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1 c labeling of afferent intramuscular arrays (gastric fundus).
2 ircular muscle layer of mouse and guinea-pig gastric fundus.
3 zed calcium release events in cells from the gastric fundus.
4 stribution occurred after injection into the gastric fundus.
5 al masses on the posteromedial border of the gastric fundus.
6 -cm-diameter polypoid, ulcerated mass in the gastric fundus.
7 link histamine and somatostatin cells in the gastric fundus.
8 C-IM mediate neural inputs in muscles of the gastric fundus.
9 y, which is mainly caused by ischemia of the gastric fundus.
10 ic defects on the superomedial aspect of the gastric fundus abutting the cardia, presumably due to bu
11 l vagal stimulation (EVS) were recorded from gastric fundus and antral regions of wild type and W/W(V
12 going discharge of unitary potentials in the gastric fundus and antrum that contributes to the overal
13                                          The gastric fundus and antrum were evaluated independently u
14  excite vagal motor neurones that project to gastric fundus and corpus.
15 rwent laparotomy and measurement of baseline gastric fundus blood flow.
16 astric vessels and subsequent measurement of gastric fundus blood flow.
17 plying fluorescent retrograde tracers to the gastric fundus, corpus or antrum/pylorus, or to the duod
18 osition of the fluorescent tracer DiI to the gastric fundus, corpus or antrum/pylorus, the duodenum o
19 ponse to nitrergic stimulation of the canine gastric fundus does not depend upon electrical hyperpola
20 enger RNA (mRNA) differential display of the gastric fundus from gastrin-deficient (GAS-KO) or wild-t
21 tric conduit despite previous surgery on the gastric fundus in 8 (20%), previous esophageal operation
22 hey involved the posteromedial border of the gastric fundus in eight patients, the central cardiac re
23 a2+]i), and tension in muscles of the canine gastric fundus in response to nitrergic stimulation by N
24                 Coinnervation of the LES and gastric fundus is logical, because the LES has similar f
25 growth factor alpha (TGFalpha) levels in the gastric fundus leads to oxyntic atrophy and massive fove
26  and visualize PDGFRalpha(+) cells in murine gastric fundus, load cells with Ca(2+) indicators, and f
27 c excitatory neural inputs was studied using gastric fundus muscles of wild-type animals and W/W(v) m
28  subunit 1 (MYPT1) phosphorylation in murine gastric fundus muscles stimulated by bath-applied carbac
29 on of purinergic enteric inhibitory input in gastric fundus muscles.
30      Slow waves were routinely recorded from gastric fundus muscles.
31 f norepinephrine, nerve growth factor in the gastric fundus muscularis externae, brain-derived neurot
32 the circular and longitudinal muscles of the gastric fundus of wild-type mice.
33  70 % (30/43) of those that projected to the gastric fundus or corpus.
34   This allows the organism to live in either gastric fundus or gastric antrum depending on the level
35 s and results in increased blood flow to the gastric fundus prior to esophagogastric anastomosis.
36 special attention to their relationship with gastric fundus-projecting neurons.
37 c dispersion of the muscle of the guinea-pig gastric fundus, single elongated cells were observed whi
38 otentials (IJP) of circular smooth muscle of gastric fundus were studied.
39            Transarterial embolization of the gastric fundus with fluoroscopic guidance was performed

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