ライフサイエンスコーパス: gastric gland

1 ulovesicles and luminal (TV/L) spaces of the gastric gland.

2 arietal cell, the acid secretory cell of the gastric gland.

3 h MUC6 secreted in concert with TFF2 by deep gastric glands.

4 clusters of Dcamkl1(+) cells at the base of gastric glands.

5 n and acid trapping, as measured in isolated gastric glands.

6 iple gastric cell lineages that populate the gastric glands.

7 gr5-positive stem cell population in pyloric gastric glands.

8 id secretion in streptolysin-O-permeabilized gastric glands.

9 ad significantly reduced numbers of infected gastric glands.

10 gestive enzyme pepsinogen into the lumina of gastric glands.

11 hanisms involved in the injury and repair of gastric glands; (2) to present a hypothesis on the devel

12 y were studied using freshly isolated rabbit gastric glands and cultured parietal cells.

13 at cell-associated H pylori can colonize the gastric glands and directly affect precursor and stem ce

14 stem cells are located in the isthmus of the gastric glands and give rise to epithelial progenitors t

15 rved consistently in epithelial cells of the gastric glands and in gastric surface cells.

16 ype and DeltaF508-deficient mice in isolated gastric glands and whole stomach preparations.

17 e ablation of parietal cells, dissolution of gastric glands, and loss of chief and mucus-producing ce

18 Organoids formed primitive gastric gland- and pit-like domains, proliferative zones

19 nd stem cell origin of normal and neoplastic gastric glands are uncertain.

20 Prior to cancer development, the oxyntic gastric glands atrophy and are replaced by metaplastic c

21 ](i) in ECL and parietal cells of superfused gastric glands, but only the parietal cell signal was in

22 Activation of SCAR in the intact rat gastric gland by divalent cations (Ca(2+) or Mg(2+)) or

23 ue-induced acid secretion in wild-type mouse gastric glands could be significantly reduced with eithe

24 the nucleus in vitro and in a novel ex vivo gastric gland culture system.

25 values; and (d) no channels carrying primary gastric gland fluid through the mucus were observed.

26 Gastric glands from animals expressing mutant beta subun

27 KCNQ1 exhibited abnormal distribution in gastric glands from kcne2 (-/-) mice, with increased exp

28 Organ cultures of gastric glands from wild-type or IL-1R1 null mice were t

29 Yet another group (e.g. gastric glands), have stem cells with well characterized

30 lls exposed to hostile environments, such as gastric glands, have no demonstrable permeability to the

31 arp contrast, they infiltrated deep into the gastric glands in Gal3-deficient mice.

32 ing were, however, unaffected in Trpml1(-/-) gastric glands, indicating that Trpml1 does not function

33 Methods: Isolated rabbit gastric glands (intact or permeabilized with S. aureus a

34 In parietal cells in isolated gastric glands, intracellular levels of Ca2+ responded t

35 y marginal reactions to bacterial infection, gastric gland lineages mounted a strong inflammatory res

36 or the generation of concentrated HCl in the gastric gland lumen.

37 ithelium, including moderate dilation of the gastric gland lumens and a reduction in the number of pa

38 t acidity within the TV/L compartment of the gastric gland may be regulated, at least in part, by its

39 the stomach epithelium before completion of gastric gland morphogenesis.

40 calpha1-->4Gal from glycans expressed in the gastric gland mucous cell-type mucin.

41 Klotho expression was detected in gastric glands, myenteric neurons and smooth muscle cell

42 pylori had successfully colonized within the gastric glands of both WT and Gal3-deficient mice, altho

43 specifically express either lineages of the gastric gland, or the gastric pit, by addition of nicoti

44 g immunohistochemistry, both mouse and human gastric glands overexpressed Cldn7 in dysplastic but not

45 Both components of the gastric gland secretome associate non-covalently and sho

46 These data provide evidence that gastric gland specification and progenitor cell maintena

47 lture surface cells and surgically dissected gastric glands, staining was observed consistently in ep

48 After 1 year, gastric glands that contained green fluorescent protein-

49 y and 3-dimensional reconstruction of entire gastric glands to determine the localizations of H pylor

50 Light microscopy indicated that gastric glands were dilated, and electron micrographs di

51 +)](i) and histamine release; its effects on gastric glands were examined by confocal microscopy of [

52 is limited to the base and lower isthmus of gastric glands, where the stem cells reside.

53 Studies of the clonal architecture of gastric glands with intestinal metaplasia are important

54 Double labeling of gastric glands with rab3D and pepsinogen antibodies conf