ライフサイエンスコーパス: gastric distention

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1 atient request or adverse effects, including gastric distention).

2 not acutely sensitize animals to subsequent gastric distention.

3 he human brain regions that are activated by gastric distention.

4 d by duodenal distention and were excited by gastric distention.

5 by duodenal distention and were inhibited by gastric distention.

6 on of the enteroenterostomy leading to acute gastric distention.

7 Symptomatic gastric distention activates structures implicated in so

8 Hypersensitivity to gastric distention, an important feature of functional d

9 dation), interlobular septal thickening, and gastric distention and contents (fluid or sediment).

10 Persistent, asymptomatic gastric distention can be seen in neonates within 48 hou

11 nine infants (both with initial asymptomatic gastric distention) developed feeding intolerance with p

12 The effect of systemic Delta(9)-THC on gastric distention-evoked LOS relaxation was reversed by

13 The effect of systemic delta9-THC on gastric distention-evoked LOS relaxation was reversed by

14 ), significantly attenuated the nadir of the gastric distention-evoked LOS relaxation, and time to re

15 g),significantly attenuated the nadir of the gastric distention-evoked LOS relaxation, and time to re

16 ophageal sphincter relaxation (tLESR) during gastric distention in patients with gastroesophageal ref

17 Gastric distention increased the frequency of reflux by

18 by the gastrointestinal system, focusing on gastric distention, intestinal and pancreatic satiation

19 Early gastric distention may be predictive of feeding intolera

20 In all cases, gastric distention resolved with cessation of prostaglan

21 ealthy volunteers during baseline and distal gastric distentions to 10 mm Hg, 20 mm Hg, threshold pai

22 prostaglandin therapy, persistent (>3 days) gastric distention was noted in four of nine infants.

23 esophageal sphincter relaxations result from gastric distention, which shortens the sphincter to the

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