ライフサイエンスコーパス: gastric motility

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1 t correlation was found between symptoms and gastric motility.

2 f the vago-vagal reflex circuitry regulating gastric motility.

3 omach of a patient with significantly slowed gastric motility.

4 al pre-motor neurons in the DMV that control gastric motility.

5 r (mu, delta, and kappa) stimulation affects gastric motility.

6 reduced chylous lymph-induced inhibition of gastric motility.

7 more potent than control lymph in inhibiting gastric motility.

8 ay be responsible for oesophageal control of gastric motility.

9 gastrin release, gastric acid secretion, and gastric motility.

10 ich may be prostaglandin dependent, inhibits gastric motility.

11 ric-NST or -DMN neuron responsiveness, or on gastric motility.

12 nsulin or central 2-DG causes an increase in gastric motility.

13 jects, including satiety or satiation (21%), gastric motility (14%), psychological factors (13%), and

14 Antidepressants and newer gastric motility agents show promise.

15 ) the pathway mediating reflex inhibition of gastric motility and (2) activation of duodenal vagal af

16 (60 nl)(-1)) in mNTS produced inhibition of gastric motility and an increase in intragastric pressur

17 eceptors in the brainstem appear to modulate gastric motility and pancreatic exocrine secretion.

18 ts that alpha-1 agonists have in suppressing gastric motility and producing anorexia.

19 od flow, and nutrient uptake and suppressing gastric motility and secretion.

20 18 mmHg) provoked a significant reduction in gastric motility and tone recorded with strain gauges.

21 est that glucoprivation-induced increases in gastric motility are dependent on intact hindbrain astro

22 It affects gastric motility by stimulating interdigestive antrum an

23 induced by erythromycin, and retardation of gastric motility caused by morphine.

24 een suggested as a therapy for patients with gastric motility disorders or morbid obesity.

25 This study evaluated gastric motility in children with AM.

26 aracterization of patterns of development of gastric motility in early infancy.

27 l for each site) of CRF in the DVC decreased gastric motility in rats pretreated with the muscarinic

28 iew focuses on progress made in the field of gastric motility in the past year, emphasizing advances

29 findings suggest a possible role of abnormal gastric motility in the pathogenesis of AM.

30 erstanding Helicobacter pylori infection and gastric motility in the pediatric population.

31 relationship between utilizable nutrient and gastric motility is well recognized, the explanation of

32 tion of CRF(2) receptors and the decrease in gastric motility observed following microinjection of CR

33 contractility; electrical activity; regional gastric motility of the fundus, antrum, and pylorus; and

34 tion in critically ill adults; however, poor gastric motility often prevents nutritional targets bein

35 tiology is likely multifactorial, related to gastric motility, respiratory secretions, and medication

36 ed by thyrotrophin-releasing hormone and the gastric motility response to DMV microinjections of TRH

37 tion of gastroparesis and other disorders of gastric motility; Society of Nuclear Medicine and Molecu

38 of inhibitory effects of central opioids on gastric motility still remains unclear.

39 ring GES can yield additional information on gastric motility to help explain patients' symptoms.

40 wnstream NST neurons that, in turn, suppress gastric motility via action on neurons in the dorsal mot

41 Gastric motility was characterized in spontaneously diab

42 Inhibition of gastric motility was measured manometrically in urethane

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