ライフサイエンスコーパス: gastric emptying

1 ory motor reflex in other gut areas (delayed gastric emptying).

2 asting, and to search for a correlation with gastric emptying.

3 thers documenting a GLP-2-dependent delay in gastric emptying.

4 Acute GLP-1 markedly slowed gastric emptying.

5 tially improved glucose tolerance and slower gastric emptying.

6 the intestine by regulating food intake and gastric emptying.

7 elate symptoms of gastroparesis with IMD and gastric emptying.

8 /- 0.6%) on insulin pump therapy with normal gastric emptying.

9 ational treatments of disorders with delayed gastric emptying.

10 cerebroventricularly significantly decreased gastric emptying.

11 d for prevention of diabetes-induced delayed gastric emptying.

12 during the muscular contractions leading to gastric emptying.

13 ration with bedside functional assessment of gastric emptying.

14 sex, body mass, symptom onset, and delay in gastric emptying.

15 ature infants, may play a functional role in gastric emptying.

16 bers of gastric dopamine neurons and delayed gastric emptying.

17 hat were resistant to development of delayed gastric emptying.

18 tyrosine kinase Kit, are required for normal gastric emptying.

19 lly antagonized GLP-1-induced delay of solid gastric emptying.

20 -3000 pmol) dose-dependently inhibited solid gastric emptying.

21 on of GLP-1 and PYY(3-36) on food intake and gastric emptying.

22 in mediating the inhibitory effect on solid gastric emptying.

23 ylorus plays an important role in regulating gastric emptying.

24 nterfere with gastric peristalsis and impede gastric emptying.

25 derive more from improved accommodation than gastric emptying.

26 lectrical activity, antral contractions, and gastric emptying.

27 y in reducing bile reflux and its effects on gastric emptying.

28 however, increase meal size and accelerated gastric emptying.

29 Nevertheless, it may also lead to delayed gastric emptying.

30 han increasing the energy density in slowing gastric emptying.

31 The primary end point was the half-time of gastric emptying.

32 c clinical syndrome characterized by delayed gastric emptying.

33 instability was associated with a change in gastric emptying.

34 oesophageal reflux, constipation and delayed gastric emptying.

35 bohydrates, 15 g protein, and 12 g fat), and gastric emptying ((13)C-acetate breath test) and blood g

36 ms of gastroparesis, 129 (72.9%) had delayed gastric emptying; 25 (14.1%) had abnormal IMD(0) Low IMD

37 er 180 min, appetite (visual analog scales), gastric emptying (3-dimensional ultrasonography), and bl

38 nic heterotypic stress significantly delayed gastric emptying (35.1+/-2.8%, n=6, P<or=0.01), compared

39 Particularly the times of 10% gastric emptying, 50% gastric emptying, maximal intestin

40 ransit slowed in mice lacking TPH2 mice, but gastric emptying accelerated.

41 n mice with intermedin led to suppression of gastric emptying activity and food intake.

42 xytocin is involved in restoring the delayed gastric emptying after paired housing, an oxytocin antag

43 tor activity and urocortin 2-induced delayed gastric emptying also indicate a safe and long-lasting a

44 eficits in olfactory discrimination, delayed gastric emptying, altered sleep latency, anxiety-like be

45 ces postprandial glycemia, partly by slowing gastric emptying, although its impact on small intestina

46 here was a protein load-dependent slowing of gastric emptying, an increase in concentrations of insul

47 Ten percent gastric emptying and 50% gastric emptying were decreased

48 s to develop a scintigraphic test to measure gastric emptying and accommodation simultaneously.

49 Gastric emptying and accommodation were measured in heal

50 s method permits simultaneous measurement of gastric emptying and accommodation.

51 2) in the intestinal lumen potently inhibits gastric emptying and acid secretion via the vagal affere

52 n, whey-protein drinks load-dependently slow gastric emptying and alter gut hormone secretion compare

53 dures under general anesthesia have impaired gastric emptying and are at high risk for aspiration of

54 Delayed gastric emptying and bile reflux are common concerns in

55 tor-mediated response) and did not influence gastric emptying and blood pressure (CRF2 receptor-media

56 Gastric emptying and colonic transit were scintigraphica

57 ooling and swallowing problems, to delays in gastric emptying and constipation.

58 NPY(-/-) mice had accelerated gastric emptying and delayed intestinal transit compared

59 d and to determine the relationships between gastric emptying and dyspeptic symptoms in inactive CD.

60 GLP-1 also modulates gastric emptying and engages neural circuits in the port

61 ency, and similar doses of ghrelin attenuate gastric emptying and feeding responses to GLP-1 and PYY(

62 inhibitory effects of GLP-1 and PYY(3-36) on gastric emptying and food intake.

63 we tested the hypothesis that inhibition of gastric emptying and gastric acid secretion in response

64 he effects of PES with various parameters on gastric emptying and gastric slow waves in 5 sessions.

65 Both processes also control gastric emptying and gastrointestinal secretions.

66 At 6 h, gastric emptying and gastrointestinal transit assessed i

67 -induced insulin secretion and inhibits both gastric emptying and glucagon secretion.

68 mittent stimulation of the GLP-1 receptor on gastric emptying and glycemia.

69 AN have slower gastric emptying and heightened visceral perception comp

70 Centrally applied opioids delay gastric emptying and inhibit intestinal transit.

71 aximal intestinal filling, indicating faster gastric emptying and intestinal filling.

72 Gastric emptying and meal appearance did not differ betw

73 Inclusion criteria included delayed gastric emptying and moderate to severe symptom scores u

74 Gastric emptying and nutritional status were maintained

75 between the viscosity and energy density on gastric emptying and perceived satiety.

76 i.p.) administration of CRF or Ucns inhibits gastric emptying and promotes colonic motility.

77 ansplantation of neural stem cells (NSCs) on gastric emptying and pyloric function in nNOS-/- mice, a

78 Ghrelin promotes gastric emptying and secretion of growth hormone and inh

79 promoting growth of the mucosa and reducing gastric emptying and secretion.

80 0 mug twice daily) significantly accelerated gastric emptying and significantly reduced vomiting, com

81 SG accelerates gastric emptying and small bowel transit of semisolids.

82 inal glucose transport but rather inhibiting gastric emptying and small intestine motility and strong

83 t mouse (m) Ucn 2, injected i.c.v. inhibited gastric emptying and stimulated distal colonic motor fun

84 Gastric emptying and volumes and satiation were not sign

85 Gastric emptying and volumes, postprandial symptoms, tot

86 relin significantly reduced the half-time of gastric emptying and vomiting, as well as nausea, abdomi

87 Appetite, gastric-emptying and orocecal transit times, and selecte

88 er and lower fullness sensations, more rapid gastric-emptying and orocecal transit times, attenuated

89 ointestinal tract symptoms, quality of life, gastric emptying, and adverse events.

90 to improve dietary absorption of fats, slow gastric emptying, and decrease food intake.

91 amate caused significant weight loss, slowed gastric emptying, and decreased calorie intake; weight l

92 with normal emptying, diabetic with delayed gastric emptying, and diabetic with delayed gastric empt

93 y weight, body mass index, body composition, gastric emptying, and genetic variation (alpha2A C1291G,

94 of resveratrol treatment on GLP-1 secretion, gastric emptying, and glycemic control in type 2 diabete

95 of whey protein on energy intake, appetite, gastric emptying, and gut hormones in healthy older men

96 iaphorase-stained myenteric neurons, delayed gastric emptying, and increased intestinal transit time.

97 fasting and postprandial glucose, decreases gastric emptying, and inhibits food intake in a GLP-1 re

98 f 4 sweet preloads on GIP and GLP-1 release, gastric emptying, and postprandial glycemia.

99 ndent insulinotropic polypeptide (GIP), slow gastric emptying, and reduce postprandial glycemic excur

100 cluding esophageal transit, liquid and solid gastric emptying, and small- and large-bowel transit, us

101 Gastric emptying, antral contractions and oro-cecal tran

102 There was no effect of sex on gastric emptying, appetite, gastrointestinal symptoms, g

103 Pasteurization did not affect gastric emptying ( approximately 30-min half time) or pH

104 ncentration of glucose (P<0.001) and delayed gastric emptying as revealed by the acetaminophen absorp

105 A relationship between body weight and gastric emptying as well as self-reported feelings of sa

106 activation, and functional reflex changes in gastric emptying associated to colon distension.

107 with moderate to severe symptoms and delayed gastric emptying at 27 clinical centers, from June 2012

108 This resulted in delayed gastric emptying, at least partially caused by impaired

109 effect on GLP-1 secretion, glycemic control, gastric emptying, body weight, or energy intake.

110 On a separate day, all patients underwent a gastric emptying breath test with assessment of postpran

111 glucose and plasma GLP-1 concentrations and gastric emptying (breath test) over 240 min.

112 food intake, hindbrain c-fos expression, or gastric emptying but improved glucose tolerance and stim

113 Current data indicate faster postoperative gastric emptying, but detailed studies on alterations in

114 .33 mum) compared with LE2 (52 mum)] delayed gastric emptying by 38 min.

115 f the proximal colon significantly inhibited gastric emptying by 82% and 34%, as measured 30 and 60 m

116 , we measured plasma GH, gastric volume, and gastric emptying by combined (99m)Tc-single-photon emiss

117 roparesis, typical symptoms alone or delayed gastric emptying by scintigraphy without gastrointestina

118 re used: (1) definite gastroparesis, delayed gastric emptying by standard scintigraphy and typical sy

119 ibition of HO-1 activity in mice with normal gastric emptying caused a loss of Kit expression and dev

120 These tests include measures of gastric emptying; contractility; electrical activity; re

121 e whether alterations in meal absorption and gastric emptying contribute to the mechanism by which in

122 ed, 105 (24.4%) of whom demonstrated delayed gastric emptying defined by 4-h gastric retention of mor

123 The primary outcome was change in gastric emptying (delay relative to baseline) of solids

124 ) mice after onset of diabetes, when delayed gastric emptying develops, and after induction of HO1 to

125 pancreatectomy hemorrhage (PPH), and delayed gastric emptying (DGE) were also graded.

126 Onset of delayed gastric emptying did not alter the total number of macro

127 n gastric emptying of solids; measurement of gastric emptying (eg, at 5 weeks of treatment) may be a

128 e, percentage emptying at 10 s), liquid-only gastric emptying (emptying half-time), liquid and solid

129 These changes include accelerated gastric emptying, enhanced postprandial cholecystokinin

130 maging assessed SPECT gastric dimensions and gastric emptying every 20 min up to 240 min.

131 lated in all the mice that developed delayed gastric emptying; expression of Kit and neuronal nitric

132 Social attachment restores delayed gastric emptying following chronic heterotypic stress, v

133 The acute effects of lipid emulsions on gastric emptying, gallbladder volume, and triglyceride a

134 in the physiologic range) versus placebo on gastric emptying, gastric volume, and postprandial sympt

135 udy was to evaluate the long-term outcome of gastric emptying, gastrointestinal symptoms and nutritio

136 the efficacy of relamorelin on symptoms and gastric emptying (GE) in a 12-week, phase 2B study of di

137 mula is thought to play an important role on gastric emptying (GE) in a variety of gastrointestinal d

138 Delayed gastric emptying (GE) induced by acute restraint stress

139 approximately 20% of patients with diabetes, gastric emptying (GE) is accelerated.

140 relationship between control of glycemia and gastric emptying (GE) is unclear.

141 Although not as well standardized as gastric emptying (GE) scintigraphy, esophageal transit s

142 ve contributions of gastric volumes (GV) and gastric emptying (GE) to meal size and postprandial symp

143 Glucagon was suppressed, and gastric emptying (GE) was decelerated.

144 fects of antidepressant therapy on symptoms, gastric emptying (GE), and meal-induced satiety in patie

145 ative parameters were determined: percentage gastric emptying (%GE) at 90 min, half-time (min) based

146 e use of mathematical modeling, we estimated gastric emptying, glucose absorption, beta-cell function

147 adequate endogenous GLP-1 release, requiring gastric emptying >2 kcal/min; and 3) the action of metfo

148 Severe delay in gastric emptying (>35% retention at 4 hours) was present

149 Patients with severely delayed gastric emptying had worse vomiting and more severe loss

150 Overall, the gastric emptying half-time (GE t50) was 54.7 +/- 3.8 min

151 tying of solids and liquids by scintigraphy (gastric emptying half-time [GE t(1/2)]); gastric volumes

152 to determine whether a study of clear liquid gastric emptying has added value for the diagnosis of ga

153 astroparesis, a 30-min study of clear liquid gastric-emptying has considerable added diagnostic value

154 associated with alterations in the rates of gastric emptying, impaired accommodation, heightened gas

155 eal (500 kcal) in 13 women without affecting gastric emptying in 6 women.

156 mes without altering postprandial volumes or gastric emptying in a predominantly female cohort.

157 nthase expression, and completely normalized gastric emptying in all mice.

158 ntral contractions and activity in promoting gastric emptying in canine models.

159 ous studies assessing the effect of GLP-2 on gastric emptying in humans have yielded inconsistent res

160 Delayed gastric emptying in inactive Crohn's disease patients se

161 oxytocin antagonist attenuated the restored gastric emptying in paired housed rats (33.2+/-4.1%, n=6

162 agonist of the ghrelin receptor that speeds gastric emptying in patients with diabetes) in patients

163 Y(3-36) [PYY(3-36)] inhibit, food intake and gastric emptying in rats.

164 mains unclear how central GLP-1 delays solid gastric emptying in rats.

165 diating central GLP-1-induced delay of solid gastric emptying in rats.

166 l (180-210 min; energy intake, appetite, and gastric emptying in the men have been published previous

167 apsule successfully detected acceleration of gastric emptying induced by erythromycin, and retardatio

168 ease in Fos expression and the inhibition of gastric emptying induced by the colon distension.

169 e of post-pancreatectomy hemorrhage, delayed gastric emptying, infectious complications, the grade of

170 Gastric emptying influences postprandial PYY levels.

171 ntestinal motility was assessed by measuring gastric emptying, intestinal transit, and isometric musc

172 is identified as the main mechanism by which gastric emptying is increased; some motilin agonists act

173 While gastric emptying is probably the most significant compon

174 Diabetic gastroparesis (delayed gastric emptying) is a well-recognized complication of d

175 afferent neurons to inhibit food intake and gastric emptying; it also increases expression of the ne

176 larly the times of 10% gastric emptying, 50% gastric emptying, maximal intestinal filling, 10% termin

177 A number of studies suggest that delayed gastric emptying may accompany a variety of disorders, o

178 We simultaneously measured gastric emptying, meal appearance, endogenous glucose pr

179 No significant change was found in gastric emptying (median half-time 137 vs. 132 vs. 125 m

180 oring (n = 27), upright esophagram (n = 90), gastric emptying (n = 26), and symptom assessment (n = 1

181 nfectious complications (n = 65) and delayed gastric emptying (n = 29).

182 Delayed gastric emptying observed in singly housed rats induced

183 Delayed gastric emptying occurs in critically ill patients and i

184 The purpose of this study was to quantify gastric emptying of 30 and 70 g of oral whey protein loa

185 Gastric emptying of a mixed nutrient liquid meal was ass

186 Gastric emptying of a non-nutrient liquid was also deter

187 Gastric emptying of a radiolabeled mashed potato meal wa

188 tion, GLP-1 receptor blockade did not affect gastric emptying of a solid meal.

189 sured by nuclear scintigraphy in awake mice, gastric emptying of an ingested whole-egg meal was signi

190 gastrointestinal transit and recipient solid gastric emptying of chrome steel balls, and marked preve

191 wn as a stress-related peptide, which delays gastric emptying of liquid and solid food via the autono

192 No significant differences in gastric emptying of liquids (acetaminophen area under th

193 glutide 4 mg/day for 10 days does not affect gastric emptying of liquids in healthy subjects as measu

194 y was to assess the effect of teduglutide on gastric emptying of liquids in healthy subjects, as meas

195 ed GLP-1-and PYY(3-36)-induced inhibition of gastric emptying of saline by 6-29%.

196 pants weighing less than 137 kg, we measured gastric emptying of solids and liquids by scintigraphy (

197 In a prospective study, we measured gastric emptying of solids and liquids, fasting and post

198 Compared with placebo, liraglutide delayed gastric emptying of solids at 5 weeks (median 70 min [IQ

199 Post-hoc analysis showed that the T1/2 of gastric emptying of solids at 5 weeks correlated with ch

200 Then, we propose to investigate whether gastric emptying of solids in patients with inactive CD

201 Gastric emptying of solids was analysed by the [13C]octa

202 On 3 separate days, we measured gastric emptying of solids, gastric volumes, postprandia

203 on weight loss are associated with delay in gastric emptying of solids; measurement of gastric empty

204 Gastric emptying of the L and H drinks was comparable wh

205 icate that, in healthy lean men, the rate of gastric emptying of whey protein is independent of load

206 inistration of leucine and isoleucine on the gastric emptying of, and blood glucose responses to, a p

207 The potential influence of gastric emptying on the "incretin effect," mediated by g

208 rovided a way to depict both bile reflux and gastric emptying on the same day in patients who underwe

209 ministration of AITC by gavage did not alter gastric emptying or small intestinal transit, but lumina

210 Vildagliptin does not alter gastric emptying or the rate of entry of ingested glucos

211 385 mL, 95% CI = -616 to -154), less delayed gastric emptying (OR = 0.6, 95% = CI 0.5-0.8), and short

212 postprandial minus fasting) gastric volumes, gastric emptying, or aggregate postprandial symptoms.

213 ected glucagon, GLP-1, GIP, cholecystokinin, gastric emptying, or energy intake.

214 asting gastric volume (P = .03), accelerated gastric emptying (P < .001 for solids and P = .011 for l

215 -daily relamorelin significantly accelerated gastric emptying (P < .03) and reduced vomiting frequenc

216 soleucine-10g, but not isoleucine-5g, slowed gastric emptying (P < 0.05), but gastric emptying was no

217 0.199), biliary leakage (P = 0.382), delayed gastric emptying (P = 0.062), burst abdomen (P = 0.480),

218 ng was significantly associated with delayed gastric emptying (p<0.05).

219 icant complications still occur (eg, delayed gastric emptying, pancreatic fistula, and biliary strict

220 Results based on standard 4-h solid gastric emptying, performed according to current Society

221 Although there was no difference in gastric emptying, plasma 3-O-methyl-glucose concentratio

222 an effect that was not due to differences in gastric emptying rate (acetaminophen test) and/or circul

223 Liquid gastric emptying rate (GE) and antral motility parameter

224 Gastric protein hydrolysis may influence gastric emptying rate and subsequent protein digestibili

225 A slower eating rate and a delayed gastric emptying rate can partly explain for the stronge

226 Gastric emptying rate of protein was a significant (p<0.

227 Gastric emptying rates and nutritional statuses were eva

228 GLP-1R agonists also decelerate gastric emptying, reduce body weight by reduction of foo

229 Stress impairs gastric emptying, reduces stomach compliance and induces

230 tration dependent insulin secretion, slowing gastric emptying, reducing food intake and stimulating b

231 substrates stimulate GLP-1 and GIP and slow gastric emptying, regardless of whether they are metabol

232 gastric emptying, and diabetic with delayed gastric emptying reversed by the HO1 inducer hemin.

233 Heart rate variability measurements and gastric emptying scintigraphies were performed in all su

234 intragastric meal distribution (IMD) during gastric emptying scintigraphy (GES) allows for a simple

235 Gastric emptying scintigraphy is considered to be the go

236 the Block Food Frequency Questionnaire, and gastric emptying scintigraphy results were analyzed.

237 istories, symptom questionnaires, and 4-hour gastric emptying scintigraphy studies were obtained from

238 f radiolabeled test meals have been used for gastric emptying scintigraphy.

239 Gastric emptying (scintigraphy, 100 mL of Ensure (Abbott

240 Smn deficiency caused constipation, delayed gastric emptying, slow intestinal transit and reduced co

241 f continuous or bolus nasogastric feeding on gastric emptying, small bowel water content, and splanch

242 ke peptide 1 (GLP-1), which is known to slow gastric emptying, stimulate insulin secretion, and suppr

243 unction alterations, including inhibition of gastric emptying, stimulation of colonic propulsive moti

244 dies can be performed alone or together with gastric emptying studies after oral administration of an

245 tudy was to evaluate the prognostic value of gastric emptying studies on the morbidity associated wit

246 The medical literature states that solid gastric-emptying studies are more sensitive for the dete

247 Gastric-emptying studies have no accepted standard metho

248 101 patients underwent both solid and liquid gastric-emptying studies, acquired sequentially on the s

249 A delayed radionucleotide gastric emptying study predicts negative health outcomes

250 All patients underwent esophageal manometry, gastric emptying study, and sham-feeding test (correspon

251 d intracerebroventricularly (icv) before the gastric emptying study.

252 , and retching) and delay in radionucleotide gastric emptying study.

253 A liquid-only 30-min gastric-emptying study followed, with ingestion of 3.7 M

254 teers (9 male, 11 female) underwent a 90-min gastric-emptying study performed with a triple-head gamm

255 ase Activity Index (CDAI) < 150, underwent a gastric emptying test by breath test using 13C octanoic

256 Advances in gastric emptying testing include wireless motility capsu

257 ive method for performing radionuclide solid gastric emptying that can be used for any dual-head gamm

258 dated a simple method for radionuclide solid gastric emptying that can be used with a dual-head gamma

259 trointestinal hormone capable of stimulating gastric emptying, the relatively recent availability of

260 and energy content have been shown to delay gastric emptying, their relative importance is not well

261 in oxidative stress, and loss of Kit delays gastric emptying; this effect is reversed by induction o

262 s motility capsule to detect drug effects on gastric emptying time (GET) and gastric contractility.

263 The gastric emptying time and small intestinal motility were

264 New onset esophageal dysmotility, delayed gastric emptying time, and abnormal sham-feeding tests w

265 disease duration, severity of constipation, gastric emptying time, and heart rate variability.

266 here was no significant difference in either gastric emptying time--23 minutes (range 5-131) in patie

267 Faster gastric-emptying times were significantly associated wit

268 One week posttransplantation, gastric emptying to a non-nutrient meal was measured usi

269 rders, such as failure to thrive and delayed gastric emptying, together accounted for 58.9% of all re

270 Nutrients in the duodenum inhibit gastric emptying via a feedback pathway that involves re

271 lucagon like peptide-1 (GLP-1) delays liquid gastric emptying via non-adrenergic, non-cholinergic neu

272 ecently showed that central CRF delays solid gastric emptying via sympathetic pathways in rats.

273 Gastric emptying was accelerated in the sleeve gastrecto

274 Severe delay in gastric emptying was associated with more severe symptom

275 Normalization of gastric emptying was associated with repopulation of CD2

276 When gastric emptying was bypassed by direct glucose administ

277 the varying degree of stomach filling during gastric emptying was compared between groups.

278 Gastric emptying was delayed in 20% of patients at 2 h a

279 Solid gastric emptying was measured after 7 consecutive days o

280 Gastric emptying was measured in nonobese diabetic mice

281 -5g, slowed gastric emptying (P < 0.05), but gastric emptying was not correlated with the blood gluco

282 -treated rats ingested food at a lower rate, gastric emptying was not significantly different between

283 ection of GLP-1 (3000 pmol)-induced delay of gastric emptying was partially antagonized by celiac gan

284 ificantly delayed with PES, and the delay in gastric emptying was significantly and negatively correl

285 The delayed gastric emptying was significantly correlated with the i

286 (2) Gastric emptying was significantly delayed with PES, and

287 Gastric emptying was significantly increased in mice tha

288 Gastric emptying was slower after both 3OMG and TIM than

289 ter of radioactivity >/=2 on day 3), but not gastric emptying, was significantly correlated with clin

290 Ten percent gastric emptying and 50% gastric emptying were decreased postoperatively as well

291 asma total GLP-1 and GIP, serum insulin, and gastric emptying were determined.

292 od glucose and plasma total GLP-1 as well as gastric emptying were similar at each assessment, and th

293 o infection, nutritional status, and delayed gastric emptying were the most common reasons for readmi

294 spepsia had significantly (p<0.05) prolonged gastric emptying when compared to patients without dyspe

295 The impact of variations in gastric emptying, which influence the magnitude of gluco

296 ike peptide 1 (GLP-1) and its agonists slows gastric emptying, which represents the major mechanism u

297 hs of continuous symptoms of CUNV but normal gastric emptying who were treated at the University of M

298 A standardized method for measuring gastric emptying with a Tc(99m)-labelled meal followed b

299 Gastric emptying (with the use of 3-dimensional ultrasou

300 d 4 (ID4) kcal/min (equating to two rates of gastric emptying within the physiological range) on the