ライフサイエンスコーパス: gastric distension

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1 ake and attenuates the hypophagic effects of gastric distension.

2 rog/kg) administration, or CCK combined with gastric distension.

3 hindbrain through a mechanism that involves gastric distension.

4 n to a meal represent satiety or feelings of gastric distension.

5 ude hormones, lipid mediators, nutrients and gastric distension.

6 ngth and pressure measured before and during gastric distension.

7 t cause further pressure increases evoked by gastric distension.

8 Gastric distension (30 ml) evoked LOS relaxation (70 +/-

9 mbly in decerebrate, unanaesthetized ferrets.Gastric distension (30 ml) evoked LOS relaxation (70 +/-

10 urons expressing GLUR2/3 was similar for the gastric distension (59.8-65.6%) and duodenal linoleic ac

11 Gastric distension (6 ml) evoked a much larger intragast

12 Gastric distension (6 ml) evoked an increase of 9.0 +/-

13 ercentage of neurons in the NTS activated by gastric distension (63.9+/-2.9%), linoleic acid (62.8+/-

14 The combination of gastric distension and cholecystokinin (CCK) enhances bo

15 Gastric distension and duodenal nutrient stimuli produce

16 f differing gastric stability would increase gastric distension and reduce appetite compared with a c

17 Similar to in vivo studies, gastric distension caused a smaller increase in intragas

18 We hypothesize that gastric distension causes progressive effacement of the

19 Gastric distension causes progressive shortening of the

20 When combined, CCK and gastric distension enhanced Fos-LI in the DVC greater th

21 ous behavioral data, these results show that gastric distension enhances CCK-induced neuronal activat

22 esult in gastro-oesophageal reflux, that is, gastric distension-evoked lower oesophageal sphincter (L

23 esult in gastro-oesophageal reflux, that is, gastric distension-evoked lower oesophageal sphincter (L

24 Graded gastric distensions (GD, 20, 40, 60 mm Hg, 20 s) were in

25 ease in intragastric pressure in response to gastric distension in the denervated, vascularly isolate

26 Likewise, ondansetron attenuated Fos-LI by gastric distension in the DVC, specifically within the n

27 overall DVC enhanced Fos-LI induced by CCK + gastric distension, in particular at the NTS and AP nucl

28 NPY (0.03--3 nmol), significantly inhibited gastric distension-induced pyloric relaxation in a dose-

29 ch, acting through the Y1 receptor, inhibits gastric distension-induced pyloric relaxation in rats ex

30 Taken together, these findings suggest that gastric distension-induced pyloric relaxation is mediate

31 modulating the effects of hyperglycaemia on gastric distension-induced pyloric relaxation was invest

32 Gastric distension-induced pyloric relaxation was signif

33 se (3 micromol) also significantly inhibited gastric distension-induced pyloric relaxation without af

34 the inhibitory effects of hyperglycaemia on gastric distension-induced pyloric relaxation.

35 from 5.4 to 12.8 mM, significantly inhibited gastric distension-induced pyloric relaxation.

36 s to assess in normal subjects the effect of gastric distension on the LES length and pressure and it

37 X) caused a significant pressure increase by gastric distension, reaching 17.0 +/- 1.7 cmH2O, suggest

38 The pressure increase evoked by gastric distension remained high 2 weeks after TV in viv

39 , semirecumbent positioning, minimization of gastric distension, subglottic suctioning, avoidance of

40 The pressure increase evoked by gastric distension was significantly enhanced by L-NAME,

41 The pressure increase evoked by gastric distension was significantly enhanced in vivo by

42 After antral denervation, gastric distension with a non-nutrient solution is an ad

43 Progressive gastric distension with air resulted in progressive shor

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