBNA1

122 (n = 317), colorectal cancer (n = 6353), and gastric carcinoma (n = 889) collected between August 24,

123 By 1996, incident cases of gastric carcinoma of the distal stomach had been diagnos

124 copathologic features between early proximal gastric carcinoma (PGC, n = 131) and distal gastric carc

125 work described here, we show that the E295K gastric carcinoma pol beta variant acts in a dominant-ne

126 The gastric carcinoma profiles resembled other tumours', sup

127 Xenografted samples of our gastric carcinomas provided optimally enriched tumors fo

128 Among the primary gastric carcinomas, rearrangement of the FHIT gene and/o

129 Human gastric carcinoma shows a higher prevalence of microsate

130 We evaluated 428 gastric carcinoma specimens after a cisplatin-based chem

131 for it being part of the hereditary diffuse gastric carcinoma spectrum of malignancies.

132 at DeltaNp73 protein is upregulated in human gastric carcinoma suggesting that DeltaNp73 may play an

133 several types of solid neoplasms, including gastric carcinoma, suggesting the presence of suppressor

134 though TFF1 expression is frequently lost in gastric carcinomas, the tumorigenic pathways this affect

135 Among the 30 assessable patients with gastric carcinoma, there were two (7%) complete responde

136 tities, such as glioblastoma, colorectal, or gastric carcinoma, this could not be confirmed.

137 sing RNA-Seq in hepatocellular carcinoma and gastric carcinoma tumors, respectively.

138 In patients with gastric carcinoma, UFT alone has a response rate of appr

139 6% of 34 informative cases) was noted in our gastric carcinomas using microsatellite markers near the

140 icobacter pylori isolates from patients with gastric carcinoma versus 78% of 18 isolates from patient

141 295K mutation of pol beta has been linked to gastric carcinoma via interference with base excision re

142 Median survival among all patients with gastric carcinoma was 10 months and 13 months for patien

143 o model of liver metastasis induced by human gastric carcinoma was established in nude mice and used

144 Invasive gastric carcinoma was observed in 6 of 8 INS-GAS mice th

145 FHIT gene alterations in the development of gastric carcinoma, we examined 8 gastric carcinoma-deriv

146 tumor stasis in a Met-dependent GTL-16 human gastric carcinoma xenograft model following oral adminis

147 vivo antitumor activity in the GTL-16 human gastric carcinoma xenograft model.

148 cytoreductive activity was demonstrated in a gastric carcinoma xenograft model.

149 In MKN-45 (gastric carcinoma) xenografts, both tracers cleared slow